Search results for "MIMICRY"

Colour alone matters : no predator generalization among morphs of an aposematic moth

2018

Local warning colour polymorphism, frequently observed in aposematic organisms, is evolutionarily puzzling. This is because variation in aposematic signals is expected to be selected against due to predators' difficulties associating several signals with a given unprofitable prey. One possible explanation for the existence of such variation is predator generalization, which occurs when predators learn to avoid one form and consequently avoid other sufficiently similar forms, relaxing selection for monomorphic signals. We tested this hypothesis by exposing the three different colour morphs of the aposematic wood tiger moth, Arctia plantaginis, existing in Finland to local wild-caught predato…

Molecular phylogeny of Malagasy poison frogs, genus Mantella (Anura: Mantellidae): homoplastic evolution of colour pattern in aposematic amphibians

2002

Abstract We studied the evolution of colour pattern in Malagasy poison frogs, genus Mantella , a group of diurnal and toxic frogs endemic to Madagascar. Based on a phylogeny reconstructed using 1130 bp of the mitochondrial 16S rRNA gene, the genus can be divided into five species groups. Within some of these groups, interspecific genetic divergences were very low (1.2–2.8% sequence divergence) while colour patterns were markedly different. In contrast, Mantella madagascariensis and M . baroni , two species which show extremely similar dorsal coloration patterns, were not included in the same clade. This conclusion was supported by high bootstrap values and by significant rejection of altern…

Safety in Numbers: How Color Morph Frequency Affects Predation Risk in an Aposematic Moth

2021

Polymorphic warning signals in aposematic systems are enigmatic because predator learning should favor the most common form, creating positive frequency-dependent survival. However, many populations exhibit variation in warning signals. There are various selective mechanisms that can counter positive frequency-dependent selection and lead to temporal or spatial warning signal diversification. Examining these mechanisms and their effects requires first confirming whether the most common morphs are favored at both local and regional scales. Empirical examples of this are uncommon and often include potentially confounding factors, such as a lack of knowledge of predator identity and behavior. …

A tale of 2 signals: signal mimicry between aposematic species enhances predator avoidance learning

2010

Mullerian mimicry, where 2 or more unrelated aposematic species resemble one another, is predicted to reduce the per capita mortality of co-mimics by allowing them to share the cost of educating nao ¨ve predators about their unpalatability. However, the specific assumptions and predictions of Muller's theory of shared resemblance have been previously unsupported; some authors have suggested that the benefits of signal similarity are undetectable or at best very small. We demonstrate clearly and un- ambiguously that mimicry between 2 defended forms can provide substantial protection from uneducated predators in the manner proposed originally by Muller. By utilizing prey signals that were des…

Is molecular mimicry the culprit in the autoimmune haemolytic anaemia affecting patients with COVID‐19?

2020

Whence pseudoscience? An epidemiological approach

2017

In this paper, we develop an epidemiological approach to account for the typical features and persistent popularity of pseudoscience. An epidemiology of pseudoscience aims at explaining why some beliefs become widely distributed whereas others do not and hence seeks to identify the factors that exert a causal effect on this distribution. We pinpoint and discuss several factors that promote the dissemination of pseudoscientific beliefs. In particular, we argue that such beliefs manage to spread widely because they are intuitively appealing, manage to hitchhike on the authority of science, and successfully immunize themselves from criticism.

The Role of Molecular Chaperones in Virus Infection and Implications for Understanding and Treating COVID-19

2020

The COVID-19 pandemic made imperative the search for means to end it, which requires a knowledge of the mechanisms underpinning the multiplication and spread of its cause, the coronavirus SARS-CoV-2. Many viruses use members of the hosts’ chaperoning system to infect the target cells, replicate, and spread, and here we present illustrative examples. Unfortunately, the role of chaperones in the SARS-CoV-2 cycle is still poorly understood. In this review, we examine the interactions of various coronaviruses during their infectious cycle with chaperones in search of information useful for future research on SARS-CoV-2. We also call attention to the possible role of molecular mimicry in the dev…

The impact of mimicry on sales – Evidence from field and lab experiments

2011

A buyer's observation that one or more people are consuming a product can lead that buyer to consume the product as well. The evidence supporting unconscious and unintentional (automatic) mimicry of consumption suggests that it is a pervasive and robust phenomenon. However, up until now most findings on the antecedents of mimicry have been obtained from lab studies. Using a field study, the current research shows that passengers in a train mimic the consumption behavior of other passengers. Two subsequent lab studies suggest that mimicry of consumption is all the more powerful the more people there are consuming and the more intense and consistent their consumption behavior is. However, the…

Analysis of p16 gene and protein status in malignant mixed Müllerian tumors of the uterus

2010

Molecular mimicry in the post-COVID-19 signs and symptoms of neurovegetative disorders?

2021

Many individuals who have severe forms of COVID-19 experience a suite of neurovegetative signs and symptoms (eg, tachycardia) after their recovery, suggesting that the imbalance of the sympathetic-parasympathetic activity of the autonomic nervous system1 could continue for many weeks or months after respiratory symptoms stop. Moreover, a reduction of the parasympathetic tone could have a role in restricting the cholinergic anti-inflammatory pathway, thus favouring hyperinflammation and cytokine storm in the most severe phases of the disease. As reported by Guglielmo Lucchese in The Lancet Microbe,2 SARS-CoV-2 can damage the nervous system via an indirect mechanism, resulting in a high preva…