Search results for "Metaplasia"

showing 10 items of 95 documents

Arrangement of myofibroblastic and smooth muscle-like cells in superficial peritoneal endometriosis and a possible role of transforming growth factor…

2018

Purpose: Superficial peritoneal endometriotic (pEM) lesions are composed of endometrial glands and stroma, in addition to a third component—myofibroblasts and smooth muscles (SM)-like cells. The latter develops secondary to a metaplasia. In this study, we characterised the third component cells in pEM according to differentiation markers in different micro-compartments. Furthermore, a possible effect of TGFβ1 on myofibroblastic metaplasia in endometriotic epithelial cells was studied. Methods: Seventy-six premenopausal patients were included. Peritoneal biopsies were excised from EM patients (n = 23), unaffected peritoneum (peritoneum from EM patients but without EM components, n = 5/23) an…

0301 basic medicineAdultPathologymedicine.medical_specialtyCalponinEndometriosisPeritoneal DiseasesTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineStromaPeritoneumMetaplasiamedicineHumansEndometriosiMyofibroblastsMyofibroblastMetaplasia030219 obstetrics & reproductive medicinebiologybusiness.industryPeritoneal endometriosiPeritoneal fluidTGFβ1Obstetrics and GynecologyCell DifferentiationMuscle SmoothGeneral MedicineTransforming growth factor beta030104 developmental biologymedicine.anatomical_structurePeritoneal Diseasebiology.proteinSmooth muscle-like cellDesminFemalemedicine.symptomPeritoneumbusinessMyofibroblastMyofibroblastic metaplasiaHumanArchives of gynecology and obstetrics
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Hnf4α is a key gene that can generate columnar metaplasia in oesophageal epithelium

2017

AbstractBarrett's metaplasia is the only known morphological precursor to oesophageal adenocarcinoma and is characterized by replacement of stratified squamous epithelium by columnar epithelium. The cell of origin is uncertain and the molecular mechanisms responsible for the change in cellular phenotype are poorly understood. We therefore explored the role of two transcription factors, Cdx2 and HNF4α in the conversion using primary organ cultures. Biopsy samples from cases of human Barrett's metaplasia were analysed for the presence of CDX2 and HNF4α. A new organ culture system for adult murine oesophagus is described. Using this, Cdx2 and HNF4α were ectopically expressed by adenoviral infe…

0301 basic medicineMalePathologyCancer ResearchEsophageal NeoplasmsBiopsyEpitheliumMice0302 clinical medicineMetaplasiaCDX2 Transcription FactorCDX2CàncerOesophageal cancerAnatomyNeoplasm ProteinsBarrett's oesophagusGene Expression Regulation Neoplasticmedicine.anatomical_structureHepatocyte Nuclear Factor 4Loricrin/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_being030211 gastroenterology & hepatologymedicine.symptomVillinHepatocyte nuclear factor 4-alphaAdultmedicine.medical_specialtyStratified squamous epitheliumBiologyAdenocarcinomaOrgan cultureArticle03 medical and health sciencesBarrett EsophagusEsophagusOrgan Culture TechniquesSDG 3 - Good Health and Well-beingmedicineAnimalsHumansMolecular BiologyHNF4αMetaplasiaHistologiaCell BiologyEpitheliumdigestive system diseases030104 developmental biologybiology.proteinEctopic expressionDevelopmental Biology
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Early miR-223 Upregulation in Gastroesophageal Carcinogenesis

2017

Objectives: To test miR-223 upregulation during gastric (intestinal-type) and Barrett esophageal carcinogenesis. Methods: miR-223 expression was assessed by quantitative reverse transcription polymerase chain reaction in a series of 280 gastroesophageal biopsy samples representative of the whole spectrum of phenotypic changes involved in both carcinogenetic cascades. The results were further validated by in situ hybridization on multiple tissue specimens obtained from six surgically treated gastroesophageal adenocarcinomas. miR-223 expression was also assessed in plasma samples from 30 patients with early stage (ie, stages I and II) gastroesophageal adenocarcinoma and relative controls. Res…

0301 basic medicineMalePathologyEsophageal NeoplasmsAtrophic gastritisCarcinogenesisPreneoplastic lesionsBarrett carcinogenesisGastroenterology0302 clinical medicineEarly Detection of CancerIn Situ HybridizationBarrett carcinogenesis; Gastric adenocarcinoma; Preneoplastic lesions; microRNA; Adenocarcinoma; Aged; Barrett Esophagus; Biomarkers Tumor; Carcinogenesis; Early Detection of Cancer; Esophageal Neoplasms; Esophagogastric Junction; Female; Humans; In Situ Hybridization; Male; MicroRNAs; Middle Aged; Retrospective Studies; Reverse Transcriptase Polymerase Chain Reaction; Stomach Neoplasms; Up-RegulationTumormedicine.diagnostic_testmicroRNAReverse Transcriptase Polymerase Chain ReactionBarrett carcinogenesiIntestinal metaplasiaGeneral MedicineMiddle AgedUp-RegulationReverse transcription polymerase chain reactionmedicine.anatomical_structure030220 oncology & carcinogenesisAdenocarcinomaBiomarker (medicine)FemaleEsophagogastric Junctionmedicine.medical_specialty2734BiologyAdenocarcinoma03 medical and health sciencesBarrett EsophagusStomach NeoplasmsInternal medicineBiopsyBiomarkers TumormedicineHumansEsophagusAgedRetrospective StudiesGastric adenocarcinomaCancermedicine.diseasedigestive system diseasesBarrett carcinogenesis; Gastric adenocarcinoma; microRNA; Preneoplastic lesions; Adenocarcinoma; Aged; Barrett Esophagus; Biomarkers Tumor; Carcinogenesis; Early Detection of Cancer; Esophageal Neoplasms; Esophagogastric Junction; Female; Humans; In Situ Hybridization; Male; MicroRNAs; Middle Aged; Retrospective Studies; Reverse Transcriptase Polymerase Chain Reaction; Stomach Neoplasms; Up-Regulation; 2734MicroRNAs030104 developmental biologyBiomarkers
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Hsp60 quantification in human gastric mucosa shows differences between pathologies with various degrees of proliferation and malignancy grade

2021

Background: Stomach diseases are an important sector of gastroenterology, including proliferative benign

0301 basic medicineTechnologyPathologymedicine.medical_specialtyQH301-705.5QC1-999intestinal metaplasia03 medical and health sciences0302 clinical medicinechaperone systemMetaplasiamedicineGastric mucosaGeneral Materials ScienceBiology (General)QD1-999Instrumentationhealth care economics and organizationsFluid Flow and Transfer Processesbusiness.industryChaperone system Gastric carcinogenesis Gastric dysplasia Gastritis Hsp60 Intestinal metaplasiaSettore BIO/16 - Anatomia UmanaTPhysicsProcess Chemistry and TechnologyStomachdigestive oral and skin physiologygastritisGeneral EngineeringIntestinal metaplasiagastric dysplasiaEngineering (General). Civil engineering (General)Hsp60medicine.diseasedigestive system diseasesComputer Science ApplicationsChemistryGastric Dysplasia030104 developmental biologymedicine.anatomical_structureHyperplastic PolypDysplasia030220 oncology & carcinogenesisgastric carcinogenesisTA1-2040medicine.symptomGastritisbusiness
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Third International Congress of Histochemistry and Cytochemistry

1968

The tissue distribution of enzymatic activities in intestinal metaplasia stomachs exhibiting chronic gastritis was compared histochemically with that of the small intestine in man.

0303 health sciencesPathologymedicine.medical_specialtybiology030302 biochemistry & molecular biologyAcridine orangeAcid phosphataseChronic gastritisIntestinal metaplasiamedicine.diseaseSmall intestine03 medical and health scienceschemistry.chemical_compoundmedicine.anatomical_structurechemistryInternational congressmedicineCytochemistrybiology.proteinImmunohistochemistry030304 developmental biology
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Different Pattern of Inflammatory and Atrophic Changes in the Gastric Mucosa of the Greater and Lesser Curvature.

2015


 
 
 
 
 
 Background & Aims: Appropriate biopsy sampling is important for the classification of gastritis, yet the extent of inflammation and atrophy of different regions of the stomach with chronic gastritis have been addressed only in a few studies. The aim of our study was to analyze the inflammatory, atrophic and metaplastic changes in the greater and lesser curvature of the antrum and corpus mucosa.
 Methods: 420 patients undergoing upper endoscopy were enrolled in the study. Four expert gastrointestinal pathologists graded biopsy specimens according to the updated Sydney classification.
 Results: The obtained results showed that the mononu…

AdultGastritis AtrophicMalePathologymedicine.medical_specialtyBiopsyChronic gastritisSeverity of Illness IndexAtrophyPredictive Value of TestsBiopsymedicinePyloric AntrumHumansProspective StudiesAntrumAgedObserver VariationMetaplasiamedicine.diagnostic_testHelicobacter pyloribusiness.industryStomachGastroenterologyIntestinal metaplasiaReproducibility of ResultsAnatomyMiddle Agedmedicine.diseaseCurvatures of the stomachmedicine.anatomical_structureGastric MucosaFemaleGastritismedicine.symptombusinessGranulocytesJournal of gastrointestinal and liver diseases : JGLD
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The phenotype of gastric mucosa coexisting with Barrett's oesophagus.

2001

Barrett's oesophagus complicates the gastro-oesophageal acid reflux. Helicobacter pylori infection, particularly with cagA positive strains, induces inflammatory/atrophic lesions of the gastric mucosa, which may impair acid output. No systematic study has investigated the phenotype of the gastric mucosa coexisting with Barrett's oesophagus. This study was designed to identify the phenotype of gastric mucosa associated with Barrett's oesophagus.In this retrospective case control study, the phenotype of the gastric mucosa was histologically characterised in 53 consecutive patients with Barrett's oesophagus and in 53 (sex and age matched) non-ulcer dyspeptic controls. Both patients and control…

AdultGastritis AtrophicMalemedicine.medical_specialtyPathologyAtrophic gastritisBiopsySpirillaceaeBarrett's oesophagus gastritis in Barrett's oesophagus Barrett's oesophagus and gastric precancerous lesionsdigestive systemGastroenterologyHelicobacter InfectionsPathology and Forensic MedicineBarrett's oesophagus and gastric precancerous lesionsBarrett EsophagusInternal medicineotorhinolaryngologic diseasesGastric mucosamedicineHumansCagAAgedRetrospective StudiesAged 80 and overHelicobacter pyloribiologybusiness.industrygastritis in Barrett's oesophagusStomachdigestive oral and skin physiologyIntestinal metaplasiaGeneral MedicineMiddle AgedHelicobacter pyloribiology.organism_classificationmedicine.diseasedigestive system diseasesBarrett's oesophagussurgical procedures operativePhenotypemedicine.anatomical_structureGastric MucosaCase-Control StudiesPapersFemaleGastritismedicine.symptombusiness
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Hiatal hernia, gastro-oesophageal reflux and oesophagitis: videofluorographic, endoscopic and histopathological correlation

2004

The aim of the study was to evaluate the correlation between hiatal hernia and gastro-oesophageal reflux and related histological abnormalities in patients without endoscopic oesophagitis. A consecutive series of 78 patients with a history of gastro-oesophageal reflux symptoms and hiatal hernia, as defined by videofluorography combined with a water siphon test, underwent oesophagogastroduodenoscopy and multiple biopsies. Hiatal hernia was confirmed endoscopically in 99% of cases. The water siphon test was positive for reflux in 72% of cases. At endoscopy 42% of patients had oesophagitis and/or Barrett's oesophagus and 58% had no lesions. In the group without endoscopic lesions, at histology…

AdultMaleEsophagitis diagnosiMetaplasiaHerniaBiopsyCineradiographyVideo RecordingWaterMiddle AgedHiatal diagnosisEndoscopy GastrointestinalDiagnosis DifferentialBarrett EsophagusEsophagusHernia HiatalFluoroscopyGastroesophageal RefluxEsophagitisHumansFemaleEsophagoscopyGastroesophageal Reflux diagnosiAged
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Proliferative Activity and Cytometric Characteristics in Polyps of the Nasal Cavity and Paranasal Sinuses

2000

Although several investigations have revealed the influence of cytokines, allergy, and environmental factors in polyp development, the etiology of nasal polyps is still unknown. To estimate the biology of this common disease the operative specimens of 50 patients who underwent surgery for polyps of the nasal cavity and the paranasal sinuses were examined; of these, 10 patients had recurrent disease and 23 patients had an allergy. The investigations included routine histology and quantitative DNA measurements, along with immunohistochemical identification of proliferation markers (i.e., MIB-1; proliferating cell nuclear antigen, PCNA). Histologically, most polyps revealed an infiltration wi…

AdultMaleNasal cavityParanasal Sinus NeoplasmPathologymedicine.medical_specialtyAdolescentAneuploidy03 medical and health sciencesNasal PolypsPolyps0302 clinical medicineProliferating Cell Nuclear Antigenotorhinolaryngologic diseasesmedicineHumansNasal polypsChild030223 otorhinolaryngologyAgedPloidiesbiologybusiness.industryHistologyDNA NeoplasmMiddle Agedmedicine.diseasedigestive system diseasesSquamous metaplasiaProliferating cell nuclear antigenmedicine.anatomical_structureParanasal sinusesOtorhinolaryngology030220 oncology & carcinogenesisbiology.proteinFemalebusinessCell DivisionParanasal Sinus NeoplasmsAmerican Journal of Rhinology
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Changes in cytokeratin expression accompany squamous metaplasia of the human respiratory epithelium.

1992

To determine the characteristics of metaplastic changes of the nasal respiratory epithelium, the distribution of individual cytokeratins (CKs) was studied immunohistochemically and by two-dimensional gel electrophoresis. The authors define four types of changes of the normal pseudostratified columnar epithelium: (1) transitional pseudostratified epithelium (first unusual CK.: no. 13); (2) stratified columnar epithelium (increased expression of CKs 4 and 13; CKs 7, 8, 18 and 19 reduced); (3) stratified squamous epithelium, non-keratinized (appearance of CK 16); and (4) stratified squamous epithelium, keratinized (expression of CKs 1 and 10, variable CK5 and 14 patterns in basal cells). These…

AdultMalePathologymedicine.medical_specialtyAdolescentStratified squamous epitheliumBiologyEpitheliumPathology and Forensic MedicineCytokeratinMetaplasiamedicineHumansChildMolecular BiologyMetaplasiaCell BiologyGeneral MedicineMiddle Agedmedicine.diseaseSquamous metaplasiaEpitheliumNasal Mucosamedicine.anatomical_structureStratified columnar epitheliumRespiratory epitheliumKeratinsFemalemedicine.symptomPseudostratified columnar epitheliumVirchows Archiv. A, Pathological anatomy and histopathology
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