Search results for "Microcephaly"

showing 10 items of 70 documents

A two base pair deletion in the PQBP1 gene is associated with microphthalmia, microcephaly, and mental retardation.

2007

X-linked mental retardation has been traditionally divided into syndromic (S-XLMR) and non-syndromic forms (NS-XLMR), although the borderlines between these phenotypes begin to vanish and mutations in a single gene, for example PQBP1, can cause S-XLMR as well as NS-XLMR. Here, we report two maternal cousins with an apparently X-linked phenotype of mental retardation (MR), microphthalmia, choroid coloboma, microcephaly, renal hypoplasia, and spastic paraplegia. By multipoint linkage analysis with markers spanning the entire X-chromosome we mapped the disease locus to a 28-Mb interval between Xp11.4 and Xq12, including the BCOR gene. A missense mutation in BCOR was described in a family with …

AdultMaleMicrocephalycongenital hereditary and neonatal diseases and abnormalitiesGermline mosaicismLocus (genetics)BiologyMicrophthalmiaFrameshift mutationGenetic linkageGenes X-LinkedIntellectual DisabilityGeneticsmedicineMissense mutationHumansMicrophthalmosAbnormalities MultipleFrameshift MutationGenetics (clinical)GeneticsChromosomes Human XNuclear ProteinsGenetic Diseases X-LinkedSyndromemedicine.diseasePedigreeLenz microphthalmia syndromeDNA-Binding ProteinsChild PreschoolMicrocephalyFemaleCarrier ProteinsGene DeletionEuropean journal of human genetics : EJHG
researchProduct

Two patients with EP300 mutations and facial dysmorphism different from the classic Rubinstein-Taybi syndrome

2009

Rubinstein-Taybi syndrome (RTS) is characterized by mental retardation, broad thumbs and great toes and a recognizable craniofacial phenotype. Causative mutations have been described in the CREBBP and EP300 genes. Here we present a 19-year-old woman and an unrelated 3-year-old boy, both with broad thumbs and halluces, but with facial aspects distinct from those of typical RTS. The woman had a marked learning disability, but no mental retardation. We identified a de novo c.7100delC mutation in EP300 (which predicts p.P2366RfsX35) in the woman and an apparently de novo c.638delG mutation in the boy, which predicts p.G213EfsX6. Mutations in EP300 are a known but rare cause of RTS. Only five ot…

AdultMaleMicrocephalymedicine.medical_specialtyMedizinmedicine.disease_causeRetrognathiaGeneticsmedicineHumansCraniofacialEP300Genetics (clinical)Rubinstein-Taybi SyndromeGeneticsMutationRubinstein–Taybi syndromebusiness.industrymedicine.diseasePhenotypeDermatologyPalpebral fissureChild PreschoolMutationFemalebusinessE1A-Associated p300 Protein
researchProduct

Prenatal head growth and child neuropsychological development at age 14 months

2014

We sought to assess the association between prenatal head growth and child neuropsychological development in the general population.We evaluated 2104 children at the age of 14 months from a population-based birth cohort in Spain. Head circumference (HC) was measured by ultrasound examinations at weeks 12, 20, and 34 of gestation and by a nurse at birth. Head growth was assessed using conditional SD scores between weeks 12-20 and 20-34. Trained psychologists assessed neuropsychological functioning using the Bayley Scales of Infant Development. Head size measurements at birth were transformed into a 3-category variable: microcephalic (10th percentile), normocephalic (≥10th and90th percentile)…

AdultMalePediatricsmedicine.medical_specialtyPopulationBayley Scales of Infant DevelopmentUltrasonography PrenatalCohort StudiesChild DevelopmentCognitionPregnancymedicineHumanseducationPsychomotor learningPregnancyeducation.field_of_studybusiness.industryInfant NewbornInfantObstetrics and GynecologyOrgan Sizemedicine.diseaseChild developmentMegalencephalyConfidence intervalMicrocephalyGestationFemalebusinessHeadCohort studyAmerican Journal of Obstetrics and Gynecology
researchProduct

Congenital neutropenia with retinopathy, a new phenotype without intellectual deficiency or obesity secondary toVPS13Bmutations

2013

Over one hundred VPS13B mutations are reported in Cohen syndrome (CS). Most cases exhibit a homogeneous phenotype that includes intellectual deficiency (ID), microcephaly, facial dysmorphism, slender extremities, truncal obesity, progressive chorioretinal dystrophy, and neutropenia. We report on a patient carrying two VPS13B splicing mutations with an atypical phenotype that included microcephaly, retinopathy, and congenital neutropenia, but neither obesity nor ID. RNA analysis of the IVS34+2T_+3AinsT mutation did not reveal any abnormal splice fragments but mRNA quantification showed a significant decrease in VPS13B expression. RNA sequencing analysis up- and downstream from the IVS57+2T>C…

AdultPathologymedicine.medical_specialtyMicrocephalyNeutropeniaDNA Mutational AnalysisVesicular Transport ProteinsNeutropeniamedicine.disease_causeRetinal DiseasesIntellectual DisabilityGene OrderGeneticsmedicineCongenital Bone Marrow Failure SyndromesHumansObesityCongenital NeutropeniaGenetics (clinical)GeneticsMutationCohen syndromebusiness.industryFaciesSyndromemedicine.diseasePhenotypePedigreeVPS13BPhenotypeMutationFemalebusinessRetinopathyAmerican Journal of Medical Genetics Part A
researchProduct

Congenital hypertrophy of the retinal pigment epithelium and familial polyposis of the colon.

1987

Inquiries to Manuel Diaz Llopis, M.D., Cl Cirilo Amoros No. 1 PTA 2, Valencia 46004, Spain. Familial polyposis of the colon is a dominant autosomal disorder that is characterized by hundreds and sometimes thousands of adenomatous polyps throughout the entire colon, which begin in adolescence. Virtually all pa­ tients with familial polyposis develop carcino­ ma of the colon by age 50 years. A total colectomy should be carried out early in adult life in affected persons. Because of the autosomal dominant inheritance pattern, an intensive sur­ vey of family members must be conducted. There is no phenotype biochemical abnormality or serologic marker that indicates whether a familial member will…

AdultPathologymedicine.medical_specialtyMicrocephalyRetinal pigment epitheliumEpidermal Cystbusiness.industryDiseaseHypertrophymedicine.diseaseMuscle hypertrophySerologyOphthalmologyCongenital hypertrophymedicine.anatomical_structureAdenomatous Polyposis ColiRetinal DiseasesmedicineHumansAbnormalityFluorescein AngiographybusinessPigment Epithelium of EyeAmerican journal of ophthalmology
researchProduct

Pathogenic correlation between mosaic variegated aneuploidy 1 (MVA1) and a novel BUB1B variant: a reappraisal of a severe syndrome.

2022

Funder: Università degli Studi di Catania

BUB1B gene Epileptic seizure Microcephaly Mosaic variegated aneuploidy 1 (MVA1) syndrome Ovary cystMosaicismCell Cycle ProteinsOvary cystDermatologyGeneral MedicineSyndromeBUB1B geneProtein Serine-Threonine KinasesAneuploidyPsychiatry and Mental healthSeizuresMosaic variegated aneuploidy 1 (MVA1) syndromeMutationEpileptic seizureMicrocephalyHumansNeurology (clinical)Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
researchProduct

Chondrodysplasia punctata — Rhizomelic form

1976

Pathologic, ultrastructural and radiologic studies are described on 3 infants with the rhizomelic form of chondrodysplasia punctata. Radiologic criteria in the young infant include radiolucent coronal clefts dividing all or most of the thoracic and lumbar vertebral bodies, short humeri with flared metaphyses and punctate calcifications commonly present adjacent to the ossified ischial and pubic bones and less commonly in other locations. In late infancy and childhood the radiologic criteria include demineralization in all bones with slow maturation, flat vertebral bodies, short humeri and femora, metaphyseal flaring, especially in the distal humerus, proximal femur and proximal tibia, immat…

Cartilage ArticularMalemusculoskeletal diseasesChondrodysplasia PunctataPathologymedicine.medical_specialtyContractureDegeneration (medical)Skin DiseasesBone and BonesCataractInfant Newborn DiseasesLumbarmedicineHumansChondrodysplasia punctataFemurChildPelvisCerebral CortexNeuronsbusiness.industryCartilageMetaphyseal flaringInfant NewbornInfantPatellaAnatomyHumerusCystic Changemedicine.diseaseCartilagemedicine.anatomical_structureChild PreschoolPediatrics Perinatology and Child HealthMicrocephalyFemalePsychomotor DisordersbusinessCancellous boneEuropean Journal of Pediatrics
researchProduct

Microcephaly and macrocephaly. A study on anthropometric and clinical data from 308 subjects

2019

Head circumference is the auxological parameter that most correlates with developmental anomalies in childhood. Head circumference (HC) two standard deviations (SD) below or above the mean defines microcephaly and macrocephaly, respectively. The aim of this retrospective study was to explore anthropometric parameters and clinical characteristics among subjects with abnormalities in HC who had been referred for developmental assessment. One hundred and sixty four subjects with microcephaly and 144 subjects with macrocephaly were enrolled from birth to 18 months of age. Head circumference at birth and the association with variables related to maternal health status, gestational age, growth pa…

Developmental delayMicrocephalyMacrocephalySyndromeMalformationMegalencephaly
researchProduct

Dysmorphic face, precociously senile appearance, microcephaly, growth retardation and neuropsychomotor delay

2005

Cockayne's syndrome is a rare recessive autosomal disorder. Its frequency at birth is very low. In the classical form (type 1) the clinical appearance becomes evident by the second year of life. Itis characterized by severe growth delay, microcephaly, progressive loss of adipose tissue, senile appearance, dysmorphic facial features, ocular anomalies (retinal pigmentary degeneration, nistagmus, cataract, optic atrophy).

Dysmorphic facegrowth retardationmicrocephaly
researchProduct

Genome-wide mapping of copy number variations in patients with both anorectal malformations and central nervous system abnormalities

2014

Background: Anorectal malformations (ARM) have a prevalence of around 1 in 2500 live births. In around 50% of patients, the malformation is isolated, while in the remainder it arises within the context of complex genetic abnormalities or a defined genetic syndrome. Recent studies have implicated rare copy number variations (CNVs) in both isolated and nonisolated ARM, and identified plausible candidate genes. Methods: In the present study, array-based molecular karyotyping was performed to identify causative CNVs in 32 sporadic ARM patients with comorbid abnormalities of the central nervous system (CNS). This phenotype was selected to enrich for rare CNVs, since previous research has implica…

EmbryologyCandidate genePathologymedicine.medical_specialtyMicrocephalyHaplotypeContext (language use)General MedicineBiologymedicine.diseaseBioinformaticsPhenotypeContiguous gene syndromePediatrics Perinatology and Child HealthmedicineCopy-number variationDevelopmental BiologySNP arrayBirth Defects Research Part A: Clinical and Molecular Teratology
researchProduct