Search results for "MDA"

showing 10 items of 275 documents

Circumdatin H, a new inhibitor of mitochondrial NADH oxidase, from Aspergillus ochraceus

2005

Circumdatin H (1), a new alkaloid from the culture broth of Aspergillus ochraceus, has been isolated, together with a known circumdatin, circumdatin E (2) and other known compounds: flavacol (3) and stephacidin A (4). The structure of 1 was established on the basis of chemical and spectral evidence. All of these alkaloids showed biological activity as inhibitors of the mammalian mitochondrial respiratory chain.

Magnetic Resonance SpectroscopyChemical PhenomenaSpectrophotometry InfraredStereochemistryCircumdatin HMass SpectrometryElectron Transportchemistry.chemical_compoundMultienzyme ComplexesDrug Discoveryheterocyclic compoundsNADH NADPH OxidoreductasesEnzyme InhibitorsPharmacologyAspergillus ochraceusBenzodiazepinonesbiologyChemistry PhysicalAlkaloidStephacidinBiological activityGeneral Medicinebiology.organism_classificationMitochondriaCircumdatin EMitochondrial respiratory chainchemistryBiochemistryFermentationNADH oxidaseSpectrophotometry UltravioletAspergillus ochraceus
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Mild systemic inflammation and moderate hypoxia transiently alter neuronal excitability in mouse somatosensory cortex

2016

During the perinatal period, the brain is highly vulnerable to hypoxia and inflammation, which often cause white matter injury and long-term neuronal dysfunction such as motor and cognitive deficits or epileptic seizures. We studied the effects of moderate hypoxia (HYPO), mild systemic inflammation (INFL), or the combination of both (HYPO + INFL) in mouse somatosensory cortex induced during the first postnatal week on network activity and compared it to activity in SHAM control animals. By performing in vitro electrophysiological recordings with multi-electrode arrays from slices prepared directly after injury (P8–10), one week after injury (P13–16), or in young adults (P28–30), we investig…

Male0301 basic medicineAction PotentialsKainate receptorStimulationPotassium ChlorideMicechemistry.chemical_compound0302 clinical medicineHypoxia6-Cyano-7-nitroquinoxaline-23-dioneNeuronsAge FactorsInterleukin-1βElectrophysiologyEpileptiform activityNeurologyAnesthesiaCNQXNMDA receptorFemalemedicine.symptommedicine.drugmedicine.medical_specialtyAMPA receptorIn Vitro TechniquesBiologyBicucullineMulti-electrode arrayArticlelcsh:RC321-57103 medical and health sciencesInternal medicinemedicineAnimalsGABA-A Receptor Antagonistslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryInflammationSystemic inflammationSomatosensory CortexHypoxia (medical)BicucullineBarrel cortexMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinology2-Amino-5-phosphonovalerateGene Expression Regulationchemistrynervous systemExploratory BehaviorExcitatory Amino Acid Antagonists030217 neurology & neurosurgeryNeurobiology of Disease
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Dopamine restores limbic memory loss, dendritic spine structure, and NMDAR-dependent LTD in the nucleus accumbens of alcohol-withdrawn rats

2018

Alcohol abuse leads to aberrant forms of emotionally salient memory, i.e., limbic memory, that promote escalated alcohol consumption and relapse. Accordingly, activity-dependent structural abnormalities are likely to contribute to synaptic dysfunctions that occur from suddenly ceasing chronic alcohol consumption. Here we show that alcohol-dependent male rats fail to perform an emotional-learning task during abstinence but recover their functioning byl-3,4-dihydroxyphenylalanin (l-DOPA) administration during early withdrawal.l-DOPA also reverses the selective loss of dendritic “long thin” spines observed in medium spiny neurons of the nucleus accumbens (NAc) shell of alcohol-dependent rats d…

Male0301 basic medicineDendritic spineDendritic SpinesAlcohol abuseDopamineDopamine AgentsAMPA receptorMotor ActivityNucleus accumbensMedium spiny neuronReceptors N-Methyl-D-AspartateNucleus AccumbensLevodopaRats Sprague-Dawley03 medical and health sciences0302 clinical medicineDopamineMemoryLimbic SystemmedicineAnimalsReceptors AMPAResearch ArticlesMemory DisordersAlcohol Abstinencebusiness.industryLong-Term Synaptic DepressionGeneral NeuroscienceDopaminergicRatsConfocal microscopyAlcoholism030104 developmental biologySynaptic plasticityLTDSettore BIO/14 - FarmacologiaNMDA receptorGlutamatebusinessNeuroscience030217 neurology & neurosurgerymedicine.drug
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Phencyclidine-induced disruption of oscillatory activity in prefrontal cortex: Effects of antipsychotic drugs and receptor ligands

2016

The non-competitive NMDA receptor (NMDA-R) antagonist phencyclidine (PCP) markedly disrupts thalamocortical activity, increasing excitatory neuron discharge and reducing low frequency oscillations (LFO, <4Hz) that temporarily group neuronal discharge. These actions are mainly driven by PCP interaction with NMDA-R in GABAergic neurons of the thalamic reticular nucleus and likely underlie PCP psychotomimetic activity. Here we report that classical (haloperidol, chlorpromazine, perphenazine) and atypical (clozapine, olanzapine, quetiapine, risperidone, ziprasidone, aripripazole) antipsychotic drugs - but not the antidepressant citalopram - countered PCP-evoked fall of LFO in the medial prefron…

Male0301 basic medicineOscillationsmedicine.drug_classDopamine AgentsAtypical antipsychoticPhencyclidineKainate receptorPharmacologyNeurotransmissionPrefrontal cortex03 medical and health scienceschemistry.chemical_compoundSerotonin Agents0302 clinical medicineHistamine AgentsmedicineAnimalsPharmacology (medical)NMDA receptor antagonistsAntipsychotic drugsRats WistarChlorpromazineEvoked PotentialsPhencyclidineBiological PsychiatryPharmacologyRacloprideAnalysis of VarianceDose-Response Relationship DrugFourier AnalysisChemistryElectroencephalographyPsychotomimeticRatsPsychiatry and Mental health030104 developmental biologyNeurologynervous systemSchizophreniaNBQXNeurology (clinical)Excitatory Amino Acid AntagonistsNeuroscience030217 neurology & neurosurgeryAntipsychotic Agentsmedicine.drug
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Relapse risk factors in anti-N-methyl-D-aspartate receptor encephalitis

2019

Aim: To identify factors that may predict and affect the risk of relapse in anti-N-methyl-D-aspartate receptor (NMDAR) encephalitis. Method: This was a retrospective study of an Italian cohort of patients with paediatric (≤18y) onset anti-NMDAR encephalitis. Results: Of the 62 children included (39 females; median age at onset 9y 10mo, range 1y 2mo–18y; onset between 2005 and 2018), 21 per cent relapsed (median two total events per relapsing patient, range 2–4). Time to first relapse was median 31.5 months (range 7–89mo). Severity at first relapse was lower than onset (median modified Rankin Scale [mRS] 3, range 2–4, vs median mRS 5, range 3–5; admission to intensive care unit: 0/10 vs 3/10…

Male030506 rehabilitationGastroenterologyCohort Studies0302 clinical medicineRetrospective StudieModified Rankin ScaleRecurrenceRisk FactorsChildrelapseAnti-N-Methyl-D-Aspartate Receptor EncephalitisHazard ratioItalyChild PreschoolCohortanti‐N‐methyl‐D‐aspartate receptor encephalitisFemale0305 other medical scienceEncephalitisHumanCohort studymedicine.medical_specialtyAdolescentSocio-culturaleanti-NMDAR antibodies03 medical and health sciencesanti-NMDARDevelopmental NeuroscienceInternal medicinemedicineAdolescent; Anti-N-Methyl-D-Aspartate Receptor Encephalitis; Child; Child Preschool; Cohort Studies; Female; Humans; Infant; Italy; Male; Recurrence; Retrospective Studies; Risk FactorsHumansPreschoolSurvival analysisRetrospective StudiesAutoimmune encephalitisbusiness.industryInfantRetrospective cohort studymedicine.diseaseanti-NMDAR antibodies autoimmune encephalitis anti‐N‐methyl‐D‐aspartate receptor encephalitisautoimmune encephalitisAnti-N-methyl-D-aspartate receptor encephalitis anti-NMDAR autoimmune encephalitis relapseAnti-N-Methyl-D-Aspartate Receptor EncephalitiPediatrics Perinatology and Child HealthNeurology (clinical)Cohort Studiebusiness030217 neurology & neurosurgery
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Phencyclidine inhibits the activity of thalamic reticular gamma-aminobutyric acidergic neurons in rat brain.

2014

Póster presentado en el IX Simposi de Neurobiologia Experimental, celebrado los días 22 y 23 de octubre de 2014 en Barcelona y organizado por la Societat Catalana de Biologia del Institut d'Estudis Catalans

MaleAction PotentialsPhencyclidinePrefrontal CortexLocal field potentialGABA AntagonistsThalamusthalamocortical networksNeural PathwaysmedicinePremovement neuronal activityAnimalsNMDA receptor antagonistsAntipsychotic drugsGABAergic NeuronsRats WistarPrefrontal cortexReceptorPhencyclidineClozapineBiological PsychiatryClozapineAnalysis of VarianceChemistryRatsschizophreniaElectrophysiologyParvalbuminspsychotic symptomsExcitatory postsynaptic potentialHallucinogensNeurosciencemedicine.drugBiological psychiatry
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Cannabinoid modulation of hippocampal long-term memory is mediated by mTOR signaling.

2009

Cognitive impairment is one of the most important negative consequences associated with cannabis consumption. We found that CB1 cannabinoid receptor (CB1R) activation transiently modulated the mammalian target of rapamycin (mTOR)/p70S6K pathway and the protein synthesis machinery in the mouse hippocampus, which correlated with the amnesic properties of delta9-tetrahydrocannabinol (THC). In addition, non-amnesic doses of either the mTOR blocker rapamycin or the protein synthesis inhibitor anisomycin abrogated the amnesic-like effects of THC, pointing to a mechanism involving new protein synthesis. Moreover, using pharmacological and genetic tools, we found that THC long-term memory deficits …

MaleCannabinoid receptormedicine.medical_treatmentGlutamic AcidHippocampusReceptors N-Methyl-D-AspartateGlutamatergicchemistry.chemical_compoundMiceCognitionReceptor Cannabinoid CB1Memorymental disordersmedicineAnimalsDronabinolPI3K/AKT/mTOR pathwayAnisomycingamma-Aminobutyric AcidMice KnockoutNeuronsProtein Synthesis InhibitorsSirolimusMemory DisordersChemistryGeneral NeuroscienceTOR Serine-Threonine KinasesRibosomal Protein S6 Kinases 70-kDanervous systemKnockout mouseNMDA receptorPhosphorylationCannabinoidNeuroscienceProtein KinasesAnisomycinCentral Nervous System AgentsSignal TransductionNature neuroscience
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Role of dopamine and glutamate receptors in cocaine-induced social effects in isolated and grouped male OF1 mice.

2005

Cocaine administration in paired male mice decreases social contacts as well as increases avoidance and flee elements. As dopamine (DA) and glutamate seem to be involved in some of cocaine's effects, an attempt was made to assess whether a range of associated receptors influenced the social impacts of this drug of abuse. The NMDA antagonist memantine (10 and 40 mg/kg); the AMPA antagonist CNQX (1 and 20 mg/kg); the DA release inhibitor CGS 10746b (2 and 8 mg/kg): the DA D1 antagonist SCH 23390 (0.05 and 0.5 mg/kg); and the DA D2/D3 antagonist raclopride (0.03 and 0.3 mg/kg) were administered prior to 25 mg/kg of cocaine and behaviour was evaluated during an encounter between an experimental…

MaleClinical BiochemistryPharmacologyToxicologyBiochemistryReceptors DopamineBehavioral Neurosciencechemistry.chemical_compoundMiceCocainemedicineAnimalsBiological PsychiatryPharmacologyRacloprideSCH-23390Behavior AnimalMemantineDopamine antagonistAntagonistchemistryReceptors GlutamateSocial IsolationDopamine receptorCNQXNMDA receptorPsychologymedicine.drugPharmacology, biochemistry, and behavior
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Role of AMPA glutamate receptors in the conditioned rewarding effects of MDMA in mice

2018

Abstract Currently, there is not an effective treatment for 3,4-methylenedioxymethamphetamine (MDMA) dependence but pharmacotherapies targeting glutamate neurotransmission are a promising strategy. Previously, we showed that blockade of glutamate NMDA and AMPA receptors impairs the conditioned rewarding effects of MDMA and cocaine, respectively. In this study we evaluated the role of AMPA receptors in the rewarding effects of MDMA in mice using the conditioned place preference (CPP) paradigm. Mice were conditioned with MDMA (1.25 mg/kg) 60 min after the treatment with saline or different doses (0.25, 1 and 5 mg/kg) of the AMPA/kainate receptor antagonist, 6-cyano-7-nitroquinoxaline-2,3-dion…

MaleHallucinogenMDMAmiceN-Methyl-34-methylenedioxyamphetamineAmphetamine-Related DisordersSpatial BehaviorKainate receptorAMPA receptorPharmacologyMice03 medical and health sciencesBehavioral Neurosciencechemistry.chemical_compound0302 clinical medicineRewardConditioning Psychologicalmental disordersmedicineAnimalsReceptors AMPAAMPA receptorsreward6-Cyano-7-nitroquinoxaline-23-dioneDose-Response Relationship Drugbusiness.industryGlutamate receptorMDMACNQXconditioned place preferenceConditioned place preference030227 psychiatrynervous systemchemistryHallucinogensCNQXNMDA receptorbusinessExcitatory Amino Acid Antagonistspsychological phenomena and processes030217 neurology & neurosurgerymedicine.drugBehavioural Brain Research
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Structural connectivity and subcellular changes after antidepressant doses of ketamine and Ro 25-6981 in the rat: an MRI and immuno-labeling study

2021

© The Author(s) 2021.

MaleHistologyDendritic spineInfralimbic cortexPrefrontal CortexNeuroimagingNeurofilamentRats Sprague-DawleyInfralimbic cortexWhite matterDorsal raphe nucleusPhenolsPiperidinesmedicineAnimalsPrefrontal cortexbiologyChemistryGeneral NeuroscienceDorsal raphe nucleusPsychotomimeticMagnetic Resonance ImagingAntidepressive AgentsRatsMyelin basic proteinMyelinizationmedicine.anatomical_structureFast-acting antidepressantbiology.proteinNMDA receptorOriginal ArticleKetamineAnatomyNeurosciencemedicine.drugBrain Structure and Function
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