Search results for "Mitochondria"

showing 10 items of 1306 documents

Comparison of telomere length between population-specific mitochondrial haplogroups among different age groups in a Latvian population

2014

Population studies have demonstrated that telomere length (TL) displays great diversity among different populations. Previously described controversial findings associated longevity with specific mitochondrial DNA haplogroups (hgs) (e.g., J and U). These observations may be influenced by population diversity, geographic location, and/or specific historic background. The aims of this study were to identify a specific hg which correlates with aging in a Latvian populating and to evaluate the possible association of TL variability with specific mitochondrial hgs. The results show no significant correlation between TL, mitochondrial DNA hgs and longevity. A slight increase in frequency was obse…

AdultMaleAgingMitochondrial DNAmedia_common.quotation_subjectLongevityPopulationBiologyDNA MitochondrialHaplogroupTelomere HomeostasisHumanseducationAgedmedia_commonAged 80 and overGeneticseducation.field_of_studyHaplotypeLongevityTelomere HomeostasisMiddle AgedTelomereLatviaTelomereHaplotypesFemaleDevelopmental BiologyHuman mitochondrial DNA haplogroupMechanisms of Ageing and Development
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PGC-1α Induction in Pulmonary Arterial Hypertension

2012

Idiopathic Pulmonary arterial hypertension (IPAH) is characterized by the obstructive remodelling of pulmonary arteries, and a progressive elevation in pulmonary arterial pressure (PAP) with subsequent right-sided heart failure and dead. Hypoxia induces the expression of peroxisome proliferator activated receptorγcoactivator-1α(PGC-1α) which regulates oxidative metabolism and mitochondrial biogenesis. We have analysed the expression of PGC-1α, cytochrome C (CYTC), superoxide dismutase (SOD), the total antioxidant status (TAS) and the activity of glutathione peroxidase (GPX) in blood samples of IPAH patients. Expression of PGC-1αwas detected in IPAH patients but not in healthy volunteers. Th…

AdultMaleAgingmedicine.medical_specialtyArticle SubjectHypertension PulmonaryPeroxisome proliferator-activated receptorBiologyBiochemistrySuperoxide dismutaseChloridesInternal medicinemedicineHumansFamilial Primary Pulmonary Hypertensionlcsh:QH573-671Heat-Shock ProteinsAgedchemistry.chemical_classificationGlutathione Peroxidaselcsh:CytologySuperoxide DismutaseGlutathione peroxidaseAge FactorsCytochromes cCell BiologyGeneral MedicineHypoxia (medical)Middle Agedmedicine.diseasePulmonary hypertensionPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaEndocrinologymedicine.anatomical_structurechemistryMitochondrial biogenesisHeart failurebiology.proteinVascular resistanceFemaleVascular Resistancemedicine.symptomTranscription FactorsResearch ArticleOxidative Medicine and Cellular Longevity
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Overcoming a “Probable” Diagnosis in Antimitochondrial Antibody Negative Primary Biliary Cirrhosis: Study of 100 Sera and Review of the Literature

2010

Serum anti-mitochondrial antibodies (AMA) are the serological hallmark of primary biliary cirrhosis (PBC), yet up to 15% of PBC sera are AMA negative at routine indirect immunofluorescence (IIF) while being referred to as “probable” cases. The diagnostic role of PBC-specific antinuclear antibodies (ANA) remains to be determined. We will report herein data on the accuracy of new laboratory tools for AMA and PBC-specific ANA in a large series of PBC sera that were AMA-negative at IIF. We will also provide a discussion of the history and current status of AMA detection methods. We included IIF AMA-negative PBC sera (n = 100) and sera from patients with other chronic liver diseases (n = 104) th…

AdultMaleAnti-nuclear antibodyNuclear dotsPBCSerologyAnticorpo antimitocondrio cirrosi biliare primitivaPrimary biliary cirrhosisAntigenparasitic diseasesHumansImmunology and AllergyMedicineskin and connective tissue diseasesAgedAutoantibodiesAged 80 and overbiologyLiver Cirrhosis Biliarybusiness.industryAutoantibodyIIfGeneral MedicineMiddle Agedmedicine.diseasedigestive system diseasesMitochondriaImmunologybiology.proteinFemaleAntibodybusinessClinical Reviews in Allergy & Immunology
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Antioxidant Activities and Oxidative Stress Byproducts in Human Hypertension

2003

The objective was to study oxidative status, antioxidant activities, and reactive oxygen species byproducts in whole blood and mononuclear peripherals cells and their relationship with blood pressure. Sixty-six hypertensive patients and 16 normotensive volunteers as a control group were studied. In both, whole blood and peripheral mononuclear cells oxidized/reduced glutathione ratio and malondialdehyde was significantly higher, and the activity of superoxide dismutase, catalase, and glutathione peroxidase was significantly lower in hypertensive patients when compared with normal subjects. The content of damaged base 8-oxo-2′-deoxyguanosine in nuclear and mitochondrial deoxyribonucleoprotein…

AdultMaleAntioxidantmedicine.medical_treatmentBlood PressureOxidative phosphorylationPharmacologymedicine.disease_causeDNA MitochondrialAntioxidantsMalondialdehydeInternal MedicinemedicineHumansWhole bloodchemistry.chemical_classificationGlutathione PeroxidaseReactive oxygen speciesGlutathione DisulfideSuperoxide DismutaseChemistryDeoxyguanosine8-Hydroxy-2'-deoxyguanosineDNAMetabolismMiddle AgedCatalaseGlutathioneDNA metabolismOxidative StressBiochemistry8-Hydroxy-2'-DeoxyguanosineHypertensionFemaleReactive Oxygen SpeciesOxidative stressHypertension
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A comprehensive untargeted metabonomic analysis of human steatotic liver tissue by RP and HILIC chromatography coupled to mass spectrometry reveals i…

2011

Steatosis, or excessive accumulation of lipids in the liver, is a generally accepted previous step to the development of more severe conditions like nonalcoholic steatohepatitis, fibrosis, and cirrhosis. We aimed, to characterize the metabolic profile that defines simple steatosis in human tissue and to identify potential disturbances in the hepatic metabolism that could favor the switch to progressive liver damage. A total of 46 samples, 23 from steatotic and 23 from nonsteatotic human livers, were analyzed following a holistic LC-MS-based metabonomic analysis that combines RP and HILIC chromatographic separations. Multivariate statistical data analysis satisfactorily classified samples an…

AdultMaleCirrhosismedicine.drug_classBiochemistryAntioxidantsMass SpectrometryBile Acids and SaltsFibrosismedicineHumansMetabolomicsPhospholipidsAgedChromatographyBile acidChemistryGene Expression ProfilingFatty liverLipid metabolismGeneral ChemistryMiddle Agedmedicine.diseaseLipid MetabolismMitochondriaGlutamineFatty LiverBiochemistryGene Expression RegulationLiverSolventsFemaleSteatosisDrug metabolismBiomarkersChromatography LiquidJournal of proteome research
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Oxidative stress markers at birth: Analyses of a neonatal population

2015

In order to further understand neonatal stress and, thus, control it efficaciously, there is a need for more information on the manifestations of stress at the molecular level in the newborn, with particular regard to oxidants, and anti-oxidant and anti-stress mechanisms, including mitochondrial heat shock protein-chaperones such as Hsp60. We investigated patterns of anti-oxidants, biomarkers of oxidative stress, and Hsp60 levels in sera from newborns and found significant associations between glutathione (GSH) levels and gestational age, delivery modality, and lipid hydroperoxydes (LOOH) level. LOOH levels and spontaneous (vaginal) delivery were independently associated with increased GSH …

AdultMaleLipid Peroxidesanimal structuresHistologyNeonatal stressPopulationNeonatal strePhysiologyOxidative-stress markerDiseaseBiologymedicine.disease_causeMitochondrial Proteinschemistry.chemical_compoundLipid hydroperoxydemedicineHumanseducationOxidative-stress markerseducation.field_of_studyfungiInfant NewbornAnti-stress moleculeGestational ageChaperonin 60Cell BiologyGeneral MedicineGlutathioneHsp60GlutathioneNeonatal stress; Oxidative-stress markers; Lipid hydroperoxydes; Anti-stress molecules; Glutathione; Hsp60Oxidative StressAdult lifeLipid hydroperoxydeschemistryAnti-stress moleculesImmunologyFemaleHSP60BiomarkersOxidative stressNeonatal stressActa Histochemica
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The link between mitochondrial DNA hypervariable segment I heteroplasmy and ageing among genetically unrelated Latvians

2011

International audience; Various studies have demonstrated that mitochondrial DNA (mtDNA) heteroplasmy tends to increase with age and that the observed frequency of heteroplasmy among populations mostly depends on the way it is measured. Therefore, we investigated age-related association on the presence of mtDNA heteroplasmy within the hypervariable segment 1 (HVS-I) in a selected study group. The study group consisted of 300 maternally unrelated Latvians ranging in age from 18 to over 90years. To determine the optimal method for mtDNA heteroplasmy detection, three approaches were used: (i) SURVEYORTM Mutation Detection Kit, (ii) sequencing and (iii) denaturing gradient-gel electrophoresis (…

AdultMaleMitochondrial DNAAgingAdolescentBiologyBiochemistryDNA MitochondrialBroad spectrumYoung AdultEndocrinologyLatviansGeneticsHumansMutation detectionheteroplasmydetection of mtDNA heteroplasmyMolecular BiologyAgedGeneticsAged 80 and overDenaturing Gradient Gel ElectrophoresismtDNA[SDV.MHEP.GEG]Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontologyCell BiologySequence Analysis DNAMiddle AgedLatviaHeteroplasmyAgeingageingMutationFemaleHuman mitochondrial DNA haplogroup
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Deciphering exome sequencing data: Bringing mitochondrial DNA variants to light

2019

The expanding use of exome sequencing (ES) in diagnosis generates a huge amount of data, including untargeted mitochondrial DNA (mtDNA) sequences. We developed a strategy to deeply study ES data, focusing on the mtDNA genome on a large unspecific cohort to increase diagnostic yield. A targeted bioinformatics pipeline assembled mitochondrial genome from ES data to detect pathogenic mtDNA variants in parallel with the "in-house" nuclear exome pipeline. mtDNA data coming from off-target sequences (indirect sequencing) were extracted from the BAM files in 928 individuals with developmental and/or neurological anomalies. The mtDNA variants were filtered out based on database information, cohort …

AdultMaleMitochondrial DNAAtaxiaAdolescentDevelopmental Disabilities[SDV]Life Sciences [q-bio]BiologyDNA MitochondrialGenomeHaplogroupYoung Adult03 medical and health sciencesExome SequencingGeneticsmedicineHumansChildExomeGenetics (clinical)Exome sequencingComputingMilieux_MISCELLANEOUSAged030304 developmental biologyAged 80 and overGenetics0303 health sciences030305 genetics & heredityInfant NewbornComputational BiologyGenetic VariationInfantMiddle AgedPhenotypeEarly DiagnosisChild PreschoolFemaleNervous System Diseasesmedicine.symptom
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Epigenetic modifications precede molecular alterations and drive human hepatocarcinogenesis

2021

Development of primary liver cancer is a multistage process. Detailed understanding of sequential epigenetic alterations is largely missing. Here, we performed Infinium Human Methylation 450k BeadChips and RNA-Seq analyses for genome-wide methylome and transcriptome profiling of cirrhotic liver (n = 7), low- (n = 4) and high-grade (n = 9) dysplastic lesions, and early (n = 5) and progressed (n = 3) hepatocellular carcinomas (HCC) synchronously detected in 8 patients with HCC with chronic hepatitis B infection. Integrative analyses of epigenetically driven molecular changes were identified and validated in 2 independent cohorts comprising 887 HCCs. Mitochondrial DNA sequencing was further em…

AdultMaleMitochondrial DNACarcinoma HepatocellularCirrhosisMolecular biologyCarcinogenesisBiologyEpigenesis GeneticHepatitis B ChronicmedicineHumansEpigeneticsAgedHepatologyGene Expression ProfilingLiver NeoplasmsDNA NeoplasmGeneral MedicineMethylationDNA MethylationMiddle AgedHCCSmedicine.diseaseGene Expression Regulation NeoplasticOncologyApoptosisDNA methylationCancer researchEpigeneticsCalmodulin-Binding ProteinsFemaleLiver cancerLiver cancerResearch ArticleJCI Insight
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Could mitochondrial haplogroups play a role in sporadic amyotrophic lateral sclerosis?

2004

Mitochondrial impairment has been implicated in the pathogenesis of the amyotrophic lateral sclerosis (ALS). Furthermore, mitochondrial-specific polymorphisms were previously related to other neurodegenerative diseases, such as Parkinson, Friedreich and Alzheimer disease. To investigate if specific genetic polymorphisms within the mitochondrial genome (mtDNA) could act as susceptibility factors and contribute to the clinical expression of sporadic ALS (sALS), we have genotyped predefined European mtDNA haplogroups in 222 Italian patients with sALS and 151 matched controls. Individuals classified as haplogroup I demonstrated a significant decrease in risk of ALS versus individuals carrying t…

AdultMaleMitochondrial DNAPathologymedicine.medical_specialtyDiseaseBiologyDNA MitochondrialHaplogroupCohort StudiesDegenerative diseaseConfidence IntervalsOdds RatiomedicineHumansAmyotrophic lateral sclerosisAgedALS; Haplogroups; mtDNA;Polymorphism GeneticmtDNAGeneral NeuroscienceAmyotrophic Lateral SclerosisOdds ratioMiddle Agedmedicine.diseaseMitochondriaALS; mtDNA; HaplogroupsHaplotypesALS; Haplogroups; mtDNAImmunologyHaplogroupsFemaleAlzheimer's diseaseALSHuman mitochondrial DNA haplogroup
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