Search results for "Lupus Erythematosu"

showing 10 items of 142 documents

SPARC regulation of PMN clearance protects from pristane induced lupus and rheumatoid arthritis

2020

AbstractOne step along the pathogenesis of Systemic lupus erythematosus (SLE) is associated with polymorphonuclear leukocyte (PMN) death and their ineffective removal by M2-macrophages. The secreted protein acidic and rich in cysteine (SPARC) is a matricellular protein with unexpected immunosuppressive function in M2-macrophages and myeloid cells. To investigate the role of SPARC in autoimmunity, we adopted a pristane–induced model of lupus in mice, which recapitulates clinical manifestations of human SLE. Sparc-/- mice developed earlier and more severe renal disease, lung and liver parenchymal damage than the WT counterpart. Most prominently, Sparc-/- mice had anticipated and severe occurr…

LungSystemic lupus erythematosusbusiness.industryMatricellular proteinArthritisDendritic cellmedicine.diseasemedicine.disease_causeAutoimmunityPathogenesismedicine.anatomical_structureRheumatoid arthritisImmunologymedicineCancer researchMacrophagebusiness
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Systemic lupus erythemathosus between clinical practice and the laboratory: state of the art and new findings on anti-DNA autoantibodies

2002

Identification of autoantibodies directed against nuclear antigens is a very important finding in the assessment of autoimmune rheumatic diseases. In particular, the anti-DNA autoantibodies have assumed a fundamental importance, both speculative and clinical, in the study of the systemic lupus erythemathous. The aim of the present review is to focalize on anti-DNA the mechanisms of both induction and production of anti-DNA autoantibodies, pathophysiologic and diagnostic and clinical aspects. For this purpose, forty years of studies on this topic have been reviewed. Aspects on different conformational shapes of double-stranded DNA have been discussed such as related pathogenetic and diagnost…

Lupus Erythematosus Systemic/immunologySettore MED/16 - ReumatologiaSettore MED/09 - Medicina InternaLupus Erythematosus Systemic/diagnosiNucleosomeAntibodies Antinuclear/immunologyLupus Erythematosus Systemic/therapyHuman
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Complement C1q and C8beta deficiency in an individual with recurrent bacterial meningitis and adult-onset systemic lupus erythematosus-like illness.

2008

Co-existing complement C8 deficiency ameliorated the SLE associated with C1q deficiency.

Lupus erythematosusLupus Erythematosusbusiness.industryComplementmedicine.diseaseLupus ErythematosuRheumatologyBacterial MeningitisImmunologymedicinePharmacology (medical)Bacterial meningitisComplement; Lupus Erythematosus; Bacterial MeningitisRecurrent bacterial meningitisbusinessMeningitisComplement C1qAnti-SSA/Ro autoantibodies
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Ādas bojājumi pacientiem ar sistēmas sarkano vilkēdi Latvijā

2015

Tēmas aktualitāte: sistēmas sarkanā vilkēde nav bieži sastopama slimība un tā bieži vien paliek nediagnosticēta, kā rezultātā pacienti nesaņem atbilstošu terapiju. Tas var veicināt slimības straujāku attīstību un būtiski samazināt pacienta dzīvildzi. Slimības ādas forma bieži tiek ārstēta kā kāda cita ādas slimība. Ļoti svarīga ir slimības agrīna diagnostika un savlaicīga terapijas uzsākšana. Darba mērķis: noteikt sistēmas sarkanās vilkēdes izplatību Latvijā, noskaidrot ādas formas izplatības biežumu, biežākās imunoloģiskās izmaiņas un pielietoto terapiju. Materiāli un metodes: Tika veikts retrospektīvs pētījums, apkopota informācija no pacientu slimības vēsturēm laika periodā no 2010. - 20…

Lupus erythematosussistēmas sarkanā vilkēdeMedicīna
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Absence of Anti-Glomerular Basement Membrane Antibodies in 200 Patients With Systemic Lupus Erythematosus With or Without Lupus Nephritis: Results of…

2020

IntroductionAnti-glomerular basement membrane (GBM) antibodies are pathogenic antibodies first detected in renal-limited anti-GBM disease and in Goodpasture disease, the latter characterized by rapidly progressive crescentic glomerulonephritis combined with intra-alveolar hemorrhage. Studies have suggested that anti-GBM antibody positivity may be of interest in lupus nephritis (LN). Moreover, severe anti-GBM vasculitis cases in patients with systemic lupus erythematosus (SLE) have been described in the literature, but few studies have assessed the incidence of anti-GBM antibodies in SLE patients.ObjectiveThe main study objective was to determine if positive anti-GBM antibodies were present …

MaleAnti-Glomerular Basement Membrane Disease[SDV]Life Sciences [q-bio]Lupus nephritisAucunurologic and male genital diseasesSeverity of Illness IndexGastroenterologyanti-glomerular basement membrane antibodies0302 clinical medicinesystemic lupus erythematosusLupus Erythematosus SystemicImmunology and Allergy030212 general & internal medicineOriginal Researchmedicine.diagnostic_testbiologyanti-GBM glomerulonephritisGlomerular basement membraneIIfMiddle Aged3. Good healthTitermedicine.anatomical_structure[SDV.IMM]Life Sciences [q-bio]/ImmunologyFemaleAntibodyVasculitisAdultlcsh:Immunologic diseases. Allergymedicine.medical_specialtyImmunology03 medical and health sciencesAntigenInternal medicineanti-GBM antibodiesmedicineHumansAutoantibodiesRetrospective Studies030203 arthritis & rheumatologylupus nephritisbusiness.industryGoodpasture diseasemedicine.diseaseCase-Control StudiesImmunoassaybiology.proteinbusinesslcsh:RC581-607BiomarkersFrontiers in Immunology
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Per-protocol repeat kidney biopsy portends relapse and long-term outcome in incident cases of proliferative lupus nephritis

2019

Abstract Objectives In patients with LN, clinical and histological responses to treatment have been shown to be discordant. We investigated whether per-protocol repeat kidney biopsies are predictive of LN relapses and long-term renal function impairment. Methods Forty-two patients with incident biopsy-proven active proliferative (class III/IV±V) LN from the database of the UCLouvain were included in this retrospective study. Per-protocol repeat biopsies were performed after a median [interquartile range (IQR)] time of 24.3 (21.3–26.2) months. The National Institutes of Health activity index (AI) and chronicity index (CI) scores were assessed in all biopsies. Results Despite a moderate corre…

MaleBiopsy030232 urology & nephrologyKidneyGastroenterologychemistry.chemical_compound0302 clinical medicinesystemic lupus erythematosusRecurrenceInterquartile rangePharmacology (medical)Proteinuriamedicine.diagnostic_testHazard ratioPrognosisLupus NephritisProteinuriaKidney TubulesCreatinineDisease ProgressionhistopathologyFemaleRenal biopsymedicine.symptomRituximabImmunosuppressive AgentsAdultlong-term outcomemedicine.medical_specialtyRenal functionMethylprednisoloneYoung Adult03 medical and health sciencesrenal biopsyRheumatologyInternal medicineBiopsymedicineHumansImmunologic FactorsCyclophosphamideGlucocorticoidsProportional Hazards ModelsRetrospective Studieslupus nephritisrepeat biopsy030203 arthritis & rheumatologyCreatininebusiness.industryrenal functionMycophenolic AcidchemistryPulse Therapy DrugHistopathologybusinessRheumatology
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Knockout of the KH-Type Splicing Regulatory Protein Drives Glomerulonephritis in MRL-Faslpr Mice

2021

KH-type splicing regulatory protein (KSRP) is an RNA-binding protein that promotes mRNA decay and thereby negatively regulates cytokine expression at the post-transcriptional level. Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by dysregulated cytokine expression causing multiple organ manifestations

MaleChemokineMice Inbred MRL lprQH301-705.5medicine.medical_treatmentLupus nephritisBiologyKidneyArticleImmune systemsystemic lupus erythematosusimmune system diseasesmedicinecytokineAnimalsCD11a AntigenRNA MessengerKSRPBiology (General)skin and connective tissue diseasesRegulation of gene expressionMice KnockoutSystemic lupus erythematosusFOXP3RNA-Binding ProteinsGlomerulonephritisGeneral Medicinemedicine.diseaseMice Inbred C57BLCytokineCancer researchbiology.proteinTrans-ActivatorsFemaleLymph NodesChemokinesBiomarkersglomerulonephritispost-transcriptional regulationCells
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Complete congenital heart block is associated with increased autoantibody titers against calreticulin.

1996

Complete congenital heart block (CCHB) is associated with anti-Ro/SS-A and anti-La/SS-B antibodies. Calreticulin, a calcium-binding, multi-functional protein of the endoplasmic reticulum with C-terminal KDEL-sequence, is not part of the Ro/SS-A ribonucleoprotein complex. In this study anti-calreticulin autoantibody responses in serum samples from 18 infants with CCHB, their mothers and in a control group of 11 anti-Ro/SS-A or anti-La/SS-B positive infants without heart block and their mothers were analysed. Specific enzyme-linked immunosorbent assays were performed. Nine out of 18 sera with CCHB contained IgG anti-calreticulin antibodies. Four sera of those with IgG antibodies also had IgM …

MaleClinical BiochemistryBlotting WesternEnzyme-Linked Immunosorbent AssayBiochemistryAutoantigensImmunopathologymedicineHumansNeonatal lupus erythematosusAutoantibodiesAutoimmune diseaseLupus erythematosusbiologybusiness.industryCalcium-Binding ProteinsAutoantibodyInfant NewbornGeneral Medicinemedicine.diseaseConnective tissue diseaseHeart BlockImmunoglobulin MRibonucleoproteinsImmunoglobulin GImmunologybiology.proteinFemaleAntibodybusinessCalreticulinCalreticulinEuropean journal of clinical investigation
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Autologous peripheral blood stem and progenitor (CD34+) cell transplantation for systemic lupus erythematosus complicated by Evans syndrome.

1998

Immunoablation followed by allogeneic stem cell (SC) transplantation has been shown to be capable of curing a large spectrum of experimental autoimmune disorders, hereditary and/or induced. Superimposable results, albeit with some exceptions, have been obtained in human patients affected by coincidental autoimmune and blood diseases. However, both because of encouraging experimental results and of the procedure's greater safety, autologous SC are being increasingly utilized worldwide. Case reports are being collected in the registry of the European Group for Blood and Marrow Transplantation (EBMT)/European League against Rheumatism (EULAR) Autoimmune Disease Stem Cell Project. Among the se…

MaleEvans syndromeAdolescentmedicine.medical_treatmentAntigens CD34Hematopoietic stem cell transplantation030204 cardiovascular system & hematologyTransplantation Autologous03 medical and health sciences0302 clinical medicineRheumatologyPrednisoneMedicineAutologous transplantationHumansLupus Erythematosus Systemicskin and connective tissue diseases030203 arthritis & rheumatologyLupus anticoagulantPurpura Thrombocytopenic IdiopathicLupus erythematosusbusiness.industryHematopoietic Stem Cell TransplantationSyndromemedicine.diseaseTransplantationImmunologyFemaleAnemia Hemolytic AutoimmunebusinessAnti-SSA/Ro autoantibodiesmedicine.drugLupus
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The systemic lupus erythematosus IRF5 risk haplotype is associated with systemic sclerosis.

2013

Systemic sclerosis (SSc) is a fibrotic autoimmune disease in which the genetic component plays an important role. One of the strongest SSc association signals outside the human leukocyte antigen (HLA) region corresponds to interferon (IFN) regulatory factor 5 (IRF5), a major regulator of the type I IFN pathway. In this study we aimed to evaluate whether three different haplotypic blocks within this locus, which have been shown to alter the protein function influencing systemic lupus erythematosus (SLE) susceptibility, are involved in SSc susceptibility and clinical phenotypes. For that purpose, we genotyped one representative single-nucleotide polymorphism (SNP) of each block (rs10488631, r…

MaleLinkage disequilibrium:Phenomena and Processes::Genetic Phenomena::Phenotype [Medical Subject Headings]Polimorfismo de nucleótido simpleSLElcsh:MedicineAutoimmunityGenome-wide association studyLinkage DisequilibriumScleroderma:Phenomena and Processes::Genetic Phenomena::Genotype::Haplotypes [Medical Subject Headings]:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]Gene Frequency:Named Groups::Persons::Population Groups::Continental Population Groups::European Continental Ancestry Group [Medical Subject Headings]Risk FactorsIRF5Genetics of the Immune SystemLupus Erythematosus Systemic:Diseases::Skin and Connective Tissue Diseases::Skin Diseases::Scleroderma Systemic [Medical Subject Headings]skin and connective tissue diseaseslcsh:ScienceMultidisciplinary:Diseases::Immune System Diseases::Autoimmune Diseases::Lupus Erythematosus Systemic [Medical Subject Headings]Predisposición genética a la enfermedad:Phenomena and Processes::Genetic Phenomena::Genetic Linkage::Linkage Disequilibrium [Medical Subject Headings]:Phenomena and Processes::Genetic Phenomena::Genotype::Genetic Predisposition to Disease [Medical Subject Headings]PhenotypeInterferon Regulatory FactorsSYSTEMIC SCLEROSISMedicineEvaluation of complex medical interventions Auto-immunity transplantation and immunotherapy [NCEBP 2]FemaleIRF5; SLE; TYPE I INTERFERON; SYSTEMIC SCLEROSISHaplotiposResearch ArticleFactores de riesgoImmunology:Chemicals and Drugs::Amino Acids Peptides and Proteins::Peptides::Intracellular Signaling Peptides and Proteins::Adaptor Proteins Signal Transducing::Interferon Regulatory Factors [Medical Subject Headings]:Check Tags::Male [Medical Subject Headings]:Health Care::Environment and Public Health::Public Health::Epidemiologic Factors::Causality::Risk Factors [Medical Subject Headings]Single-nucleotide polymorphismHuman leukocyte antigenBiologyPolymorphism Single NucleotideWhite PeopleAutoimmune DiseasesRheumatologyLupus eritematoso sistémicoGeneticsHumansGenetic Predisposition to DiseaseGrupo de ascendencia continental europeaAlleleBiologyAllele frequencyAllelesGenetic Association Studies:Phenomena and Processes::Genetic Phenomena::Genetic Structures::Genome::Genome Components::Genes::Alleles [Medical Subject Headings]Scleroderma SystemicHaplotypelcsh:R:Phenomena and Processes::Genetic Phenomena::Genetic Structures::Genome::Genome Components::Genetic Loci [Medical Subject Headings]Human Genetics:Phenomena and Processes::Genetic Phenomena::Genetic Variation::Polymorphism Genetic [Medical Subject Headings]Factores reguladores del interferónHaplotypesDesequilibrio de ligamiento:Check Tags::Female [Medical Subject Headings]Genetic LociTYPE I INTERFERONGenetics of DiseaseImmunologyGenetic PolymorphismClinical Immunologylcsh:Q:Phenomena and Processes::Genetic Phenomena::Gene Frequency [Medical Subject Headings]Population GeneticsIRF5PLoS ONE
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