Search results for "Map"

showing 10 items of 3484 documents

Human cationic amino acid transporter gene hCAT-2 is assigned to 8p22 but is not the causative gene in lysinuric protein intolerance

1997

Lysinuric protein intolerance (LPI) is a recessively inherited amino acid disorder characterized by defective efflux of cationic amino acids at the basolateral membrane of the intestinal and renal tubular epithelium. Recently, cDNAs encoding the related proteins hCAT-2A and hCAT-2B have been cloned. These two carrier proteins are most likely the product of the same gene, hCAT-2. Using the hCAT-2B cDNA, we assigned the hCAT-2 gene to chromosome 8p22. Furthermore, by linkage analysis in Finnish LPI families, we ruled out that hCAT-2B is involved in LPI disease.

Genetic LinkageBiologyGene mappingGenetic linkageComplementary DNAGeneticsmedicineHumansAmino acid transporterAmino Acid Metabolism Inborn ErrorsGeneGenetics (clinical)chemistry.chemical_classificationLysineChromosome MappingMembrane Proteinsmedicine.diseaseLysinuric protein intoleranceAmino acidchemistryBiochemistryAmino Acid Transport Systems BasicEffluxCarrier ProteinsChromosomes Human Pair 8Microsatellite RepeatsHuman Genetics
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Polymerase chain reaction analysis of the Xba I polymorphism of the human complement C4 genes provides evidence for strong haplotype conservation.

1995

The genes coding for the two isotypes of the fourth component of human complement, C4A and C4B, are located between the HLA-B and -DR loci of the MHC. We studied the linkage relationship of the previously described XbaI RFLP to obtain further insight into the evolution of the tandemly arranged C4 genes. Using exon-specific PCR amplification followed by restriction analysis and direct DNA sequencing, the polymorphic site could be located in exon 40 of the C4 gene (cDNA position 5095). The polymorphism does not change an amino acid residue. Using nested PCR amplification with isotype-specific primers to amplify either C4A or C4B alleles the haplotype arrangement of the XbaI sites in both isot…

Genetic LinkageImmunologyMolecular Sequence DataBiologyPolymerase Chain Reactionlaw.inventionExonlawComplementary DNAImmunology and AllergyHumansDeoxyribonucleases Type II Site-SpecificGenePolymerase chain reactionGeneticsPolymorphism GeneticBase SequenceHaplotypeIntronChromosome MappingComplement C4General MedicineMolecular biologyRestriction siteHaplotypesRestriction fragment length polymorphismPolymorphism Restriction Fragment LengthHuman immunology
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Human type I cytokeratin genes are a compact cluster

1997

A YAC clone (211F11) containing approximately 0.5 Mb of human DNA was isolated from a human genomic library by PCR-based screening with cytokeratin (KRT) 13-specific primers. The YAC clone was mapped by FISH to the long arm of chromosome 17 (17q12→q21), a region to which several other type I KRT genes had been mapped previously. We now show by Southern blot hybridization and PFGE analyses that KRT13, 14, 15, and 16 are all contained within YAC clone 211F11. Long-range restriction mapping analysis of clone 211F11 and of two smaller YAC clones that were also isolated with KRT13-specific primers, suggests that KRT13, 14, 15, 16 and their linked type I genes KRT17 and 19, are contained in less …

Genetic LinkageLocus (genetics)BiologyPolymerase Chain ReactionRestriction mapGene mappingGene clusterGeneticsHumansGenomic libraryCloning MolecularChromosomes Artificial YeastMolecular BiologyIn Situ Hybridization FluorescenceGenetics (clinical)Southern blotGeneticsBase SequenceChromosome MappingMolecular biologyChromosome 17 (human)genomic DNAMultigene FamilyKeratinsDNA ProbesChromosomes Human Pair 17
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Sequence and evolution of the gene for the monomeric globin I and its linkage to genes coding for dimeric globins in the insect Chironomus thummi.

1995

We isolated genomic clones containing sequences encoding globins I and IA from a Chironomus thummi thummi genomic library. Three clones contain globin IA (ctt-1A) genes, while one contains a globin I (ctt-1) gene. The coding regions of the four genes are identical except for the single base substitution accounting for the globin I/IA polymorphism. The noncoding DNA flanking the coding region is more than 98% similar, confirming a previous hypothesis that the globin ctt-1 and ctt-1A genes are alleles. Hemoglobins I and IA are monomeric in the insect hemolymph. Earlier in situ hybridization studies suggested that monomeric and dimeric globin genes are clustered at different chromosomal loci. …

Genetic LinkageMolecular Sequence DataGenes InsectBiologyChironomidaechemistry.chemical_compoundMolecular evolutionhemic and lymphatic diseasesGeneticsCoding regionAnimalsGenomic libraryGlobinAmino Acid SequenceMolecular BiologyGeneEcology Evolution Behavior and SystematicsIn Situ HybridizationGeneticsPolytene chromosomeBase SequenceSequence Homology Amino AcidChromosome MappingMolecular biologyNoncoding DNABiological EvolutionGlobinschemistrySequence AlignmentDNAJournal of molecular evolution
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Genetic variability at neutral markers, quantitative trait loci and trait in a subdivided population under selection

2003

Abstract Genetic variability in a subdivided population under stabilizing and diversifying selection was investigated at three levels: neutral markers, QTL coding for a trait, and the trait itself. A quantitative model with additive effects was used to link genotypes to phenotypes. No physical linkage was introduced. Using an analytical approach, we compared the diversity within deme (HS) and the differentiation (FST) at the QTL with the genetic variance within deme (VW) and the differentiation (QST) for the trait. The difference between FST and QST was shown to depend on the relative amounts of covariance between QTL within and between demes. Simulations were used to study the effect of se…

Genetic Markers0106 biological sciencesGenotypeQuantitative Trait LociPopulation[SDV.GEN] Life Sciences [q-bio]/GeneticsQuantitative trait locusBiology010603 evolutionary biology01 natural sciences03 medical and health sciencesFamily-based QTL mappingGeneticsComputer SimulationGenetic variabilitySelection Genetic10. No inequalityeducationSelection (genetic algorithm)ComputingMilieux_MISCELLANEOUS030304 developmental biologyDemeGenetics0303 health scienceseducation.field_of_study[SDV.GEN]Life Sciences [q-bio]/GeneticsModels GeneticDisruptive selectionGenetic VariationGenetic architectureGenetics PopulationPhenotypeEvolutionary biologyResearch Article
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Development and implementation of high-throughput SNP genotyping in barley

2009

Abstract Background High density genetic maps of plants have, nearly without exception, made use of marker datasets containing missing or questionable genotype calls derived from a variety of genic and non-genic or anonymous markers, and been presented as a single linear order of genetic loci for each linkage group. The consequences of missing or erroneous data include falsely separated markers, expansion of cM distances and incorrect marker order. These imperfections are amplified in consensus maps and problematic when fine resolution is critical including comparative genome analyses and map-based cloning. Here we provide a new paradigm, a high-density consensus genetic map of barley based…

Genetic Markers0106 biological sciencesGenotypelcsh:QH426-470Genetic Linkagelcsh:BiotechnologyPopulationSingle-nucleotide polymorphismBiologyPolymorphism Single Nucleotide01 natural sciences03 medical and health sciencesGene mappinglcsh:TP248.13-248.65Research articleGeneticseducationAlleles030304 developmental biology2. Zero hungerGenetics0303 health scienceseducation.field_of_studyfood and beveragesHordeumSNP genotypingMinor allele frequencylcsh:GeneticsGenetic TechniquesGenetic distanceGenetic markerDoubled haploidy010606 plant biology & botanyBiotechnology
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Polycystin-1 downregulation induces ERK-dependent mTOR pathway activation in a cellular model of psoriasis

2018

Psoriatic plaques tend to localize to the knees and elbows, areas that are particularly subject to mechanical stress resulting from bending and friction. Moreover, plaques often develop at sites of mechanical trauma or injury (Koebner phenomenon). Nevertheless, mechanotransduction has never been linked to psoriasis. Polycystins (polycystin-1, PC1; polycystin-2, PC2) are mechanosensitive molecules that function as key regulators of cellular mechanosensitivity and mechanotransduction. The aim of this in vitro study was to investigate the role of polycystins in the development of psoriasis. We showed that PC1 knockdown in HaCaT cells led to an elevated mRNA expression of psoriasis-related biom…

Genetic Markers0301 basic medicineMAPK/ERK pathwayendocrine systemTRPP Cation ChannelsMAP Kinase Signaling SystemDown-RegulationModels BiologicalCell Line03 medical and health sciences0302 clinical medicineDownregulation and upregulationCell MovementPsoriasismedicineHumansPsoriasisMechanotransductionMolecular BiologyPI3K/AKT/mTOR pathwayCell ProliferationGene knockdownCell growthChemistryTOR Serine-Threonine Kinasesmedicine.diseaseCell biologyHaCaT030104 developmental biologyGene Knockdown Techniques030220 oncology & carcinogenesisMolecular MedicineBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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Replication of linkage of familial hypobetalipoproteinemia to chromosome 3p in six kindreds

2002

Familial hypobetalipoproteinemia (FHBL) is a genetically heterogeneous condition characterized by very low apolipoprotein B (apoB) concentrations in plasma and/or low levels of LDL-cholesterol (LDL-C) with a propensity to developing fatty liver. In a minority of cases, truncation-specifying mutations of the apoB gene (APOB) are etiologic, but the genetic basis of most cases is unknown. We previously reported linkage of FHBL to a 10 cM region on 3p21.1-22 in one kindred. The objectives of the current study were to identify other FHBL families with linkage to 3p and to narrow the FHBL susceptibility region on 3p. Six additional FHBL kindreds unlinked to the APOB region on chromosome 2 were ge…

Genetic MarkersAdultMaleMeiosiSettore MED/09 - Medicina InternaApolipoprotein BGenotypeGenetic LinkageQD415-436BiologyBiochemistryChromosomal crossoverHypobetalipoproteinemiasEndocrinologyQuantitative Trait HeritableGenetic linkageGenetic MarkerHaplotypeHumanslinkage analysisCrossing Over GeneticChildAgedAdult; Aged; Aged 80 and over; Child; Chromosome Mapping; Chromosomes Human Pair 3; Crossing Over Genetic; Female; Genetic Linkage; Genetic Markers; Genotype; Haplotypes; Humans; Hypobetalipoproteinemias; Male; Meiosis; Middle Aged; Pedigree; Quantitative Trait HeritableGeneticsAged 80 and overGenetic heterogeneityHaplotypeChromosomeChromosome MappingCell BiologyoligogenicMiddle AgedPedigreeMeiosisMarkov chain Monte CarloChromosome 3HaplotypesGenetic markerbiology.proteinvariance componentslipids (amino acids peptides and proteins)FemaleChromosomes Human Pair 3geneticHypobetalipoproteinemiaHuman
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Complete karyotype characterization of the K562 cell line by combined application of G-banding, multiplex-fluorescence in situ hybridization, fluores…

2001

This study combines conventional cytogenetics, fluorescence in situ hybridization (FISH), multiplex-FISH and comparative genomic hybridization (CGH). In applying this multimodal approach on the human leukemia cell line K562, the chromosome composition was refined in detail and compared with data from the literature. A hypotriploid karyotype with a modal chromosome number of 67, and 21 unique marker chromosomes were identified. The classification of six markers was identical to published data and the composition of five further markers from the literature could be fully clarified for the first time. The composition of another five markers, which have been interpreted in divergent ways in dif…

Genetic MarkersCancer Researchmedicine.medical_specialtyG bandingIn situ hybridizationComputational biologyBiologyChromosome PaintingCytogeneticsmedicineHumansIn Situ Hybridization FluorescenceGeneticsmedicine.diagnostic_testCytogeneticsChromosome MappingNucleic Acid HybridizationKaryotypeHematologyModal Chromosome NumberOncologyKaryotypingK562 CellsVirtual karyotypeComparative genomic hybridizationFluorescence in situ hybridizationLeukemia research
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Fine mapping of the 2p11 dyslexia locus and exclusion of TACR1 as a candidate gene.

2003

Developmental dyslexia, or reading disability, is a multigenic complex disease for which at least five loci, i.e. DYX1-3 and DYX5-6, have been clearly identified from the human genome. To date, DYX1C1 is the only dyslexia candidate gene cloned. We have previously reported linkage to 2p11 and 7q32 in 11 Finnish pedigrees. Here, we report the fine mapping of the approximately 40-cM linked region from chromosome 2 as we increased marker density to one per 1.8 cM. Linkage was supported with the highest NPL score of 3.0 (P=0.001) for marker D2S2216. Association analysis using the six pedigrees showing linkage pointed to marker D2S286/rs3220265 (P value0.001) in the near vicinity of D2S2216. We w…

Genetic MarkersCandidate geneLocus (genetics)Quantitative trait locusBiologyPolymorphism Single NucleotideDyslexia03 medical and health sciences0302 clinical medicineGene mappingGenetic linkageGeneticsmedicineHumansGenetics (clinical)FinlandReceptors Tachykinin030304 developmental biologyGenetics0303 health sciencesGene Expression ProfilingHaplotypeDyslexiaChromosome Mappingmedicine.diseaseBlotting NorthernPedigreeGenetic markerChromosomes Human Pair 2030217 neurology & neurosurgeryMicrosatellite RepeatsHuman genetics
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