Search results for "Liver Failure"

showing 10 items of 60 documents

Scrub typhus, acute respiratory distress, and hemophagocytic lymphohistiocytosis

2013

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Microbiology (medical)MalePediatricsmedicine.medical_specialtyHemophagocytic lymphohistiocytosisRespiratory Distress SyndromeScrub typhubusiness.industryScrub typhus; acute respiratory distress; hemophagocytic lymphohistiocytosis.General MedicineScrub typhusAcute respiratory distresshemophagocytic lymphohistiocytosis.Liver Failure Acutemedicine.diseaseInfectious Diseaseshemophagocytic lymphohistiocytosisScrub typhusmedicineHumansbusinessacute respiratory distreacute respiratory distressInternational Journal of Infectious Diseases
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Safety and clinical activity of a combination therapy comprising two antibody-based targeting agents for the treatment of non-Hodgkin lymphoma: resul…

2013

Purpose Inotuzumab ozogamicin (INO) is an antibody-targeted chemotherapy agent composed of a humanized anti-CD22 antibody conjugated to calicheamicin, a potent cytotoxic agent. We performed a phase I/II study to determine the maximum-tolerated dose (MTD), safety, efficacy, and pharmacokinetics of INO plus rituximab (R-INO) for treatment of relapsed/refractory CD20+/CD22+ B-cell non-Hodgkin lymphoma (NHL). Patients and Methods A dose-escalation phase to determine the MTD of R-INO was followed by an expanded cohort to further evaluate the efficacy and safety at the MTD. Patients with relapsed follicular lymphoma (FL), relapsed diffuse large B-cell lymphoma (DLBCL), or refractory aggressive NH…

OncologyAdultLiver CirrhosisMaleCancer Researchmedicine.medical_specialtyNeutropeniamedicine.medical_treatmentFollicular lymphomaPharmacologyAntibodies Monoclonal HumanizedDrug Administration Schedulechemistry.chemical_compoundAntibodies Monoclonal Murine-Derivedimmune system diseasesRecurrenceRisk Factorshemic and lymphatic diseasesInternal medicineCalicheamicinAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansInotuzumab OzogamicinMolecular Targeted TherapyB-cell lymphomaAgedHyperbilirubinemiaInotuzumab ozogamicinChemotherapybusiness.industryLymphoma Non-HodgkinMiddle Agedmedicine.diseasePrognosisThrombocytopeniaPolatuzumab vedotinLymphomaTreatment OutcomeOncologychemistryLiverRituximabFemalebusinessRituximabLiver Failuremedicine.drugJournal of clinical oncology : official journal of the American Society of Clinical Oncology
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Mucopolysaccharidosis Type VII (Sly disease) survivors

2013

treatment. Onset of neurological symptoms at age 8 and in adolescence. Pair 4: L.M. died at age 5 months due to liver failure. P.M. (7 years): earlyinfantile form, despite treatment start at age 2 progressive neurological deterioration. Pair 5: R.K.: late-infantile form, untreated, died at age 9 due to progressive neurological involvement. M.K.: late infantileform, start of treatment at age 5, died at age 13 due to epileptic encephalopathy. These cases reveal that disease onset and progression in siblings with NPC vary, and that miglustat can slow disease progression.

Pediatricsmedicine.medical_specialtyDisease onsetbusiness.industryEndocrinology Diabetes and MetabolismEpileptic encephalopathyMucopolysaccharidosisLiver failuremedicine.diseaseBiochemistryEndocrinologyMiglustatGeneticsmedicineSly diseaseSlow disease progressionbusinessMolecular Biologymedicine.drugMolecular Genetics and Metabolism
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N-Acetylcysteine for the Management of Non-Acetaminophen Drug-Induced Liver Injury in Adults: A Systematic Review

2022

Introduction: Idiosyncratic drug-induced liver injury (DILI) is a rare adverse reaction to drugs and other xenobiotics. DILI has different grades of severity and may lead to acute liver failure (ALF), for which there is no effective therapy. N-acetylcysteine (NAC) has been occasionally tested for the treatment of non-acetaminophen drug-induced ALF. However, limited evidence for its efficacy and safety is currently available. Our aim was to elucidate the benefit and safety of NAC in DILI and evaluate its hepatoprotective effect.Methods: We conducted a systematic review to evaluate the management and prevention focused on NAC in idiosyncratic DILI. The main outcomes included mortality due to …

PharmacologyPharmacology (medical)DILInon-acetaminophenacute liver failureacute liver injuryN-acetylcysteine
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Acute organ failure following the loss of anti-apoptotic cellular FLICE-inhibitory protein involves activation of innate immune receptors

2014

Apoptosis signaling is involved in both physiological tissue homeostasis and acute and chronic diseases. The role of regulatory apoptosis signaling molecules and their organ-specific functions are less defined. Therefore, we investigated the loss of the anti-apoptotic cellular FLICE-inhibitory protein (cFLIP) and the mechanisms of the resulting lethal organ failure in vivo using inducible knockout mice. These were generated by crossing floxed cFLIP mice to a tamoxifen inducible Rosa26-creERT2 mouse strain. Death following global loss of cFLIP resulted from liver failure, accumulation of M1-polarized macrophages and accompanying hepatic cell death and inflammation. Apoptosis was also promine…

Programmed cell deathCASP8 and FADD-Like Apoptosis Regulating ProteinMice TransgenicInflammationBiologyMiceImmune systemmedicineAnimalsMolecular BiologyTissue homeostasisOriginal PaperInnate immune systemMacrophagesMembrane ProteinsCell BiologyLiver Failure AcuteImmunity InnateCell biologyToll-Like Receptor 4TransplantationApoptosisToll-Like Receptor 9Stimulator of interferon genesHepatocytesmedicine.symptomCell Death & Differentiation
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Hepassocin as a treatment for fulminant hepatic failure: will it translate from rats to human?

2010

Acute liver failure (ALF) is defined as the abrupt loss of hepatic cellular function in a patient without pre-existing liver disease, with the subsequent development of coagulopathy, jaundice and encephalopathy. It remains one of the most challenging medical emergencies, due to the multiorgan nature of the disease, the rapid evolution of the clinical condition and the need for multidisciplinary supportive interventions in order to assess the necessity for liver transplantation (LT) correctly.1 Despite different causes of ALF, the mode of cell death typically follows one of two patterns: necrosis or apoptosis; apoptosis is manifest by nuclear and cytoplasmic shrinkage without disturbance of …

Programmed cell deathNecrosismedicine.medical_treatmenthepassocinLiver transplantationProinflammatory cytokineTranslational Research BiomedicalLiver diseaseFulminant hepatic failureSpecies SpecificitymedicineAnimalsHumansbusiness.industryLiver cellGastroenterologyFibrinogenLiver Failure Acutemedicine.diseaseRecombinant ProteinsNeoplasm ProteinsRatsDisease Models AnimalCytokineImmunologymedicine.symptombusiness
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New Therapeutic Aspects in Fulminant Hepatic Failure

1991

Pulmonary and Respiratory Medicinemedicine.medical_specialtyPathologybusiness.industryFulminantLiver failureInsuficiencia hepaticaCritical Care and Intensive Care MedicineGastroenterologyLiver RegenerationLiver TransplantationFulminant hepatic failureHepatic EncephalopathyInternal medicinemedicineHumansGABA-A Receptor AntagonistsCardiology and Cardiovascular MedicinebusinessChest
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Portal Vein Thrombosis Relevance on Liver Cirrhosis: Italian Venous Thrombotic Events Registry

2016

Portal vein thrombosis may occur in cirrhosis; nevertheless, its prevalence, and predictors are still elusive. To investigate this issue, the Italian Society of Internal Medicine undertook the “Portal vein thrombosis Relevance On Liver cirrhosis: Italian Venous thrombotic Events Registry” (PRO-LIVER). This prospective multicenter study includes consecutive cirrhotic patients undergoing Doppler ultrasound examination of the portal area to evaluate the prevalence and incidence of portal vein thrombosis over a 2-year scheduled follow-up. Seven hundred and fifty-three (68 % men; 64 ± 12 years) patients were included in the present analysis. Fifty percent of the cases were cirrhotic outpatients.…

RegistrieLiver CirrhosisMaleCirrhosisHepatocellular carcinoma030204 cardiovascular system & hematologyGastroenterology0302 clinical medicineEsophageal varicesPrevalenceMedicineProspective StudiesRegistriesProspective cohort studyMultivariate AnalysiVenous ThrombosisPortal VeinAnticoagulants; Esophageal varices; Hepatocellular carcinoma; Liver failure; Splanchnic venous thrombosis; Emergency Medicine; Internal MedicineMiddle AgedPortal vein thrombosisVenous thrombosisSplanchnic venous thrombosis Anticoagulants Liver failure Hepatocellular carcinoma Esophageal varicesSplanchnic venous thrombosisItalyLiverEmergency MedicineFemale030211 gastroenterology & hepatologymedicine.symptomSettore SECS-S/01 - StatisticaHumanmedicine.medical_specialtyLiver CirrhosiSocio-culturaleEsophageal varicesAsymptomatic03 medical and health sciencesAnticoagulants Esophageal varices Hepatocellular carcinoma Liver failure Splanchnic venous thrombosisInternal medicineInternal MedicineHumansAgedHepatologyEsophageal varicebusiness.industryAnticoagulantLiver failureAnticoagulants; Esophageal varices; Hepatocellular carcinoma; Liver failure; Splanchnic venous thrombosis;AnticoagulantsSplanchnic venous thrombosimedicine.diseaseSurgeryProspective StudieSplanchnic venous thrombosis Anticoagulants Liver failure Hepatocellular carcinoma Esophageal varicesMultivariate AnalysisUpper gastrointestinal bleedingComplicationbusinessJournal of Hepatology
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Weight-loss supplementation and acute liver failure: the case of Garcinia Cambogia

2018

Traditional medicinebusiness.industrydiagnosisGarcinia cambogiaGarcinia cambogiaLiver failureMEDLINELiver Failure Acute03 medical and health sciences0302 clinical medicineWeight lossWeight LossEmergency MedicineInternal MedicineMedicineHumans030211 gastroenterology & hepatology030212 general & internal medicinemedicine.symptombusinessHerbs induced liver injury
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The role of mycotoxins in neurodegenerative diseases: current state of the art and future perspectives of research

2021

Abstract Mycotoxins are fungal metabolites that can cause various diseases in humans and animals. The adverse health effects of mycotoxins such as liver failure, immune deficiency, and cancer are well-described. However, growing evidence suggests an additional link between these fungal metabolites and neurodegenerative diseases. Despite the wealth of these initial reports, reliable conclusions are still constrained by limited access to human patients and availability of suitable cell or animal model systems. This review summarizes knowledge on mycotoxins associated with neurodegenerative diseases and the assumed underlying pathophysiological mechanisms. The limitations of the common in vivo…

business.industryClinical BiochemistryFungiLiver failureNeurotoxicityfood and beveragesNeurodegenerative DiseasesMycotoxinsBioinformaticsmedicine.diseaseBiochemistryLimited accesschemistry.chemical_compoundAnimal modelchemistryAdverse health effectAnimalsHumansMedicinebusinessMycotoxinMolecular BiologyBiological Chemistry
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