Search results for "Macrophage"

showing 10 items of 781 documents

Insights on Kawasaki disease and multisystem inflammatory syndrome: relationship with COVID-19 infection.

2021

At the beginning of coronavirus disease 2019 (COVID-19) children seemed to be less affected and with milder symptoms than adults. Afterward, however, a warning was released regarding the possible association between COVID-19 and Kawasaki disease (KD) or Kawasaki-like disease. Thereafter, labels of Paediatric Inflammatory Multisystem Syndrome Temporally associated with SARS-CoV-2 (PIMS-TS) in Europe and Multisystem Inflammatory Syndrome in Children (MIS-C) in the USA were coined to refer to this new disease entity. The reality is that PIMS-TS/MIS-C resembles certain KD complications such as toxic shock syndrome and macrophage activation syndrome than to classic KD. PIMS-TS/MIS-C and KD share…

MyocarditisCoronavirus disease 2019 (COVID-19)AdolescentDiseaseMucocutaneous Lymph Node Syndrome03 medical and health sciences0302 clinical medicinehemic and lymphatic diseases030225 pediatricsmedicineHumansChildInnate immune systembusiness.industryToxic shock syndromeCOVID-19Infantmedicine.diseaseSystemic Inflammatory Response Syndrome030228 respiratory systemInfectious disease (medical specialty)Macrophage activation syndromeChild PreschoolPediatrics Perinatology and Child HealthImmunologyKawasaki diseasebusinessMinerva pediatrics
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Phytochemical indicaxanthin suppresses 7-ketocholesterol-induced THP-1 cell apoptosis by preventing cytosolic Ca++ increase and oxidative stress

2013

7-Ketocholesterol (7-KC)-induced apoptosis of macrophages is considered a key event in the development of human atheromas. In the present study, the effect of indicaxanthin (Ind), a bioactive pigment from cactus pear fruit, on 7-KC-induced apoptosis of human monocyte/macrophage THP-1 cells was investigated. A pathophysiological condition was simulated by using amounts of 7-KC that can be reached in human atheromatous plaque. Ind was assayed within a micromolar concentration range, consistent with its plasma level after dietary supplementation with cactus pear fruit. Pro-apoptotic effects of 7-KC were assessed by cell cycle arrest, exposure of phosphatidylserine at the plasma membrane, varia…

NADPH oxidase-4MacrophageSettore BIO/10 - BiochimicaApoptosiIndicaxanthin7-Ketocholesterol
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Zebrafish Embryos Allow Prediction of Nanoparticle Circulation Times in Mice and Facilitate Quantification of Nanoparticle–Cell Interactions

2020

The zebrafish embryo is a vertebrate well suited for visualizing nanoparticles at high resolution in live animals. Its optical transparency and genetic versatility allow noninvasive, real-time observations of vascular flow of nanoparticles and their interactions with cells throughout the body. As a consequence, this system enables the acquisition of quantitative data that are difficult to obtain in rodents. Until now, a few studies using the zebrafish model have only described semiquantitative results on key nanoparticle parameters. Here, a MACRO dedicated to automated quantitative methods is described for analyzing important parameters of nanoparticle behavior, such as circulation time and…

NANOCARRIERSEmbryo Nonmammalianmiceanimal structurescirculation timeCellNanoparticleLIPOSOMES02 engineering and technology010402 general chemistry01 natural sciencesSEQUENCEBiomaterialsMiceDELIVERYmedicineMedicine and Health SciencesAnimalsGeneral Materials ScienceZebrafishZebrafishbiologyChemistryMacrophagesEndothelial CellsOptical transparencyPLGAGeneral ChemistryTARGETING MACROPHAGES021001 nanoscience & nanotechnologybiology.organism_classificationzebrafishCANCER0104 chemical sciencesCell biologymacrophagesChemistrymedicine.anatomical_structureCell cultureembryonic structuresZebrafish embryoNanoparticlesCirculation timenanoparticlesNanocarriers0210 nano-technologyANTIBIOTICSBiotechnology
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Loss of Nrf2 in bone marrow-derived macrophages impairs antigen-driven CD8+ T cell function by limiting GSH and Cys availability

2015

NF-E2-related factor 2 (Nrf2), known to protect against reactive oxygen species, has recently been reported to resolve acute inflammatory responses in activated macrophages. Consequently, disruption of Nrf2 promotes a proinflammatory macrophage phenotype. In the current study, we addressed the impact of this macrophage phenotype on CD8(+) T cell activation by using an antigen-driven coculture model consisting of Nrf2(-/-) and Nrf2(+/+) bone marrow-derived macrophages (BMDMΦ) and transgenic OT-1 CD8(+) T cells. OT-1 CD8(+) T cells encode a T cell receptor that specifically recognizes MHC class I-presented ovalbumin OVA(257-264) peptide, thereby causing a downstream T cell activation. Interes…

NF-E2-Related Factor 2OvalbuminAntiporterT cellBlotting WesternReceptors Antigen T-CellApoptosisMice TransgenicCD8-Positive T-LymphocytesBiologyReal-Time Polymerase Chain Reactionenvironment and public healthBiochemistryAntioxidantsImmunoenzyme TechniquesMicechemistry.chemical_compoundBone MarrowPhysiology (medical)MHC class ImedicineAnimalsCytotoxic T cellRNA MessengerCells CulturedCell ProliferationMice KnockoutReverse Transcriptase Polymerase Chain ReactionGCLMMacrophagesHistocompatibility Antigens Class IGlutathionerespiratory systemFlow CytometryGlutathioneMolecular biologyMice Inbred C57BLOxidative Stressmedicine.anatomical_structurechemistrybiology.proteinCystineReactive Oxygen SpeciesIntracellularCD8Signal TransductionFree Radical Biology and Medicine
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The IFN-γ-Inducible GTPase, Irga6, Protects Mice against Toxoplasma gondii but Not against Plasmodium berghei and Some Other Intracellular Pathogens

2011

Clearance of infection with intracellular pathogens in mice involves interferon-regulated GTPases of the IRG protein family. Experiments with mice genetically deficient in members of this family such as Irgm1(LRG-47), Irgm3(IGTP), and Irgd(IRG-47) has revealed a critical role in microbial clearance, especially for Toxoplasma gondii. The in vivo role of another member of this family, Irga6 (IIGP, IIGP1) has been studied in less detail. We investigated the susceptibility of two independently generated mouse strains deficient in Irga6 to in vivo infection with T. gondii, Mycobacterium tuberculosis, Leishmania mexicana, L. major, Listeria monocytogenes, Anaplasma phagocytophilum and Plasmodium …

Naturwissenschaftliche Fakultät -ohne weitere Spezifikation-Plasmodium bergheiScience-medicine.disease_causeMicrobiologyLeishmania mexicanaGTP PhosphohydrolasesMicrobiologyMiceListeria monocytogenesIn vivoddc:570Parasite Groupsparasitic diseasesmedicineAnimalsLeishmania majorPlasmodium bergheiBiologyMicrobial PathogensPathogenMultidisciplinarybiologyIntracellular parasiteQRImmunityToxoplasma gondiiImmune DefenseMacrophage Activationbiology.organism_classificationVirologyInnate ImmunityBacterial PathogensHost-Pathogen InteractionMice Inbred C57BLMedicineParasitologyToxoplasmaToxoplasmosisResearch ArticlePLoS ONE
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Homeostasis of Microglia in the Adult Brain: Review of Novel Microglia Depletion Systems.

2015

Microglia are brain macrophages that emerge from early erythro-myeloid precursors in the embryonic yolk sac and migrate to the brain mesenchyme before the blood brain barrier is formed. They seed the brain, and proliferate until they have formed a grid-like distribution in the central nervous system that is maintained throughout lifespan. The mechanisms through which these embryonic-derived cells contribute to microglia homoeostasis at steady state and upon inflammation are still not entirely clear. Here we review recent studies that provided insight into the contribution of embryonically-derived microglia and of adult 'microglia-like' cells derived from monocytes during inflammation. We ex…

NeuroimmunomodulationCellular differentiationMesenchymeImmunologyCentral nervous systemEmbryonic DevelopmentInflammation610 Medicine & healthBiologyBlood–brain barrier10263 Institute of Experimental ImmunologymedicineImmunology and AllergyAnimalsHomeostasisHumansNeuroinflammationInflammation2403 ImmunologyMicrogliaMacrophagesBrainCell DifferentiationEmbryonic stem cellDisease Models Animalmedicine.anatomical_structureImmunologyModels Animal2723 Immunology and Allergy570 Life sciences; biologyMicrogliamedicine.symptomTrends in immunology
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Cycloamphilectenes, a new type of potent marine diterpenes: inhibition of nitric oxide production in murine macrophages.

2003

The inhibitory effect of a series of 6 cycloamphilectenes, novel marine diterpenes based on amphilectene skeletons and isolated from the Vanuatu sponge Axinella sp., on NO, PGE(2) and TNFalpha production in murine peritoneal macrophages was studied. These compounds reduced potently nitric oxide production in a concentration-dependent manner with IC(50) values in the submicromolar range (0.1-4.3 microM). Studies on intact cells and Western blot analysis showed that the more potent cycloamphilectenes reduced the expression of inducible nitric oxide synthase without affecting cyclo-oxygenase-2 expression. Among them cycloamphilectene 2, the unique compound bearing an exocyclic methylene group,…

NeutrophilsBlotting WesternNitric Oxide Synthase Type IIElectrophoretic Mobility Shift AssayIn Vitro TechniquesNitric OxideGeneral Biochemistry Genetics and Molecular BiologyDinoprostoneNitric oxidechemistry.chemical_compoundMiceStructure-Activity RelationshipWestern blotmedicineAnimalsEdemaHumansGeneral Pharmacology Toxicology and PharmaceuticsMethylenebiologymedicine.diagnostic_testPancreatic ElastaseTumor Necrosis Factor-alphaMacrophagesZymosanAnti-Inflammatory Agents Non-SteroidalAxinellaNF-kappa BMembrane ProteinsGeneral Medicinebiology.organism_classificationPoriferaNitric oxide synthaseIsoenzymesSpongechemistryBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide Synthasesbiology.proteinMacrophages PeritonealTumor necrosis factor alphaFemaleMarine ToxinsDiterpenesNitric Oxide SynthaseLife sciences
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Diaryl-dithiolanes and -isothiazoles: COX-1/COX-2 and 5-LOX-inhibitory, OH scavenging and anti-adhesive activities

2008

Three series of non-steroidal anti-inflammatory drugs (NSAIDs) inhibiting the cyclooxygenase/5-lipoxygenase (COX/5-LOX) pathways as such as formation of hydroxyl radicals and adhesion were prepared: 4,5-diaryl isothiazoles, 4,5-diaryl 3H-1,2-dithiole-3-thiones and 4,5-diaryl 3H-1,2-dithiole-3-ones. The aim of the present study was to develop substances which can intervene into the inflammatory processes via different mechanisms of action as multiple target non-steroidal anti-inflammatory drugs (MTNSAIDs) with increased anti-inflammatory potential. The current lead 11a was evaluated in COX-1/2, 5-LOX and (*)OH scavenging in vitro assays and in a static adhesion assay where it proved to inhib…

NeutrophilsClinical BiochemistryMacrophage-1 AntigenPharmaceutical SciencePeritonitisPharmacologyBiochemistryMiceIn vivoDrug DiscoveryCell AdhesionAnimalsCyclooxygenase InhibitorsLipoxygenase InhibitorsSulfhydryl CompoundsCell adhesionMolecular BiologybiologyHydroxyl RadicalChemistryAnti-Inflammatory Agents Non-SteroidalOrganic ChemistryIn vitro toxicologyBiological activityFree Radical ScavengersAdhesionIn vitroThiazolesBiochemistryCyclooxygenase 2Arachidonate 5-lipoxygenaseCyclooxygenase 1biology.proteinMolecular MedicineCyclooxygenaseBioorganic & Medicinal Chemistry
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Pharmacology of Ischemia-Reperfusion. Translational Research Considerations.

2016

Ischemia-reperfusion (IRI) is a complex physiopathological mechanism involving a large number of metabolic processes that can eventually lead to cell apoptosis and ultimately tissue necrosis. Treatment approaches intended to reduce or palliate the effects of IRI are varied, and are aimed basically at: inhibiting cell apoptosis and the complement system in the inflammatory process deriving from IRI, modulating calcium levels, maintaining mitochondrial membrane integrity, reducing the oxidative effects of IRI and levels of inflammatory cytokines, or minimizing the action of macrophages, neutrophils, and other cell types. This study involved an extensive, up-to-date review of the bibliography …

NeutrophilsIschemiaApoptosis030204 cardiovascular system & hematologyPharmacologyurologic and male genital diseasesAntioxidantsProinflammatory cytokineTranslational Research Biomedical03 medical and health sciences0302 clinical medicinemedicineHumanscardiovascular diseasesIschemic PreconditioningOpiate alkaloidurogenital systemMechanism (biology)business.industryTumor Necrosis Factor-alphaMacrophagesOpiate AlkaloidsfungiNF-kappa BComplement System Proteinsmedicine.diseaseApoptosis030220 oncology & carcinogenesisReperfusion InjuryAnesthetics InhalationIschemic preconditioningCytokinesSurgeryTumor necrosis factor alphaInflammation MediatorsbusinessReperfusion injuryJournal of investigative surgery : the official journal of the Academy of Surgical Research
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Inhibition of 5-lipoxygenase activity by the natural anti-inflammatory compound aethiopinone

2001

Objetive and Design: We have investigated the mechanisms of action of aethiopinone, an anti-inflammatory compound from Salvia aethiopis L. roots.¶Material and Subjects: Human neutrophils from healthy volunteers and murine peritoneal macrophages. Swiss mice were randomly divided into groups of six animals.¶Treatment: Test compounds were applied topically in the mouse ear oedema test. In the air pouch, mice received aethiopinone (0.001-0.5 μmol/pouch or 12.5-50 mg/kg p.o.).¶Methods: LTB4 production was assayed in human neutrophils and COX-2 and iNOS activities in murine macrophages. Air pouches were induced subcutaneously in mice and injected with zymosan on the day six. Mouse ear oedema was …

Neutrophilsmedicine.drug_classImmunologyNitric Oxide Synthase Type IIPharmacologyLeukotriene B4DinoprostonePhospholipases AAnti-inflammatoryMicechemistry.chemical_compoundIn vivoAnimalsEdemaHumansMedicineLipoxygenase InhibitorsIC50InflammationPharmacologyArachidonic Acidbiologybusiness.industryAnti-Inflammatory Agents Non-SteroidalZymosanMembrane ProteinsEarbiology.organism_classificationIn vitroIsoenzymeschemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesArachidonate 5-lipoxygenaseImmunologyCyclooxygenase 1Macrophages PeritonealSalvia aethiopisbiology.proteinArachidonic acidNitric Oxide SynthasebusinessNaphthoquinonesInflammation Research
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