Search results for "MitoP"

showing 10 items of 27 documents

Autophagy as a rescue mechanism in efavirenz-induced mitochondrial dysfunction: a lesson from hepatic cells.

2011

Efavirenz (EFV) is the most widely used non-nucleoside reverse transcriptase inhibitor applied in highly active antiretroviral therapy (HAART), the combined pharmacological treatment of the human immunodeficiency virus infection. Its use has been associated with the development of several adverse events including hepatotoxicity. The molecular pathogenesis of this effect is poorly understood but recent reports have highlighted features of mitochondrial dysfunction in hepatic cells exposed to clinically relevant concentrations of EFV. In this study, we investigated the activation of autophagy and, in particular, mitophagy, in human hepatic cells exposed to EFV. We detected the presence of alt…

CyclopropanesEfavirenzCell SurvivalMitochondrionBiologyModels Biologicalchemistry.chemical_compoundMitophagymedicineAutophagyHumansMolecular BiologyReverse-transcriptase inhibitorAutophagyCell BiologyBenzoxazinesMitochondriachemistryApoptosisAlkynesImmunologyCancer researchHepatic stellate cellHepatocytesReverse Transcriptase InhibitorsHomeostasismedicine.drugAutophagy
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Compromising mitochondrial function with the antiretroviral drug efavirenz induces cell survival-promoting autophagy

2011

Hepatotoxicity is a very common side effect associated with the pharmacological treatment of human immunodeficiency virus (HIV) infection and its pathogenesis is poorly understood. Efavirenz (EFV) is the most widely used nonnucleoside reverse transcriptase inhibitor administered for the control of HIV and some of its toxic effects in hepatic cells have been recently shown to display features of mitochondrial dysfunction. Here we studied the activation of autophagy and, in particular, mitophagy, the main mitochondrial turnover mechanism, in human hepatic cells treated with clinically relevant concentrations of this drug. EFV-treated cells had altered mitochondria, characterized by a relative…

CyclopropanesEfavirenzHepatologyAnti-HIV AgentsCell SurvivalMitochondrial TurnoverAutophagyVacuoleMitochondrionBiologyBenzoxazinesMitochondriaCell biologychemistry.chemical_compoundchemistryApoptosisAlkynesMitophagyAutophagyCancer researchHepatic stellate cellHumansChemical and Drug Induced Liver InjuryHeLa CellsHepatology
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Archetipolatria e summa iconologica

2015

Simboli ed attributi intesi quali vulgata iconografica delle virtù della città di Palermo, posti a corredo del suo nume tutelare, secondo il distico strofico epigrafico del gruppo scultoreo del Marabitti, nella mitopoietica Flora Julia, assurgono, insieme ai loro significati opposti, a chiave di interpretazione ermeneutica dell'opera di Enzo Venezia. L’applicazione di attente e ricercate strutture compositive, e l'abilità (da architetto) di "architetturare" la visione e, soprattutto, la rappresentazione delle cose, genera la cura per l'insieme e per il particolare verboso e magniloquente, in grado di divenire sostanza principale e tema conduttore di ogni struttura narrativa; un registro est…

Enzo Venezia mitopoietica Palermo Genius lociSettore ICAR/14 - Composizione Architettonica E Urbana
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Mitophagy in myeloid cells : role in infection with gram-negative bacteria

2017

Sepsis and related organ dysfunctions remain a leading cause of mortality in intensive care units. Increasing evidences have shed light on an unexpected link between mitochondria and immune cell functions. Alterations in mitochondrial functions have been reported in peripheral blood cells in sepsis. We hypothesize here that mitophagy might impact on phagocyte functions in the context of bacterial infection. Mitophagy is a mitochondria-dedicated autophagy that governs the elimination of dysfunctional mitochondria. We demonstrated here in vivo and in vitro that macrophages exposed to Gram-negative bacteria or their cell wall component LPS display a marked inhibition of mitophagy that constitu…

LipopolysaccharidesInflammation[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyMacrophagesSepsisMyeloid cellsMitophagyCellules myeloidesMitophagie[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Mitochondrial oxidative injury: a key player in nonalcoholic fatty liver disease.

2020

Nonalcoholic fatty liver disease (NAFLD) has become the most prevalent liver disease worldwide. NAFLD is tightly linked to the metabolic syndrome, insulin resistance, and oxidative stress. Globally, its inflammatory form, nonalcoholic steatohepatitis (NASH), has become the main cause of liver-related morbidity and mortality, mainly due to liver cirrhosis and primary liver cancer. One hallmark of NASH is the presence of changes in mitochondrial morphology and function that are accompanied by a blocked flow of electrons in the respiratory chain, which increases formation of mitochondrial reactive oxygen species in a self-perpetuating vicious cycle. Consequences are oxidation of DNA bases and …

Liver Cirrhosismedicine.medical_specialtyMitochondrial DNAMitochondrial DiseasesPhysiologyMitochondrial TurnoverRespiratory chainBiologymedicine.disease_causedigestive system03 medical and health sciences0302 clinical medicineNon-alcoholic Fatty Liver DiseasePhysiology (medical)Internal medicineMitochondrial unfolded protein responseMitophagyNonalcoholic fatty liver diseasemedicineAnimalsHumans030304 developmental biology0303 health sciencesHepatologyGastroenterologynutritional and metabolic diseasesmedicine.diseasedigestive system diseases3. Good healthFatty LiverOxidative StressEndocrinologyMitochondrial biogenesis030211 gastroenterology & hepatologyOxidative stressAmerican journal of physiology. Gastrointestinal and liver physiology
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Altered insulin pathway compromises mitochondrial function and quality control both in in vitro and in vivo model systems.

2021

Abstract Altered insulin signaling and insulin resistance are considered the link between Alzheimer's disease (AD) and metabolic syndrome. Here, by using an in vitro and an in vivo model, we investigated the relationship between these disorders focusing on neuronal mitochondrial dysfunction and mitophagy. In vitro Aβ insult induced the opening of mitochondrial permeability transition pore (mPTP), mitochondrial membrane potential (ΔΨm) loss, and apoptosis while insulin addition ameliorated these dysfunctions. The same alterations were detected in a 16 weeks of age mouse model of diet-induced obesity and insulin resistance. In addition, we detected an increase of fission related proteins and …

MaleAgingAmyloid beta-Peptidemedicine.medical_treatmentMetabolic diseasePINK1Insulin pathway Neurodegeneration Metabolic diseases Mitochondrion Mitophagy AgingMitochondrionDiet High-FatParkinNOMiceInsulin resistanceMetabolic DiseasesCell Line TumorMitophagymedicineAnimalsHumansInsulinMitochondrionNeurodegenerationMolecular BiologyAmyloid beta-PeptidesbiologyAnimalChemistryInsulinMitophagyCell Biologymedicine.diseaseCell biologyMitochondriaMice Inbred C57BLInsulin receptorMitochondrial permeability transition porebiology.proteinMolecular MedicineInsulin ResistanceInsulin pathwayHumanSignal TransductionMitochondrion
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Mitochondrial dynamics in type 2 diabetes: Pathophysiological implications

2017

Mitochondria play a key role in maintaining cellular metabolic homeostasis. These organelles have a high plasticity and are involved in dynamic processes such as mitochondrial fusion and fission, mitophagy and mitochondrial biogenesis. Type 2 diabetes is characterised by mitochondrial dysfunction, high production of reactive oxygen species (ROS) and low levels of ATP. Mitochondrial fusion is modulated by different proteins, including mitofusin-1 (MFN1), mitofusin-2 (MFN2) and optic atrophy (OPA-1), while fission is controlled by mitochondrial fission 1 (FIS1), dynamin-related protein 1 (DRP1) and mitochondrial fission factor (MFF). PARKIN and (PTEN)-induced putative kinase 1 (PINK1) partici…

MiD51 mitochondrial dynamics proteins of 51 kDaΔΨm mitochondrial membrane potential0301 basic medicineMitochondrial fission factorClinical BiochemistryMitochondrial DegradationMFN2Review ArticleTXNIP thioredoxin interacting proteinMitochondrial DynamicsBiochemistryAdenosine TriphosphateGRP78 78 kDa glucose-regulated proteinMFF mitochondrial fission factorMFN2 mitofusin 2TRX2 thioredoxin 2Redox biologylcsh:QH301-705.5NF-κB nuclear factor kappa Blcsh:R5-920MitophagyType 2 diabetesDRP1 dynamin-related protein 1FIS1 fission protein 1BNIP3 BCL2/adenovirus E1B 19 kDa interacting protein 3MitochondriaOPA1 optic atrophy 1SIRT1/3 sirtuin 1/3Biochemistrymitochondrial fusionTGF-β1 transforming growth factor-β1Mitochondrial fissionOMM outer mitochondrial membranelcsh:Medicine (General)MiD49 mitochondrial dynamics proteins of 49Nox 4 NADPH oxidase-4IMM inner mitochondrial membraneFIS1ATF6 activating transcription factor 6PINK1mTOR mammalian target of rapamycinCHOP C/EBP homologous proteinBiologymdivi-1 mitochondrial division inhibitor-1Mitochondrial Proteins03 medical and health sciencesROS reactive oxygen speciessXBP1 spliced X-box binding protein 1UCP-1 uncoupling protein-1MFN1 mitofusin 1SOD superoxide dismutaseLC3 1 A/1B-light chain 3HumansPINK1 (PTEN)-induced putative kinase 1S3 15-OxospiramilactoneOrganic ChemistrymtDNA mitochondrial DNAAMPK AMP-activated protein kinase030104 developmental biologyDiabetes Mellitus Type 2Mitochondrial biogenesislcsh:Biology (General)Oxidative stressp38 MAPK p38 mitogen-activated protein kinasep62/SQSTM1 ubiquitin and sequestosome-1Reactive Oxygen SpeciesRedox Biology
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Mitochondrial dysfunction and host immune response during pneumococcal pneumonia : impact of mechanical ventilation?

2020

Lung damage induced by mechanical ventilation (MV) worsens the prognosis of pneumonia. Mitochondrial dysfunctions could account for such deleterious effects. Accordingly, data obtained from both in vitro and in vivo experimental studies, as well as in ventilated patients with acute respiratory distress syndrome, show that mitochondrial alarmins, especially mitochondrial DNA, are released in the alveolar compartment as a result of the cyclic stretch, and could thus represent one link between the mechanical insult and the sterile inflammation leading to VILI (ventilator-induced lung injury). In rabbits, we show herein that adverse and prolonged MV worsens the prognosis of pneumococcal pneumon…

Pneumopathie[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyStreptococcus pneumoniaeMechanical ventilationBiogénèse mitochondrialeMitochondrial biogenesisVentilation mécaniqueMitophagyDysfonction mitochondrialePneumoniaMitochondrial dysfunctionMitophagie[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Mediterraneo: mito, paisajes y arquitecturas silenciosas

2019

Il Mediterraneo è certamente l’ambito geografico compiuto e definito che, più di ogni altro, ha visto nascere, sviluppare ed evolvere le civiltà in grado di segnare la storia dell’Uomo. Punto di confronto tra oriente ed occidente, l’identità e la riconoscibilità del mito mediterraneo sta proprio nella molteplicità e nelle diversità delle culture e dei popoli che nel “bacino” si affacciano. L’articolo individua alcuni degli aspetti attraverso i quali è possibile leggere l’influenza del mito mediterraneo nell’architettura contemporanea. Aspetti da intendersi quale monumentum nel senso del valore del monere, del ricordare l’inesauribile concettualizzazione del mythos la cui attualità sta propr…

Settore ICAR/14 - Composizione Architettonica E UrbanaMediterráneo mito paisajes arquitectura Le Corbusier Casa Malaparte en Capri monumento.Mediterraneo mito mitopoiesi paesaggi architettura Le Corbusier Casa Malaparte a Capri monumento.Mediterranean area myth landscape architecture Le Corbusier Villa Malaparte at Capri monument.
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Figure reali e fantastiche nel primo Spielmannsepos della tradizione letteraria tedesca, il König Rother

2013

Settore L-FIL-LET/15 - Filologia GermanicaLetteratura tedesca mitopoiesi germanica
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