Search results for "MUTATION"

showing 10 items of 2830 documents

Clinical and Genetic Aspects of Juvenile Onset Pompe Disease

2021

AbstractLittle is known about clinical symptomatology and genetics of juvenile onset Pompe disease (JOPD). The aims of this study were to analyze how these children are diagnosed, what clinical problems they have, and how phenotype is related to genotype. To accomplish this, we analyzed retrospectively data of 34 patients diagnosed after their first and before completion of their 18th birthday. Median age at diagnosis was 3.9 (range 1.1–17) years. Eight patients (23.5%) developed initial symptoms in the first year, 12 (35%) between 1 and 7 years, and 6 (18%) thereafter. Eight (23.5%) had no clinical symptoms at the time of diagnosis. Indications for diagnostics were a positive family histor…

Pediatricsmedicine.medical_specialtyGeneralized muscle weaknessDisease03 medical and health sciences0302 clinical medicineGenotypeHumansMedicineFamily historyRetrospective Studies030304 developmental biology0303 health sciencesGlycogen Storage Disease Type IIbusiness.industryHypertrophic cardiomyopathyMuscle weaknessalpha-GlucosidasesGeneral Medicinemedicine.disease3. Good healthPhenotypeJuvenile onsetMutationPediatrics Perinatology and Child HealthFailure to thriveNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryNeuropediatrics
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Long-term follow-up and novel genotype-phenotype analysis of monozygotic twins with ATP1A3 mutation in Alternating Hemiplegia of Childhood-2

2020

Alternating Hemiplegia of Childhood (AHC) is a rare disorder characterized by frequent, transient attacks of hemiplegia involving either side of the body or both in association to several other disturbances including dystonic spells, abnormal ocular movements, autonomic manifestations, epileptic seizures and cognitive impairment. The clinical manifestations usually start before the age of 18 months. Two forms of the disorder known as AHC-1 (MIM#104290) and AHC-2 (MIM#614820) depends on mutations in ATP1A2 and ATP1A3 genes respectively, with over 75% of AHC caused by a mutation in the ATP1A3 gene. Herewith, we report serial clinical follow-up data of monozygotic (MZ) twin sisters, who presen…

Pediatricsmedicine.medical_specialtyGenotype-phenotype correlationGenotypeTwinsHemiplegiaMonozygoticEpilepsyYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticaATP1A2Alternating Hemiplegia of Childhood (AHC)ATP1A3GenotypeGeneticsmedicineHumansYoung adultATPase Na+/K+ transporting subunit alpha 2 (ATP1A2)Genetics (clinical)DystoniaATPase Na+/K+ transporting subunit alpha 3 (ATP1A3)business.industryAlternating hemiplegia of childhoodp.Asn773SerGeneral MedicineTwins Monozygoticmedicine.diseasePhenotypePhenotypeMutationFemaleSodium-Potassium-Exchanging ATPasebusiness
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Autosomal recessive polycystic kidney disease: case report of a newborn with rare PKHD1 mutation, rapid renal enlargement and early fatal outcome

2020

Abstract Introduction Autosomal recessive polycystic kidney disease (ARPKD; MIM#263200) is one of the most frequent pediatric renal cystic diseases, with an incidence of 1:20,000. It is caused by mutations of the PKHD1 gene, on chromosome 6p12. The clinical spectrum is highly variable, ranging from late-onset milder forms to severe perinatal manifestations. The management of newborns with severe pulmonary insufficiency is challenging, and causes of early death are sepsis or respiratory failure. In cases of massive renal enlargement, early bilateral nephrectomy and peritoneal dialysis may reduce infant mortality. However, there is no conclusive data on the role of surgery, and decision-makin…

Pediatricsmedicine.medical_specialtyGenotype-phenotype correlationGenotypemedicine.medical_treatmentARPKDPulmonary insufficiencyReceptors Cell SurfaceCase ReportPeritoneal dialysisSepsis03 medical and health sciencesLiver diseaseConsanguinity0302 clinical medicineFatal OutcomeNext generation sequencingmedicineHumansGenetic Predisposition to DiseaseEthicPotter sequencePolycystic Kidney Autosomal RecessiveEthicsbusiness.industrylcsh:RJ1-570Infant Newbornlcsh:Pediatricsmedicine.diseaseAutosomal Recessive Polycystic Kidney DiseaseRespiratory failure030220 oncology & carcinogenesisMutationFemalebusiness030217 neurology & neurosurgeryInfant PrematureBilateral NephrectomyPotter sequence
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Trouble always comes in threes: three mutations for three auto inflammatory genes in a child and in his father

2014

The coexistence of mutations in more than one gene, linked to Autoinflammatory Diesases, can confuse and make difficult the diagnosis and management of these patients, especially in childhood, when the clinical history is still brief.

Pediatricsmedicine.medical_specialtyauto inflammatory geneBioinformaticsSettore MED/38 - Pediatria Generale E SpecialisticaRheumatologyClinical historyInternal medicinemedicineImmunology and AllergyPediatrics Perinatology and Child HealthGeneInflammatory genesHeterozygous mutationgene mutationsbusiness.industryMultiple sclerosisfood and beveragesmedicine.diseaseRheumatologyPharyngitisCanakinumabPediatrics Perinatology and Child HealthPoster Presentationmedicine.symptombusinessmedicine.drug
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Nonsense-mediated decay mechanism is a possible modifying factor of clinical outcome in nonsense cd39 beta thalassemia genotype

2012

Nonsense-mediated mRNA decay (NMD) is a surveillance system to prevent the synthesis of non-functional proteins. In β-thalassemia, NMD may have a role in clinical outcome. An example of premature translation stop codons appearing for the first time is the β-globin cd39 mutation; when homozygous, this results in a severe phenotype. The aim of this study was to determine whether the homozygous nonsense cd39 may have a milder phenotype in comparison with IVS1,nt110/cd39 genotype. Genotypes have been identified from a cohort of 568 patients affected by β-thalassemia. These genotypes were compared with those found in 577 affected fetuses detected among 2292 prenatal diagnoses. The…

Pediatricsmedicine.medical_specialtymedia_common.quotation_subjectNonsense-mediated decayNonsenseBeta thalassemiaBiologynonsense-mediated mRNA decay; beta-thalassemia; clinical outcame; beta-globin gene mutationsmedicine.diseaseGastroenterologynonsense-mediated mRNA decay beta-thalassemia beta-globin gene mutationsnonsense-mediated mRNA decay beta-thalassemia clinical outcame beta-globin gene mutations.Internal medicineGenotypemedicineDiseases of the blood and blood-forming organsRC633-647.5media_commonThalassemia Reports
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Single Peptide Backbone Surrogate Mutations to Regulate Angiotensin GPCR Subtype Selectivity

2020

Mutating the side-chains of amino acids in a peptide ligand, with unnatural amino acids, aiming to mitigate its short half-life is an established approach. However, it is hypothesized that mutating specific backbone peptide bonds with bioisosters can be exploited not only to enhance the proteolytic stability of parent peptides, but also to tune its receptor subtype selectivity. Towards this end, four [Y]6-Angiotensin II analogues are synthesized where amide bonds have been replaced by 1,4-disubstituted 1,2,3-triazole isosteres in four different backbone locations. All the analogues possessed enhanced stability in human plasma in comparison with the parent peptide, whereas only two of them a…

PeptidomimeticStereochemistryChemistry Multidisciplinary[SDV]Life Sciences [q-bio]G-protein-coupled receptorsPeptide[CHIM.THER]Chemical Sciences/Medicinal ChemistryLigandsClick chemistry; Competition-binding experiments; G-protein-coupled receptors; Neurotrophic effects; Peptidomimetics010402 general chemistry01 natural sciencesCatalysisSubstrate Specificityneurotrophic effectscompetition-binding experimentsAnimalsHumansPeptide bondAmino AcidsComputingMilieux_MISCELLANEOUSG protein-coupled receptorchemistry.chemical_classificationReceptors AngiotensinScience & TechnologyAngiotensin II receptor type 1010405 organic chemistry[CHIM.ORGA]Chemical Sciences/Organic chemistryAngiotensin IIOrganic ChemistryGeneral ChemistryAngiotensin II0104 chemical sciencesAmino acidChemistryHEK293 CellschemistrypeptidomimeticsMutationPhysical Sciencesclick chemistryPeptides03 Chemical SciencesTwo-dimensional nuclear magnetic resonance spectroscopy
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Las matemáticas del cubo de Rubik

2013

[ES] En este artículo mostramos cómo podemos utilizar el cubo de Rubik para presentar algunos conceptos básicos de la teoría de grupos y cómo podemos usar esta para resolver el cubo de Rubik.

PermutationConjugationGrups Teoria deGrupoConjugaciónCubo de RubikPermutaciónOrderGroupRubik's cubeMATEMATICA APLICADAOrdenMatemàtica
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Two Simple Constructive algorithms for the Distributed Assembly Permutation Flowshop Scheduling Problem

2014

Nowadays, it is necessary to improve the management of complex supply chains which are often composed of multi-plant facilities. This paper proposes a Distributed Assembly Permutation Flowshop Scheduling Problem (DAPFSP). This problem is a generalization of the Distributed Permutation Flowshop Scheduling Problem (DPFSP) presented by Naderi and Ruiz (Comput Oper Res, 37(4):754–768, 2010). The first stage of the DAPFSP is composed of f identical production factories. Each center is a flowshop that produces jobs that have to be assembled into final products in a second assembly stage. The objective is to minimize the makespan. Two simple constructive algorithms are proposed to solve the proble…

PermutationMathematical optimizationJob shop schedulingSimple (abstract algebra)GeneralizationSupply chainConstructive algorithmsProduction (computer science)Mathematics
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Transmission of Genetic Properties in Permutation Problems: Study of Lehmer Code and Inversion Table Encoding

2021

Solution encoding describes the way decision variables are represented. In the case of permutation problems, the classical encoding should ensure that there are no duplicates. During crossover operations, repairs may be carried out to correct or avoid repetitions. The use of indirect encoding aims to define bijections between the classical permutation and a different representation of the decision variables. These encodings are not sensitive to duplicates. However, they lead to a loss of genetic properties during crossbreeding. This paper proposes a study of the impact of this loss both in the space of decision variables and in that of fitness values. We consider two indirect encoding: the …

PermutationTransmission (telecommunications)Computer scienceEncoding (memory)Lehmer codeGenetic algorithmCrossoverArithmeticRepresentation (mathematics)Bijection injection and surjection
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Scatter Search vs. Genetic Algorithms

2005

The purpose of this work is to compare the performance of a scatter search (SS) implementation and an implementation of a genetic algorithm (GA) in the context of searching for optimal solutions to permutation problems. Scatter search and genetic algorithms are members of the evolutionary computation family. That is, they are both based on maintaining a population of solutions for the purpose of generating new trial solutions. Our computational experiments with four well-known permutation problems reveal that in general a GA with local search outperforms one without it. Using the same problem instances, we observed that our specific scatter search implementation found solutions of a higher …

Permutationeducation.field_of_studybusiness.industryComputer scienceGenetic algorithmPopulationCombinatorial optimizationLocal search (optimization)Context (language use)businesseducationAlgorithmEvolutionary computation
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