Search results for "MICE"

showing 10 items of 6027 documents

In vitro and in vivo antileishmanial and trypanocidal studies of new N-benzene- and N-naphthalenesulfonamide derivatives.

2013

We report in vivo and in vitro antileishmanial and trypanocidal activities of a new series of N-substituted benzene and naphthalenesulfonamides 1-15. Compounds 1-15 were screened in vitro against Leishmania infantum , Leishmania braziliensis , Leishmania guyanensis , Leishmania amazonensis , and Trypanosoma cruzi . Sulfonamides 6e, 10b, and 10d displayed remarkable activity and selectivity toward T. cruzi epimastigotes and amastigotes. 6e showed significant trypanocidal activity on parasitemia in a murine model of acute Chagas disease. Moreover, 6e, 8c, 9c, 12c, and 14d displayed interesting IC50 values against Leishmania spp promastigotes as well as L. amazonensis and L. infantum amastigot…

Trypanosoma cruziLeishmania guyanensisAntiprotozoal AgentsParasitemiaMicrobiologyCell LineMiceStructure-Activity RelationshipIn vivoparasitic diseasesDrug DiscoverymedicineAnimalsHumansComputer SimulationAmastigoteTrypanosoma cruziLeishmaniaSulfonamidesbiologyChemistryBenzenebiology.organism_classificationmedicine.diseaseLeishmaniaLeishmania braziliensisDrug DesignMolecular MedicineFemaleLeishmania infantumJournal of medicinal chemistry
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Synthesis and biological evaluation of papain-family cathepsin L-like cysteine protease inhibitors containing a 1,4-benzodiazepine scaffold as antipr…

2014

Novel papain-family cathepsin L-like cysteine protease inhibitors endowed with antitrypanosomal and antimalarial activity were developed, through an optimization study of previously developed inhibitors. In the present work, we studied the structure-activity relationships of these derivatives, with the aim to develop new analogues with a simplified and more synthetically accessible structure and with improved antiparasitic activity. The structure of the model compounds was significantly simplified by modifying or even eliminating the side chain appended at the C3 atom of the benzodiazepine scaffold. In addition, a simple methylene spacer of appropriate length was inserted between the benzod…

Trypanosomamedicine.drug_classPeptidomimeticStereochemistryAntiparasiticCell SurvivalCathepsin LAntiprotozoal AgentsCysteine Proteinase InhibitorsBiochemistryCathepsin BCell LineCathepsin Lchemistry.chemical_compoundBenzodiazepinesMiceStructure-Activity RelationshipDrug DiscoverymedicineMoietyAnimalsGeneral Pharmacology Toxicology and PharmaceuticsPharmacologyCathepsinbiologyOrganic ChemistryCombinatorial chemistryCysteine proteasePapainantiprotozoal agents; inhibitors; Malaria; Peptidomimetics; structure-activity relationshipsCysteine EndopeptidaseschemistryAntiprotozoalbiology.proteinMolecular MedicineProtein BindingChemMedChem
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Damping excessive inflammation and tissue damage in Mycobacterium tuberculosis infection by Toll IL-1 receptor 8/single Ig IL-1-related receptor, a n…

2007

Abstract Toll IL-1R 8/single Ig IL-1-related receptor (TIR8/SIGIRR) is a member of the IL-1R family, expressed by epithelial tissues and immature dendritic cells, and is regarded as a negative regulator of TLR/IL-1R signaling. Tir8-deficient mice were rapidly killed by intranasal administration of low doses of Mycobacterium tuberculosis, despite controlling efficiently the number of viable bacilli in different organs. Tir8−/−-infected mice showed an increased number of neutrophils and macrophages in the lungs; however, mycobacteria-specific CD4 and CD8 T cells were similar in Tir8−/− and Tir8+/+ mice. Exaggerated mortality of Tir8−/− mice was due to massive liver necrosis and was accompanie…

TuberculosisNeutrophilsImmunologyInterleukin-1betaInflammationBiologyPeripheral blood mononuclear cellAntibodiesMycobacterium tuberculosisMiceNecrosisCell MovementMacrophages AlveolarmedicineImmunology and AllergyCytotoxic T cellAnimalsReceptorLungTuberculosis PulmonaryInflammationLungTumor Necrosis Factor-alphaToll-Like ReceptorsReceptors Interleukin-1Dendritic CellsMycobacterium tuberculosismedicine.diseasebiology.organism_classificationIn vitroMice Mutant Strainsmedicine.anatomical_structureLiverImmunologyCytokinesmedicine.symptomToll IL-1 Receptor 8/Single Ig IL-1-Related Receptor Inlfammation Mycobacterium tuberculosisInterleukin-1Signal Transduction
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Biology of gama delta T Cells in Tuberculosis and Malaria

2002

Tuberculosis and malaria remain the leading causes of mortality among human infectious diseases in the world. It is estimated that 3 to 5 million people die from tuberculosis and malaria each year. Although it is traditionally believed that CD4 and CD8 alphabeta T lymphocytes are mandatory for protective immune responses against Mycobacterium tuberculosis and Plasmodium falciparum (the ethiologic agents of tuberculosis and the most severe form of malaria, respectively), there is still incomplete understanding of the mechanisms of immune protection and of the causes of its failure in the affected patients. Several studies in humans and animal models have suggested that Vgamma9/Vdelta2 T cell…

TuberculosisT cellPlasmodium falciparumBiochemistryMycobacterium tuberculosisMiceImmune systemAntigenT-Lymphocyte Subsetsparasitic diseasesmedicineAnimalsHumansTuberculosisMalaria FalciparumMolecular BiologybiologyReceptors Antigen T-Cell gamma-deltaPlasmodium falciparumMycobacterium tuberculosisGeneral Medicinemedicine.diseasebiology.organism_classificationVirologyDisease Models Animalmedicine.anatomical_structureImmunologyMolecular MedicineCD8MalariaCurrent Molecular Medicine
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IL-4 depletion enhances host resistance and passive IgA protection against tuberculosis infection in BALB/c mice.

2007

The influence of Th2 cytokines in tuberculosis has been a matter of dispute. Here we report that IL-4 has a profound regulatory effect on the infection of BALB/c mice with Mycobacterium tuberculosis. Depletion of IL-4 with a neutralizing mAb caused only evanescent reduction of lung infection, but when combined with i.n. inoculations of IgA anti-mycobacterial alpha-crystallin mAb and mouse rIFN-gamma, we observed a 40-fold reduction of the bacterial counts in the lungs at 3 wks following i.n. infection (p<0.001). In genetically deficient IL-4-/- BALB/c mice, infection in both lung and spleen was substantially reduced for up to 8 wks without further treatment. Reconstitution of IL-4-/- mice w…

Tuberculosismedicine.medical_treatmentImmunologySpleenNitric OxideBALB/cMicrobiologyProinflammatory cytokineMycobacterium tuberculosisInterferon-gammaMiceImmunitymedicineImmunology and AllergyAnimalsTuberculosis PulmonaryMice KnockoutMice Inbred BALB CbiologyMacrophagesImmunization PassiveImmunotherapyMycobacterium tuberculosisbiology.organism_classificationmedicine.diseaseAntibodies Bacterial infections Cytokines TuberculosisImmunity InnateImmunoglobulin Amedicine.anatomical_structureImmunologybiology.proteinInterleukin-4AntibodyEuropean journal of immunology
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Synthesis and biological evaluation of 2-(3 ',4 ',5 '-trimethoxybenzoyl)-3-amino 5-aryl thiophenes as a new class of tubulin inhibitors

2006

2-(3',4',5'-Trimethoxybenzoyl)-3-amino-5-aryl/heteroaryl thiophene derivatives were synthesized and evaluated for antiproliferative activity, inhibition of tubulin polymerization, and cell cycle effects. SARs were elucidated with various substitutions on the aryl moiety 5-position of the thienyl ring. Substituents at the para-position of the 5-phenyl group showed antiproliferative activity in the order of F=CH(3) > OCH(3)=Br=NO(2) > CF(3)=I > OEt. Several of these compounds led to arrest of HL-60 cells in the G2/M phase of the cell cycle and induction of apoptosis.

Tubulin ModulatorsStereochemistryArylCell CycleApoptosisBiological activityThiophenesCell cycleChemical synthesisTubulin ModulatorsIn vitro- Tubulin Inhibitors -Antiproliferative activity -5-Aryl TiophenesMiceStructure-Activity Relationshipchemistry.chemical_compoundchemistryCell Line TumorDrug DiscoveryAnimalsHumansMolecular MedicineStructure–activity relationshipMoietyDrug Screening Assays Antitumor
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Bezmugurkaulnieku daudzveidība Heterobasidion spp. augļķermeņos uz trupējušas egles koksnes

2017

Sakņu piepe Heterobasidion spp. ir koksni noārdošas sēnes, kas rada ievērojamus zaudējumus mežsaimniecībai. Informācija par tās apdzīvojošiem bezmugurkaulniekiem ir fragmentāra. Darba mērķis ir izpētīt bezmugurkaulnieku daudzveidību sakņu piepes augļķermeņos uz trupējušiem egļu baļķiem un noskaidrot tos ietekmējošos faktorus. Divos parauglaukumos analizēti 134 H. annosum s.l. augļķermeņu, egles koksnes bez piepes un augsnes paraugi. Posmkāju izpētē ievākti 7341 bezmugurkaulnieku indivīdi. Pētītajos substrātos atšķīrās konstatēto bezmugurkaulnieku skaits un sastāvs. Dominēja ērces, kolembolas, vaboles, tūkstoškāji un divspārņi. Bezmugurkaulnieku daudzveidība sakņu piepes augļķermeņos saistīt…

Tullgren lamatasbezmugurkaulniekiparastā egleBioloģijasakņu piepemicetofāgija
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Capturing colorectal cancer inter-tumor heterogeneity in patient-derived xenograft (PDX) models

2019

Patient‐derived xenograft (PDX) models have become an important asset in translational cancer research. However, to provide a robust preclinical platform, PDXs need to accommodate the tumor heterogeneity that is observed in patients. Colorectal cancer (CRC) can be stratified into four consensus molecular subtypes (CMS) with distinct biological and clinical features. Surprisingly, using a set of CRC patients, we revealed the partial representation of tumor heterogeneity in PDX models. The epithelial subtypes, the largest subgroups of CRC subtype, were very ineffective in establishing PDXs, indicating the need for further optimization to develop an effective personalized therapeutic approach …

Tumor Markers and SignaturesCMSShort Reportcolorectal cancerXenograft Model Antitumor AssaysDisease Models AnimalMicecell proliferationxenograft CMStumor subtypeAnimalsHeterograftsHumansxenograftColorectal NeoplasmsPDX
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Analysis of p53 and mdm2 proteins in malignant fibrous histiocytoma in absence of gene alteration: prognostic significance.

2000

TP53 and MDM2 genes and their protein expression were evaluated in frozen and paraffin-embedded tissue from 27 patients with malignant fibrous histiocytoma to elucidate the relationship between them, their implication in tumor progression mechanisms and their possible diagnostic-prognostic value in malignant fibrous histiocytoma. Single-strand conformation polymorphism analysis and direct sequencing of polymerase chain reaction-amplified DNA were used to establish two TP53 mutations (7.4%): a point mutation and a 63-bp duplication. Amplification of the MDM2 gene was observed in two tumors (7.4%) by means of Southern-blot analysis, one of them also carrying the TP53 point mutation. Immunohis…

Tumor suppressor geneBlotting WesternSoft Tissue NeoplasmsBiologyPolymerase Chain ReactionPathology and Forensic MedicineImmunoenzyme TechniquesMiceProto-Oncogene ProteinsGene duplicationGene expressionAnimalsHumansneoplasmsMolecular BiologyGeneTP53 Gene MutationPolymorphism Single-Stranded ConformationalCell NucleusMice Inbred BALB CHistiocytoma Benign FibrousPoint mutationNuclear ProteinsSingle-strand conformation polymorphismProto-Oncogene Proteins c-mdm2Cell BiologyGeneral MedicineDNA NeoplasmMolecular biologyNeoplasm ProteinsSurvival RateBlotting SouthernTumor progressionMutationCancer researchNeoplasm Recurrence LocalTumor Suppressor Protein p53Virchows Archiv : an international journal of pathology
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Direct Activation of Bax by p53 Mediates Mitochondrial Membrane Permeabilization and Apoptosis

2004

The tumor suppressor p53 exerts its anti-neoplastic activity primarily through the induction of apoptosis. We found that cytosolic localization of endogenous wild-type or trans-activation–deficient p53 was necessary and sufficient for apoptosis. p53 directly activated the proapoptotic Bcl-2protein Bax in the absence of other proteins to permeabilize mitochondria and engage the apoptotic program. p53 also released both proapoptotic multidomain proteins and BH3-only proteins [Proapoptotic Bcl-2family proteins that share only the third Bcl-2homology domain (BH3)] that were sequestered by Bcl-xL. The transcription-independent activation of Bax by p53 occurred with similar kinetics and concentra…

Tumor suppressor geneProtein ConformationUltraviolet RaysWheat Germ AgglutininsRecombinant Fusion Proteinsbcl-X ProteinApoptosisEndogenyMitochondrionBiologyPermeabilityHomology (biology)law.inventionMiceCytosollawProto-Oncogene ProteinsMitochondrial membrane permeabilizationAnimalsHumansCells CulturedCell Line Transformedbcl-2-Associated X ProteinCell NucleusMultidisciplinaryCytochromes cIntracellular MembranesGenes p53MitochondriaCell biologyCytosolGene Expression RegulationProto-Oncogene Proteins c-bcl-2ApoptosisLiposomesMutationSuppressorTumor Suppressor Protein p53biological phenomena cell phenomena and immunityCarrier ProteinsBH3 Interacting Domain Death Agonist ProteinHeLa CellsScience
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