Search results for "MT2"

showing 8 items of 18 documents

Synergistic targeting of FLT3 mutations in AML via combined menin-MLL and FLT3 inhibition

2020

Abstract The interaction of menin (MEN1) and MLL (MLL1, KMT2A) is a dependency and provides a potential opportunity for treatment of NPM1-mutant (NPM1mut) and MLL-rearranged (MLL-r) leukemias. Concomitant activating driver mutations in the gene encoding the tyrosine kinase FLT3 occur in both leukemias and are particularly common in the NPM1mut subtype. In this study, transcriptional profiling after pharmacological inhibition of the menin-MLL complex revealed specific changes in gene expression, with downregulation of the MEIS1 transcription factor and its transcriptional target gene FLT3 being the most pronounced. Combining menin-MLL inhibition with specific small-molecule kinase inhibitors…

NPM1Transcription GeneticImmunologyApoptosisBiochemistryMiceRandom AllocationMice Inbred NODCell Line TumorProto-Oncogene Proteinshemic and lymphatic diseasesAntineoplastic Combined Chemotherapy ProtocolsGene expressionmedicineAnimalsHumansMEN1PhosphorylationMyeloid Ecotropic Viral Integration Site 1 ProteinProtein Kinase InhibitorsneoplasmsbiologyGene Expression Regulation LeukemicKinaseNuclear ProteinsMyeloid leukemiaDrug SynergismHistone-Lysine N-MethyltransferaseCell BiologyHematologymedicine.diseaseCoculture TechniquesNeoplasm ProteinsLeukemia Myeloid AcuteLeukemiaKMT2Afms-Like Tyrosine Kinase 3biology.proteinCancer researchNucleophosminProtein Processing Post-TranslationalTyrosine kinaseMyeloid-Lymphoid Leukemia ProteinBlood
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Synthesis of new melatoninergic hexahydroindenopyridines

2014

Hexahydroindenopyridine (HHIP) is an interesting heterocyclic framework that contains an indene core similar to ramelteon. This type of tricyclic piperidines aroused our interest as potential melatoninergic ligands. Melatonin receptor ligands have applications in insomnia and depression. We report herein an efficient two-step method to prepare new HHIP by the reaction of an enamine with 3-bromopropylamine hydrobromide. Some synthesized compounds showed moderate affinity for melatonin receptors in the nanomolar or low micromolar range. Furthermore, the methylenedioxy HHIPs 2d (N-phenylacetamide) and 2f (N,N-diethylacetamide), exhibited high selectivity at MT1 or MT2 receptors, respectively, …

StereochemistryClinical BiochemistryRamelteonPharmaceutical ScienceLigandsHeterocyclic Compounds 4 or More RingsBiochemistryMelatonin receptorMethylenedioxyEnamineMelatoninStructure-Activity Relationshipchemistry.chemical_compoundDrug DiscoverymedicineHumansIndeneMolecular Biologychemistry.chemical_classificationDose-Response Relationship DrugMolecular StructureReceptor Melatonin MT2HydrobromideReceptor Melatonin MT1Organic ChemistryHEK293 CellschemistryMolecular MedicineTricyclicmedicine.drugBioorganic & Medicinal Chemistry Letters
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Synthesis and Characterization of New Bivalent Agents as Melatonin- and Histamine H3-Ligands

2014

Melatonin is an endogenous molecule involved in many pathophysiological processes. In addition to the control of circadian rhythms, its antioxidant and neuroprotective properties have been widely described. Thus far, different bivalent compounds composed by a melatonin molecule linked to another neuroprotective agent were synthesized and tested for their ability to block neurodegenerative processes in vitro and in vivo. To identify a novel class of potential neuroprotective compounds, we prepared a series of bivalent ligands, in which a prototypic melatonergic ligand is connected to an imidazole-based H3 receptor antagonist through a flexible linker. Four imidazolyl-alkyloxy-anilinoethylami…

StereochemistryHistamine AntagonistsLigandsMelatonin receptorMT<sub>2</sub>ArticleCatalysisInorganic Chemistrylcsh:ChemistryHistamine receptorPiperidinesH<sub>3</sub> antagonistsHumansReceptors Histamine H3Physical and Theoretical ChemistryBinding siteReceptormelatonin receptorMolecular Biologylcsh:QH301-705.5SpectroscopyBinding SitesReceptor Melatonin MT2ChemistryReceptor Melatonin MT1MT1Organic ChemistryMT2ImidazolesHistaminergicMT<sub>1</sub>General Medicinemelatonin receptor; MT1; MT2; H3 antagonists; bivalent ligandsLigand (biochemistry)Protein Structure TertiaryComputer Science ApplicationsMelatonergicMolecular Docking SimulationBiochemistrylcsh:Biology (General)lcsh:QD1-999bivalent ligandsHistamine H3 receptorH3 antagonistsProtein BindingInternational Journal of Molecular Sciences
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Efficient synthesis of hexahydroindenopyridines and their potential as melatoninergic ligands.

2014

Hexahydroindenopyridine (HHIP) is an interesting tricyclic piperidine nucleus that is structurally related to melatonin, a serotonin-derived neurohormone. Melatonin receptor ligands have applications in several cellular, neuroendocrine and neurophysiological disorders, including depression and/or insomnia. We report herein an efficient two-step method to prepare new HHIP via enamine C-alkylation-cyclization. The influence of substituents on the benzene ring and the nitrogen atom on melatoninergic receptors has been studied. Among the 25 synthesized HHIPs, some of them containing methylenedioxy (series 2) and 8-chloro-7-methoxy substituents (series 4) on the benzene ring revealed affinity fo…

StereochemistryPyridinesRing (chemistry)LigandsMelatonin receptorMethylenedioxyEnaminechemistry.chemical_compoundStructure-Activity RelationshipDrug DiscoveryHumansReceptorCells CulturedPharmacologychemistry.chemical_classificationBinding SitesDose-Response Relationship DrugMolecular StructureReceptor Melatonin MT2Receptor Melatonin MT1Organic ChemistryGeneral MedicineHEK293 CellschemistryPiperidineAcetamideTricyclicEuropean journal of medicinal chemistry
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Bases genéticas y celulares de neuropatías periféricas hereditarias

2015

Tesis doctoral; 208 págs.

UNESCO::CIENCIAS DE LA VIDA::GenéticaModificadores genéticosSMYD4:CIENCIAS DE LA VIDA::Genética [UNESCO]Enfermedades rarasCharcot Marie ToothCMT2KJunctophilinGDAP1Genética humanaNeuropatías periféricas hereditariasJPH1
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Clinical Utility of a Unique Genome-Wide DNA Methylation Signature for KMT2A-Related Syndrome

2022

Wiedemann–Steiner syndrome (WDSTS) is a Mendelian syndromic intellectual disability (ID) condition associated with hypertrichosis cubiti, short stature, and characteristic facies caused by pathogenic variants in the KMT2A gene. Clinical features can be inconclusive in mild and unusual WDSTS presentations with variable ID (mild to severe), facies (typical or not) and other associated malformations (bone, cerebral, renal, cardiac and ophthalmological anomalies). Interpretation and classification of rare KMT2A variants can be challenging. A genome-wide DNA methylation episignature for KMT2A-related syndrome could allow functional classification of variants and provide insights into the pathoph…

Wiedemann–Steiner syndromeQH301-705.5Intellectual disability[SDV.BC]Life Sciences [q-bio]/Cellular BiologyCatalysisInorganic ChemistryKMT2A geneNeurodevelopmental disorderGrowth DisorderAbnormalities Multiple[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Biology (General)Physical and Theoretical ChemistryEpisignatureQD1-999[SDV.BC] Life Sciences [q-bio]/Cellular BiologyMolecular BiologySpectroscopyDNA methylationOrganic ChemistryNeurodevelopmental disordersCraniofacial AbnormalitieEpigeneticHypertrichosiGeneral MedicineFacieComputer Science Applications<i>KMT2A</i> geneChemistryepigenetics; DNA methylation; episignature; Wiedemann–Steiner syndrome; <i>KMT2A</i> gene; intellectual disability; neurodevelopmental disordersPhenotype[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]EpigeneticsHuman
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Cytosine methylation of tRNA-Asp by DNMT2 has a role in translation of proteins containing poly-Asp sequences

2015

AbstractThe Dnmt2 RNA methyltransferase catalyses the methylation of C38 in the anticodon loop of tRNA-Asp, but the molecular role of this methylation is unknown. Here, we report that mouse aspartyl-tRNA synthetase shows a four to fivefold preference for C38-methylated tRNA-Asp. Consistently, a 30% reduced charging level of tRNA-Asp was observed in Dnmt2 knockout (KO) murine embryonic fibroblast cells. Gene expression analysis with fluorescent reporter proteins fused to an N-terminal poly-Asp sequence showed that protein synthesis of poly-Asp-tagged reporter proteins was reduced in Dnmt2 KO cells as well. The same effect was observed with endogenous proteins containing poly-Asp sequences, i…

aminoacylationTRNA methylationRNATranslation (biology)Cell BiologyMethylationBiologyBiochemistryMolecular biologyregulation of translationArticleBiochemistrytRNA methylationTransfer RNADNA methylationGene expressionGeneticsProtein biosynthesisDnmt2Molecular BiologyAsp-rich proteinsCell Discovery
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Effects of 5-Azacitidine on Dnmt2/Trdmt1 expression levels and endoplasmic reticulum stress in cellular models of insulinoma

2021

Diabetes mellitus affects people all over the world of all ages or social groups making it a worldwide healthcare challenge. Moreover, untreated or badly treated diabetes carries a risk of serious complications including premature death (IDF Diabetes Atlas 9th edition, 2019). The main symptom of diabetes is insulin secretion and/or action disorder leading to hyperglycemia what results in impaired carbohydrate, fat, and protein metabolism (“Diagnosis and Classification of Diabetes Mellitus,” 2013). Dnmt2/ Trdmt1 in its structure and sequence is similar to DNA methyltransferases, however, it has been shown that mainly methylates aspartic acid transfer RNA, specifically at the cytosine-38 resi…

insulinoma diabetes mellitus senolytic pancreas senescence ER stress Trdmt1/Dnmt2 tRNA methylation 5-Azacitidine
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