Search results for "Molecular target"

showing 10 items of 187 documents

Nanomaterial-based biosensors for detection of pathogenic virus

2020

Viruses are real menace to human safety that cause devastating viral disease. The high prevalence of these diseases is due to improper detecting tools. Therefore, there is a remarkable demand to identify viruses in a fast, selective and accurate way. Several biosensors have been designed and commercialized for detection of pathogenic viruses. However, they present many challenges. Nanotechnology overcomes these challenges and performs direct detection of molecular targets in real time. In this overview, studies concerning nanotechnology-based biosensors for pathogenic virus detection have been summarized, paying special attention to biosensors based on graphene oxide, silica, carbon nanotub…

High prevalenceComputer science010401 analytical chemistryOptical detectionNanotechnologymacromolecular substances02 engineering and technologyNanomaterial021001 nanoscience & nanotechnology01 natural sciencesArticleVirusVirus0104 chemical sciencesAnalytical ChemistryVirus detectionNanomaterialsElectrochemistryMolecular targetsViral diseaseHuman safety0210 nano-technologyBiosensorBiosensorSpectroscopyTrAC Trends in Analytical Chemistry
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Cell uptake enhancement of folate targeted polymer coated magnetic nanoparticles.

2013

Dual targeted drug delivery systems represent a potential platform for developing efficient vector to tumor sites. In this study we evaluated a folate- and magnetic-targeted nanocarriers based on 10 nm iron oxide nanodomais coated with the properly synthesized and characterized folic acid (FA)-functionalized amphiphilic copolymer PHEA-PLA-PEG-FA. FA was chemically conjugated to one end of diamino-polyethylene glycol of 2000 Da, in order to ensure its exposition on the polymer coated magnetic nanoparticles (MNPs-FA). The prepared nanoparticles have been exhaustively characterized by different methods, including DLS, SEM, FT-IR and magnetic measurements. Magnetic nanoparticles showed dimensio…

IRON-OXIDE NANOPARTICLES; DRUG-DELIVERY; COPOLYMERSPolymersmedia_common.quotation_subjectBiomedical EngineeringPharmaceutical ScienceMedicine (miscellaneous)NanoparticleBioengineeringFolic AcidCoated Materials BiocompatibleCell Line TumorMaterials TestingHumansGeneral Materials ScienceViability assayMolecular Targeted TherapyInternalizationMagnetite Nanoparticlesmedia_commonChemistryNeoplasms Experimentalequipment and suppliesTreatment OutcomeTargeted drug deliveryCancer cellBiophysicsMCF-7 CellsMagnetic nanoparticlesNanocarriershuman activitiesFolate Targeting; Magnetic Nanoparticles; Cell Uptake; Ferrozine Assay; Polymer CoatingSuperparamagnetismJournal of biomedical nanotechnology
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HSP90 and HSP70: Implication in Inflammation Processes and Therapeutic Approaches for Myeloproliferative Neoplasms.

2015

Myeloproliferative neoplasms (MPN) are clonal stem cell disorders that lead to the excessive production of one or more blood cell lineages. It has been reported that, in most MPN, inflammatory cytokines are frequently increased, indicating that inflammation plays a crucial role in these disorders. Heat shock proteins (HSP) are induced in response to many stressful conditions from heat shock to hypoxia and inflammation. Besides their chaperone and cytoprotective functions, HSPs are key players during inflammation, hence the term “chaperokine.” Through their chaperone activity, HSP90, a stabilizer of many oncogenes (e.g., JAK2), and HSP70, a powerful antiapoptotic chaperone, tightly regulate …

ImmunologyInflammationReview ArticleBiologyModels BiologicalProinflammatory cytokineMyeloproliferative DisordersHeat shock proteinlcsh:PathologymedicineHumansHSP70 Heat-Shock ProteinsHSP90 Heat-Shock ProteinsMolecular Targeted TherapyMyeloproliferative DisordersCell BiologyHsp90Chaperone (protein)ImmunologyCancer researchbiology.proteinmedicine.symptomSignal transductionStem cellInflammation Mediatorslcsh:RB1-214Signal TransductionMediators of inflammation
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Disulfide stress and its targets in acute pancreatitis

2014

Under physiological conditions, the balance between ROS production and removal properly maintains the intracellular redox-sensitive signaling as well as the appropriate status of protein thiols and disulfides. However, inflammation among other factors can modify this balance causing a rapid increase in intracellular ROS levels and hence thiol oxidation, eventually leading to oxidative stress. In the case of acute pancreatitis, both redox signaling and oxidative stress seem to contribute to the progression of the severe form of the disease. In this review we will focus on the reversible oxidation of protein cysteines during the course of acute pancreatitis. We describe disulfide stress in an…

ImmunologyInflammationmedicine.disease_causechemistry.chemical_compoundmedicineAnimalsHumansImmunology and AllergyCysteineDisulfidesMolecular Targeted TherapyCysteine metabolismPharmacologychemistry.chemical_classificationReactive oxygen speciesGeneral MedicineGlutathionemedicine.diseaseOxidative StressPancreatitischemistryBiochemistryAcute DiseaseAcute pancreatitismedicine.symptomSignal transductionOxidation-ReductionIntracellularOxidative stressSignal Transduction
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HSP70 sequestration by free α-globin promotes ineffective erythropoiesis in β-thalassaemia

2014

International audience; β-Thalassaemia major (β-TM) is an inherited haemoglobinopathy caused by a quantitative defect in the synthesis of β-globin chains of haemoglobin, leading to the accumulation of free α-globin chains that form toxic aggregates. Despite extensive knowledge of the molecular defects causing β-TM, little is known of the mechanisms responsible for the ineffective erythropoiesis observed in the condition, which is characterized by accelerated erythroid differentiation, maturation arrest and apoptosis at the polychromatophilic stage. We have previously demonstrated that normal human erythroid maturation requires a transient activation of caspase-3 at the later stages of matur…

Ineffective erythropoiesisCytoplasmErythroblastsCell SurvivalMutantApoptosis[ SDV.BBM.BM ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyalpha-globin[SDV.BC.BC]Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]Biologymedicine.disease_causeProtein Refolding03 medical and health sciences0302 clinical medicinealpha-GlobinsBone Marrowhemic and lymphatic diseasesmedicineHumans[ SDV.MHEP.HEM ] Life Sciences [q-bio]/Human health and pathology/HematologyErythropoiesisGATA1 Transcription FactorHSP70 Heat-Shock ProteinsMolecular Targeted TherapyCells CulturedHSP70030304 developmental biologyRegulation of gene expressionCell Nucleus0303 health sciencesMultidisciplinaryCaspase 3beta-Thalassemia[ SDV.BC.BC ] Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]GATA1[SDV.MHEP.HEM]Life Sciences [q-bio]/Human health and pathology/Hematology[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyMolecular biologyHsp70Enzyme ActivationKineticsGene Expression RegulationCytoplasm030220 oncology & carcinogenesisChaperone (protein)biology.proteinErythropoiesisbeta-ThalassaemiaProtein Binding
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Host-directed therapies for COVID-19

2021

Purpose of review Severe acute respiratory syndrome coronavirus-2-induced hyperinflammation is a major cause of death or end-organ dysfunction in COVID-19 patients. We review adjunct host-directed therapies (HDTs) for COVID-19 management. Recent findings The use of umbilical cord-derived mesenchymal stem cells as HDT for COVID-19 has been shown to be safe in phase 1 and 2 trials. Trials of anti-interleukin-6 receptor antibodies show promising mortality benefit in hospitalized COVID-19 patients. Repurposed drugs and monoclonal antibodies targeting specific cytokines acting on different aspects of the pro- and anti-inflammatory cascades are under evaluation. Summary A range of HDTs shows prom…

InflammationPulmonary and Respiratory Medicine2019-20 coronavirus outbreakCoronavirus disease 2019 (COVID-19)SARS-CoV-2medicine.drug_classbusiness.industryMesenchymal stem cellCOVID-19InflammationLong term disabilityMesenchymal Stem Cell TransplantationBioinformaticsMonoclonal antibodymedicineHumansImmunologic FactorsIn patientMolecular Targeted Therapymedicine.symptombusinessCause of deathCurrent Opinion in Pulmonary Medicine
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Molecular dynamics, dynamic site mapping, and highthroughput virtual screening on leptin and the Ob receptor as anti-obesity target.

2014

Body weight control is a mechanism finely regulated by several hormonal, metabolic, and nervous pathways. The leptin receptor (Ob-R) is crucial for energy homeostasis and regulation of food uptake. Leptin is a 16 kDa hormone that is mainly secreted by fat cells into the bloodstream, and under normal circumstances, circulating levels are proportionate to the fat body mass. Sensing of elevated leptin levels by the hypothalamic neurocircutry activates a negative feedback loop resulting in reduced food intake and increased energy expenditure. Decreased concentrations lead to opposite effects. Therefore rational design of leptin agonists constitute an appealing challenge in the battle against ob…

Leptinmedicine.medical_specialtyProtein ConformationAdipose tissueDrug designBiologyMolecular Dynamics SimulationDynamic SiteMapping HTVS Leptin Molecular Dynamics Obesity Protein/protein docking Multivariate analysis Ob ReceptorCatalysisEnergy homeostasisInorganic ChemistryStructure-Activity RelationshipInternal medicinemedicineMolecular Targeted TherapyPhysical and Theoretical ChemistryReceptorVirtual screeningLeptin receptorBinding SitesMolecular StructureLeptindigestive oral and skin physiologyOrganic ChemistryHydrogen BondingSettore CHIM/08 - Chimica FarmaceuticaComputer Science ApplicationsHigh-Throughput Screening AssaysMolecular Docking SimulationEndocrinologyComputational Theory and MathematicsDocking (molecular)Drug DesignMultivariate AnalysisComputer-Aided DesignReceptors LeptinAnti-Obesity AgentsHydrophobic and Hydrophilic InteractionsProtein BindingJournal of molecular modeling
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Plasma PLTP (phospholipid-transfer protein): an emerging role in ‘reverse lipopolysaccharide transport’ and innate immunity

2011

Plasma PLTP (phospholipid-transfer protein) is a member of the lipid transfer/LBP [LPS (lipopolysaccharide)-binding protein] family, which constitutes a superfamily of genes together with the short and long PLUNC (palate, lung and nasal epithelium clone) proteins. Although PLTP was studied initially for its involvement in the metabolism of HDL (high-density lipoproteins) and reverse cholesterol transport (i.e. the metabolic pathway through which cholesterol excess can be transported from peripheral tissues back to the liver for excretion in the bile), it displays a number of additional biological properties. In particular, PLTP can modulate the lipoprotein association and metabolism of LPS …

Lipopolysaccharidesmedicine.medical_specialtyInflammationPluncBiologyBiochemistryLipopolysaccharide transportchemistry.chemical_compoundInternal medicinePhospholipid transfer proteinmedicineAnimalsBileHumansMolecular Targeted TherapyPhospholipid Transfer ProteinsInnate immune systemCholesterolReverse cholesterol transportShock SepticImmunity InnateEndocrinologyLiverchemistrylipids (amino acids peptides and proteins)medicine.symptomMetabolic Networks and PathwaysLipoproteinBiochemical Society Transactions
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Liver fibrosis: Direct antifibrotic agents and targeted therapies

2018

Liver fibrosis and in particular cirrhosis are the major causes of morbidity and mortality of patients with chronic liver disease. Their prevention or reversal have become major endpoints in clinical trials with novel liver specific drugs. Remarkable progress has been made with therapies that efficiently address the cause of the underlying liver disease, as in chronic hepatitis B and C. Highly effective antiviral therapy can prevent progression or even induce reversal in the majority of patients, but such treatment remains elusive for the majority of liver patients with advanced alcoholic or nonalcoholic steatohepatitis, genetic or autoimmune liver diseases. Moreover, drugs that would speed…

Liver Cirrhosis0301 basic medicineCirrhosisDiseaseChronic liver disease03 medical and health sciencesLiver diseaseTransforming Growth Factor betaFibrosisAnimalsHumansMedicineMolecular Targeted TherapyMolecular BiologyExtracellular Matrix ProteinsDDR1business.industrymedicine.disease3. Good healthBiomarker (cell)030104 developmental biologyDisease ProgressionCancer researchHepatic stellate cellbusinessSignal TransductionMatrix Biology
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The nonalcoholic steatohepatitis (NASH) drug development graveyard: established hurdles and planning for future success

2020

Contains fulltext : 229341.pdf (Publisher’s version ) (Open Access) INTRODUCTION: Numerous pharmacological compounds that target the different molecular targets involved in the pathobiology of nonalcoholic steatohepatitis (NASH) are currently in clinical testing. So far, there are no regulatory approvals. AREAS COVERED: This paper sheds light on the molecular pathways involved in NASH and the drugs targeting these pathways. We have identified 10 compounds whose clinical development program has been halted. Moreover, we explore early phase clinical trials and dissect the reasons for termination of development. EXPERT OPINION: The main goal of NASH pharmacotherapy is to halt or reverse hepati…

Liver Cirrhosis0301 basic medicineNonalcoholic steatohepatitisAnti-Inflammatory AgentsPhases of clinical researchBioinformaticsdigestive system03 medical and health sciences0302 clinical medicineDrug DevelopmentNon-alcoholic Fatty Liver DiseasemedicineAnimalsHumansPharmacology (medical)Molecular Targeted TherapyPharmacologybusiness.industryFatty liverGeneral Medicinemedicine.diseasedigestive system diseasesRenal disorders Radboud Institute for Molecular Life Sciences [Radboudumc 11]030104 developmental biologyDrug development030220 oncology & carcinogenesisMolecular targetsbusinessExpert Opinion on Investigational Drugs
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