Search results for "Lung"

showing 10 items of 2389 documents

Ultrastructural study on human lung in alveolitis versus pulmonary fibrosis

1993

Lung specimens of 21 patients with diffuse interstitial lung disease were examined. The present ultrastructural study outlines the topography and distribution of inflammatory changes in the interstitium, endothelium, and in pneumocytes and phagocytes. Alveolitis is characterized by marked regenerative activity of type II pneumocytes (cuboid metaplasia), intraluminal macrophage accumulation, endothelial swelling, multilamination of the endothelial basement membrane, pericapillary edema, and primarily by cellular infiltrates in the interstitial space. The most prominent feature of the interstitium in pulmonary fibrosis is the lack of immunoinflammatory cells. In some areas there is a marked a…

AdultMalePathologymedicine.medical_specialtyEndotheliumPulmonary FibrosisEpitheliumInterstitial spaceEdemaMacrophages AlveolarDrug DiscoveryPulmonary fibrosismedicineHumansLymphocytesLungGenetics (clinical)InflammationBasement membraneMetaplasiaLungbusiness.industryPneumonia PneumocystisType-II PneumocytesInterstitial lung diseaseGeneral MedicineMiddle Agedrespiratory systemmedicine.diseaseCapillariesmedicine.anatomical_structureImmunologyMolecular MedicineFemalemedicine.symptombusinessAlveolitis Extrinsic AllergicThe Clinical Investigator
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Oncogene overexpression in non-small-cell lung cancer tissue: prevalence and clinicopathological significance.

1994

In contrast to small-cell lung cancer, few data are available on the role of oncogene overexpression in non-small-cell lung cancers (NSCLC). To determine the prevalence and extent of the transcriptional activation of cancer genes in NSCLC we investigated the level of mRNA of the three important cellular oncogenes — erbB2, Ki-ras, and c-myc — in 39 surgically or endoscopically obtained tumor samples and 24 samples of normal bronchopulmonary tissue taken from the same patients. Tissue RNA was prepared and the specific mRNA analyzed by the highly sensitive nuclease S1 protection assay. Oncogene mRNA in the tumors was quantified by comparison with the homogeneously weak signals in normal lung t…

AdultMalePathologymedicine.medical_specialtyLung NeoplasmsAdolescentBiologyCarcinoma Non-Small-Cell LungDrug DiscoveryGene expressionmedicineCarcinomaHumansLung cancerGenetics (clinical)AgedAged 80 and overMessenger RNAOncogeneCancerOncogenesMiddle Agedmedicine.diseaseMolecular medicineGene Expression Regulation NeoplasticCancer researchMolecular MedicineAdenocarcinomaFemaleThe Clinical investigator
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Functional, biochemical and morphological studies on human bronchi after cryopreservation

1995

1. Human isolated bronchi have been investigated as fresh tissue or after storage (7 and 30 days) at -196 degrees C in foetal calf serum containing 1.8 M dimethyl sulphoxide. 2. After cryopreservation, the maximal contractile response to acetylcholine (3 mM) was reduced (approximately 25%) but the difference did not reach significance statistically. Maximal responses to other spasmogens tested (histamine, [Nle10]NKA(4-10), bradykinin, leukotriene D4, U46619, and KCl) did not differ between unfrozen and frozen/thawed tissues. The sensitivity of cryopreserved tissues to the constrictor agents tested was similar to that of fresh tissues. 3. The accumulation of inositol phosphates produced by a…

AdultMalePathologymedicine.medical_specialtyLung NeoplasmsInositol PhosphatesBronchiIn Vitro TechniquesBiologyTritiumCryopreservationAndrologychemistry.chemical_compoundFresh TissueIsoprenalinemedicineHumansDimethyl SulfoxideRolipramAgedCryopreservationPharmacologyMicroscopyMiddle AgedAcetylcholinechemistryFemaleSodium nitroprussidemedicine.symptomAcetylcholineHistamineMuscle ContractionResearch Articlemedicine.drugMuscle contractionBritish Journal of Pharmacology
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CT-guided intratumoral gene therapy in non-small-cell lung cancer.

1999

The objective of this study was to prove the principle of CT-guided gene therapy by intratumoral injection of a tumor suppressor gene as an alternative treatment approach of incurable non-small-cell lung cancer. In a prospective clinical phase I trial six patients with non-small-cell lung cancer and a mutation of the tumor suppressor gene p53 were treated by CT-guided intratumoral gene therapy. Ten milliliters of a vector solution (replication-defective adenovirus with complete wild-type p53 cDNA) were injected under CT guidance. In four cases the vector solution was completely applied to the tumor center, whereas in two cases 2 ml aliquots were injected into different tumor areas. For the …

AdultMalePathologymedicine.medical_specialtyLung NeoplasmsTumor suppressor geneAdolescentGenetic enhancementGenetic VectorsDNA RecombinantInjections IntralesionalPolymerase Chain ReactionAdenoviridaeCarcinoma Non-Small-Cell LungBiopsyCarcinomaMedicineHumansRadiology Nuclear Medicine and imagingProspective StudiesProspective cohort studyAdverse effectLung cancerAgedmedicine.diagnostic_testbusiness.industryGene Transfer TechniquesGeneral MedicineGenetic TherapyMiddle Agedmedicine.diseaseGenes p53Clinical trialTreatment OutcomeMutationFemalebusinessTomography X-Ray ComputedFollow-Up StudiesEuropean radiology
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A phase I study of adenovirus-mediated wild-type p53 gene transfer in patients with advanced non-small cell lung cancer.

1998

Mutations of the tumor suppressor gene p53 are the most common genetic alterations observed in human cancer. Loss of wild-type p53 function impairs cell cycle arrest as well as repair mechanisms involved in response to DNA damage. Further, apoptotic pathways as induced by radio- or chemotherapy are also abrogated. Gene transfer of wild-type p53 was shown to reverse these deficiencies and to induce apoptosis in vitro and in preclinical in vivo tumor models. A phase I dose escalation study of a single intratumoral injection of a replication-defective adenoviral expression vector encoding wild-type p53 was carried out in patients with incurable non-small cell lung cancer. All patients enrolled…

AdultMalePathologymedicine.medical_specialtyLung NeoplasmsTumor suppressor geneAdolescentmedicine.medical_treatmentGenetic enhancementGenetic Vectorsmedicine.disease_causeAdenoviridaeInjectionsIn vivoCarcinoma Non-Small-Cell LungGeneticsMedicineHumansRNA MessengerMortalityLung cancerMolecular BiologyAgedRegulation of gene expressionChemotherapyExpression vectorbusiness.industryGene Transfer TechniquesGenetic TherapyMiddle Agedmedicine.diseaseGenes p53AdenoviridaeGene Expression Regulation NeoplasticTreatment OutcomeCancer researchMolecular MedicineFemalebusinessHuman gene therapy
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A macrophage-suppressing 40-kD protein in a case of pulmonary alveolar proteinosis.

1987

Pulmonary alveolar proteinosis (PAP) is a rare disease of unknown etiology. Macrophage dysfunctions are claimed to be involved in the pathogenesis. We investigated phagocytosis and oxidative metabolism of alveolar macrophages in a case of pulmonary alveolar proteinosis. These cells phagocytize normally and phagocytizable stimulants cause a normal oxidative burst. In response to the membrane signals phorbolmyristate acetate and aggregated immunoglobulin, however, no stimulated turnover of the oxidative metabolism can be observed. A 40-kD protein found in the lavage fluid mediates this macrophage-inhibiting effect. This phenomenon may contribute to the frequent opportunistic infections seen i…

AdultMalePathologymedicine.medical_specialtyPhagocytosisOpportunistic InfectionsPulmonary Alveolar ProteinosisPathogenesisPhagocytosisDrug DiscoverymedicineMacrophageHumansMacrophage Migration-Inhibitory FactorsGenetics (clinical)Lungmedicine.diagnostic_testbiologyMacrophagesfood and beveragesProteinsGeneral MedicineMacrophage Activationmedicine.diseaseRespiratory burstMolecular WeightPulmonary AlveoliBronchoalveolar lavagemedicine.anatomical_structureImmunologyLuminescent Measurementsbiology.proteinMolecular MedicineAntibodyPulmonary alveolar proteinosisEnergy MetabolismBronchoalveolar Lavage FluidKlinische Wochenschrift
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Ewing-like sarcoma with CIC-DUX4 gene fusion in a patient with neurofibromatosis type 1. A hitherto unreported association.

2015

Sarcoma with CIC-DUX4 gene fusion is emerging as the most prevalent subset of Ewing-like undifferentiated small round cell sarcomas with around 50 cases published. We report hereby the case of a 40-year-old male who presented a CIC-DUX4 sarcoma in deep soft tissues in his thigh. He had been diagnosed with neurofibromatosis type 1 at age 19 and over the years underwent resection of multiple neural neoplasms, including two malignant peripheral nerve sheath tumors with classical spindle-cell histopathology. The CIC-DUX4 sarcoma was treated with surgical resection, radiation and chemotherapy, but lung and brain metastases developed and the patient died from the disease 14 months after diagnosis…

AdultMalePathologymedicine.medical_specialtySoft Tissue NeoplasmLung NeoplasmsNeurofibromatosis 1Oncogene Proteins Fusionmedicine.medical_treatmentSoft Tissue NeoplasmsThighBiologyPathology and Forensic MedicineFusion geneFatal OutcomemedicineHumansNeurofibromatosisChemotherapyBrain NeoplasmsSoft tissueCell Biologymedicine.diseasemedicine.anatomical_structureSarcoma Small CellHistopathologySarcomaPathology, research and practice
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Morphological Alterations and Stress Protein Variations in Lung Biopsies Obtained from Autopsies of COVID-19 Subjects

2021

Molecular chaperones, many of which are heat shock proteins, play a role in cell stress response and regulate the immune system in various ways, such as in inflammatory/autoimmune reactions. It would be interesting to study the involvement of these molecules in the damage done to COVID-19-infected lungs. In our study, we performed a histological analysis and an immunomorphological evaluation on lung samples from subjects who succumbed to COVID-19 and subjects who died from other causes. We also assessed Hsp60 and Hsp90 distribution in lung samples to determine their location and post-translational modifications. We found histological alterations that could be considered pathognomonic for CO…

AdultMalePathologymedicine.medical_specialtyendotheliumEndotheliumQH301-705.5Hsp90InflammationArticleImmune systemCOVID-19EndotheliumHsp60Hsp90InflammationSARS-CoV-2Heat shock proteinSARS-CoV-2; COVID-19; Hsp60; Hsp90; endothelium; inflammationmedicineHumansBiology (General)LungHeat-Shock ProteinsAgedInflammationLungSARS-CoV-2Settore BIO/16 - Anatomia Umanabusiness.industryCOVID-19Endothelial CellsColocalizationGeneral MedicineMiddle AgedHsp60medicine.anatomical_structureImmunohistochemistryFemaleHSP60Autopsymedicine.symptombusinessCells; Volume 10; Issue 11; Pages: 3136
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Diffusion-weighted MRI of the lung with hyperpolarized helium-3: A study of reproducibility

2005

Purpose To determine the reproducibility of several parameters of the ADC measurement by calculating the scan-to-scan intrasubject variability. Materials and Methods Measurements were performed using a gradient-echo sequence with a bipolar gradient for diffusion weighting (b = 3.89 sec/cm2). Five patients with pulmonary emphysema, and six healthy-lung volunteers were included in the study. Images were acquired after inspiration of 3He during a single inspiratory breath-hold. To assess the reproducibility, the measurement was performed twice (time between measurements = 20 minutes) without repositioning the subjects. Analysis was performed on the basis of region-of-interest (ROI) analysis an…

AdultMalePercentileHyperpolarized Helium 3HeliumStatistics NonparametricStandard deviationAdministration InhalationImage Processing Computer-AssistedHumansMedicineRadiology Nuclear Medicine and imagingAgedReproducibilityLungDiffusion weightingbusiness.industrySignificant differenceReproducibility of ResultsMiddle AgedDiffusion Magnetic Resonance Imagingmedicine.anatomical_structurePulmonary EmphysemaFemaleNuclear medicinebusinessDiffusion MRIJournal of Magnetic Resonance Imaging
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Apoptosis resistance in epithelial tumors is mediated by tumor-cell-derived interleukin-4

2008

We investigated the mechanisms involved in the resistance to cell death observed in epithelial cancers. Here, we identify that primary epithelial cancer cells from colon, breast and lung carcinomas express high levels of the antiapoptotic proteins PED, cFLIP, Bcl-xL and Bcl-2. These cancer cells produced interleukin-4 (IL-4), which amplified the expression levels of these antiapoptotic proteins and prevented cell death induced upon exposure to TRAIL or other drug agents. IL-4 blockade resulted in a significant decrease in the growth rate of epithelial cancer cells and sensitized them, both in vitro and in vivo, to apoptosis induction by TRAIL and chemotherapy via downregulation of the antia…

AdultMaleProgrammed cell deathLung NeoplasmsTime Factorsapoptosis interleukin-4 cancer stem cells cancer chemiotherapy cytokinesCASP8 and FADD-Like Apoptosis Regulating Proteinbcl-X ProteinAntineoplastic AgentsApoptosisBreast NeoplasmsBiologyTNF-Related Apoptosis-Inducing LigandTumor Cells CulturedmedicineHumansAutocrine signallingMolecular BiologyInterleukin 4AgedCell ProliferationSettore MED/04 - Patologia GeneraleCell DeathDose-Response Relationship DrugCell growthCarcinomaIntracellular Signaling Peptides and ProteinsAntibodies MonoclonalInterleukin-4 Receptor alpha SubunitCorrectionCancerCell BiologyMiddle AgedPhosphoproteinsmedicine.diseaseUp-RegulationCell biologyAutocrine CommunicationProto-Oncogene Proteins c-bcl-2Drug Resistance NeoplasmApoptosisColonic NeoplasmsCancer cellFemaleInterleukin-4Interleukin-4 Cancer stem cellsSignal transductionApoptosis Regulatory ProteinsSignal Transduction
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