Search results for "MITO"

showing 10 items of 2513 documents

Fas signaling-mediated T

2018

Fas induces apoptosis in activated T cell to maintain immune homeostasis, but the effects of non-apoptotic Fas signaling on T cells remain unclear. Here we show that Fas promotes TH9 cell differentiation by activating NF-κB via Ca2+-dependent PKC-β activation. In addition, PKC-β also phosphorylates p38 to inactivate NFAT1 and reduce NFAT1-NF-κB synergy to promote the Fas-induced TH9 transcription program. Fas ligation exacerbates inflammatory bowel disease by increasing TH9 cell differentiation, and promotes antitumor activity in p38 inhibitor-treated TH9 cells. Furthermore, low-dose p38 inhibitor suppresses tumor growth without inducing systemic adverse effects. In patients with tumor, rel…

Mice Inbred BALB CNFATC Transcription FactorsNF-kappa BMice NudeCell DifferentiationCancer immunotherapySignal transductionInflammatory Bowel DiseasesT-Lymphocytes Regulatoryp38 Mitogen-Activated Protein KinasesArticleMice Inbred C57BLMiceLymphocyte differentiationNeoplasmsProtein Kinase C betaAnimalsCytokinesHumansFemalefas ReceptorCD4-positive T cellsNature communications
researchProduct

The thiol switch C684 in Mitofusin-2 mediates redox-induced alterations of mitochondrial shape and respiration

2017

Mitofusin-2 (MFN2) is a GTPase in the outer mitochondrial membrane involved in the regulation of mitochondrial fusion and bioenergetics. MFN2 also plays a role in mitochondrial fusion induced by changes in the intracellular redox state. Adding oxidized glutathione (GSSG), the core cellular stress indicator, to mitochondrial preparations stimulates mitochondrial fusion by inducing disulphide bond-mediated oligomer formation of MFN2 and its homolog MFN1 which involve cysteine 684 (C684) of MFN2. Mitochondrial hyperfusion represents an adaptive stress response that confers transient protection by increasing mitochondrial ATP production but how this depends on the thiol switch C684 in MFN2 has …

Mice Knockout0301 basic medicineCell RespirationMFN2Cell BiologyOxidative phosphorylationMitochondrionBiologyMitochondrial apoptosis-induced channelGTP PhosphohydrolasesMitochondriaCell biologyMice03 medical and health sciencesCellular and Molecular NeuroscienceMitofusin-2030104 developmental biologymitochondrial fusionAnimalsMFN1Sulfhydryl CompoundsATP–ADP translocaseCell ShapeOxidation-ReductionCells CulturedNeurochemistry International
researchProduct

Epidemiological cutoff values for fluconazole, itraconazole, posaconazole, and voriconazole for six Candida species as determined by the colorimetric…

2013

ABSTRACT In the absence of clinical breakpoints (CBP), epidemiological cutoff values (ECVs) are useful to separate wild-type (WT) isolates (without mechanisms of resistance) from non-WT isolates (those that can harbor some resistance mechanisms), which is the goal of susceptibility tests. Sensititre YeastOne (SYO) is a widely used method to determine susceptibility of Candida spp. to antifungal agents. The CLSI CBP have been established, but not for the SYO method. The ECVs for four azoles, obtained using MIC distributions determined by the SYO method, were calculated via five methods (three statistical methods and based on the MIC 50 and modal MIC). Respectively, the median ECVs (in mg/lit…

Microbiology (medical)AzolesPosaconazolemedicine.medical_specialtyAntifungal AgentsItraconazoleMycologyMicrobial Sensitivity TestsBiologyMicrobiology:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]EpidemiologymedicineHumansCutoffCandida albicansPruebas de sensibilidad microbianaCandidaVoriconazoleCandidiasisLiter:Chemicals and Drugs::Heterocyclic Compounds::Heterocyclic Compounds 1-Ring::Azoles [Medical Subject Headings]:Diseases::Bacterial Infections and Mycoses::Mycoses::Candidiasis [Medical Subject Headings]biology.organism_classification:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Therapeutic Uses::Anti-Infective Agents::Antifungal Agents [Medical Subject Headings]:Analytical Diagnostic and Therapeutic Techniques and Equipment::Diagnosis::Diagnostic Techniques and Procedures::Clinical Laboratory Techniques::Microbiological Techniques::Microbial Sensitivity Tests [Medical Subject Headings]:Organisms::Eukaryota::Fungi::Mitosporic Fungi::Candida [Medical Subject Headings]AntifúngicosFluconazolemedicine.drug
researchProduct

Efavirenz alters mitochondrial respiratory function in cultured neuron and glial cell lines.

2015

Abstract Background The NNRTI efavirenz is among the most widely employed antiretroviral drugs. Although it is considered safe, efavirenz has been linked with several adverse effects including neurological manifestations, which appear in the majority of the patients on efavirenz-containing regimens. The molecular mechanisms responsible for these manifestations are not understood, but mounting evidence points to altered brain bioenergetics. Methods We evaluated the effect of short-term efavirenz treatment on the mitochondrial respiratory function of cultured glioblastoma and differentiated neuroblastoma cell lines using a Seahorse Extracellular Flux Analyzer. Results Incubation with efaviren…

Microbiology (medical)CyclopropanesCell typeEfavirenzCell RespirationBiologyPharmacologyMitochondrionCell Linechemistry.chemical_compoundAdenosine TriphosphateRespirationExtracellularmedicineHumansPharmacology (medical)Respiratory functionPharmacologyNeuronsNeurotoxicityvirus diseasesmedicine.diseaseVirologyBenzoxazinesMitochondriaInfectious Diseasesmedicine.anatomical_structurechemistryAnti-Retroviral AgentsAlkynesNeurogliaEnergy MetabolismNeurogliaThe Journal of antimicrobial chemotherapy
researchProduct

Lack of mitochondrial toxicity of darunavir, raltegravir and rilpivirine in neurons and hepatocytes: a comparison with efavirenz.

2014

Objectives Growing evidence associates the non-nucleoside reverse transcriptase inhibitor efavirenz with several adverse events. Newer antiretrovirals, such as the integrase inhibitor raltegravir, the non-nucleoside reverse transcriptase inhibitor rilpivirine and the protease inhibitor darunavir, claim to have a better toxicological profile than efavirenz while producing similar levels of efficacy and virological suppression. The objective of this study was to determine the in vitro toxicological profile of these three new antiretrovirals by evaluating their effects on the mitochondrial and cellular parameters altered by efavirenz in hepatocytes and neurons. Methods Hep3B cells and primary …

Microbiology (medical)CyclopropanesEfavirenzAnti-HIV AgentsIntegrase inhibitorBiologyMitochondrionPharmacologychemistry.chemical_compoundCell Line TumorRaltegravir PotassiumDrug Resistance ViralNitrilesmedicineAnimalsHumansPharmacology (medical)DarunavirCells CulturedDarunavirPharmacologyNeuronsSulfonamidesReverse-transcriptase inhibitorRilpivirinemedicine.diseaseRaltegravirPyrrolidinonesBenzoxazinesMitochondriaRatsMitochondrial toxicityInfectious DiseasesPyrimidineschemistryRilpivirineAlkynesHepatocytesReverse Transcriptase Inhibitorsmedicine.drugThe Journal of antimicrobial chemotherapy
researchProduct

T lymphocyte-stimulating microbial toxins as ?superantigens?

1991

Infectious pathogens generally have to cope with the host's adaptive immune system, i.e., T and B lymphocytes. Common evasion mechanisms in this complex interaction are antigenic variations, the escape to immunologically priviledged sites or the use of immunosuppressive mechanisms. Many bacteria and other microorganisms eleborate soluble factors or toxins that act suppressively on cells of the immune system, such as pore-forming molecules or proteins that interfere with the function of G proteins. Gram-positive cocci and a mycoplasma have developed an extremely potent mechanism of T cell stimulation by closely mimicking recognition of specific antigen. From the functional similarity to anti…

Microbiology (medical)G proteinT-LymphocytesT cellBacterial ToxinsImmunologyBiologyLymphocyte Activationmedicine.disease_causeMicrobiologyMiceMycoplasmaImmune systemAntigenmedicineSuperantigenAnimalsImmunology and AllergyAntigens BacterialGeneral MedicineMycoplasmaT lymphocyteAcquired immune systemmedicine.anatomical_structureImmunologyMitogensMedical Microbiology and Immunology
researchProduct

Lack of Functional Trehalase Activity in Candida parapsilosis Increases Susceptibility to Itraconazole

2022

Central metabolic pathways may play a major role in the virulence of pathogenic fungi. Here, we have investigated the susceptibility of a Candida parapsilosis mutant deficient in trehalase activity (atc1Δ/ntc1Δ strain) to the azolic compounds Fluconazole and Itraconazole. A time-course exposure to Itraconazole but not Fluconazole induced a significant degree of cell-killing in mutant cells compared to the parental strain. Flow cytometry determinations indicated that Itraconazole was able to induce a marked production of endogenous ROS together with a simultaneous increase in membrane potential, these effects being irrelevant after Fluconazole addition. Furthermore, only …

Microbiology (medical)Medicaments antifúngicsbiology_otherPlant ScienceFongs patògensfluconazole; itraconazole; ROS; mitochondrial activity; trehalase; trehalose; <i>Candida parapsilosis</i>Ecology Evolution Behavior and Systematics
researchProduct

Systematics of Mepraia (Hemiptera-Reduviidae): cytogenetic and molecular variation.

2009

The haematophagous insects of the subfamily Triatominae (Hemiptera-Reduviidae) have great epidemiological importance as vectors of Trypanosoma cruzi, the causative agent of Chagas disease. Mepraia was originally described as a monotypic genus comprised of Mepraia spinolai, distributed along coastal areas of northern Chile (from Region I to the Metropolitan Region). Recently, some M. spinolai populations have been ranked as a new species named Mepraia gajardoi. Several populations along the distribution range of the genus were sampled, and genetic differentiation was studied based upon the analysis of three molecular markers: cytogenetics (karyotype and chromosome behaviour during meiosis us…

Microbiology (medical)SystematicsMaleMitochondrial DNAMolecular Sequence DataMicrobiologyPolymerase Chain ReactionMepraiaCytogeneticsIntergenic regionGeneticsAnimalsChagas DiseaseChileGonadsMolecular BiologyRibosomal DNAEcology Evolution Behavior and SystematicsPhylogenyGeneticsPolymorphism GeneticbiologyMolecular epidemiologyBase SequenceGeographyChromosomebiology.organism_classificationInsect VectorsInfectious DiseasesReduviidaeEvolutionary biologyDNA IntergenicFemaleTriatominaeSequence AlignmentInfection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases
researchProduct

Hybridization of mouse lemurs: different patterns under different ecological conditions

2011

Abstract Background Several mechanistic models aim to explain the diversification of the multitude of endemic species on Madagascar. The island's biogeographic history probably offered numerous opportunities for secondary contact and subsequent hybridization. Existing diversification models do not consider a possible role of these processes. One key question for a better understanding of their potential importance is how they are influenced by different environmental settings. Here, we characterized a contact zone between two species of mouse lemurs, Microcebus griseorufus and M. murinus, in dry spiny bush and mesic gallery forest that border each other sharply without intermediate habitats…

Microcebus murinusEvolutionMolecular Sequence DataPopulationIntrogressionLemurCheirogaleidaeDNA MitochondrialLinkage DisequilibriumHybrid zonebiology.animalMadagascarQH359-425AnimalseducationEcosystemPhylogenyEcology Evolution Behavior and SystematicsDNA Primerseducation.field_of_studyBase SequenceModels GeneticbiologyEcologyBayes TheoremSequence Analysis DNAbiology.organism_classificationGenetics PopulationHaplotypesHabitatEvolutionary biologyHybridization GeneticCheirogaleidaeMicrocebus griseorufusMicrosatellite RepeatsResearch ArticleBMC Evolutionary Biology
researchProduct

Mutations in KATNB1 Cause Complex Cerebral Malformations by Disrupting Asymmetrically Dividing Neural Progenitors

2014

SummaryExome sequencing analysis of over 2,000 children with complex malformations of cortical development identified five independent (four homozygous and one compound heterozygous) deleterious mutations in KATNB1, encoding the regulatory subunit of the microtubule-severing enzyme Katanin. Mitotic spindle formation is defective in patient-derived fibroblasts, a consequence of disrupted interactions of mutant KATNB1 with KATNA1, the catalytic subunit of Katanin, and other microtubule-associated proteins. Loss of KATNB1 orthologs in zebrafish (katnb1) and flies (kat80) results in microcephaly, recapitulating the human phenotype. In the developing Drosophila optic lobe, kat80 loss specificall…

Microtubule-associated proteinNeurogenesisNeuroscience(all)Cell CountKataninSpindle ApparatusBiologymedicine.disease_causeArticleMice03 medical and health sciences0302 clinical medicineNeural Stem CellsNeuroblastmedicineAnimalsDrosophila ProteinsHumansProgenitor cellZebrafishMitosisZebrafishAdenosine TriphosphatasesMutationGeneral NeuroscienceOptic Lobe NonmammalianBrainDendritesbiology.organism_classificationSpindle apparatusmedicine.anatomical_structureCentrosome030220 oncology & carcinogenesisCerebral malformationsMutationMicrocephalybiology.proteinDrosophilaNeuronKataninMicrotubule-Associated ProteinsNeuroscienceCell Division030217 neurology & neurosurgery
researchProduct