Search results for "MUTATION"

showing 10 items of 2830 documents

Dynamics of gene regulatory networks and their dependence on network topology and quantitative parameters – the case of phage λ

2019

Background Gene regulatory networks can be modelled in various ways depending on the level of detail required and biological questions addressed. One of the earliest formalisms used for modeling is a Boolean network, although these models cannot describe most temporal aspects of a biological system. Differential equation models have also been used to model gene regulatory networks, but these frameworks tend to be too detailed for large models and many quantitative parameters might not be deducible in practice. Hybrid models bridge the gap between these two model classes – these are useful when concentration changes are important while the information about precise concentrations and binding…

Gene Expression Regulation ViralHybrid systemsComputer scienceGene regulatory networklcsh:Computer applications to medicine. Medical informaticsNetwork topologyModels BiologicalBiochemistryGene regulatory networks03 medical and health sciences0302 clinical medicineStructural BiologyLysogenic cycleStable behavioursOperonPhage λlcsh:QH301-705.5LysogenyMolecular BiologyTopology (chemistry)030304 developmental biology0303 health sciencesModel validationApplied MathematicsBacteriophage lambdaComputer Science ApplicationsBoolean networkOrder (biology)lcsh:Biology (General)030220 oncology & carcinogenesisHybrid systemMutationlcsh:R858-859.7Biological systemSoftwareResearch ArticleBMC Bioinformatics
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Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice

2002

The serine-threonine kinase Akt seems to be central in mediating stimuli from different classes of receptors. In fact, both IGF-1 and IL6-like cytokines induce hypertrophic and antiapoptotic signals in cardiomyocytes through PI3K-dependent Akt activation. More recently, it was shown that Akt is involved also in the hypertrophic and antiapoptotic effects of β-adrenergic stimulation. Thus, to determine the effects of Akt on cardiac function in vivo, we generated a model of cardiac-specific Akt overexpression in mice. Transgenic mice were generated by using the E40K, constitutively active mutant of Akt linked to the rat α-myosin heavy chain promoter. The effects of cardiac-selective Akt overex…

Gene ExpressionTransgenicGlycogen Synthase Kinase 3MiceGSK-3Receptorsgenetics/physiologycytology/metabolismMultidisciplinaryBiological SciencesProtein-Serine-Threonine KinasesDNA-Binding Proteinsenzymology/genetics/pathologyAdrenergicPhosphorylationSignal transductionMitogen-Activated Protein KinasesSignal Transductionmedicine.medical_specialtyCardiomyopathyAnimals; Calcium-Calmodulin-Dependent Protein Kinases; metabolism; Cardiomyopathy; Hypertrophic; enzymology/genetics/pathology; Cell Size; physiology; DNA-Binding Proteins; GATA4 Transcription Factor; Gene Expression; Glycogen Synthase Kinase 3; Mice; Transgenic; Mitogen-Activated Protein Kinases; Myocardial Contraction; Myocardium; cytology/metabolism; Point Mutation; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt; Proto-Oncogene Proteins; genetics/physiology; Rats; Receptors; Adrenergic; beta; Signal Transduction; Transcription FactorsMice TransgenicBiologyProtein Serine-Threonine KinasesContractilityIn vivoInternal medicineProto-Oncogene ProteinsReceptors Adrenergic betamedicineAnimalsPoint MutationGlycogen synthaseProtein kinase BPI3K/AKT/mTOR pathwayCell SizeMyocardiumCardiomyopathy HypertrophicMyocardial ContractionGATA4 Transcription FactorRatsEndocrinologyHypertrophicphysiologyCalcium-Calmodulin-Dependent Protein Kinasesbiology.proteinbetametabolismProto-Oncogene Proteins c-aktTranscription Factors
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Muscleblind isoforms are functionally distinct and regulate α-actinin splicing

2007

Drosophila Muscleblind (Mbl) proteins control terminal muscle and neural differentiation, but their molecular function has not been experimentally addressed. Such an analysis is relevant as the human Muscleblind-like homologs (MBNL1-3) are implicated in the pathogenesis of the inherited muscular developmental and degenerative disease myotonic dystrophy. The Drosophila muscleblind gene expresses four protein coding splice forms (mblA to mblD) that are differentially expressed during the Drosophila life cycle, and which vary markedly in their ability to rescue the embryonic lethal phenotype of muscleblind mutant flies. Analysis of muscleblind mutant embryos reveals misregulated alternative sp…

Gene isoformCancer ResearchMolecular Sequence DataBiologyKidneyChlorocebus aethiopsAnimalsDrosophila ProteinsHumansProtein IsoformsActininMuscle Skeletal3' Untranslated RegionsMolecular BiologyGeneCells CulturedCell NucleusGeneticsBase SequenceAlternative splicingGene Expression Regulation DevelopmentalNuclear ProteinsRNA-Binding ProteinsRNAKidney metabolismCell BiologyAlternative SplicingDrosophila melanogasterCOS CellsMutationRNA splicingTrinucleotide Repeat ExpansionTrinucleotide repeat expansionDevelopmental BiologyMinigeneDifferentiation
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Different La/SS-B mRNA isoforms are expressed in salivary gland tissue of patients with primary Sjogren's syndrome

1996

Recently we isolated a La/SS-B mRNA isoform from a cDNA library made from peripheral blood lymphocytes of a patient with primary Sjögren's Syndrome. In the La/SS-B mRNA isoform the exon 1 was replaced. The alternative exon was termed exon 1'. Genomic analysis showed that the exon 1' La mRNA was the result of a promoter-switch in combination with alternative splicing. Due to the unusual structure of the exon 1' La/SS-B mRNA, the function and the behaviour under physiological and pathophysiological conditions in tissue of patients with primary Sjögren's syndrome or Systemic Lupus Erythematosus remained obscure. Therefore assays were established allowing a qualitative and quantitative estimati…

Gene isoformCytoplasmDNA ComplementaryMolecular Sequence DataImmunologyGene ExpressionIn situ hybridizationBiologyAutoantigensPolymerase Chain ReactionEpitheliumSalivary GlandsExonGene expressionmedicineHumansImmunology and AllergyRNA MessengerRNA Processing Post-TranscriptionalIn Situ HybridizationDNA PrimersMessenger RNABase SequencecDNA libraryAlternative splicingExonsMolecular biologyEpitheliumSjogren's Syndromemedicine.anatomical_structureRibonucleoproteinsMutationEndothelium Vascular
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Identification of Cysteine Residues in Human Cationic Amino Acid Transporter hCAT-2A That Are Targets for Inhibition by N-Ethylmaleimide

2013

In most cells, cationic amino acids such as l-arginine, l-lysine, and l-ornithine are transported by cationic (CAT) and y(+)L (y(+)LAT) amino acid transporters. In human erythrocytes, the cysteine-modifying agent N-ethylmaleimide (NEM) has been shown to inhibit system y(+) (most likely CAT-1), but not system y(+)L (Devés, R., Angelo, S., and Chávez, P. (1993) J. Physiol. 468, 753-766). We thus wondered if sensitivity to NEM distinguishes generally all CAT and y(+)LAT isoforms. Transport assays in Xenopus laevis oocytes established that indeed all human CATs (including the low affinity hCAT-2A), but neither y(+)LAT isoform, are inhibited by NEM. hCAT-2A inhibition was not due to reduced tran…

Gene isoformMutantMutation MissenseXenopusBiologyBiochemistryXenopus laevischemistry.chemical_compoundMembrane BiologyAnimalsHumansheterocyclic compoundsCysteineAmino acid transporterEnzyme InhibitorsMolecular Biologychemistry.chemical_classificationN-EthylmaleimideTransporterCell Biologybiology.organism_classificationMolecular biologyProtein Structure TertiaryAmino acidAmino Acid SubstitutionchemistryBiochemistryEthylmaleimideOocytesAmino Acid Transport Systems BasicCysteineJournal of Biological Chemistry
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Gene Repair of an Usher Syndrome Causing Mutation by Zinc-Finger Nuclease Mediated Homologous Recombination

2012

PURPOSE. Human Usher syndrome (USH) is the most frequent cause of inherited deaf-blindness. It is clinically and genetically heterogeneous, assigned to three clinical types of which the most severe type is USH1. No effective treatment for the ophthalmic component of USH exists. Gene augmentation is an attractive strategy for hereditary retinal diseases. However, several USH genes, like USH1C, are expressed in various isoforms, hampering gene augmentation. As an alternative treatment strategy, we applied the zinc-finger nuclease (ZFN) technology for targeted gene repair of an USH1C, causing mutation by homologous recombination. METHODS. We designed ZFNs customized for the p.R31X nonsense mut…

Gene isoformNonsense mutationCell Cycle ProteinsBiologyRetinaCell Linechemistry.chemical_compoundHumansDNA Breaks Double-StrandedDNA CleavageHomologous RecombinationGeneAdaptor Proteins Signal TransducingZinc fingerGeneticsTargeted Gene RepairfungiZinc FingersDNAEndonucleasesZinc finger nucleaseCytoskeletal ProteinschemistryCodon NonsenseHomologous recombinationUsher SyndromesDNATargeted Gene RepairInvestigative Opthalmology & Visual Science
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β1-Integrin Cytoplasmic Subdomains Involved in Dominant Negative Function

1998

The beta1-integrin cytoplasmic domain consists of a membrane proximal subdomain common to the four known isoforms ("common" region) and a distal subdomain specific for each isoform ("variable" region). To investigate in detail the role of these subdomains in integrin-dependent cellular functions, we used beta1A and beta1B isoforms as well as four mutants lacking the entire cytoplasmic domain (beta1TR), the variable region (beta1COM), or the common region (beta1 deltaCOM-B and beta1 deltaCOM-A). By expressing these constructs in Chinese hamster ovary and beta1 integrin-deficient GD25 cells (Wennerberg et al., J Cell Biol 132, 227-238, 1996), we show that beta1B, beta1COM, beta1 deltaCOM-B, a…

Gene isoformTalinCytoplasmProtein ConformationIntegrinMolecular Sequence DataCHO CellsIntegrin alpha5Platelet Membrane GlycoproteinsArticleFocal adhesionchemistry.chemical_compoundMiceAntigens CDCricetinaeCell AdhesionAnimalsActininAmino Acid SequencePhosphorylationCell adhesionMolecular BiologyBinding SitesbiologyCell adhesion moleculeChinese hamster ovary cellIntegrin beta1Integrin beta3Tyrosine phosphorylationCell BiologyIntegrin alphaVProtein-Tyrosine KinasesRecombinant ProteinsCell biologyFibronectinsFibronectinchemistryFocal Adhesion Kinase 1Focal Adhesion Protein-Tyrosine KinasesMutationbiology.proteinCell Adhesion MoleculesSignal Transduction
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Allelic age of the USH2A c.2299delG mutation

2010

24 p., figuras y bibliografía

Gene isoformUsher syndromePopulationc.2299delGSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideArticleLinkage DisequilibriumWhite PeopleExonUSH2Aotorhinolaryngologic diseasesGeneticsmedicineHaplotypeHumansAlleleeducationGeneAllelesPhylogenyGenetics (clinical)GeneticsExtracellular Matrix Proteinseducation.field_of_studyHaplotypemedicine.diseaseHaplotypesMutationDatingUsher Syndromes
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BRAF mutation influences hypoxia-inducible factor-1α expression levels in papillary thyroid cancer

2010

Hypoxia-inducible factor-1α is found frequently overexpressed in solid tumors cells, exerting an important role in angiogenesis, glucose metabolism, cell proliferation, survival and invasion. In thyroid carcinomas, hypoxia-inducible factor-1α expression was found increased in differentiated, poorly differentiated, medullary and anaplastic variants. Hypoxia represents the principal stimulus responsible for hypoxia-inducible factor-1α induction. Other nonhypoxic stimuli increase hypoxia-inducible factor-1α synthesis through the activation of phosphatidylinositol 3-kinase and mitogen-activated protein kinase pathways in a cell-type-specific manner. We have previously shown the role of BRAFV600…

Gene knockdownPathologymedicine.medical_specialtyMutationCell growthAngiogenesisBiologymedicine.diseasemedicine.disease_causePathology and Forensic MedicinePapillary thyroid cancerThyroid carcinomaCancer researchmedicineSignal transductionPAX8Modern Pathology
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Disparity between Inter-Patient Molecular Heterogeneity and Repertoires of Target Drugs Used for Different Types of Cancer in Clinical Oncology

2020

Inter-patient molecular heterogeneity is the major declared driver of an expanding variety of anticancer drugs and personalizing their prescriptions. Here, we compared interpatient molecular heterogeneities of tumors and repertoires of drugs or their molecular targets currently in use in clinical oncology. We estimated molecular heterogeneity using genomic (whole exome sequencing) and transcriptomic (RNA sequencing) data for 4890 tumors taken from The Cancer Genome Atlas database. For thirteen major cancer types, we compared heterogeneities at the levels of mutations and gene expression with the repertoires of targeted therapeutics and their molecular targets accepted by the current guideli…

Gene mutationMedical OncologychemotherapyGenomeTranscriptomelcsh:ChemistryDrug Delivery SystemsProstateNeoplasmstumor heterogeneityMedicineCluster AnalysisMolecular Targeted TherapyPathology MolecularPrecision Medicinelcsh:QH301-705.5targeted therapeuticscancer drugsSpectroscopyExome sequencingGeneral MedicineGenomicspersonalized medicineComputer Science ApplicationsDrug repositioningmedicine.anatomical_structureAntineoplastic AgentsComputational biologyCatalysisArticleInorganic Chemistrymolecular diagnosticsGenetic HeterogeneityDrug TherapyExome SequencingHumansPhysical and Theoretical ChemistryMolecular Biologygenomeclinical oncologybusiness.industryOrganic ChemistryMolecular diagnosticsmutationslcsh:Biology (General)lcsh:QD1-999MutationPersonalized medicinebusinesstranscriptomeInternational Journal of Molecular Sciences
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