Search results for "Macro"

showing 10 items of 3471 documents

Defective Induction of COX-2 Expression by Psoriatic Fibroblasts Promotes Pro-inflammatory Activation of Macrophages

2019

Fibroblasts play an important role as members of the innate immune system through the secretion of COX-2-derived inflammatory mediators such as prostaglandin E-2 (PGE(2)). However, it has been described that dermal fibroblasts behave like mesenchymal stem cells reducing lymphocyte recruitment and dendritic cell activation through PGE(2) release. As the role of fibroblasts in psoriasis remains poorly characterized, in the present study we have evaluated the possible influence of PGE(2) derived from dermal fibroblasts as modulator of the immune response in psoriatic skin. Our results indicate that under inflammatory conditions, psoriatic fibroblasts showed defective induction of COX-2, which …

lcsh:Immunologic diseases. AllergyAdultMale0301 basic medicineTHP-1 CellsLymphocyteImmunologyInflammationDinoprostoneYoung Adult03 medical and health sciences0302 clinical medicineImmune systemPsoriasismedicineHumansImmunology and AllergyPsoriasisProstaglandin E2SkinOriginal ResearchInflammationInnate immune systemChemistryMacrophagesMesenchymal stem cellpsoriasisDendritic cellMiddle AgedFibroblastsmedicine.diseaseCyclooxygenasecyclooxygenase030104 developmental biologymedicine.anatomical_structureCyclooxygenase 2inflammationCancer researchFemalemedicine.symptomlcsh:RC581-607030215 immunologymedicine.drug
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Innate Immune Cells' Contribution to Systemic Lupus Erythematosus

2019

Systemic lupus erythematosus (SLE) is a chronic autoimmune disease characterized by the presence of autoantibodies against nuclear antigens, immune complex deposition, and tissue damage in the kidneys, skin, heart and lung. Because of the pathogenic role of antinuclear antibodies and autoreactive T cells in SLE, extensive efforts have been made to demonstrate how B cells act as antibody-producing or as antigen-presenting cells that can prime autoreactive T cell activation. With the discovery of new innate immune cells and inflammatory mediators, innate immunity is emerging as a key player in disease pathologies. Recent work over the last decade has highlighted the importance of innate immun…

lcsh:Immunologic diseases. AllergyAnti-nuclear antibodyMini ReviewT cellImmunologyPathogenesisAntigenimmune system diseasesmedicineAnimalsHumansLupus Erythematosus SystemicImmunology and Allergydendritic cellsskin and connective tissue diseasesinnate immunitylupus (SLE)Autoimmune diseaseInnate immune systembusiness.industryInnate lymphoid cellAutoantibodymedicine.diseaseImmunity Innatemacrophage-cellmedicine.anatomical_structureImmunologyinnate lymphoid celllcsh:RC581-607businessFrontiers in Immunology
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Metabolism via arginase or nitric oxide synthase: two competing arginine pathways in macrophages

2014

Macrophages play a major role in the immune system, both as antimicrobial effector cells and as immunoregulatory cells, which induce, suppress or modulate adaptive immune responses. These key aspects of macrophage biology are fundamentally driven by the phenotype of macrophage arginine metabolism that is prevalent in an evolving or ongoing immune response. M1 macrophages express the enzyme nitric oxide synthase (NOS), which metabolizes arginine to nitric oxide (NO) and citrulline. NO can be metabolized to further downstream reactive nitrogen species, while citrulline might be reused for efficient NO synthesis via the citrulline-NO cycle. M2 macrophages are characterized by expression of the…

lcsh:Immunologic diseases. AllergyArginineMOUSE MACROPHAGESImmunologyReview ArticlemacrophageM1 and M2BiologyArginineamino acid transporterchemistry.chemical_compoundImmune systemALTERNATIVELY ACTIVATED MACROPHAGESCitrullineImmunology and AllergyMacrophageALVEOLAR MACROPHAGESIN-VIVOReactive nitrogen speciesMARROW-DERIVED MACROPHAGESScience & TechnologyT-CELL RESPONSESMOLECULAR-CLONINGArginaseImmunoregulationAcquired immune systemM2 MacrophageArginaseTUMOR-ASSOCIATED MACROPHAGESchemistryBiochemistryMURINE MACROPHAGESAMINO-ACID TRANSPORTERSNitric Oxide Synthaselcsh:RC581-607Life Sciences & BiomedicineFrontiers in Immunology
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Role of Peripheral Immune Cells-Mediated Inflammation on the Process of Neurodegenerative Diseases

2020

Neurodegenerative diseases are characterized by progressive loss of selectively vulnerable neuronal populations, which contrasts with selectively static loss of neurons due to toxic or metabolic disorders. The mechanisms underlying their progressive nature remain unknown. To date, a timely and well-controlled peripheral inflammatory reaction is verified to be essential for neurodegenerative diseases remission. The influence of peripheral inflammation on the central nervous system is closely related to immune cells activation in peripheral blood. The immune cells activation participated in the uncontrolled and prolonged inflammation that drives the chronic progression of neurodegenerative di…

lcsh:Immunologic diseases. AllergyCentral Nervous System0301 basic medicinedendritic cellT-LymphocytesT cellImmunologyCentral nervous systemInflammationReviewmacrophageNatural killer cell03 medical and health sciences0302 clinical medicineImmune systemAnimalsHumansImmunology and AllergyMedicineMacrophageB cellInflammationB-LymphocytesB cellbusiness.industryMacrophagesT cellNeurodegenerative DiseasesDendritic cellnatural killer cellKiller Cells Natural030104 developmental biologymedicine.anatomical_structuremonocyteImmunologyNeurogenic Inflammationmedicine.symptomlcsh:RC581-607peripheral immune cellsbusiness030215 immunologyFrontiers in Immunology
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CD11b Regulates Fungal Outgrowth but Not Neutrophil Recruitment in a Mouse Model of Invasive Pulmonary Aspergillosis

2019

Abstract Background and Aims: In immunosuppressed individuals Aspergillus (A.) fumigatus is a frequent cause of invasive pulmonary aspergillosis (IPA) which is highly associated with relevant morbidity and mortality. Moreover, it often occurs in patients suffering from leukocyte-adhesion deficiency type 1 (LAD1) which is triggered by a functional loss of CD18 in ß2 integrin receptors as these receptors consist of an alpha subunit (CD11a-CD11d) and CD18 as the common beta subunit. ß2 integrin receptors are differentially expressed by leukocytes, and are required for cell-cell interaction, transendothelial migration, uptake of opsonized pathogens, and cell signaling processes. Here, we asked …

lcsh:Immunologic diseases. AllergyChemokineNeutrophilsPhagocytosisImmunology610 MedizinMedizinMacrophage-1 AntigenCD18InflammationKaplan-Meier EstimateBronchoalveolar LavageBiochemistryMicrobiologyAspergillus fumigatusProinflammatory cytokinecomplement receptor 3MicePhagocytosis610 Medical sciencesmedicineAnimalspneumoniaCC-chemokine ligand 5LungOriginal ResearchInflammationInvasive Pulmonary AspergillosisMice KnockoutCD11b Antigenbiologymedicine.diagnostic_testAspergillus fumigatusCD11bpolymorphonuclear neutrophilsCell BiologyHematologybiology.organism_classificationMice Inbred C57BLDisease Models AnimalBronchoalveolar lavageNeutrophil InfiltrationIntegrin alpha Mβ2 integrinsbiology.proteinCytokinesFemalemedicine.symptomlcsh:RC581-607
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Blockade of MIF–CD74 Signalling on Macrophages and Dendritic Cells Restores the Antitumour Immune Response Against Metastatic Melanoma

2018

Mounting an effective immune response against cancer requires the activation of innate and adaptive immune cells. Metastatic melanoma is the most aggressive form of skin cancer. While immunotherapies have shown a remarkable success in melanoma treatment, patients develop resistance by mechanisms that include the establishment of an immune suppressive tumor microenvironment. Thus, understanding how metastatic melanoma cells suppress the immune system is vital to develop effective immunotherapies against this disease. In this study, we find that macrophages (MOs) and dendritic cells (DCs) are suppressed in metastatic melanoma and that the Ig-CDR-based peptide C36L1 is able to restore MOs and …

lcsh:Immunologic diseases. Allergymacrophage migration inhibitory factorchemical and pharmacologic phenomenadendritic cellspeptide-based immunotherapylcsh:RC581-607immune responsemetastatic melanomamacrophagesFrontiers in Immunology
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Leukotriene B4-loaded microspheres: a new therapeutic strategy to modulate cell activation

2008

AbstractBackgroundLeukotriene B4(LTB4) is a potent inflammatory mediator that also stimulates the immune response. In addition, it promotes polymorphonuclear leukocyte phagocytosis, chemotaxis, chemokinesis and modulates cytokines release. Regarding chemical instability of the leukotriene molecule, in the present study we assessed the immunomodulatory activities conferred by LTB4released from microspheres (MS). A previous oil-in-water emulsion solvent extraction-evaporation method was chosen to prepare LTB4-loaded MS.ResultsIn the mice cremasteric microcirculation, intraescrotal injection of 0.1 ml of LTB4-loaded MS provoked significant increases in leukocyte rolling flux, adhesion and emig…

lcsh:Immunologic diseases. Allergymedicine.medical_specialtyLeukotriene B4ImmunologyChemokinesisLeukocyte RollingBiologyPharmacologyNitric OxideLeukotriene B4Nitric oxideMicechemistry.chemical_compoundCell MovementInternal medicineCell AdhesionLeukocytesmedicineAnimalsHumansLeukocyte RollingPPAR alphaLungChemokine CCL2NitritesLeukotrieneMonocyteEndothelial CellsChemotaxisMicrospheresEndocrinologymedicine.anatomical_structurechemistryMacrophages Peritoneallipids (amino acids peptides and proteins)Cell activationlcsh:RC581-607Research ArticleBMC Immunology
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Nivolumab Enhances In Vitro Effector Functions of PD-1+ T-Lymphocytes and Leishmania-Infected Human Myeloid Cells in a Host Cell-Dependent Manner

2017

Functional impairment of T-cells and a concomitant augmented expression of programmed death-1 (PD-1) have been observed in visceral leishmaniasis patients, as well as in experimental models for visceral and cutaneous leishmaniasis. The PD-1/PD-1-ligand (PD-1/PD-L) interaction negatively regulates T-cell effector functions, which are required for parasite control during leishmaniasis. The aim of this study was to elucidate the impact of the PD-1/PD-L axis in a human primary in vitro infection model of Leishmania major (Lm). Blocking the PD-1/PD-L interaction with nivolumab increased T-cell proliferation and release of the proinflammatory cytokines TNFα and IFNγ during the cocultivation of Lm…

lcsh:Immunologic diseases. Allergyprogrammed death-1 ligand 10301 basic medicineprogrammed death-1 ligand 2ImmunologyProinflammatory cytokine03 medical and health sciencesCutaneous leishmaniasisPD-L1medicineImmunology and AllergyLeishmania majorGranulysinOriginal Researchprogrammed death-1Leishmanianivolumabhuman macrophagesbiologyT-cellsmedicine.diseasebiology.organism_classification030104 developmental biologyGranzymePerforinImmunologybiology.proteinTumor necrosis factor alphahuman dendritic cellslcsh:RC581-607Frontiers in Immunology
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Macrophage: SHIP of Immunity

2014

Immunology. Why does it exist? Two words. Cure disease. People get diseases. “Test tubes” do not. People fund immunologists for solutions to their health problems. But, immunologists often study leukocytes in test tubes – the laboratory – away from diseases. Why? Because much can be learned from analyzing cellular biochemistry and behaviors in vitro that cannot be ascertained when leukocytes are in animals. At the same time, isolated leukocyte reactions often do not reflect how the immune system operates as a unit. So, it is critical to verify in vitro observations in vivo. Among leukocytes, macrophages are the central initiating and directing element in immune systems, and serve this role …

lcsh:Immunologic diseases. AllergywoundImmunologyM1DiseasemacrophageBiologyM2Immune systemAntigenIn vivoImmunitynitric oxideImmunology and AllergyMacrophagecancerCancerWound HealingInnate immune systemMacrophagesarginaseOpinion ArticleIn vitroiNOSImmunologylcsh:RC581-607Frontiers in Immunology
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Blueprint for a minimal photoautotrophic cell: conserved and variable genes in Synechococcus elongatus PCC 7942.

2011

This is an Open Access article distributed under the terms of the Creative Commons Attribution License.-- et al.

lcsh:QH426-470Gene Transfer HorizontalGenomic IslandsOperonlcsh:BiotechnologyComputingMilieux_LEGALASPECTSOFCOMPUTINGmacromolecular substancesGenome03 medical and health sciencesOpen Reading FramesBacterial ProteinsBacterial proteinslcsh:TP248.13-248.65Genomic islandGeneticsGenomic islandsGeneGenome sizeGene transferPhylogeny030304 developmental biologyGeneticsSynechococcus0303 health sciencesBase CompositionGenomebiology030302 biochemistry & molecular biologySynechococcusbiology.organism_classification3. Good healthlcsh:GeneticsCodon usage biasHorizontal gene transferbacteriaGenome BacterialBiotechnologyResearch ArticleBMC genomics
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