Search results for "Lymphocyte"

showing 10 items of 2280 documents

Time to activin on pathogenic T cells

2020

In multiple sclerosis (MS), Th17 cells are critical drivers of autoimmune central nervous system (CNS) inflammation and demyelination. Th17 cells exhibit functional heterogeneity fostering both pathogenic and nonpathogenic, tissue-protective functions. Still, the factors that control Th17 pathogenicity remain incompletely defined. Here, using experimental autoimmune encephalomyelitis, an established mouse MS model, we report that therapeutic administration of activin-A ameliorates disease severity and alleviates CNS immunopathology and demyelination, associated with decreased activation of Th17 cells. In fact, activin-A signaling through activin-like kinase-4 receptor represses pathogenic t…

0301 basic medicineT-Lymphocytesmedicine.medical_treatmentAutoimmune Diseases03 medical and health sciences0302 clinical medicineCerebrospinal fluidImmune systemCommentariesDemyelinating diseaseMedicineCytotoxic T cellNeuroinflammationInflammationMultidisciplinaryVirulencebusiness.industryMultiple sclerosisBiological Sciencesmedicine.diseaseActivins030104 developmental biologyCytokineImmunologybusinessCD8030215 immunologyProceedings of the National Academy of Sciences
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Novel Opportunities for Cathepsin S Inhibitors in Cancer Immunotherapy by Nanocarrier-Mediated Delivery

2020

Cathepsin S (CatS) is a secreted cysteine protease that cleaves certain extracellular matrix proteins, regulates antigen presentation in antigen-presenting cells (APC), and promotes M2-type macrophage and dendritic cell polarization. CatS is overexpressed in many solid cancers, and overall, it appears to promote an immune-suppressive and tumor-promoting microenvironment. While most data suggest that CatS inhibition or knockdown promotes anti-cancer immunity, cell-specific inhibition, especially in myeloid cells, appears to be important for therapeutic efficacy. This makes the design of CatS selective inhibitors and their targeting to tumor-associated M2-type macrophages (TAM) and DC an attr…

0301 basic medicineT-Lymphocytesmedicine.medical_treatmentReview02 engineering and technologyCancer immunotherapyNeoplasmsTumor-Associated MacrophagesTumor Microenvironmentcysteine proteaseMolecular Targeted TherapySulfoneslcsh:QH301-705.5Cathepsin SAntigen PresentationDrug Carrierscysteine cathepsintumor-associated macrophage (TAM)ChemistrynanoparticleAzepinesDipeptidesGeneral Medicine021001 nanoscience & nanotechnologyGene Expression Regulation NeoplasticImmunotherapy0210 nano-technologydendritic cellAntigen presentationAntineoplastic AgentsTumor-associated macrophageM2 macrophage03 medical and health sciencesLeucinemedicineHumansProtease InhibitorsAntigen-presenting celltargetingtherapypolarizationTumor microenvironmentT cellDendritic CellsDendritic cellextracellular matrix (ECM)Cathepsinstumor associated macrophage030104 developmental biologylcsh:Biology (General)antigen presenting cellCancer researchNanoparticlesimmune suppressionNanocarriers
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Targeting Neoepitopes to Treat Solid Malignancies: Immunosurgery

2020

Successful outcome of immune checkpoint blockade in patients with solid cancers is in part associated with a high tumor mutational burden (TMB) and the recognition of private neoantigens by T-cells. The quality and quantity of target recognition is determined by the repertoire of ‘neoepitope’-specific T-cell receptors (TCRs) in tumor-infiltrating lymphocytes (TIL), or peripheral T-cells. Interferon gamma (IFN-γ), produced by T-cells and other immune cells, is essential for controlling proliferation of transformed cells, induction of apoptosis and enhancing human leukocyte antigen (HLA) expression, thereby increasing immunogenicity of cancer cells. TCR αβ-dependent therapies should account f…

0301 basic medicineT-Lymphocytesmedicine.medical_treatmentprecision medicineImmunologyEpitopes T-LymphocyteReviewHuman leukocyte antigenBiologyMajor histocompatibility complexCancer Vaccines03 medical and health sciencesLymphocytes Tumor-Infiltrating0302 clinical medicineImmune systemAntigenAntigens NeoplasmantigensNeoplasmsmedicineAnimalsHumansImmunology and AllergyT-cell receptorTumor microenvironmentneoepitopesWhole Genome SequencingT-cellsT-cell receptorComputational BiologyImmunotherapyTILRC581-607vaccinationImmune checkpoint030104 developmental biology030220 oncology & carcinogenesisCancer researchbiology.proteinimmunotherapyImmunologic diseases. AllergyFrontiers in Immunology
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Activation and selective IL-17 response of human Vγ9Vδ2 T lymphocytes by TLR-activated plasmacytoid dendritic cells.

2016

// Elena Lo Presti 1,2 , Nadia Caccamo 1,2 , Valentina Orlando 1,2 , Francesco Dieli 1,2 and Serena Meraviglia 1,2 1 Central Laboratory of Advanced Diagnosis and Biomedical Research (CLADIBIOR), University of Palermo, Palermo, Italy 2 Department of Biopathology and Medical Biotechnologies (DIBIMED), University of Palermo, Palermo, Italy Correspondence to: Serena Meraviglia, email: // Keywords : γδ T cells, plasmacytoid dendritic cells, IL-17, TLR activation, proliferation, Immunology and Microbiology Section, Immune response, Immunity Received : July 20, 2016 Accepted : August 02, 2016 Published :August 31, 2016 Abstract Vγ9Vδ2 T cells and plasmacytoid dendritic cells (pDCs) are two distinc…

0301 basic medicineTLR activationCellCell CommunicationLigandsLymphocyte Activation0302 clinical medicineT-Lymphocyte SubsetsCoculture TechniqueAntigen PresentationInterleukin-17Research Paper: Immunologyhemic and immune systemsIL-17medicine.anatomical_structurePhenotypeOncologyplasmacytoid dendritic cellsImmunology and Microbiology SectionInterleukin 17HumanCell typeproliferationCD40 LigandLigandBiologyDendritic Cellγδ T cells03 medical and health sciencesInducible T-Cell Co-Stimulator LigandInterferon-gammaImmune systemImmunityplasmacytoid dendritic cellmedicineHumansImmune responseCell Proliferationγδ T cellCD40Innate immune systemImmunityTLR9Dendritic CellsReceptors OX40Coculture TechniquesImmunity Innate030104 developmental biologyImmunologybiology.proteinLeukocytes MononuclearCpG IslandsCpG IslandImmunologic Memory030215 immunologyOncotarget
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Cytomegalovirus vector expressing RAE-1γ induces enhanced anti-tumor capacity of murine CD8+ T cells

2017

Designing of CD8 T cell vaccines which would provide protection against tumors is still considered a great challenge in immunotherapy. Here we show a robust potential of a cytomegalovirus (CMV) vector expressing the NKG2D ligand RAE-1γ as CD8 T cell-based vaccine against malignant tumors. Immunization with the CMV vector expressing RAE-1γ delayed tumor growth or even provided complete protection against tumor challenge in both prophylactic and therapeutic settings. Moreover, a potent tumor control in mice vaccinated with this vector can be further enhanced by blocking the immune checkpoints TIGIT and PD-1. Expression of RAE-1γ by the CMV vector potentiated expansion of KLRG1+ CD8 T cells wi…

0301 basic medicineTumor vaccine [RAE-1γ]medicine.medical_treatmentT cellImmunologyGenetic VectorsProgrammed Cell Death 1 ReceptorMelanoma ExperimentalCytomegalovirusEpitopes T-LymphocyteBiologyCD8-Positive T-LymphocytesCancer VaccinesArticleCMV vectorNKG2DImmunomodulation03 medical and health sciencesMiceImmune systemTIGITKLRG1+ CD8+ T cellsNeoplasmsmedicineImmunology and AllergyCytotoxic T cellAnimalsHumansRAE-1γ : Tumor vaccineLectins C-TypeReceptors ImmunologicαTIGIT ; CMV vector ; KLRG1+ CD8+ T cells ; NKG2D ; RAE-1γ : Tumor vaccineBIOMEDICINE AND HEALTHCARE. Basic Medical Sciences.Membrane ProteinsImmunotherapyNKG2DVirology3. Good healthKiller Cells NaturalDisease Models Animal030104 developmental biologymedicine.anatomical_structureImmunizationAnimals NewbornFemaleαTIGITImmunotherapyBIOMEDICINA I ZDRAVSTVO. Temeljne medicinske znanosti.CD8
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Deciphering human γδ T cell response in cancer: Lessons from tumor‐infiltrating γδ T cells

2020

The finding that γδ T cells are present among tumor-infiltrating lymphocytes in humans suggests they participate in tumor immune surveillance, but their relevance is unclear because the relative abundance of tumor-infiltrating γδ T cells correlates with positive or negative, or even do not correlate with prognosis. This likely depends on the fact that tumor-infiltrating γδ T cells may play substantially different effector or regulatory functions, and correlation with patient's prognosis relies on distinct γδ T cell subsets in the context of the tumor. There is interest to exploit γδ T cells in tumor immunotherapy, but to make this approach successful there is urgent need to fully understand…

0301 basic medicine[SDV]Life Sciences [q-bio]medicine.medical_treatmentT cellImmunologyContext (language use)BiologyTumor-infiltrating lymphocytesclinical correlationcolon cancer tumor microenvironment tumor-infiltrating lymphocytes γδ T lymphocytesClinical correlazion03 medical and health sciencesLymphocytes Tumor-Infiltrating0302 clinical medicineT-Lymphocyte SubsetsNeoplasmsmedicineHumansImmunology and AllergyComputingMilieux_MISCELLANEOUSTumor microenvironmentTumor-infiltrating lymphocytesEffectorCancerReceptors Antigen T-Cell gamma-deltaImmunotherapyGamma-delta T lymphocytesmedicine.diseaseColon cancer3. Good health030104 developmental biologymedicine.anatomical_structureTumor microenvironmentCancer researchEx vivo030215 immunologyImmunological Reviews
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Enzymatic Activity of HPGD in Treg Cells Suppresses Tconv Cells to Maintain Adipose Tissue Homeostasis and Prevent Metabolic Dysfunction.

2019

Summary Regulatory T cells (Treg cells) are important for preventing autoimmunity and maintaining tissue homeostasis, but whether Treg cells can adopt tissue- or immune-context-specific suppressive mechanisms is unclear. Here, we found that the enzyme hydroxyprostaglandin dehydrogenase (HPGD), which catabolizes prostaglandin E2 (PGE2) into the metabolite 15-keto PGE2, was highly expressed in Treg cells, particularly those in visceral adipose tissue (VAT). Nuclear receptor peroxisome proliferator-activated receptor-γ (PPARγ)-induced HPGD expression in VAT Treg cells, and consequential Treg-cell-mediated generation of 15-keto PGE2 suppressed conventional T cell activation and proliferation. C…

0301 basic medicineanalogs & derivatives [Dinoprostone]Malemetabolism [Diabetes Mellitus Type 2]Adipose tissueLymphocyte Activation15-ketoprostaglandin E2T-Lymphocytes RegulatoryJurkat cellsJurkat CellsMice0302 clinical medicineimmunology [Lymphocyte Activation]genetics [Insulin Resistance]STAT5 Transcription FactorHomeostasisImmunology and AllergyTissue homeostasisgenetics [Hydroxyprostaglandin Dehydrogenases]Mice Knockoutcytology [Intra-Abdominal Fat]enzymology [T-Lymphocytes Regulatory]FOXP3hemic and immune systems3T3 CellsCell biologyInfectious Diseases030220 oncology & carcinogenesisHydroxyprostaglandin Dehydrogenasesmedicine.symptomimmunology [T-Lymphocytes Regulatory]metabolism [STAT5 Transcription Factor]Immunologymetabolism [Dinoprostone]chemical and pharmacologic phenomenaInflammationIntra-Abdominal FatBiologyDinoprostoneCell Line03 medical and health sciencesmetabolism [Hydroxyprostaglandin Dehydrogenases]immunology [Homeostasis]medicineAnimalsHumansddc:610immunology [Intra-Abdominal Fat]HEK 293 cells030104 developmental biologyHEK293 CellsDiabetes Mellitus Type 2Cell cultureInsulin ResistanceHomeostasis
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Herpes simplex virus 1 induces egress channels through marginalized host chromatin

2016

AbstractLytic infection with herpes simplex virus type 1 (HSV-1) induces profound modification of the cell nucleus including formation of a viral replication compartment and chromatin marginalization into the nuclear periphery. We used three-dimensional soft X-ray tomography, combined with cryogenic fluorescence, confocal and electron microscopy, to analyse the transformation of peripheral chromatin during HSV-1 infection. Our data showed an increased presence of low-density gaps in the marginalized chromatin at late infection. Advanced data analysis indicated the formation of virus-nucleocapsid-sized (or wider) channels extending through the compacted chromatin of the host. Importantly, co…

0301 basic medicineanalysisvirusesHerpesvirus 1 Humanmedicine.disease_causeVirus Replicationlaw.inventionRussia[ SDV.CAN ] Life Sciences [q-bio]/CancerMicelaw2.1 Biological and endogenous factorsAetiologynuclear organisationTomographyB-LymphocytesMicroscopyMultidisciplinaryMicroscopy ConfocalTomography X-Rayta3141Chromatin3. Good healthCell biologyChromatinOther Physical SciencesInfectious Diseasesmedicine.anatomical_structureLytic cycleConfocalHost-Pathogen InteractionsVirusesFranceInfectionHumanConfocal030106 microbiology[SDV.CAN]Life Sciences [q-bio]/CancerBiologyta3111ElectronTime-Lapse ImagingArticleCell Line03 medical and health sciencesMicroscopy Electron TransmissionmedicineHerpes virusTransmissionAnimalsHumansCell Nucleusta114Herpesvirus 1ta1182VirionHerpes SimplexCell nucleus030104 developmental biologyHerpes simplex virusViral replicationCell cultureX-RaySexually Transmitted InfectionsBiochemistry and Cell BiologyElectron microscopeLaboratories
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Finding the right biomarker for renal cell carcinoma (RCC): Nivolumab treatment induces the expression of specific peripheral lymphocyte microRNAs in…

2019

Abstract Background The variability of clinical response to immune checkpoint inhibitors in RCC patients makes necessary the discovery of predictive biomarkers for patient selection. Emerging evidence has revealed a multitude of silenced genes and deregulated signalling pathways. These findings point towards extensive microRNAs (miRNAs) regulation and imply epigenetic reprogramming as a key feature of RCC. The aim of this study was to analyze the peripheral lymphocyte miRNA expression profile in metastatic RCC patients undergoing nivolumab treatment, to identify a lymphocyte miRNA signature specifically expressed in patients with partial or complete response (RP; RC) >12 months. Methods miR…

0301 basic medicinebiologybusiness.industryMicroarray analysis techniquesLymphocyteHematologymedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureOncologyRenal cell carcinoma030220 oncology & carcinogenesismicroRNAmedicinebiology.proteinCancer researchPTENBiomarker (medicine)NivolumabbusinessPI3K/AKT/mTOR pathwayAnnals of Oncology
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Γδ T Cell-Based Immunotherapy in Melanoma: State of the Art

2019

Metastatic melanoma is still associated with a poor prognosis, and there is increasing interest in immunotherapy alone or in combination with other adjuvant therapies. Γδ T lymphocytes play a pivot role in the immune response against cancer, but while γδ-based immunotherapy is already a clinical reality for several solid tumors, data on melanoma are still limited and fragmented. This systematic review presents preclinical and clinical evidence for a role of γδ T lymphocytes in immunotherapeutic strategies for advanced melanoma and discusses research state of the art and future perspectives. Current strategies focus on in vivo stimulation, and ex vivo adoptive therapy and vaccination; result…

0301 basic medicinebusiness.industrymedicine.medical_treatmentMelanomaT cellCancerReview ArticleImmunotherapymedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenslcsh:RC254-28203 medical and health sciences030104 developmental biology0302 clinical medicineImmune systemmedicine.anatomical_structureOncologyAntigen030220 oncology & carcinogenesismedicineCancer researchbusinessT-Lymphocytes | Receptors Antigen T-Cell gamma-delta | Cell subsetsAdjuvantEx vivoJournal of Oncology
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