Search results for "Low-Density Lipoprotein"

showing 10 items of 172 documents

LDL size: does it matter?

2004

The atherogenic lipoprotein phenotype is characterised by a moderate increase in plasma triglycerides, a decrease in high density lipoprotein cholesterol and the prevalence of smaller denser low density lipoprotein particles. The prevalence of this partially inheritable phenotype is approximately 30% and is a feature of the metabolic syndrome associated with an increased risk for cardiovascular events. The predominance of small dense LDL has been accepted as an emerging cardiovascular risk factor by the adult treatment panel (ATP) III.

AdultMalemedicine.medical_specialtyArteriosclerosisCoronary DiseaseCohort Studieschemistry.chemical_compoundHigh-density lipoproteinSex FactorsRisk FactorsInternal medicineDiabetes mellitusmedicineDiabetes MellitusPrevalenceAnimalsHumansatherogenic lipoprotein phenotypeProspective Studiescoronary heart diseaseRisk factorsmall dense LDLTriglyceridesAgedHypolipidemic AgentsMetabolic SyndromeClinical Trials as Topicdiabetesbusiness.industryCholesterolSmokingAge FactorsGeneral MedicineCholesterol LDLAtherogenic lipoprotein phenotypeMiddle Agedmedicine.diseasePhenotypeLipoproteins LDLEndocrinologyLogistic ModelsPhenotypechemistryLow-density lipoproteinFemaleMetabolic syndromeInsulin ResistancebusinessSwiss medical weekly
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Changes in plasma lipids and low-density lipoprotein peak particle size during and after acute myocardial infarction

2002

In summary, LDL peak particle size variations occur very early after AMI, and therefore this parameter, which may be considered a genetically determined risk factor, must be measured not before 2 to 3 months after the acute event.

AdultMalemedicine.medical_specialtyMyocardial Infarctionchemistry.chemical_compoundInternal medicineBlood plasmaPlasma lipidsmedicineHumansMyocardial infarctionLow-Density Lipoprotein Acute Myocardial InfarctionRisk factorAgedAged 80 and overbusiness.industryCholesterolCholesterol HDLCholesterol LDLMiddle Agedmedicine.diseaseLipidsCoronary heart diseaseEndocrinologychemistryLow-density lipoproteinCardiologyParticle sizeCardiology and Cardiovascular MedicinebusinessThe American Journal of Cardiology
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Low-density lipoproteins generated during an oral fat load in mild hypertriglyceridemic and healthy subjects are smaller, denser, and have an increas…

2005

Triglyceride-rich lipoproteins generated during the postprandial phase are atherogenic. Large very low-density lipoproteins (LDLs) or chylomicrons (CMs) are not as atherogenic as their remnants (Rem). Small and dense LDLs are associated with cardiovascular disease. Low-density lipoprotein size is partly under genetic control and is considered as a relatively stable LDL feature. In this article, we present data on retinyl palmitate kinetics correlated with the modification of LDL features in terms of size, density, and in vitro receptor binding affinity after an oral fat load. Six nondiabetic, hypertriglyceridemic (HTG) patients and 6 healthy controls were examined. Low-density lipoprotein s…

AdultMalemedicine.medical_specialtyRetinyl EstersSettore MED/09 - Medicina InternaEndocrinology Diabetes and MetabolismBinding CompetitiveModels Biologicalchemistry.chemical_compoundEndocrinologyInternal medicineRetinyl palmitateCell Line TumorChylomicronsmedicineHumansReceptorVitamin AHypertriglyceridemiaLow-density lipoproteins hypertriglyceridemia Fasting and postprandial LDLsTriglycerideCatabolismChemistryFastingFibroblastsPostprandial PeriodDietary FatsLipidsLipoproteins LDLKineticsEndocrinologyPostprandialReceptors LDLlipids (amino acids peptides and proteins)Density gradient ultracentrifugationElectrophoresis Polyacrylamide GelFemaleDiterpenesUltracentrifugationLipoproteinChylomicronMetabolism: clinical and experimental
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Atherogenic lipoprotein phenotype and low-density lipoprotein size and subclasses in patients with growth hormone deficiency before and after short-t…

2007

Objective: Patients with growth hormone deficiency (GHD) have increased cardiovascular risk and may show elevated triglyceride and reduced high density lipoprotein (HDL) cholesterol concentrations, two lipid abnormalities usually accompanied by increased small dense LDL in the ‘atherogenic lipoprotein phenotype’ (ALP). In the present study, we directly investigated (1) whether hypopituitary patients with GHD have increased small dense LDL; (2) whether growth hormone replacement therapy (GHRT) beneficially impact on such particles; (3) the prevalence of ALP in GHD and GHRT patients. Design and methods: In 14 hypopituitary patients with GHD (44 ± 13 years, body mass index (BMI) 27 ± 3) before…

AdultMalemedicine.medical_specialtyTime FactorsArteriosclerosisHormone Replacement TherapyEndocrinology Diabetes and MetabolismBiologyHypopituitarismGrowth hormone deficiencychemistry.chemical_compoundEndocrinologyInternal medicinemedicineHumansIn patientInsulin-Like Growth Factor ITriglyceridesTriglycerideHuman Growth HormoneCholesterolVascular diseaseCholesterol HDLGeneral MedicineMiddle AgedAtherogenic lipoprotein phenotypemedicine.diseaseLipoproteins LDLCholesterolTreatment OutcomeEndocrinologyAtheromachemistryLow-density lipoproteinElectrophoresis Polyacrylamide GelFemalelipids (amino acids peptides and proteins)European Journal of Endocrinology
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Distinct patterns of heparin affinity chromatography VLDL1 and VLDL2 subfractions in the different dyslipidaemias

2007

Very low density lipoprotein (VLDL) 1 and 2 were fractionated by heparin affinity chromatography into a bound and an unbound fraction and the different subfractions were quantified in 17 normolipidaemic (NL), 13 hypercholesterolaemic (HC), 10 hypertriglyceridaemic (HTG) and 11 combined hyperlipidaemic subjects (CHL). Unbound VLDL1 and VLDL2 were, respectively, 1.9- and 2.2-fold richer in triglycerides than bound VLDL1 and VLDL2. In HTG and CHL the concentration of all the VLDL subfractions was increased and plasma triglyceride level was correlated to unbound VLDL1 and to bound VLDL1 (respectively, r=0.86 (p<0.001) and r=0.77 (p<0.01) in HTG and r=0.73 (p<0.001) and r=0.62 (p<0.05) in CHL). …

AdultMalemedicine.medical_specialtyVery low-density lipoproteinHypercholesterolemiaHyperlipidemia Familial CombinedLipoproteins VLDLChromatography AffinityGlycosaminoglycanchemistry.chemical_compoundAffinity chromatographyInternal medicinemedicineHumansApolipoproteins BDyslipidemiasHypertriglyceridemiaTriglycerideHeparinCholesterolHypertriglyceridemiaAnticoagulantsHeparinMiddle Agedmedicine.diseaseEndocrinologychemistryFemaleCardiology and Cardiovascular MedicineUltracentrifugationProtein BindingLipoproteinmedicine.drugAtherosclerosis
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Association of Serum Retinol Binding Protein 4 with Atherogenic Dyslipidemia in Morbid Obese Patients

2013

Retinol binding protein 4 (RBP4) is an adipokine that may contribute to the development of insulin resistance. However, how this adipokine is affected and its possible involvement in lipid metabolism in obese patients with varying degrees of insulin resistance is yet to be determined. A total of 299 middle-aged morbid obese patients (BMI>40 kg/m(2)) were divided in euglycemic, metabolic syndrome or type 2 diabetic. Anthropometric measurements, biochemical variables and systemic RBP4 levels were determined. RBP4 levels were significantly higher in patients with metabolic syndrome and type 2 diabetes than in euglycemic subjects (42.9±14.6; 42.3±17.0 and 37.4±11.7 µg/ml, respectively) and corr…

AdultMalemedicine.medical_specialtyVery low-density lipoproteinScienceCholesterol VLDLAdipokineType 2 diabetesBody Mass Indexchemistry.chemical_compoundInsulin resistanceInternal medicinemedicineHumansTriglyceridesDyslipidemiasMetabolic SyndromeRetinol binding protein 4MultidisciplinarybiologyCholesterolbusiness.industryCholesterol HDLQRCholesterol LDLMiddle AgedAtherosclerosisLipid Metabolismmedicine.diseaseObesity MorbidRetinol binding proteinEndocrinologyDiabetes Mellitus Type 2chemistryMultivariate Analysisbiology.proteinMedicineFemaleInsulin ResistanceMetabolic syndromebusinessRetinol-Binding Proteins PlasmaResearch ArticlePLoS ONE
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Prevalence of dyslipidemia and associated risk factors in Turkish adults.

2013

Background and Objectives Dyslipidemia is a modifiable major risk factor for coronary heart disease. The objective of this study was to determine the prevalence of dyslipidemia among Turkish adults and its associations with other cardiovascular risk factors. Methods This study included 4309 people ages 20 to 83 years old from 7 provinces of Turkey. People from the city centers, districts, and villages were selected by a stratified sampling method. Weight, height, and waist and hip circumferences were measured. Blood samples were obtained to determine glucose, total cholesterol (TC), high-density lipoprotein cholesterol (HDL-C), low-density lipoprotein cholesterol (LDL-C), and triglycerides …

AdultMalemedicine.medical_specialtyWaistTurkeygenetic structuresEndocrinology Diabetes and MetabolismCoronary DiseaseBody Mass Indexchemistry.chemical_compoundHigh-density lipoproteinCholesterol Dyslipidemia High-density lipoprotein Low-density lipoprotein Prevalence Triglycerides TurkeyRisk FactorsInternal medicinePrevalenceInternal MedicinemedicineHumansRisk factorNational Cholesterol Education ProgramTriglyceridesAgedDyslipidemiasAged 80 and overNutrition and Dieteticsbusiness.industryCholesterolBody WeightCholesterol HDLnutritional and metabolic diseasesCholesterol LDLMiddle Agedmedicine.diseaseCholesterolBlood pressurechemistryPhysical therapyFemalelipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicinebusinessBody mass indexDyslipidemia
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Effect of fish oil on LDL oxidation and plasma homocysteine concentrations in health.

2003

Oxidation of low-density lipoprotein (LDL) and hyperhomocysteinemia are believed to play a role in therogenesis. Whether n-3 polyunsaturated fatty acids increase LDL susceptibility to oxidation or influence homocysteine (Hcy) metabolism has long been a subject of controversy. In this study, we evaluated the effect of 8 weeks of dietary supplementation with 6 g/day of fish oil (FO; 3 g of n-3 fatty acids) on plasma lipoproteins, in vitro LDL peroxidation, antioxidant status, and plasma Hcy concentrations in 16 normolipidemic subjects. FO rapidly and significantly (P < .01) decreased plasma total and very low density lipoprotein triglyceride concentrations and had no effect on LDL or high-den…

AdultMalemedicine.medical_specialtyalpha-TocopherolNitric OxideThiobarbituric Acid Reactive SubstancesPathology and Forensic Medicinechemistry.chemical_compoundHigh-density lipoproteinFish OilsDietary Fats UnsaturatedInternal medicineFatty Acids Omega-3medicineHumansHomocysteineNitritesTriglycerideschemistry.chemical_classificationNitratesTriglycerideCholesterolCholesterol HDLGeneral MedicineCholesterol LDLMiddle Agedbeta CaroteneEicosapentaenoic acidGlutathioneLipoproteins LDLEndocrinologychemistryBiochemistryDocosahexaenoic acidLow-density lipoproteinFemaleLipid PeroxidationCopperPolyunsaturated fatty acidLipoproteinThe Journal of laboratory and clinical medicine
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Nitric oxide production and endothelium-dependent vasorelaxation ameliorated by N1-methylnicotinamide in human blood vessels.

2012

N 1 -methylnicotinamide (MNA + ) has until recently been thought to be a biologically inactive product of nicotinamide metabolism in the pyridine nucleotides pathway. However, the latest observations imply that MNA + may exert antithrombotic and anti-inflammatory effects through direct action on the endothelium. We examined both in vivo and in vitro whether the compound might induce vasorelaxation in human blood vessels through the improvement of nitric oxide (NO) bioavailability and a reduction of oxidative stress mediated by endothelial NO synthase (eNOS) function. MNA + treatment (100 mg/m 2 orally) in healthy normocholesterolemic and hypercholesterolemic subjects increased the l-argini…

AdultNiacinamidemedicine.medical_specialtyEndotheliumBrachial ArteryNitric Oxide Synthase Type IIIHypercholesterolemiachemistry.chemical_elementCalciumIn Vitro Techniquesmedicine.disease_causeNitric OxideNitric oxidechemistry.chemical_compoundN^{1}-methylnicotinamideDouble-Blind MethodEnosnitric oxideInternal medicineInternal MedicinemedicineHumansflow-mediated dilationCalcimycinCells Culturedendothelial nitric oxide synthaseoxidized low-density lipoproteinbiologyDose-Response Relationship DrugChemistrySuperoxidebiology.organism_classificationendothelial cellsAcetylcholineOxygenVasodilationOxidative Stressmedicine.anatomical_structureEndocrinologysuperoxideEndothelium VascularAcetylcholineOxidative stressmedicine.drugLipoproteinHypertension (Dallas, Tex. : 1979)
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Influence of obesity on atherogenic dyslipidemia in women with polycystic ovary syndrome.

2013

Background Obesity is known to underlie, at least partially, dyslipidemia in polycystic ovary syndrome (PCOS), but it is unclear whether PCOS status per se increases the risk of alterations of lipoprotein subfractions, which differ in size and atherogenic potential. Our objective was to evaluate whether PCOS influences lipoprotein profile and LDL and HDL subfractions and to study the impact of obesity on these parameters. Materials and methods This was a case–control study conducted in an academic medical centre. The study population consisted of 54 women of fertile age with PCOS and 60 controls adjusted for age and BMI. Biochemical lipid profile and LDL and HDL lipoprotein subfractions (me…

AdultVery low-density lipoproteinmedicine.medical_specialtyendocrine system diseasesClinical BiochemistryBiochemistryBody Mass Indexchemistry.chemical_compoundRisk FactorsInternal medicinemedicineHumansObesityDyslipidemiasmedicine.diagnostic_testCholesterolbusiness.industryCholesterol HDLCase-control studynutritional and metabolic diseasesCholesterol LDLGeneral MedicineAtherosclerosismedicine.diseasePolycystic ovaryfemale genital diseases and pregnancy complicationsEndocrinologychemistryCase-Control StudiesFemalelipids (amino acids peptides and proteins)Lipid profilebusinessBody mass indexDyslipidemiaPolycystic Ovary SyndromeLipoprotein
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