Search results for "Models"

showing 10 items of 8211 documents

Sex steroids, carcinogenesis, and cancer progression

2004

The relationship between sex steroids and cancer has been studied for more than a century. Using an original intact cell analysis, we investigated sex steroid metabolism in a panel of human cancer cell lines, either hormone responsive or unresponsive, originating from human breast, endometrium, and prostate. We found that highly divergent patterns of steroid metabolism exist and that the catalytic preference (predominantly reductive or oxidative) is strictly associated with the steroid receptor status of cells. We explored intra-tissue concentrations and profiles of estrogens in a set of human breast tumors as compared to normal mammary tissues, also in relation to their estrogen receptor s…

Receptor StatusTime FactorsIntratumor estrogenCatecholsBreast cancer; Intratumor estrogens; Sex steroids; Adsorption; Androstenedione; Animals; Breast Neoplasms; Catalysis; Catechols; Cell Line Tumor; Chromatography High Pressure Liquid; Disease Progression; Estradiol; Estrogens; Humans; In Vitro Techniques; Ions; Kinetics; Models Biological; Neoplasms; Steroids; Time Factors; Biochemistry Genetics and Molecular Biology (all)Sex steroidmedicine.disease_causeEndometriumCatalysiBreast cancerNeoplasmsEstrogen Receptor StatusChromatography High Pressure LiquidEstradiolGeneral NeuroscienceSex hormone receptormedicine.anatomical_structureDisease ProgressionSteroidsBreast NeoplasmHumanmedicine.medical_specialtyTime FactorBreast NeoplasmsIn Vitro TechniquesBiologyModels BiologicalCatalysisGeneral Biochemistry Genetics and Molecular BiologyBreast cancerHistory and Philosophy of ScienceCell Line TumorInternal medicinemedicineAnimalsHumansIonSteroidKineticIonsBiochemistry Genetics and Molecular Biology (all)AnimalIn Vitro TechniqueAndrostenedioneCancerEstrogensmedicine.diseaseEstrogenKineticsEndocrinologySex steroidCatecholNeoplasmAdsorptionCarcinogenesis
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Lipid presentation by the protein C receptor links coagulation with autoimmunity.

2021

A lipid-protein autoimmunity target Several autoimmune diseases, including systemic lupus erythematosus and primary antiphospholipid syndrome, are characterized by the presence of antiphospholipid antibodies (aPLs). These molecules can activate the complement and coagulation cascades, which contributes to pathologies such as thrombosis, stroke, and pregnancy complications. Müller-Calleja et al. found that endothelial protein C receptor (EPCR) in complex with lysobisphosphatidic acid (LBPA) is the cell-surface target for aPL and mediates its internalization (see the Perspective by Kaplan). aPL binding to EPCR-LBPA resulted in the activation of tissue factor–mediated coagulation and interfero…

Receptor complexAntigen presentationAutoimmunityEndosomesmedicine.disease_causeArticleAutoimmunityMiceInterferonimmune system diseasesmedicineAnimalsHumansLupus Erythematosus SystemicneoplasmsBlood CoagulationAutoantibodiesAutoimmune diseaseEndothelial protein C receptorAntigen PresentationMultidisciplinaryInnate immune systemLupus erythematosusEndothelial Protein C ReceptorThrombosismedicine.diseaseAntiphospholipid SyndromeImmunity InnateMice Mutant StrainsDisease Models AnimalSphingomyelin PhosphodiesteraseToll-Like Receptor 7ImmunologyAntibodies AntiphospholipidEmbryo LossMonoglyceridesEndothelium VascularLysophospholipidsmedicine.drugScience (New York, N.Y.)
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Reelin and CXCL12 regulate distinct migratory behaviors during the development of the dopaminergic system.

2014

The proper functioning of the dopaminergic system requires the coordinated formation of projections extending from dopaminergic neurons in the substantia nigra (SN), ventral tegmental area (VTA) and retrorubral field to a wide array of forebrain targets including the striatum, nucleus accumbens and prefrontal cortex. The mechanisms controlling the assembly of these distinct dopaminergic cell clusters are not well understood. Here, we have investigated in detail the migratory behavior of dopaminergic neurons giving rise to either the SN or the medial VTA using genetic inducible fate mapping, ultramicroscopy, time-lapse imaging, slice culture and analysis of mouse mutants. We demonstrate that…

Receptors CXCR4Cell Adhesion Molecules NeuronalDopamineEmbryonic DevelopmentSubstantia nigraNerve Tissue ProteinsStriatumBiologyNucleus accumbensLigandsModels BiologicalTime-Lapse ImagingMiceCell MovementDopaminergic CellmedicineAnimalsCell LineageReelinMolecular BiologyMice KnockoutExtracellular Matrix ProteinsDopaminergic NeuronsDopaminergicSerine EndopeptidasesVentral Tegmental AreaAnatomyChemokine CXCL12Ventral tegmental areaSubstantia NigraReelin Proteinmedicine.anatomical_structurenervous systemForebrainbiology.proteinNeuroscienceDevelopmental BiologySignal TransductionDevelopment (Cambridge, England)
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C-X-C Motif Chemokine Receptor 4 Blockade Promotes Tissue Repair After Myocardial Infarction by Enhancing Regulatory T Cell Mobilization and Immune-R…

2019

Background: Acute myocardial infarction (MI) elicits an inflammatory response that drives tissue repair and adverse cardiac remodeling. Inflammatory cell trafficking after MI is controlled by C-X-C motif chemokine ligand 12 (CXCL12) and its receptor, C-X-C motif chemokine receptor 4 (CXCR4). CXCR4 antagonists mobilize inflammatory cells and promote infarct repair, but the cellular mechanisms are unclear. Methods: We investigated the therapeutic potential and mode of action of the peptidic macrocycle CXCR4 antagonist POL5551 in mice with reperfused MI. We applied cell depletion and adoptive transfer strategies using lymphocyte-deficient Rag1 knockout mice; DEREG mice, which express a diphth…

Receptors CXCR4Regulatory T cellCXCR4 antagonistSus scrofaAnti-Inflammatory AgentsMyocardial InfarctionNeovascularization PhysiologicMice TransgenicInflammation030204 cardiovascular system & hematologyT-Lymphocytes RegulatoryVentricular Function Left03 medical and health sciencesChemokine receptor0302 clinical medicineImmune systemPhysiology (medical)medicineAnimalsMyocardial infarction030304 developmental biology0303 health sciencesMobilizationVentricular Remodelingbusiness.industryMyocardiumProteinsDendritic CellsRecovery of FunctionRegulatory T cellsTissue repairmedicine.diseaseMyocardial ContractionBlockadeMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureCancer researchmedicine.symptomCardiology and Cardiovascular MedicinebusinessSignal TransductionCirculation
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Macrophage migration inhibitory factor is critically involved in basal and fluoxetine-stimulated adult hippocampal cell proliferation and in anxiety,…

2011

Intensive research is devoted to unravel the neurobiological mechanisms mediating adult hippocampal neurogenesis, its regulation by antidepressants, and its behavioral consequences. Macrophage migration inhibitory factor (MIF) is a pro-inflammatory cytokine that is expressed in the CNS, where its function is unknown. Here, we show, for the first time, the relevance of MIF expression for adult hippocampal neurogenesis. We identify MIF expression in neurogenic cells (in stem cells, cells undergoing proliferation, and in newly proliferated cells undergoing maturation) in the subgranular zone of the rodent dentate gyrus. A causal function for MIF in cell proliferation was shown using genetic (M…

Receptors SteroidStem-Cellsanimal diseasesmedicine.medical_treatmentHippocampusExpressionHippocampal formationHippocampusSubgranular zonememoryMice0302 clinical medicineConditioning PsychologicalCyclin D2Rat Dentate GyrusMice KnockoutNeurons0303 health sciencesMicroscopy ConfocalChronic StressMifNeurogenesisBrainFearrespiratory systemanxietyPsychiatry and Mental healthC-Reactive ProteinCytokinemedicine.anatomical_structuredepressionAntidepressive Agents Second-GenerationStem cellPsychologyAnimal-ModelNeurogenesisSpatial BehaviorNerve Tissue Proteinschemical and pharmacologic phenomena03 medical and health sciencesCellular and Molecular Neurosciencemedicineotorhinolaryngologic diseasesAnimalsRats WistarMaze LearningMacrophage Migration-Inhibitory FactorsMolecular BiologyCell Proliferation030304 developmental biologyMemory DisordersDentate gyrusfluoxetineFactor Mifbiological factorsRatsDisease Models AnimalAcoustic StimulationBromodeoxyuridineMacrophage migration inhibitory factorCorticosteroneNeuroscience030217 neurology & neurosurgery
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Specific Regulation of T Helper Cell 1–mediated Murine Colitis by CEACAM1

2004

Carcinoembryonic antigen-related cellular adhesion molecule 1 (CEACAM1) is a cell surface molecule that has been proposed to negatively regulate T cell function. We have shown that CEACAM1 is associated with specific regulation of T helper cell (Th)1 pathways, T-bet–mediated Th1 cytokine signaling, and Th1-mediated immunopathology in vivo. Mice treated with anti–mouse CEACAM1-specific monoclonal antibody (mAb) CC1 during the effector phase exhibited a reduced severity of trinitrobenzene sulfonic acid colitis in association with decreased interferon (IFN)-γ production. Although oxazolone colitis has been reported as Th2 mediated, mice treated with the CC1 mAb or a CEACAM1-Fc chimeric protein…

Recombinant Fusion Proteinsmedicine.medical_treatmentT cellImmunologyBiologyArticleOxazoloneInterferon-gammaMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAntigeninflammatory bowel diseaseInterferonmedicineAnimalsImmunology and AllergyColitisCell adhesionCEACAM1030304 developmental biologyInflammationMice KnockoutMice Inbred BALB C0303 health sciencesT cell immunityOxazoloneAntibodies MonoclonalT-Lymphocytes Helper-InducerT helper cellTh1 CellsColitismedicine.diseaseMolecular biologyCarcinoembryonic AntigenImmunoglobulin Fc Fragments3. Good healthMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureCytokinechemistry030220 oncology & carcinogenesisFemaleTh1 cytokineInterleukin-1hapten-induced colitismedicine.drugJournal of Experimental Medicine
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Comparative cytogenetics of human chromosome 3q21.3 reveals a hot spot for ectopic recombination in hominoid evolution

2004

Fluorescence in situ hybridization mapping of fully integrated human BAC clones to primate chromosomes, combined with precise breakpoint localization by PCR analysis of flow-sorted chromosomes, was used to analyze the evolutionary rearrangements of the human 3q21.3-syntenic region in orangutan, siamang gibbon, and silvered-leaf monkey. Three independent evolutionary breakpoints were localized within a 230-kb segment contained in BACs RP11-93K22 and RP11-77P16. Approximately 200 kb of the human 3q21.3 sequence was not present on the homologous orangutan, siamang, and Old World monkey chromosomes, suggesting a genomic DNA insertion into the breakpoint region in the lineage leading to humans a…

Recombination GeneticGeneticsGenome evolutionModels GeneticGenome HumanEndogenous RetrovirusesBreakpointChromosome MappingChromosome BreakageHominidaeBiologyGenomeEvolution MolecularMolecular evolutionCytogenetic AnalysisGeneticsAnimalsHumansEctopic recombinationHuman genomeChromosomes Human Pair 3Segmental duplicationSyntenyGenomics
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Virtual lock-and-key approach: The in silico revival of Fischer model by means of molecular descriptors

2010

Abstract In the last years the application of computational methodologies in the medicinal chemistry fields has found an amazing development. All the efforts were focused on the searching of new leads featuring a close affinity on a specific biological target. Thus, different molecular modeling approaches in simulation of molecular behavior for a specific biological target were employed. In spite of the increasing reliability of computational methodologies, not always the designed lead, once synthesized and screened, are suitable for the chosen biological target. To give another chance to these compounds, this work tries to resume the old concept of Fischer lock-and-key model. The same can …

Record lockingInhibitorProcess (engineering)Chemistry PharmaceuticalNanotechnologycomputer.software_genreSet (abstract data type)Molecular descriptorDrug DiscoveryProtocol (object-oriented programming)PharmacologyChemistryOrganic ChemistryLock-and-keyGeneral MedicineSettore CHIM/08 - Chimica FarmaceuticaRange (mathematics)Models ChemicalDrugs re-purposingBiological targetTest setBiological targetData miningcomputerSoftwareMolecular descriptor
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Study on New Product Development, by Networking

2015

The paper presents a situation of working in network in order to design or improve a new product. To implement theoretical models and to validate a working algorithm into a virtual research, there was proposed a specific theme from drilling and chamfering tool. The facilities offered by the research centers – database, software - led to rapid selection and configuration solutions, demonstrating achieving research productivity growth and a substantial reduction of times redesign.

Reduction (complexity)Engineering drawingEngineeringSoftwarebusiness.industryOrder (exchange)New product developmentTheoretical modelsGeneral MedicinebusinessTheme (computing)Selection (genetic algorithm)Manufacturing engineeringApplied Mechanics and Materials
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Goethe's dream

2009

The German philosopher Immanuel Kant (1724–1804) defined living organisms as objects with an intrinsic purpose, which are self‐organized in such a way that every part is a function of the whole and the whole is a function of every part, and in which “nothing is for nothing”. Kant already anticipated the tension between agency and structure, and between forward and backward causation. He also perceived living beings as entities that, being extremely complex, are not amenable to descriptions based on laws that are similar to the fundamental laws of physics: “There will never be a Newton of a grass blade,” he wrote. Less metaphorically, Kant believed that science would not be able to understan…

ReductionismScience and SocietySystems Biologymedia_common.quotation_subjectBiomedical EngineeringComputational BiologyBiologyStructure and agencyModels BiologicalBiochemistryEpistemologyNothingIncarnationGeneticsCausationFunction (engineering)Molecular BiologyNaturalismBiotechnologyPhysical lawmedia_commonEMBO reports
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