Search results for "Lung"

showing 10 items of 2389 documents

2′O-galloylhyperin attenuates LPS-induced acute lung injury via up-regulation antioxidation and inhibition of inflammatory responses in vivo

2019

2'O-galloylhyperin, an active flavonol glycoside compound with remarkable anti-immune activity, was isolated from Pyrola [P. incarnata Fisch.]. However, the evidence of anti-inflammatory activity in pulmonary diseases was still not convincing. The aim of the present study was (1) to investigate the effect of 2'O-galloylhyperin on LPS-induced acute lung injury in mice, and (2) to identify the mechanisms of attenuation of inflammatory responses. The results demonstrated that 2'O-galloylhyperin significantly reduced LPS-induced inflammation damage in a dose-dependent manner. After LPS challenge, treatment with 2'O-galloylhyperin reduced the production of pro-inflammatory cytokines and chemokin…

LipopolysaccharidesMale0301 basic medicineMAPK/ERK pathwayp38 mitogen-activated protein kinasesAcute Lung InjuryMolecular ConformationInflammationPharmacologyLung injuryToxicologyAntioxidantsMiceStructure-Activity Relationship03 medical and health sciences0302 clinical medicineGallic AcidmedicineAnimalsProtein kinase AInflammationMice Inbred ICRDose-Response Relationship DrugChemistryAnti-Inflammatory Agents Non-SteroidalAMPKGeneral MedicineUp-Regulation030104 developmental biology030220 oncology & carcinogenesisPhosphorylationQuercetinmedicine.symptomSignal transductionChemico-Biological Interactions
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CD11c+ Alveolar Macrophages are a Source of IL-23 During Lipopolysaccharide-Induced Acute Lung Injury

2013

Acute lung injury (ALI) is a severe pulmonary disease causing high numbers of fatalities worldwide. Innate immune responses are an integral part of the pathophysiologic events during ALI. Interleukin 23 (IL-23) is a proinflammatory mediator known to direct the inflammatory responses in various settings of infection, autoimmunity, and cancer. Interleukin 23 has been associated with proliferation and effector functions in T(H)17 cells. Surprisingly, little is known about production of IL-23 during ALI. In this study, we found expression of mRNA for IL-23p19 to be 10-fold elevated in lung homogenates of C57BL/6 mice after lipopolysaccharide (LPS)-induced ALI. Likewise, concentrations of IL-23 …

LipopolysaccharidesMaleLipopolysaccharideAcute Lung InjuryCD11cBiologyLung injuryCritical Care and Intensive Care MedicineInterleukin-23ArticleProinflammatory cytokineMicechemistry.chemical_compoundMacrophages AlveolarmedicineAnimalsInnate immune systemmedicine.diagnostic_testrespiratory systemCD11c Antigenrespiratory tract diseasesBronchoalveolar lavagechemistryImmunologyEmergency MedicineAlveolar macrophageInterleukin 17Shock
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Cationic Proteins Inhibit l-Arginine Uptake in Rat Alveolar Macrophages and Tracheal Epithelial Cells

1999

Eosinophil-derived cationic proteins play an essential role in the pathogenesis of bronchial asthma. We tested whether cationic proteins interfere with the cationic amino-acid transport in alveolar macrophages (AMPhi) and tracheal epithelial cells, and whether L-arginine-dependent pathways were affected. The effect of cationic polypeptides on cellular uptake of [(3)H]-L-arginine, nitrite accumulation, and the turnover of [(3)H]-L-arginine by nitric oxide (NO) synthase and arginase (formation of [(3)H]-L-citrulline and [(3)H]-L-ornithine, respectively) were studied. Poly-L-arginine reduced [(3)H]-L-arginine uptake in rat AMPhi and tracheal epithelial cells in a concentration-dependent manner…

LipopolysaccharidesMalePulmonary and Respiratory MedicineTime FactorsClinical BiochemistryGene ExpressionArginineNitric OxideNitric oxideRats Sprague-DawleyPathogenesischemistry.chemical_compoundRibonucleasesFibrinolytic AgentsMacrophages AlveolarAnimalsNitriteLungMolecular BiologyNitritesArginaseDose-Response Relationship DrugbiologyATP synthaseHeparinLysineCationic polymerizationEpithelial CellsBlood ProteinsCell BiologyEosinophil Granule ProteinsProtamineRatsTracheaArginaseBiochemistrychemistryMajor basic proteinbiology.proteinCitrullineFemaleAmerican Journal of Respiratory Cell and Molecular Biology
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LTB4 is present in exudative pleural effusions and contributes actively to neutrophil recruitment in the inflamed pleural space

2004

SUMMARY The pleural space is a virtual compartment between the lung and chest wall that becomes filled with fluid and inflammatory cells during a variety of respiratory diseases. Here, we study the potential role of the eicosanoid metabolite leukotriene B4 (LTB4) in disparate diseases leading to acute (pneumonia) or chronic (tuberculosis, cancer) inflammation of the pleural space. LTB4 concentrations were significantly higher in pleural fluid due to pneumonia, tuberculosis and cancer with respect to congestive heart failure and correlated with neutrophil elastase, which is used as an indication of state of activation of neutrophils in the pleural space. Moreover, pleural LTB4 was biological…

LipopolysaccharidesPathologyHot TemperatureNeutrophilsLeukotriene B4Gene ExpressionEpitheliumchemistry.chemical_compoundNeoplasmsClinical StudiesImmunology and AllergyMedicineRespiratory systemPancreatic ElastasebiologyNeutrophilMiddle Agedrespiratory systemChemotaxis Leukocytemedicine.anatomical_structureNeutrophil InfiltrationNeutrophil elastaseLTB4Pleuralipids (amino acids peptides and proteins)medicine.symptomAdultmedicine.medical_specialtyImmunologyInflammationGranulocyteLeukotriene B4HumansRNA MessengerTuberculosis PulmonaryAgedArachidonate 5-LipoxygenaseLungbusiness.industryPneumoniaMacrophage Activationmedicine.diseaserespiratory tract diseasesPleural EffusionPneumoniaEicosanoidchemistryImmunologybiology.proteinbusinessClinical and Experimental Immunology
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Overview of the pathology of three widely used animal models of acute lung injury

2007

Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are syndromes of acute diffuse damage to the pulmonary parenchyma by a variety of local or systemic insults. Increased alveolar capillary membrane permeability was recognized as the common end organ injury and a central feature in all forms of ALI/ARDS. Although great strides have been made in understanding the pathogenesis of ALI/ARDS and in intensive care medicine, the treatment approach to ARDS is still relying on ventilatory and cardiovascular support based on the recognition of the clinical picture. In the course of evaluating novel treatment approaches to ARDS, 3 models of ALI induced in different species, i.e. the…

LipopolysaccharidesPathologymedicine.medical_specialtyARDSSwineAcute respiratory distressLung injuryBronchoalveolar LavagePathogenesisDogsAnimalsHumansMedicineDiffuse alveolar damageIntensive care medicineLungPathologicalRespiratory Distress SyndromeSheepmedicine.diagnostic_testbusiness.industryrespiratory systemmedicine.diseaseDisease Models AnimalBronchoalveolar lavageSurgerybusinessOleic AcidAcute diffuse
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Navigating the liquid biopsy Minimal Residual Disease (MRD) in Non-Small Cell Lung Cancer: Making the Invisible Visible.

2022

Liquid biopsy has gained increasing interest in the growing era of precision medicine as minimally invasive technique. Recent findings demonstrated that detecting minimal or molecular residual disease (MRD) in NSCLC is a challenging matter of debate that need multidisciplinary competencies, avoiding the overtreatment risk along with achieving a significant survival improvement. This review aims to provide practical consideration for solving data interpretation questions about MRD in NSCLC thanks to the close cooperation between biologists and oncology clinicians. We discussed with a translational approach the critical point of view from benchside, bedside and bunchside to facilitate the fut…

Liquid biopsyNon-small cell lung cancerOncologyMinimal residual diseaseNGSHematologyCancer diagnosticsCritical reviews in oncology/hematology
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Liver Fibrosis and Metabolic Alterations in Adults With alpha-1-antitrypsin Deficiency Caused by the Pi*ZZ Mutation

2019

Background & Aims Alpha-1 antitrypsin deficiency (AATD) is among the most common genetic disorders. Severe AATD is caused by a homozygous mutation in the SERPINA1 gene that encodes the Glu342Lys substitution (called the Pi*Z mutation, Pi*ZZ genotype). Pi*ZZ carriers may develop lung and liver diseases. Mutation-associated lung disorders have been well studied, but less is known about the effects in liver. We assessed the liver disease burden and associated features in adults with this form of AATD. Methods We collected data from 554 Pi*ZZ adults (403 in an exploratory cohort, 151 in a confirmatory cohort), in 9 European countries, with AATD who were homozygous for the Pi*Z mutation, and 234…

Liver CirrhosisMale0301 basic medicineALTLiver diseasechemistry.chemical_compound0302 clinical medicineLiver Function TestsRisk FactorsGenotypeRare Liver DiseaseAlpha 1-antitrypsin deficiencyHomozygoteAge FactorsGastroenterologyMiddle AgedEuropeEditorial CommentaryPhenotypemedicine.anatomical_structureLiverElasticity Imaging TechniquesFemale030211 gastroenterology & hepatologyTEAdultmedicine.medical_specialty610Mice Transgenic03 medical and health sciencesSex Factorsalpha 1-Antitrypsin DeficiencyInternal medicinemedicinePiAnimalsHumansGenetic Predisposition to DiseaseASTAgedLungHepatologyTriglyceridebusiness.industryLipid Metabolismmedicine.diseaseFatty Liver030104 developmental biologyEndocrinologychemistryCase-Control Studiesalpha 1-AntitrypsinMutationSteatosisTransient elastographybusinessGastroenterology
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Coprology of Panthera tigris altaica and Felis bengalensis euptilurus from the Russian Far East

2007

Fecal samples from the Siberian tiger (Panthera tigris altaica) and the Amur cat (Felis bengalensis euptilurus) from Far Eastern Russia, were examined for parasites. A natural sedimentation methodology was used and a complete examination of all the sediment was performed. This fecal investigation allowed us to isolate and identify several developmental stages of gastrointestinal, hepatic, and respiratory parasites. Five parasites were found from P. t. altaica: 11 trematodes (Platynosomum fastosum) and 4 nematodes (Strongyloides sp., Ancylostomatidae, Toxascaris leonina, and Toxocara cati). Five parasites were found from F. b. euptilurus: 1 cestode (Diplopylidium sp.) and 4 nematodes (Trichu…

Liver Diseases ParasiticLung Diseases ParasiticParasitic Diseases AnimalZoologyFecesToxocara catibiology.animalFelisPrevalenceAnimalsParasitologíaZoologíaIntestinal Diseases ParasiticTigersEcology Evolution Behavior and SystematicsAmur cat (Felis bengalensis euptilurus)Toxascaris leoninaAelurostrongylus abstrususbiologyAncylostomatidaeEcologyFelisSiberian tiger (Panthera tigris altaica)Ecologíabiology.organism_classificationSiberiaStrongyloidesParasitologyPantheraCoprologySiberian tiger
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Zur Erinnerung an Julius Gustav Petersen, Livländischen Gouvernements-Procureur, geb. am 17. August 1806, gest. am 17. Mai 1844

1844

"von C.A. Berkholz, Oberpastor zu St. Jacob in Riga"

Livländischen Gouvernements - Procureur:HUMANITIES and RELIGION [Research Subject Categories]TrauerversammlungTeoloģijaPetersen Julius Gustav - TrauerversammlungBēru sprediķis
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Materialien zur Kenntniss der livländischen Bauer-Verhältnisse: veröffentlicht von dem livländischen Landraths-Collegium

1883

Inhalt: Vorbemerkung... 5 Erster Abschnitt: Die agrare Entwickelurg Livlands... 7 I. Die Höhe der livländischen Bauerlandpachten... 8 II. Die Höhe des Kaufpreises der livländischen Bauerlandgesinde... 11 III. Das Verhältniss des verpachteten zum verkauften Bauerlande w 14 Zweiter Abschnitt: Die Organisation der Selbstverwaltung... 17 I. Die Selbstverwaltung... 17 II. Die livländische Volksschule... 21 III. Die Steuer-Verhältnisse... 25 IV. Die Agrar-Gesetzgebung... 35 Tabelle

Livonijas lauksaimniecībaAgrāra politika - Vidzemes guberņaLivlands GeschichteLivlands Bauerverhältnisse:HUMANITIES and RELIGION::History and philosophy subjects::History subjects [Research Subject Categories]Livlands AgrarentwicklungZemnieki - Vidzemes guberņaLivonian historyLivonijas vēstureLivlands BauernLivonian agriculture
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