Search results for "METASTASIS"

showing 10 items of 986 documents

Ileal Melanoma, A Rare Cause of Small Bowel Obstruction: Report of a Case, and Short Literature Review.

2020

Background: Malignant melanoma frequently spreads to the gastrointestinal tract, with 60% of patients with advanced metastatic disease showing digestive involvement; however, primary MM of the small intestine is a controversial diagnosis. In fact, whether these lesions arise as true small bowel primary neoplasms or represent metastases from unidentified cutaneous melanomas remains debatable. The most common complications are intestinal obstruction, massive gastrointestinal bleeding, and perforation. Objective & Methods: We report a case of a 64-year-old patient, with an unremarkable medical history, in which a late diagnosis of primary ileal malignant melanoma in the setting of an emer…

MaleCT scanmedicine.medical_specialtyGastrointestinal bleedingmedicine.medical_treatmentPerforation (oil well)Metastasis03 medical and health sciences0302 clinical medicineIleumLaparotomymedicineHumansRadiology Nuclear Medicine and imagingMedical historymetastatic disease.Ileal MelanomaSmall Bowel ObstructionAntineoplastic Agents AlkylatingMelanomaPharmacologyGastrointestinal tractCurrent Radiopharmaceuticalsbusiness.industryGeneral surgeryMelanomaMiddle Agedmedicine.diseaseBowel obstructionDacarbazineIleal NeoplasmsSettore MED/18 - Chirurgia GeneralePEThypercaptation030220 oncology & carcinogenesisPositron-Emission Tomography030211 gastroenterology & hepatologybusinessmetastatic diseaseIntestinal ObstructionCurrent radiopharmaceuticals
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Role of S128R polymorphism of E-selectin in colon metastasis formation.

2007

The extravasation of cancer cells is a key step of the metastatic cascade. Polymorphisms in genes encoding adhesion molecules can facilitate metastasis by increasing the strength of interaction between tumor and endothelial cells as well as impacting other properties of cancer cells. We investigated the Ser128Arg (a561c at the nucleotide level) polymorphism in the E-selectin gene in patients with metastatic colon cancer and its functional significance. Genotyping for a561c polymorphism was performed on 172 cancer patients and on an age-matched control population. The colon cancer group was divided into groups with (M+) and without observable metastasis (M−). For in vitro functional assays, …

MaleCancer ResearchColorectal cancerBiologyArginineTransfectionMetastasise-SELECTIN; COLON CANCER METASTASISSettore BIO/13 - Biologia ApplicataCell MovementE-selectinmedicineCell AdhesionSerineTumor Cells CulturedHumansNeoplasm MetastasisPolymorphism GeneticCell adhesion moleculeCancerTransfectionMiddle Agedmedicine.diseaseExtravasationColon Carcinoma E-Selectin Metastasis PolymorphismPhenotypeOncologyImmunologyCancer cellColonic NeoplasmsCancer researchbiology.proteinFemaleE-SelectinSignal TransductionInternational journal of cancer
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Phase I study of FOLFIRI plus pimasertib as second-line treatment for KRAS-mutated metastatic colorectal cancer

2015

BACKGROUND: The mitogen-activated protein kinase (MAPK) pathway has been implicated in the molecular pathogenesis of human cancers, including metastatic colorectal cancer (mCRC). This provides a rationale for the development of MAPK-targeted agents such as pimasertib. METHODS: Patients with KRAS mutant mCRC were treated in the second-line setting with FOLFIRI (5-fluorouracil/folinic acid/irinotecan) plus pimasertib. The primary objective of the safety run-in phase was to determine the maximum-tolerated dose (MTD) and the recommended phase II dose of pimasertib combined with FOLFIRI. RESULTS: Sixteen patients were enrolled in the trial. Ten and six patients were treated daily with 45 and 60 …

MaleCancer ResearchColorectal cancermedicine.medical_treatmentLeucovorinColorectal NeoplasmPharmacologymedicine.disease_causepimasertibcombination therapyAntineoplastic Combined Chemotherapy ProtocolsNeoplasm Metastasiscombination therapy second-line treatmentAged 80 and overProto-Oncogene ProteinCetuximabKRAS-mutated metastatic colorectal cancerMedicine (all)MEK inhibitorMiddle AgedNeoplasm MetastasiTreatment OutcomeOncologyFluorouracilFOLFIRISecond-line treatmentFemaleFluorouracilKRASColorectal NeoplasmsPimasertibHumanmedicine.drugNiacinamideProto-Oncogene Proteins p21(ras)FOLFIRIProto-Oncogene ProteinsmedicineHumansCombination therapyneoplasmsAgedChemotherapyMEK inhibitorAntineoplastic Combined Chemotherapy Protocolbusiness.industryKRAS -mutated metastatic colorectal cancerGenes raras Proteinmedicine.diseasedigestive system diseasesGenes rassecond-line treatmentMutationras ProteinsClinical StudyCancer researchCamptothecinHuman medicinebusinessCamptothecinThe British journal of cancer
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Neuroblastoma after Childhood: Prognostic Relevance of Segmental Chromosome Aberrations, ATRX Protein Status, and Immune Cell Infiltration

2014

AbstractNeuroblastoma (NB) is a common malignancy in children but rarely occurs during adolescence or adulthood. This subgroup is characterized by an indolent disease course, almost uniformly fatal, yet little is known about the biologic characteristics. The aim of this study was to identify differential features regarding DNA copy number alterations, α-thalassemia/mental retardation syndrome X-linked (ATRX) protein expression, and the presence of tumor-associated inflammatory cells. Thirty-one NB patients older than 10 years who were included in the Spanish NB Registry were considered for the current study; seven young and middle-aged adult patients (range 18-60 years) formed part of the c…

MaleCancer ResearchHet heterogeneousGene ExpressionNeuroblastomaImmunophenotypingRegistriesYoung adultNeoplasm MetastasisMLPA multiplex ligation probe amplificationChildIn Situ Hybridization FluorescenceNuclear ProteinsMiddle AgedAYA adolescent and young adultsPrognosislcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensNCA numerical chromosome aberrationImmunohistochemistryFemaleSCA segmental chromosome aberrationIHC immunohistochemistryNB neuroblastomaAdultX-linked Nuclear ProteinAdolescentaSNP single nucleotide polymorphism arrayBiologyMalignancyChromosome aberrationPolymorphism Single Nucleotidelcsh:RC254-282ArticleImmunophenotypingYoung AdultLymphocytes Tumor-InfiltratingNeuroblastomacnLOH copy-neutral loss of heterozygositymedicineHumansHom homogeneousATRXNeoplasm StagingChromosome AberrationsDNA Helicasesmedicine.diseaseSpainMNNA MYCN not amplifiedCancer researchFSCA focal segmental chromosome aberrationCD8MNA MYCN amplifiedNeoplasia
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Vinflunine in Metastatic Urothelial Carcinoma of the Bladder in Progression after a Platinum-Containing Regimen

2019

<b><i>Background:</i></b> Vinflunine is a microtubule inhibitor of the vinca alkaloid class approved for the treatment of urothelial bladder carcinoma after a platinum-containing regimen. <b><i>Methods:</i></b> To evaluate the effectiveness of vinflunine, we enrolled 80 subjects with a histologically confirmed diagnosis of metastatic urothelial bladder carcinoma that had previously undergone chemotherapy with a platinum-containing regimen and had measurable lesions according to the Response Evaluation Criteria in Solid Tumors (RECIST). The patients (<i>n</i> = 80) received vinflunine (Javlor®) every 3 weeks at 320 mg/m<sup>2&…

MaleCancer ResearchJavlor®medicine.medical_treatmentMetastasichemistry.chemical_compound0302 clinical medicineRetrospective StudieAntineoplastic Combined Chemotherapy Protocols030212 general & internal medicineNeoplasm MetastasisAged 80 and overVinflunineGeneral MedicineMiddle AgedTolerabilityNeoplasm MetastasiOncologyTolerabilityResponse Evaluation Criteria in Solid Tumors030220 oncology & carcinogenesisUrinary Bladder NeoplasmUrothelial carcinomaFemaleHumanmedicine.medical_specialtyMetastatic Urothelial Carcinomamedicine.drug_classBladderUrologyPainVinblastineVinca alkaloid03 medical and health sciencesCarcinomamedicineHumansChemotherapyRetrospective StudiesAgedChemotherapyAntineoplastic Combined Chemotherapy ProtocolToxicitybusiness.industrymedicine.diseaseRegimenUrinary Bladder NeoplasmschemistryCisplatinbusiness
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Cluster of differentiation 44 promotes osteosarcoma progression in mice lacking the tumor suppressor Merlin.

2019

Merlin is a versatile tumor suppressor protein encoded by the NF2 gene. Several lines of evidence suggest that Merlin exerts its tumor suppressor activity, at least in part, by forming an inhibitory complex with cluster of differentiation 44 (CD44). Consistently, numerous NF2 mutations in cancer patients are predicted to perturb the interaction of Merlin with CD44. We hypothesized that disruption of the Merlin-CD44 complex through loss of Merlin, unleashes putative tumor- or metastasis-promoting functions of CD44. To evaluate the relevance of the Merlin-CD44 interaction in vivo, we compared tumor growth and progression in Cd44-positive and Cd44-negative Nf2-mutant mice. Heterozygous Nf2-mut…

MaleCancer ResearchLung NeoplasmsIntegrin610 MedizinBone NeoplasmsBiologyBone and Boneslaw.inventionMetastasis03 medical and health sciencesMice0302 clinical medicineDownregulation and upregulationlaw610 Medical sciencesCell Line TumormedicineCell AdhesionAnimalsHumansLungCell ProliferationMice KnockoutNeurofibromin 2OsteosarcomaCluster of differentiationCD44medicine.diseaseMerlin (protein)Disease Models AnimalHyaluronan ReceptorsOncology030220 oncology & carcinogenesisbiology.proteinCancer researchDisease ProgressionOsteosarcomaSuppressorInternational journal of cancerREFERENCES
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DNA image cytometry. A prognostic tool in squamous cell carcinoma of the esophagus?

1991

In 45 patients who underwent an esophagus resection due to a squamous cell carcinoma, in addition to the TNM classification and usual morphologic criteria, the paraffin-embedded material underwent deparaffinization, was rehydrated, and was mechanically and enzymatically processed into a single-cell solution. For evaluating the DNA histogram this was analyzed with the help of automatic single-cell cytophotometric study. The method, contrary to that of flow cytometric study, allows for the selective analysis of tumor cells due to the electronically, previously given selection criteria, whereas artifacts, stroma, and infection cells remain excluded from analysis. The multivariate analysis show…

MaleCancer ResearchPathologymedicine.medical_specialtyEsophageal NeoplasmsTumor cellsResectionStromaEsophagus CarcinomamedicineHumansBasal cellEsophagusDNA Image CytometryNeoplasm StagingPloidiesbusiness.industryDNA NeoplasmPrognosisSurvival Analysismedicine.anatomical_structureOncologyLymphatic MetastasisCarcinoma Squamous CellFemaleCytophotometrybusinessMedian survivalCancer
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Influence of class M1 glutathione S-transferase (GST Mu) polymorphism on GST M1 gene expression level and tumor size in oral squamous cell carcinoma.

2010

Glutathione S-transferases (GST) are antioxidant enzymes and oxidative stress markers in oral carcinogenesis. They present a system of polymorphic proteins. Some variants are associated with increased sensitivity to toxic compounds, as it is known for the GSTM1-null variant allele. However, the influence of the GSTM1 allele variant genotype on GSTM1-mRNA quantity in oral squamous cell carcinoma (OSCC) and normal mucosa as well as the impact on prognosis remains unclear. The genotype for GSTM1 (mutation vs. wild type) was determined by polymerase chain reaction (PCR) using genomic DNA extracted from peripheral blood from 28 OSCC patients. From the same patients, 28 pairs of OSCC cells and no…

MaleCancer ResearchPathologymedicine.medical_specialtyGenotypemedicine.disease_causeGenotypeGene expressionmedicineCarcinomaBiomarkers TumorHumansAlleleneoplasmsAgedGlutathione TransferaseNeoplasm StagingRegulation of gene expressionPolymorphism Geneticintegumentary systembiologyWild typeMouth Mucosamedicine.diseaseMolecular biologyTumor BurdenGene Expression Regulation NeoplasticGlutathione S-transferaseOncologyLymphatic Metastasisbiology.proteinCarcinoma Squamous CellFemaleMouth NeoplasmsOral SurgeryCarcinogenesisOral oncology
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Chemokine receptor CCR7 enhances intrahepatic and lymphatic dissemination of human hepatocellular cancer.

2006

Despite many pathophysiological analyses, the process of tumor dissemination of hepatocellular carcinoma (HCC) remains vague. In diverse tumor entities, expression of the chemokine receptor, CCR7, has been linked to tumor dissemination and poor prognosis. Therefore, we evaluated, whether CCR7 exerts similar effects in human HCC. CCR7 expression analysis was performed in vitro on human hepatoma cell lines (Huh7, Hep3B, wt HepG2, p53 dominant negative transfected HepG2). In addition, CCR7 expression was evaluated in 39 patients with hepatocellular cancer and correlated with both, tumor and patients characteristics. Human hepatocellular carcinoma samples and hepatoma cell lines displayed varia…

MaleCancer ResearchPathologymedicine.medical_specialtyReceptors CCR7Carcinoma Hepatocellularchemical and pharmacologic phenomenaC-C chemokine receptor type 7BiologyMetastasisChemokine receptorimmune system diseasesCell Line TumormedicineCarcinomaHumansGenetic Predisposition to DiseaseAgedGenes DominantOncogeneLiver NeoplasmsCancerhemic and immune systemsGeneral MedicineMiddle Agedmedicine.diseasePrognosisdigestive system diseasesGene Expression Regulation NeoplasticLymphatic systemOncologyLiverHepatocellular carcinomaLymphatic MetastasisFemaleReceptors ChemokineOncology reports
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Overexpression of cyclin D1 and interaction between p27Kip1 and tumour thickness predict lymph node metastases occurrence in lower lip squamous cell …

2005

We have attempted to identify those subgroups of patients most likely to develop lymph node metastases from squamous cell carcinoma of the lower lip (LLSSC). A total of 97 subjects, who did not undergo elective neck dissection, were recruited into the 60-month disease-free survival study. After univariate analysis, tumour size, histological grading, maximal thickness, perineural invasion and immunoreactivity to cyclin D1 and p27Kip1 proteins proved to be significant factors. Tests of the effect of interaction between p27Kip1 LI and tumour thickness yielded that the impact of tumour thickness on the risk of lymph node metastases was modified by the percentage of p27Kip1 positive cells. Subse…

MaleCancer ResearchPathologymedicine.medical_specialtyp27Kip1medicine.medical_treatmentPerineural invasionCell Cycle ProteinsSettore MED/08 - Anatomia PatologicaMetastasisLymph node metastasiCyclin D1Squamous cell carcinomaBiomarkers TumorCarcinomaHumansMedicineCyclin D1Lymph nodeTumour thickness.AgedAged 80 and overbusiness.industryTumor Suppressor ProteinsNeck dissectionMiddle AgedPrognosismedicine.diseaseImmunohistochemistryLymph node metastasis; Squamous cell carcinoma; Lower lip; Cyclin D1; p27Kip1; Tumour thickness.medicine.anatomical_structureOncologyEpidermoid carcinomaLymphatic MetastasisLip NeoplasmsLower lipCarcinoma Squamous CellRegression AnalysisImmunohistochemistryFemaleOral SurgerybusinessCyclin-Dependent Kinase Inhibitor p27Oral Oncology
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