Search results for "Mesenteric Arteries"

showing 10 items of 28 documents

Role of endothelial nitric oxide in pulmonary and systemic arteries during hypoxia

2014

Abstract Our aim was to investigate the role played by endothelial nitric oxide (NO) during acute vascular response to hypoxia, as a modulator of both vascular tone (through guanylate cyclase (sGC) activation) and mitochondrial O2 consumption (through competitive inhibition of cytochrome-c-oxydase (CcO)). Organ bath experiments were performed and O2 consumption (Clark electrode) was determined in isolated aorta, mesenteric and pulmonary arteries of rats and eNOS-knockout mice. All pre-contracted vessels exhibited a triphasic hypoxic response consisting of an initial transient contraction (not observed in vessels from eNOS-knockout mice) followed by relaxation and subsequent sustained contra…

MaleCancer ResearchContraction (grammar)Nitric Oxide Synthase Type IIIEndotheliumPhysiologyClinical BiochemistryVasodilationPulmonary ArteryMitochondrionPharmacologyNitric OxideBiochemistryNitric oxideRats Sprague-DawleyMicechemistry.chemical_compoundNon-competitive inhibitionEnosmedicineAnimalsHypoxiaAortaMice KnockoutbiologyMyxothiazolEndothelial Cellsbiology.organism_classificationMesenteric ArteriesRatsMice Inbred C57BLmedicine.anatomical_structurechemistryAnesthesiacardiovascular systemNitric Oxide
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Role of the Endothelium in the Relaxation Induced by Propofol and Thiopental in Isolated Arteries from Man

1997

Abstract Induction of anaesthesia with intravenous propofol and thiopental is often accompanied by hypotension. This study evaluates whether propofol and thiopental induce relaxation of isolated arteries from man and whether this effect is modulated by the endothelium. Mesenteric artery rings (with and without endothelium) from 12 patients were placed in organ baths and precontracted with phenylephrine before addition of propofol (10−3 M) or thiopental (10−3 M). Relaxation induced by propofol and thiopental was evaluated for rings with intact endothelium in the presence of the nitric oxide synthase inhibitor NG-nitro-l-arginine methyl ester (l-NAME; 10−4 M) or the cyclooxygenase inhibitor i…

MaleEndotheliumMuscle RelaxationIndomethacinPharmaceutical ScienceVasodilationIn Vitro TechniquesPharmacologyMuscle Smooth VascularNitric oxidePhenylephrinechemistry.chemical_compoundmedicineHumansCyclooxygenase InhibitorsThiopentalPropofolMesenteric arteriesPhenylephrineAgedPharmacologyAnalysis of VarianceThiopental Sodiumbusiness.industryMiddle AgedMesenteric ArteriesNG-Nitroarginine Methyl Estermedicine.anatomical_structurechemistryAnesthesiaFemaleEndothelium VascularTissue PreservationNitric Oxide SynthasePropofolbusinessAnesthetics IntravenousMuscle Contractionmedicine.drugBlood vesselJournal of Pharmacy and Pharmacology
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Endothelial kinin B1‐receptors are induced by myocardial ischaemia‐reperfusion in the rabbit

2001

Kinin B1-receptors are induced by various inflammatory stimuli. Since myocardial ischaemia-reperfusion results in inflammation, we questioned whether it could induce B1-receptor-dependent responses to des-Arg9-bradykinin (DBK). Thirty-six rabbits were submitted either to a 30 min coronary occlusion followed by a 3 h reperfusion or to a sham operation. The response to DBK was then tested in vivo on mean arterial pressure (MAP) and in vitro on isolated hearts and arterial rings. DBK induced a dose-dependent decrease in MAP in the ischaemia-reperfusion group (DBK, 10 μg kg−1, intra-arterial: -12 ± 2 vs. -5 ± 2 mmHg in the sham group, P < 0.02), which was significantly antagonised by [Leu8]-des…

MaleMean arterial pressuremedicine.medical_specialtyEndotheliumPhysiologyBlotting WesternBradykininBlood PressureMyocardial Reperfusion InjuryInflammationIn Vitro TechniquesBradykininReceptor Bradykinin B1Muscle Smooth Vascularchemistry.chemical_compoundIsometric ContractionInternal medicinemedicineAnimalsMesenteric arteriesDose-Response Relationship Drugbiologybusiness.industryReceptors BradykininAngiotensin-converting enzymeOriginal ArticlesKininImmunohistochemistryMyocardial ContractionPerfusionmedicine.anatomical_structureEndocrinologychemistryImmunologybiology.proteinCoronary perfusion pressureEndothelium VascularRabbitsmedicine.symptombusinessThe Journal of Physiology
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Heterogeneity of atherosclerosis in mesenteric arteries and outgrowth remodeling

2009

Abstract Background In patients with acute mesenteric ischemia by occlusive thrombo-embolism, the superior mesenteric artery (SMA) is more affected than the inferior mesenteric artery (IMA). Methods This study investigated postmortem mesenteric arteries from aged subjects (n=21). Four atherosclerotic stages were defined by signs of degeneration and inflammation in sections stained with Elastica-van-Gieson and immunohistology, respectively. Results In females and males, Stages 3 and 4 were found in 62% of the SMA and 24% of the IMA. Lumenal areas based on diameter measurements remained essentially unchanged between Stages 1 and 4. Compared to a Stage 1 reference, remodeling was associated wi…

MalePathologymedicine.medical_specialtyInflammationSeverity of Illness IndexInferior mesenteric arteryPathology and Forensic MedicineIschemiaMesenteric Artery Superiormedicine.arteryAdventitiaMesenteric Vascular OcclusionLeukocytesmedicineHumansSuperior mesenteric arteryProgenitor cellMesenteric arteriesAgedAged 80 and overInflammationStaining and Labelingbusiness.industryMesenteric Artery InferiorGeneral MedicineAnatomyMiddle AgedAtherosclerosisSMA*Immunohistochemistrymedicine.anatomical_structureVasa vasorumFemaleAutopsyInflammation Mediatorsmedicine.symptomCardiology and Cardiovascular MedicinebusinessCardiovascular Pathology
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Comparative effects of the novel vasotocin analogue F-180 vs. vasopressin and terlipressin on systemic and splanchnic isolated vessels from portal hy…

2001

F-180 has been proposed as a new vasopressin analogue for the treatment of portal hypertension. This study investigates the contractile profile of F-180 compared to vasopressin and its analogue terlipressin on isolated systemic and splanchnic vessels from sham-operated and partial portal vein ligated (PPVL) rats. F-180 (10(-9)-10(-6) M), vasopressin (10(-11)-10(-8) M) and terlipressin (10(-9)-10(-4) M) induced contraction of the mesenteric vein, aorta, iliac, tail and mesenteric arteries. The order of potency in these vessels was vasopressin (pD2 approximately 9)F-180 (pD2 approximately 8)terlipressin (pD2 approximately 6). Significant (P0.01) differences between sham-operated and PPVL rats…

MaleVasopressinmedicine.medical_specialtymedicine.drug_classVasopressinsPharmacology toxicologyPortal veinLypressinVasotocinMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundMesenteric VeinsInternal medicineHypertension PortalmedicineAnimalsVasoconstrictor AgentsPharmacologybusiness.industryGeneral Medicinemedicine.diseaseMesenteric ArteriesRatsEndocrinologychemistryVasoconstrictioncardiovascular systemPortal hypertensionVasopressin AnalogueSplanchnicbusinessTerlipressinTerlipressinmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Arrhythmic sudden cardiac death in a 3-year-old child with intimal fibroplasia of coronary arteries, aorta, and its branches

2001

Abstract We report an unusual case of “arrhythmic” sudden cardiac death in a 3-year-old child who died of ischemic myocardial lesions as a result of intimal fibroplasia of the coronary arteries. Also affected were the aorta and its major branches, whereas renal and mesenteric arteries, celiac trunk, and systemic veins were normal. Histopathologic examination showed severe concentric thickening of intima because of a proliferation of spindle-shaped cells (mesenchymal cells) set in an abundant extracellular matrix. In some vascular segments the intima was densely fibrotic and hyalinized. No significant inflammation, foam cells, cholesterol clefts, or other evidence of atheroma were present. T…

Malemedicine.medical_specialtyHeart VentriclesFibromuscular dysplasiaPathology and Forensic MedicineSudden cardiac deathPolypsAdventitiaInternal medicinemedicine.arterymedicineFibromuscular DysplasiaHumansVimentinMesenteric arteriesAortaAortabusiness.industryMyocardiumArrhythmias CardiacGeneral Medicinemedicine.diseaseCoronary VesselsImmunohistochemistryActinsCoronary arteriesDeath Sudden CardiacAtheromamedicine.anatomical_structureChild Preschoolcardiovascular systemCardiologyDesminTunica IntimaCardiology and Cardiovascular MedicinebusinessCardiovascular Pathology
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Milrinone is preferred to levosimendan for mesenteric perfusion in hypoxia-reoxygenated newborn piglets treated with dopamine

2012

INTRODUCTION There is little information regarding the comparative hemodynamic effects of adding milrinone or levosimendan to dopamine infusion in hypoxia-reoxygenated (H-R) newborns. RESULTS Severely hypoxic piglets had cardiogenic shock with depressed cardiac index (CI) and mean arterial pressure (MAP). The hemodynamics deteriorated gradually after initial recovery upon reoxygenation. Heart rate and CI improved with milrinone (D+M) and levosimendan (D+L) administration (P < 0.05 vs. control). Both regimens improved carotid arterial flow and carotid vascular resistance; D+M additionally improved superior mesentric arterial flow (all P < 0.05 vs. control). No effect was found on renal arter…

Mean arterial pressureCardiotonic AgentsSwineDopamineVasodilator AgentsHemodynamicsMyocardial ReperfusionMyocardial Reperfusion Injury030204 cardiovascular system & hematologyArticle03 medical and health sciences0302 clinical medicine030225 pediatricsMedicineAnimalsHypoxiaSimendanbusiness.industryCardiogenic shockHemodynamicsHydrazonesLevosimendanHypoxia (medical)medicine.disease3. Good healthMesenteric ArteriesPyridazinesOxidative Stressmedicine.anatomical_structureAnimals NewbornAnesthesiaPediatrics Perinatology and Child HealthModels AnimalVascular resistanceMilrinoneDrug Therapy Combinationmedicine.symptombusinessPerfusionmedicine.drugMilrinonePediatric research
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Increased endothelin-1 vasoconstriction in mesenteric resistance arteries after superior mesenteric ischaemia-reperfusion

2012

BACKGROUND AND PURPOSE Endothelin-1 (ET-1) plays an important role in the maintenance of vascular tone. We aimed to evaluate the influence of superior mesenteric artery (SMA) ischaemia-reperfusion (I/R) on mesenteric resistance artery vasomotor function and the mechanism involved in the changes in vascular responses to ET-1. EXPERIMENTAL APPROACH SMA from male Sprague-Dawley rats was occluded (90 min) and following reperfusion (24 h), mesenteric resistance arteries were dissected. Vascular reactivity was studied using wire myography. Protein and mRNA expression, superoxide anion (O2•−) production and ET-1 plasma concentration were evaluated by immunofluorescence, real-time quantitative PCR,…

Pharmacologymedicine.medical_specialtyEndotheliumElectrical impedance myographyChemistryEndothelial NOSEndothelin 1medicine.anatomical_structureEndocrinologymedicine.arteryInternal medicinecardiovascular systemmedicineSuperior mesenteric arterymedicine.symptomReceptorMesenteric arteriesVasoconstrictionBritish Journal of Pharmacology
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Different β-adrenoceptor subtypes coupling to cAMP or NO/cGMP pathways: implications in the relaxant response of rat conductance and resistance vesse…

2013

Background and Purpose To analyse the relative contribution of β1-, β2- and β3-adrenoceptors (Adrb) to vasodilatation in conductance and resistance vessels, assessing the role of cAMP and/or NO/cGMP signalling pathways. Experimental Approach Rat mesenteric resistance artery (MRA) and aorta were used to analyse the Adrb expression by real-time-PCR and immunohistochemistry, and for the pharmacological characterization of Adrb-mediated activity by wire myography and tissue nucleotide accumulation. Key Results The mRNAs and protein for all Adrb were identified in endothelium and/or smooth muscle cells (SMCs) in both vessels. In MRA, Adrb1 signalled through cAMP, Adrb3 through both cAMP and cGMP…

Pharmacologymedicine.medical_specialtyEndotheliumElectrical impedance myographyVasodilationBiologyAdenylyl cyclasechemistry.chemical_compoundmedicine.anatomical_structureEndocrinologychemistryInternal medicineIsoprenalinecardiovascular systemmedicinecAMP-dependent pathwaySoluble guanylyl cyclaseMesenteric arteriesmedicine.drugBritish Journal of Pharmacology
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Relaxant effects of antidepressants on human isolated mesenteric arteries

1999

Aims The therapeutic action of tricyclic agents may be accompanied by unwanted effects on the cardiovascular system. The evidence for the effects on vascular and nonvascular smooth muscle comes from animal studies. Whether these studies can be extrapolated to human vessels remains to be determined. Therefore, the present study was designed to investigate the influence of amitriptyline, nortriptyline and sertraline on the contractile responses of human isolated mesenteric arteries to electrical field stimulation, noradrenaline and potassium chloride. Methods Arterial segments (lumen diameter 0.8–1.2 mm) were obtained from portions of the human omentum during the course of 41 abdominal operat…

Pharmacologymedicine.medical_specialtybusiness.industryVasodilationSmooth muscle contractionMuscle relaxationEndocrinologymedicine.anatomical_structureInternal medicineCirculatory systemmedicinePharmacology (medical)AmitriptylineNortriptylinemedicine.symptombusinessMesenteric arteriesmedicine.drugMuscle contractionBritish Journal of Clinical Pharmacology
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