Search results for "Lupus"

showing 10 items of 187 documents

Thymoma and pure red cell aplasia in a patient with systemic lupus erythematosus.

1995

We present the case of a female patient with a diagnosis of systemic lupus erythematosus (SLE) at the age of 54 years. At the age of 63 years, she suffered from malignant thymoma and 3 years after removal of the thymoma a diagnosis of pure red cell aplasia (PRCA) was established. This is, to our knowledge, the first report of the occurrence of SLE, thymoma and PRCA in the same patient. The case is discussed with regard to the already known associations between these diseases.

Adultmedicine.medical_specialtySystemic diseasePathologyThymomaThymomaImmunologyPure red cell aplasiaBone Marrow Aplasiaurologic and male genital diseasesRed-Cell Aplasia PureRheumatologyimmune system diseasesBone Marrowhemic and lymphatic diseasesmedicineImmunology and AllergyHumansLupus Erythematosus Systemicskin and connective tissue diseasesMalignant ThymomaLupus erythematosusThymus Neoplasmbusiness.industryGeneral MedicineThymus Neoplasmsmedicine.diseaseConnective tissue diseaseDermatologyFemalebusinessScandinavian journal of rheumatology
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Immunoregulatory role of Jα281 T cells in aged mice developing lupus-like nephritis

2007

Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the emergence of autoreactive T cells. Humans and mice with SLE have reduced numbers of CD1d-restricted invariant natural killer T (iNKT) cells, suggesting a key role for these cells in its immunopathogenesis. This subset uses an invariant TCR constituted by Valpha14 Jalpha281 chains paired with some Vbeta domains. The regulatory role for iNKT cells in non-autoimmune mice was suggested by our previous results showing that aged Jalpha281 knockout (KO) mice produce anti-dsDNA. Here we show that old Jalpha281 KO mice have proteinuria and antibodies against dsDNA and cardiolipin. Histological analysis of Jalpha281 KO m…

AgingImmunologyReceptors Antigen T-CellEnzyme-Linked Immunosorbent AssayLymphocyte Activationmedicine.disease_causeAutoimmunity Knockout NKT cellsAutoimmunityMicemedicineAnimalsLupus Erythematosus SystemicImmunology and AllergyAutoantibodiesMice KnockoutSettore MED/04 - Patologia GeneraleB-LymphocytesSystemic lupus erythematosusbiologyT-cell receptorAutoantibodyNatural killer T cellMarginal zonemedicine.diseaseImmunohistochemistryLupus NephritisKiller Cells NaturalImmunologybiology.proteinAntibodyNephritisSpleenEuropean Journal of Immunology
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Data from: Moving in the Anthropocene: global reductions in terrestrial mammalian movements

2019

Animal movement is fundamental for ecosystem functioning and species survival, yet the effects of the anthropogenic footprint on animal movements have not been estimated across species. Using a unique GPS-tracking database of 803 individuals across 57 species, we found that movements of mammals in areas with a comparatively high human footprint were on average one-half to one-third the extent of their movements in areas with a low human footprint. We attribute this reduction to behavioral changes of individual animals and to the exclusion of species with long-range movements from areas with higher human impact. Global loss of vagility alters a key ecological trait of animals that affects no…

Alces alcesPapio cynocephalusOdocoileus hemionusSus scrofaSaiga tataricaMartes pennantimedicine and health careAnthropocenePuma concolorConnochaetes taurinusDasypus novemcinctusChrysocyon brachyurusOvibos moschatusPanthera pardusEquus hemionusTrichosurus vulpeculaLife SciencesLynx lynxPapio anubisUrsus arctosNDVI; diet; movement ecologyTolypeutes matacusmovement ecologyMedicineCapreolus capreolusEquus quaggaCanis latransPropithecus verreauxiBeatragus hunteriOdocoileus virginianusTamandua mexicanaSyncerus cafferLepus europaeusNDVICervus elaphusEquus grevyiEuphractus sexcinctusLoxodonta africanaOdocoileus hemionus columbianusProcyon lotorAntilocapra americanaMyrmecophaga tridactylaMadoqua guentheriGulo guloTapirus terrestrisPanthera oncaCerdocyon thousFelis silvestrisCanis aureusEulemur rufifronsSaguinus geoffroyiHuman FootprintRangifer tarandusCanis lupusCercocebus galeritusAepyceros melampusChlorocebus pygerythrusProcapra gutturosaLoxodonta africana cyclotisGiraffa camelopardalisdiet
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Inducible Co-Stimulator Null MRL-Fas lpr Mice

2005

MRL/MpJ-Tnfrsf6lpr (MRL-Faslpr) mice develop a spontaneous T cell-dependent autoimmune disease that shares features with human lupus, including fatal nephritis, systemic pathology, and autoantibodies (autoAb). The inducible co-stimulator (ICOS) is upregulated on activated T cells and modulates T cell-mediated responses. To investigate whether ICOS has an essential role in regulating autoimmune lupus nephritis and the systemic illness in MRL-Faslpr mice, ICOS null (-/-) MRL Faslpr and ICOS intact (+/+) MRL-Faslpr strains (wild-type [WT]) were generated and compared. It was determined that in ICOS-/- MRL-Faslpr as compared with the WT strain, (1) there is a significant reduction in circulatin…

Antigens Differentiation T-LymphocyteMice Inbred MRL lprT-LymphocytesT cellLupus nephritismedicine.disease_causeBlood Urea NitrogenAutoimmunityInducible T-Cell Co-Stimulator ProteinInterferon-gammaMiceImmune systemimmune system diseasesmedicineAnimalsskin and connective tissue diseasesAutoantibodiesMice Inbred C3HSystemic lupus erythematosusTumor Necrosis Factor-alphabusiness.industryAutoantibodyGeneral Medicinemedicine.diseaseLupus NephritisIsotypeInterleukin-10Mice Inbred C57BLProteinuriamedicine.anatomical_structureNephrologyImmunoglobulin GImmunologyInterleukin-4businessNephritisJournal of the American Society of Nephrology
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Histopathology of the gut in rheumatic diseases

2018

The gastrointestinal tract regulates the trafficking of macromolecules between the environment and the host through an epithelial barrier mechanism and is an important part of the immune system controlling the equilibrium between tolerance and immunity to non-self-antigens. Various evidence indicates that intestinal inflammation occurs in patients with rheumatic diseases. In many rheumatic diseases intestinal inflammation appears to be linked to dysbiosis and possibly represents the common denominator in the pathogenesis of different rheumatic diseases. The continuative interaction between dysbiosis and the intestinal immune system may lead to the aberrant activation of immune cells that ca…

Arthritislcsh:MedicineIntestinal inflammationPathogenesisSystemic sclerosiBehçet’s diseaseIntestinal MucosaConnective Tissue DiseasesGastrointestinal tractBehcet SyndromeIntestineIntestinesSymbiosimedicine.symptomHumanAnkylosing spondylitislcsh:Internal medicineInflammationSystemic lupus erythematosuRheumatic DiseaseImmune systemSystemic lupus erythematosusRheumatologyImmunityRheumatic DiseasesSpondylarthritismedicineHumansSpondylitis AnkylosingRheumatoid arthritisSymbiosislcsh:RC31-1245Rheumatoid arthritiConnective Tissue DiseaseInflammationAnkylosing spondylitisbusiness.industryArthritis PsoriaticSpondylarthritilcsh:RMuscle Smoothmedicine.diseaseBehget’s diseaseDysbiosiAnkylosing spondylitiSettore MED/16 - ReumatologiaImmunologyDysbiosisbusinessDysbiosisReumatismo
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Genetics and novel aspects of therapies in systemic lupus erythematosus.

2015

Autoimmune diseases, such as rheumatoid arthritis, multiple sclerosis, autoimmune hepatitis and inflammatory bowel disease, have complex pathogeneses and the factors which cause these disorders are not well understood. But all have in common that they arise from a dysfunction of the immune system, interpreting self components as foreign antigens. Systemic lupus erythematosus (SLE) is one of these complex inflammatory disorders that mainly affects women and can lead to inflammation and severe damage of virtually any tissue and organ. Recently, the application of advanced techniques of genome-wide scanning revealed more genetic information about SLE than previously possible. These case-contro…

Autoimmune diseaseMultiple sclerosisImmunologyGenome-wide association studyAutoimmune hepatitisBiologymedicine.diseaseInflammatory bowel diseaseImmune systemAutologous stem-cell transplantationTreatment OutcomeRheumatoid arthritisHistocompatibility AntigensImmunologymedicineImmunology and AllergyAnimalsHumansLupus Erythematosus SystemicGenetic Predisposition to Diseaseskin and connective tissue diseasesImmunosuppressive AgentsGenome-Wide Association StudyAutoimmunity reviews
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PS5:100 Patophysiological role of type i and iii interferons in systemic lupus erythematosus (sle)

2018

Systemic Lupus erythematosus (SLE) is an autoimmune disease characterised by activated autoreactive lymphocytes and autoantibodies, resulting in tissue damage in multiple organs. An important factor for the disease´s mortality is the development of Lupus nephritis (LN). Type I and III interferons, which are both part of the antiviral defense, have both been associated with the disease´s activity. In sera and urine of SLE patients an enhanced level of IL28/29 was described, but their distinct functional role in the course of disease need to be further investigated. To determine the role of type I and III interferons during onset and progression of autoimmunity – with focus on the development…

Autoimmune diseaseSystemic lupus erythematosusbusiness.industryLupus nephritisAutoantibodyGlomerulonephritisSpleenmedicine.diseasemedicine.disease_causeAutoimmunitymedicine.anatomical_structureimmune system diseasesImmunologyMedicineskin and connective tissue diseasesbusinessReceptorPoster session 5: Innate and adaptive immunity
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Cutaneous Lupus Erythematosus

2008

Lupus erythematosus (LE) is an inflammatory autoimmune disease, characterized by a heterogeneous clinical presentation. The skin lesions are one of the most frequent symptoms of the disease and present with a broad spectrum of LE-nonspecific and LE-specific cutaneous manifestations. Therefore, the development of a classification for skin lesions in the disease has proven difficult. For example, the LE-nonspecific cutaneous manifestations include livedo racemosa, thrombophlebitis, and leukocytoclastic vasculitis and can be associated with high disease activity and systemic organ involvement. The LE-specific cutaneous manifestations encompass the subtypes of cutaneous lupus erythematosus (CLE…

Autoimmune diseasemedicine.medical_specialtyLupus erythematosusmedicine.diagnostic_testbusiness.industryfungiDiseaseLivedo racemosamedicine.diseaseThrombophlebitisDermatologySkin biopsyPhototestingmedicineCutaneous Lupus ErythematosusAntimalarial Agentmedicine.symptombusinessPanniculitisSkin lesionTherapeutic strategyAnti-SSA/Ro autoantibodies
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Successful Treatment of Catastrophic Antiphospholipid Antibody Syndrome (CAPS) Associated With Splenic Marginal-zone Lymphoma With Low-molecular Weig…

2008

ABSTRACT Case report A 69-year-old woman with splenic marginal-zone lymphoma was admitted with progressive abdominal pain and splenomegaly as the suspected cause of pain. Rituximab treatment (375 mg/m 2 ) had been initiated on the day of admission. Abdominal computerized tomography revealed splenic infarction. Laboratory tests showed elevation of liver enzymes and creatinine, low platelet count, prolonged partial thromboplastin time, and lupus anticoagulant positivity. The diagnosis of catastrophic antiphospholipid antibody syndrome was made. Weight-adjusted low-molecular weight heparin therapy was initiated. Freedom from symptoms and normalization of liver enzymes and creatinine occurred w…

Bendamustinemedicine.medical_specialtyLymphoma B-Cellmedicine.drug_classLow molecular weight heparinAntineoplastic AgentsGastroenterologyAntibodies Monoclonal Murine-Derivedimmune system diseaseshemic and lymphatic diseasesInternal medicineBendamustine HydrochlorideHumansMedicineSplenic marginal zone lymphomaAgedLupus anticoagulantbusiness.industrySplenic NeoplasmsAnticoagulantAntibodies MonoclonalAnticoagulantsGeneral MedicineHeparinHeparin Low-Molecular-WeightAntiphospholipid Syndromemedicine.diseaseSurgerySplenic infarctionNitrogen Mustard CompoundsFemaleRituximabRituximabbusinessmedicine.drugThe American Journal of the Medical Sciences
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Label-free piezoelectric biosensor for prognosis and diagnosis of Systemic Lupus Erythematosus

2017

[EN] An autoantigen piezoelectric sensor to quantify specific circulating autoantibodies in human serum is developed. The sensor consisted on a quartz crystal microbalance with dissipation monitoring (QCM-D) where TRIM21 and TROVE2 autoantigens were covalently immobilized, allowing the selective determination of autoantibodies for diagnosis and prognosis of Systemic Lupus Erythematosus (SLE). The sensitivity of the biosensor, measured as IC50 value, was 1.51 U/mL and 0.32 U/mL, for anti-TRIM21 and anti-TROVE2 circulating autoantibodies, respectively. The sensor is also able to establish a structural interaction fingerprint pattern or profile of circulating autoantibodies, what allows scorin…

Biomedical EngineeringBiophysicsEarly detectionBiosensing Techniques02 engineering and technologyImmunosensorDissipation monitoringAutoantigensSensitivity and SpecificitySystemic Lupus Erythematosus01 natural sciencesQuartz crystal microbalanceRNA Small CytoplasmicDiagnosisQUIMICA ANALITICAElectrochemistryHumansLupus Erythematosus SystemicMedicineMultiplexPiezoelectric biosensorAutoantibodiesLabel freeRibonucleoproteinbusiness.industry010401 analytical chemistryAutoantibodyGeneral MedicineQuartz crystal microbalancePrognosis021001 nanoscience & nanotechnology0104 chemical sciencesInteraction fingerprintRibonucleoproteinsImmunologyQuartz Crystal Microbalance Techniques0210 nano-technologybusinessBiosensorBiotechnologyBiosensors and Bioelectronics
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