Search results for "MACROPHAGES"

showing 10 items of 533 documents

Novel anti-inflammatory chalcone derivatives inhibit the induction of nitric oxide synthase and cyclooxygenase-2 in mouse peritoneal macrophages

1999

AbstractIn a previous work, we tested a series of chalcone derivatives as possible anti-inflammatory compounds. We now investigate the effects of three of those compounds, CH1, CH8 and CH12, on nitric oxide and prostanoid generation in mouse peritoneal macrophages stimulated with lipopolysaccharide and in the mouse air pouch injected with zymosan, where they showed a dose-dependent inhibition with inhibitory concentration 50% values in the μM range. This effect was not the consequence of a direct inhibitory action on enzyme activities. Our results demonstrated that chalcone derivatives inhibited de novo inducible nitric oxide synthase and cyclooxygenase-2 synthesis, being a novel therapeuti…

LipopolysaccharidesChalconeLipopolysaccharidemedicine.drug_classBiophysicsNitric Oxide Synthase Type IILipopolysaccharidePharmacologyBiochemistryDinoprostoneAnti-inflammatoryNitric oxideMicechemistry.chemical_compoundChalconeStructural BiologyGeneticsmedicineAnimalsCyclooxygenase-2Mouse air pouchInducible nitric oxide synthaseMolecular BiologyNitritesDose-Response Relationship DrugbiologyAnti-Inflammatory Agents Non-SteroidalZymosanZymosanProstanoidMouse peritoneal macrophageCell BiologyIsoenzymesNitric oxide synthasechemistryBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesEnzyme InductionMacrophages Peritonealbiology.proteinFemaleCyclooxygenaseNitric Oxide SynthaseFEBS Letters
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Mitochondrial G protein coupled receptor kinase 2 regulates proinflammatory responses in macrophages.

2013

G-protein-coupled receptor kinase 2 (GRK2) levels are elevated in inflammation but its role is not clear yet. Here we show that GRK2 expression is dependent on NFκB transcriptional activity. In macrophages, LPS induces GRK2 accumulation in mitochondria increasing biogenesis. The overexpression of the carboxy-terminal domain of GRK2 (βARK-ct), known to displace GRK2 from plasma membranes, in macrophages induces earlier localization of GRK2 in mitochondria in response to LPS leading to increased mt-DNA transcription, reduced ROS production and cytokines expression. Our study shows the relevance of GRK2 subcellular localization in macrophage’s biology and its potential therapeutic properties i…

LipopolysaccharidesG-Protein-Coupled Receptor Kinase 2BiophysicsβARK-ctGRK2InflammationMitochondrionBiochemistryArticleProinflammatory cytokineMiceStructural BiologyGeneticsmedicineAnimalsHumansReceptorMolecular BiologyInflammationG protein-coupled receptor kinasebiologyKinaseSubcellular localizationBeta adrenergic receptor kinaseMacrophagesCell BiologySubcellular localizationCell biologyMitochondriabiology.proteinmedicine.symptomReactive Oxygen SpeciesSignal Transduction
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The macrophage-activating tetrapeptide tuftsin induces nitric oxide synthesis and stimulates murine macrophages to kill Leishmania parasites in vitro.

1994

The macrophage-activating tetrapeptide tuftsin was able to activate, in a dose-dependent manner, murine macrophages to express nitric oxide (NO) synthase and to produce NO. Tuftsin required lipopolysaccharides for the optimal induction of NO production and synergized with gamma interferon in the induction of NO synthesis. Tuftsin-dependent NO production was sensitive to inhibition by dexamethasone and the NO synthase specific inhibitor LGN-monomethylarginine (L-NMMA). Murine peritoneal macrophages activated by tuftsin were able to kill the amastigotes of the intracellular protozoan parasite Leishmania major in vitro.

LipopolysaccharidesImmunologyTuftsinArginineNitric OxideMicrobiologyDexamethasoneNitric oxidechemistry.chemical_compoundInterferon-gammaMicemedicineMacrophageAnimalsLeishmania majorInterferon gammaLeishmania majorMice Inbred BALB Comega-N-MethylargininebiologyMacrophagesMacrophage Activationbiology.organism_classificationLeishmaniaCell biologyNitric oxide synthaseInfectious DiseaseschemistryBiochemistrybiology.proteinOmega-N-MethylarginineTuftsinParasitologyFemalemedicine.drugResearch ArticleInfection and immunity
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Study of Potential Anti-Inflammatory Effects of Red Wine Extract and Resveratrol through a Modulation of Interleukin-1-Beta in Macrophages

2018

Inflammation has been described as an initiator event of major diseases with significant impacts in terms of public health including in cardiovascular disease, autoimmune disorders, eye diseases, age-related diseases, and the occurrence of cancers. A preventive action to reduce the key processes leading to inflammation could be an advantageous approach to reducing these associated pathologies. Many studies have reported the value of polyphenols such as resveratrol in counteracting pro-inflammatory cytokines. We have previously shown the potential of red wine extract (RWE) and the value of its qualitative and quantitative polyphenolic composition to prevent the carcinogenesis process. In thi…

LipopolysaccharidesInflammasomesred wine extractInterleukin-1betainflammation;interleukins;polyphenols;red wine extract;resveratrolAnti-Inflammatory AgentsWinelcsh:TX341-641resveratrolArticleCell LineMiceNLR Family Pyrin Domain-Containing 3 ProteinAnimalspolyphenolsPlant ExtractsMacrophagesfood and beveragesCARD Signaling Adaptor Proteins[SDV.AEN] Life Sciences [q-bio]/Food and NutritionRAW 264.7 Cellsinterleukinsinflammation[SDV.AEN]Life Sciences [q-bio]/Food and Nutritionlcsh:Nutrition. Foods and food supplyPhytotherapyNutrients
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Mitophagy in myeloid cells : role in infection with gram-negative bacteria

2017

Sepsis and related organ dysfunctions remain a leading cause of mortality in intensive care units. Increasing evidences have shed light on an unexpected link between mitochondria and immune cell functions. Alterations in mitochondrial functions have been reported in peripheral blood cells in sepsis. We hypothesize here that mitophagy might impact on phagocyte functions in the context of bacterial infection. Mitophagy is a mitochondria-dedicated autophagy that governs the elimination of dysfunctional mitochondria. We demonstrated here in vivo and in vitro that macrophages exposed to Gram-negative bacteria or their cell wall component LPS display a marked inhibition of mitophagy that constitu…

LipopolysaccharidesInflammation[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyMacrophagesSepsisMyeloid cellsMitophagyCellules myeloidesMitophagie[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Phenolic Substances from Phagnalon rupestre Protect against 2,4,6-Trinitrochlorobenzene-Induced Contact Hypersensitivity

2011

2-isoprenylhydroquinone-1-glucoside (1), 3,5-dicaffeoylquinic acid (2), and 3,5-dicaffeoylquinic acid methyl ester (3), isolated from Phagnalon rupestre, improved the contact hypersensitivity response to 2,4,6-trinitrochlorobenzene in mice. These phenolics reduced ear swelling and IL-1β content by 50% 24 h after challenge; in addition, 2 inhibited tumor necrosis factor-α by 53%. All three compounds also reduced interleukin-2 content by 50% 72 h after challenge. Both 2 and 3 inhibited metalloproteinase-9 levels in the skin lesions by 66% and 41%, respectively, and lowered cyclooxygenase-2 expression by 44% and 49%, respectively, at 24 h. Moreover, 2 was effective against atopic dermatitis in…

LipopolysaccharidesInterleukin-1betaPharmaceutical SciencePicryl ChloridePharmacognosyDermatitis ContactAnalytical ChemistryMicechemistry.chemical_compoundPhenolsDrug DiscoverymedicineAnimalsEdemaPhenolsSkinPharmacologychemistry.chemical_classificationCyclooxygenase 2 InhibitorsDose-Response Relationship DrugTraditional medicineTumor Necrosis Factor-alphaMacrophagesOrganic ChemistryContact hypersensitivityGlycosidePhagnalon rupestreEarmedicine.diseaseDisease Models AnimalComplementary and alternative medicineBiochemistrychemistryInterleukin-2Molecular MedicineContact dermatitisJournal of Natural Products
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Inhibition of NF-κB Activation and iNOS Induction by ent-Kaurane Diterpenoids in LPS-Stimulated RAW264.7 Murine Macrophages

2009

Xerophilusin A (1), xerophilusin B (2), longikaurin B (3), and xerophilusin F (4) from Isodon xerophylus inhibit LPS-induced NO production in RAW 264.7 macrophages, with IC(50) values of 0.60, 0.23, 0.44, and 0.67 muM, respectively, and they all inhibited mRNA production in these same cells. They decreased the luciferase activity in RAW 264.7 cells transiently transfected with the NF-kappaB-dependent luciferase reporter, with IC(50) values of 1.8, 0.7, 1.2, and 1.6 muM, respectively. Compounds 1-3 reduced NF-kappaB activation, with compound 4 showing no effect, but p65 translocation from the cytoplasm to the nucleus and the LPS-induced degradation of IkappaB were inhibited by all four test …

LipopolysaccharidesIsodonNitric Oxide Synthase Type IIPharmaceutical ScienceChromosomal translocationNitric OxideAnalytical ChemistryMiceDrug DiscoverymedicineAnimalsLuciferaseLuciferasesPharmacologyPlants MedicinalbiologyMolecular StructureReverse Transcriptase Polymerase Chain ReactionMonocyteMacrophagesAnti-Inflammatory Agents Non-SteroidalOrganic ChemistryNF-kappa BBiological activityTransfectionbiology.organism_classificationMolecular biologyIn vitromedicine.anatomical_structureBiochemistryComplementary and alternative medicineCell cultureIsodonMolecular MedicineDiterpenesDiterpenes KauraneJournal of Natural Products
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Pro-oxidant activity of indicaxanthin from Opuntia ficus indica modulates arachidonate metabolism and prostaglandin synthesis through lipid peroxide …

2014

Macrophages come across active prostaglandin (PG) metabolism during inflammation, shunting early production of pro-inflammatory towards anti-inflammatory mediators terminating the process. This work for the first time provides evidence that a phytochemical may modulate the arachidonate (AA) metabolism in lipopolysaccharide (LPS)-stimulated RAW 264.7 macrophages, promoting the ultimate formation of anti-inflammatory cyclopentenone 15deoxy-PGJ2. Added 1 h before LPS, indicaxanthin from Opuntia Ficus Indica prevented activation of nuclear factor-κB (NF-κB) and over-expression of PGE2 synthase-1 (mPGES-1), but up-regulated cyclo-oxygenase-2 (COX-2) and PGD2 synthase (H-PGDS), with final product…

LipopolysaccharidesLipid PeroxidesLipopolysaccharidePyridinesPhytochemicalsClinical BiochemistryProstaglandinIndicaxanthinmedicine.disease_causeBiochemistryCell LineMiceStructure-Activity Relationshipchemistry.chemical_compoundmedicineAnimalslcsh:QH301-705.5Inflammationlcsh:R5-920Arachidonic AcidNADPH oxidaseDose-Response Relationship DrugLipid peroxidebiologyMacrophagesOrganic ChemistryOpuntiaMetabolismOxidantsPro-oxidantBetaxanthinslcsh:Biology (General)chemistryBiochemistryOxidative stressFruitIndicaxanthin Phytochemicals Eicosanoids Inflammation Oxidative stress.Prostaglandinsbiology.proteinEicosanoidslipids (amino acids peptides and proteins)lcsh:Medicine (General)IndicaxanthinOxidative stressResearch PaperRedox Biology
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Dual anti-oxidant and anti-inflammatory actions of the electrophilic cyclooxygenase-2-derived 17-oxo-DHA in lipopolysaccharide- and cigarette smoke-i…

2014

Abstract Background 17-Oxo-DHA is an endogenous electrophilic derivative of the omega-3 fatty acid docosahexaenoic acid (DHA) which is generated in activated macrophages by the action of cyclooxygenase-2. Methods The ability of 17-oxo-DHA to control inflammation and oxidative stress was tested in human macrophages (THP-1) and bronchial epithelial cell line (16HBE) stimulated with cigarette smoke extract (CSE) and lipopolysaccharide (LPS). All data were further confirmed using primary bronchial epithelial cells, alveolar macrophages and peripheral blood mononuclear cells. Results 17-Oxo-DHA was a strong inducer of the anti-oxidant response promoting Nrf2 nuclear accumulation, leading to the …

LipopolysaccharidesLipopolysaccharideDocosahexaenoic Acidsmedicine.drug_classBiophysicsAnti-Inflammatory AgentsInflammationPharmacologymedicine.disease_causeBiochemistryAnti-inflammatoryAntioxidantsCell Linechemistry.chemical_compoundImmune systemmedicineHumansOmega 3 fatty acidMolecular BiologyInflammationChemistryMacrophagesSmokingfood and beveragesEpithelial CellsHeme oxygenaseBiochemistryCyclooxygenase 2Leukocytes Mononuclearlipids (amino acids peptides and proteins)Tumor necrosis factor alphamedicine.symptomOxidative stressBiochimica et biophysica acta
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Polymeric proanthocyanidins from Sicilian pistachio (Pistacia vera L.) nut extract inhibit lipopolysaccharide-induced inflammatory response in RAW 26…

2011

Positive effects of pistachio nut consumption on plasma inflammatory biomarkers have been described; however, little is known about molecular events associated with these effects. We studied the anti-inflammatory activity of a hydrophilic extract from Sicilian Pistacia L. (HPE) in a macrophage model and investigated bioactive components relevant to the observed effects. HPE oligomer/polymer proanthocyanidin fractions were isolated by adsorbance chromatography, and components quantified as anthocyanidins after acidic hydrolysis. Isoflavones were measured by gradient elution HPLC analysis. RAW 264.7 murine macrophages were pre-incubated with either HPE (1- to 20-mg fresh nut equivalents) or i…

LipopolysaccharidesLipopolysaccharideInflammation Isoflavones Macrophages Nut Proanthocyanidins Sicilian pistachioCell SurvivalAnti-Inflammatory AgentsNitric Oxide Synthase Type IIMedicine (miscellaneous)Nitric OxideCell LineNitric oxideMicechemistry.chemical_compoundWestern blotmedicineAnimalsNutsProanthocyanidinsViability assayFood scienceProstaglandin E2InflammationNutrition and DieteticsPistaciabiologymedicine.diagnostic_testPlant ExtractsTumor Necrosis Factor-alphaNF-kappa BIsoflavonesbiology.organism_classificationProanthocyanidinchemistryCyclooxygenase 2Pistaciamedicine.drug
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