Search results for "MACROPHAGES"

showing 10 items of 533 documents

In vitro fusion of phagosomes with different endocytic organelles from J774 macrophages.

1998

We describe novel biochemical and electron microscopy assays to investigate in vitro fusion of latex bead phagosomes with three different endocytic organelle fractions from J774 macrophages. After formation, early phagosomes fuse avidly with early and late endosomes and for a longer period of time with lysosomes, but they subsequently become fusion-incompetent. The fusion of early, but not late, phagosomes with all three endocytic fractions could be significantly stimulated by Rab5. In contrast to other cell types investigated, this Rab is uniquely enriched on both early and late endosomes in J774 macrophages. Moreover, exogenous Rab5 stimulates homotypic fusion between both sets of organel…

Cell typeEndosomeMacrophagesEndocytic cycleCell BiologyBiologyBiochemistryIn vitroEndocytosisCell biologyCell LineCell FusionMiceCricetinaePhagosomesOrganelleAnimalsHumansRabMolecular BiologyFusion mechanismPhagosomeThe Journal of biological chemistry
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Mast cells as rapid innate sensors of cytomegalovirus by TLR3/TRIF signaling-dependent and -independent mechanisms

2014

The succinct metaphor, ‘the immune system's loaded gun', has been used to describe the role of mast cells (MCs) due to their storage of a wide range of potent pro-inflammatory and antimicrobial mediators in secretory granules that can be released almost instantly on demand to fight invaders. Located at host–environment boundaries and equipped with an arsenal of pattern recognition receptors, MCs are destined to be rapid innate sensors of pathogens penetrating endothelial and epithelial surfaces. Although the importance of MCs in antimicrobial and antiparasitic defense has long been appreciated, their role in raising the alarm against viral infections has been noted only recently. Work on cy…

MaleChemokineImmunologyCytomegalovirusBiologyCD8-Positive T-LymphocytesCCL5MiceImmune systemImmunology and AllergyCytotoxic T cellAnimalsMast CellsMice KnockoutIntegrasesMacrophagesDegranulationPattern recognition receptorhumanitiesToll-Like Receptor 3Killer Cells NaturalMice Inbred C57BLAdaptor Proteins Vesicular TransportInfectious DiseasesTRIFImmunologyTLR3Cytomegalovirus Infectionsbiology.proteinFemaleResearch Article
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Humanized mice in cutaneous leishmaniasis—Suitability analysis of human PBMC transfer into immunodeficient mice

2019

Humanized mice represent a suitable preclinical test system for example therapeutic interventions in various disease settings, including infections. Here, we intended to establish such system for cutaneous leishmaniasis by infecting T, B and NK cell-deficient mice adoptively transferred with human peripheral blood mononuclear cells (PBMC). L major infection led to the establishment of parasite lesions harbouring viable parasites and human T cells, but parasite elimination was not seen due to a species-specific activity of T cell-derived human IFNγ. In addition, up to 50% of infected mice succumbed to severe graft-versus-host disease. In summary, even though long-term disease outcome assessm…

0301 basic medicineT cellGraft vs Host DiseaseLeishmaniasis CutaneousDermatologyDiseaseBiochemistryPeripheral blood mononuclear cellLesionInterferon-gammaMice030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicineSpecies SpecificityCutaneous leishmaniasisT-Lymphocyte SubsetsIn vivoAnimalsHumansMedicineParasite hostingMolecular Biologybusiness.industryMacrophagesLeishmaniasismedicine.diseaseAdoptive Transfer030104 developmental biologymedicine.anatomical_structureModels AnimalImmunologyDisease ProgressionLeukocytes MononuclearHeterograftsmedicine.symptombusinessExperimental Dermatology
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Sputum metalloproteinase-9/tissue inhibitor of metalloproteinase-1 ratio correlates with airflow obstruction in asthma and chronic bronchitis

1998

Asthma and chronic bronchitis are inflammatory diseases with extracellular matrix (ECM) remodeling and collagen deposition. Collagen homeostasis is controlled by metalloproteinases (MMPs) and tissue inhibitors of metalloproteinases (TIMPs). We evaluated MMP and TIMP balance in induced sputum of 10 control, 31 untreated asthmatic, and 16 chronic bronchitic subjects. We first performed zymographic analysis to identify the profile of MMPs. Zymography revealed a similar MMPs profile in all populations studied and that MMP-9 was the major enzyme released. We then measured, using enzyme immunoassay, the concentrations of MMP-9 and of its inhibitor TIMP-1 and evaluated whether airflow limitation m…

AdultPulmonary and Respiratory MedicineChronic bronchitisAdolescentNeutrophilsCell CountEnzyme-Linked Immunosorbent AssayMatrix metalloproteinaseCritical Care and Intensive Care MedicinePathogenesisLeukocyte CountSurface-Active AgentsForced Expiratory VolumemedicineHomeostasisHumansProtease InhibitorsCollagenasesBronchitisAgedAsthmaTissue Inhibitor of Metalloproteinase-1business.industryMacrophagesRespiratory diseaseSputumSodium Dodecyl SulfateMiddle AgedTissue inhibitor of metalloproteinasemedicine.diseaseAsthmaExtracellular Matrixrespiratory tract diseasesAirway ObstructionMatrix Metalloproteinase 9Chronic DiseaseImmunologyBronchitisSputumElectrophoresis Polyacrylamide GelCollagenmedicine.symptomPulmonary Ventilationbusiness
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Opsonizing activities of IgG, IgM antibodies and the C3b inactivator-cleaved third component of complement in macrophage phagocytosis

1976

Phagocytosis of SRBC by guinea-pig peritoneal macrophages is enhanced by opsonizing IgG antibody alone. IgM antibody requires the presence of bound C3. Treatment of C3b coated SRBC with purified C3b inactivator (yielding EAIgM C1423d) does not reduce attachment to, and phagocytosis by, peritoneal macrophages. This finding suggests the existence of a C3d receptor on peritoneal macrophages. EC43b intermediates which have been produced by removing IgM antibody by mercaptoethanol treatment and by subsequent removal of C1 and C2, are phagocytosed despite the absence of IgM antibody. Furthermore, treatment of EC43b with C3b inactivator does not change phagocytosis. Thus, IgM antibody does not app…

Igm antibodyReceptors DrugPhagocytosisGuinea PigsImmunologychemical and pharmacologic phenomenaStimulationToxicologyMicrobiologyMacrophage phagocytosisPhagocytosisOpsonin ProteinsC3b inactivatorAnimalsPharmacology (medical)ReceptorPharmacologybiologyChemistryMacrophagesCell MembraneComplement C3Complement System ProteinsOpsonin ProteinsImmunoglobulin MImmunoglobulin GImmunologybiology.proteinAntibodyAgents and Actions
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Nivolumab Enhances In Vitro Effector Functions of PD-1+ T-Lymphocytes and Leishmania-Infected Human Myeloid Cells in a Host Cell-Dependent Manner

2017

Functional impairment of T-cells and a concomitant augmented expression of programmed death-1 (PD-1) have been observed in visceral leishmaniasis patients, as well as in experimental models for visceral and cutaneous leishmaniasis. The PD-1/PD-1-ligand (PD-1/PD-L) interaction negatively regulates T-cell effector functions, which are required for parasite control during leishmaniasis. The aim of this study was to elucidate the impact of the PD-1/PD-L axis in a human primary in vitro infection model of Leishmania major (Lm). Blocking the PD-1/PD-L interaction with nivolumab increased T-cell proliferation and release of the proinflammatory cytokines TNFα and IFNγ during the cocultivation of Lm…

lcsh:Immunologic diseases. Allergyprogrammed death-1 ligand 10301 basic medicineprogrammed death-1 ligand 2ImmunologyProinflammatory cytokine03 medical and health sciencesCutaneous leishmaniasisPD-L1medicineImmunology and AllergyLeishmania majorGranulysinOriginal Researchprogrammed death-1Leishmanianivolumabhuman macrophagesbiologyT-cellsmedicine.diseasebiology.organism_classification030104 developmental biologyGranzymePerforinImmunologybiology.proteinTumor necrosis factor alphahuman dendritic cellslcsh:RC581-607Frontiers in Immunology
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A rhamnose-binding lectin from sea bass (Dicentrarchus labrax) plasma agglutinates and opsonizes pathogenic bacteria

2014

Abstract The discovery of rhamnose-binding lectins (RBLs) in teleost fish eggs led to the identification of a novel lectin family characterized by a unique sequence motif and a structural fold, and initially proposed to modulate fertilization. Further studies of the RBL tissue localization and gene organization were also suggestive of role(s) in innate immunity. Here we describe the purification, and biochemical and functional characterization of a novel RBL (DlRBL) from sea bass (Dicentrarchus labrax) serum. The purified DlRBL had electrophoretic mobilities corresponding to 24 kDa and 100 kDa under reducing and non-reducing conditions, respectively, suggesting that in plasma the DlRBL is p…

AgglutinationGram-negative bacteriaErythrocytesRhamnoselectin; D. labraxImmunologyAmino Acid MotifsMolecular Sequence DataRhamnoseArticlechemistry.chemical_compoundPlasmaPhagocytosisLectinsEscherichia coliAnimalsAmino Acid SequenceSea bassPeptide sequencePhylogenybiologyD. labraxLectinRhamnose bindingBacterial Infectionsbiology.organism_classificationImmunity InnateProtein Structure TertiaryBiochemistrychemistrybiology.proteinMacrophages PeritoneallectinBassRabbitsProtein MultimerizationSequence motifDevelopmental BiologyHomotetramerProtein Binding
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Synthesis and expression of MHC class II molecules in the absence of attached invariant chains by recombinant-interferon-gamma-activated bone-marrow-…

1987

Pure populations of in vitro propagated bone marrow-derived macrophages are constitutively Ia negative. Co-culturing of these cells with recombinant interferon-gamma (rIFN-gamma) resulted in the appearance of high amounts of Ia antigens at the cell surface of essentially all cells. The continuous presence of the stimulus was a prerequisite for sustained Ia expression because removal of the stimulus resulted in rapid decline of surface Ia. Two-dimensional (2D) gel analysis (1D isoelectric focusing, 2D sodium dodecyl sulfate-polyacrylamide gel electrophoresis) of class II molecules synthesized by rIFN-gamma-stimulated bone marrow macrophages (BMM phi) revealed that, in contrast to class II co…

ImmunologyBone Marrow Cellslaw.inventionInterferon-gammaMicelawImmunology and AllergyAnimalsNorthern blotRNA MessengerGel electrophoresisMessenger RNAMHC class IIMice Inbred C3HPolymorphism GeneticbiologyIsoelectric focusingMacrophagesHistocompatibility Antigens Class IIDNAMacrophage ActivationMolecular biologyIn vitroRecombinant ProteinsGene Expression RegulationRecombinant DNAbiology.proteinIntracellularEuropean journal of immunology
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The neuro-B cell link of peptidergic innervation in the Bursa Fabricii

1991

The Bursa Fabricii, restricted to birds, specifically provides the microenvironment for B-cell maturation. The presence of nerve fibers containing immunopotent neuropeptides in immune organs opens interesting perspectives on the understanding of neuroimmune communication. As an organ for the development of only B-lymphocytes is not known in mammals, the contribution of a peptidergic innervation to the microenvironment of B-cells is not known. Therefore, we studied the peptidergic innervation of the Bursa Fabricii as an organ of B-cell maturation. Four different neuropeptides were found in nerve fibers of the Bursa Fabricii: tachykinins (TK), vasoactive intestinal peptide (VIP), galanin (GAL…

medicine.medical_specialtyNeuroimmunomodulationCalcitonin Gene-Related PeptideImmunologyVasoactive intestinal peptideNeuropeptideGalaninNerve fiberBiologyCalcitonin gene-related peptideMonocytesImmunoenzyme TechniquesBehavioral NeuroscienceBursa of FabriciusNerve FibersTachykininsInternal medicinemedicineAnimalsGalaninMedullaB cellB-LymphocytesEndocrine and Autonomic SystemsMacrophagesNeuropeptidesCell DifferentiationCell biologyEndocrinologymedicine.anatomical_structurePeripheral nervous systemPeptidesChickensVasoactive Intestinal PeptideBrain, Behavior, and Immunity
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Homeostasis of Microglia in the Adult Brain: Review of Novel Microglia Depletion Systems.

2015

Microglia are brain macrophages that emerge from early erythro-myeloid precursors in the embryonic yolk sac and migrate to the brain mesenchyme before the blood brain barrier is formed. They seed the brain, and proliferate until they have formed a grid-like distribution in the central nervous system that is maintained throughout lifespan. The mechanisms through which these embryonic-derived cells contribute to microglia homoeostasis at steady state and upon inflammation are still not entirely clear. Here we review recent studies that provided insight into the contribution of embryonically-derived microglia and of adult 'microglia-like' cells derived from monocytes during inflammation. We ex…

NeuroimmunomodulationCellular differentiationMesenchymeImmunologyCentral nervous systemEmbryonic DevelopmentInflammation610 Medicine & healthBiologyBlood–brain barrier10263 Institute of Experimental ImmunologymedicineImmunology and AllergyAnimalsHomeostasisHumansNeuroinflammationInflammation2403 ImmunologyMicrogliaMacrophagesBrainCell DifferentiationEmbryonic stem cellDisease Models Animalmedicine.anatomical_structureImmunologyModels Animal2723 Immunology and Allergy570 Life sciences; biologyMicrogliamedicine.symptomTrends in immunology
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