Search results for "Move"

showing 10 items of 2153 documents

Ultramicro-trauma in the endometrial-myometrial junctional zone and pale cell migration in adenomyosis

2015

Objective To determine if ultrastructural tissue trauma occurs in the junctional zone in uteri in adenomyosis. Design A case-control experimental study. Setting Endometriosis research center. Patient(s) Twelve uteri with adenomyosis, and 9 uteri without adenomyosis, were gained during laparoscopy-assisted vaginal hysterectomy. Intervention(s) Transmission electron microscopic study of the junctional zone, as well as immunohistochemical staining for epithelial cadherin, and van Gieson staining and immunofluorescence for CD45 and CD68. Main Outcome Measure(s) Analysis of the electron microscopy photos and the immunoreactive scores of the staining. Result(s) The inner myometrial muscle fibers …

PathologyEndometriosisFluorescent Antibody TechniqueEndometriumBasal (phylogenetics)EndometriumMyofibrilsCell MovementVan Gieson's stainHysterectomy VaginalMedicine (all)MyometriumObstetrics and GynecologyAntigens CD45AnatomyMiddle AgedCadherinsultrastructureImmunohistochemistrymedicine.anatomical_structurePhenotypeMyometriumImmunohistochemistryFemaleAdenomyosis pathogenesiCase-Control StudieAdenomyosisHumanAdultmedicine.medical_specialtyAntigens Differentiation MyelomonocyticBiologypale cellMicroscopy Electron TransmissionAntigens CDmedicineHumansAdenomyosisendometrial-myometrial junctional zoneEpithelial CellCadherinEpithelial CellsBiomarkermedicine.diseaseReproductive MedicineCase-Control StudiesCadherinLeukocyte Common AntigensLaparoscopyAdenomyosiBiomarkersMyofibril
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Multiple congenital contractures (Congenital multiple arthrogryposis)

2002

Arthrogryposis, is the occurrence of joint contractures of variable etiology that start prenatally. Arthrogryposis may result from neurologic deficit, neuromuscular disorders, connective tissue abnormalities, amniotic bands, [figure: see text] or fetal crowding. Arthrogryposis may result from no apparent hereditary causes (neuropathic, for example) or may be the result of hereditary factors (myopathic form, for example). Ultrasound diagnosis depends on observation of scant or absent motion of fetal extremities. Prognosis depends on the specific etiology of the contractures.

Pathologymedicine.medical_specialtyAmniotic BandConnective tissueNeurological disorderUltrasonography PrenatalPregnancyHumansMedicineAbnormalities MultipleJoint ContractureFetal MovementMuscle contractureArthrogryposisArthrogryposisbusiness.industryObstetrics and GynecologyExtremitiesSyndromemedicine.diseasemedicine.anatomical_structurePediatrics Perinatology and Child HealthFetal movementEtiologyFemalemedicine.symptombusinessJournal of Perinatal Medicine
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Myofibroblasts and increased angiogenesis contribute to periapical cystic injury containment and repair

2019

Background Myofibroblasts (MF) and angiogenesis are important factors in the development and expansion of cystic lesions, where these cells secrete growth factors and proteases, stimulating angiogenesis, matrix deposition and cell migration, affecting the growth of these periapicopathies. The present study aimed to evaluate the immunohistochemical expression of CD34 and α-SMA in radicular cysts (RC) and residual radicular cysts (RRC), with the purpose of contributing to a better understanding of the expansion and progression of these periapical lesions. Material and Methods The present study os a descriptive, quantitative and comparative analysis of positive CD34 and α-SMA immunohistochemic…

Pathologymedicine.medical_specialtyAngiogenesisCD34Neovascularization03 medical and health sciences0302 clinical medicineCell MovementmedicineHumansMyofibroblastsGeneral DentistryMicrovesselRadicular CystOral Medicine and PathologyNeovascularization Pathologicbusiness.industryResearchCell migration030206 dentistry:CIENCIAS MÉDICAS [UNESCO]OtorhinolaryngologyMicrovesselsUNESCO::CIENCIAS MÉDICASImmunohistochemistrySurgerymedicine.symptombusinessMyofibroblastImmunostaining
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Extracellular calcium-sensing receptor mediates human bronchial epithelial wound repair

2010

The airway epithelium routinely undergoes damage that requires repair to restore epithelial barrier integrity. Cell migration followed by proliferation are necessary steps to achieve epithelial repair. Calcium-sensing receptor (CaSR) is implicated in cell migration and proliferation processes. Thus we hypothesized that CaSR mediates lung epithelial wound repair. We detected CaSR expression in human lung and in well-differentiated human bronchial epithelial cells (HBEC). To test the CaSR functionality, HBEC loaded with fura-2 were stimulated with extracellular Ca(2+) ([Ca(2+)](out)) which resulted in a concentration-dependent intracellular Ca(2+) ([Ca(2+)](i)) increase (potency approximately…

Pathologymedicine.medical_specialtyBronchiBiologyBiochemistryCell MovementmedicineExtracellularHumansCalcium SignalingEnzyme InhibitorsEstrenesReceptorEgtazic AcidCell ProliferationPharmacologyWound HealingPhospholipase CCell growthEpithelial CellsCell migrationPyrrolidinonesEpitheliumCell biologymedicine.anatomical_structureRespiratory epitheliumCalciumCalcium-sensing receptorFura-2Receptors Calcium-SensingBiochemical Pharmacology
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Expression of T-cadherin in tumor cells influences invasive potential of human hepatocellular carcinoma

2006

Overexpression of T-cadherin (T-cad) transcripts occurs in approximately 50% of human hepatocellular carcinomas (HCCs). To elucidate T-cad functions in HCC, we examined T-cad protein expression in normal and tumoral human livers and hepatoma cell lines and investigated its influence on invasive potential of HCC using RNA interference silencing of T-cad expression in Mahlavu cells. Whereas T-cad expression was restricted to endothelial cells (EC) from large blood vessels in normal livers, it was up-regulated in sinusoidal EC from 8/15 invasive HCCs. Importantly, in three of them (38%) T-cad was detected in tumor cells within regions in which E-cadherin expression was absent. Among six hepato…

Pathologymedicine.medical_specialtyCarcinoma HepatocellularTranscription GeneticLiver cytologyCell Culture TechniquesMotilityBiologyTransfectionBiochemistryRNA interferenceCell MovementCell Line TumorGeneticsmedicineGene silencingAnimalsHumansNeoplasm Invasivenesscardiovascular diseasesRNA Small InterferingMolecular BiologyDNA PrimersWound Healingprimary tumors cadherin switch cell invasion hepatoma cell lines RNA interferenceLiver NeoplasmsEndothelial CellsTransfectionHCCSFibroblastsCadherinsdigestive system diseasesT-cadherinLiverCell cultureCancer researchHepatocytesRabbitsCell DivisionBiotechnology
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18F-fluorodeoxyglucose hypometabolism in cerebellar tonsil and flocculus in downbeat nystagmus.

2006

A patient with downbeat nystagmus was examined by F-fluorodeoxyglucose-positron emission tomography once while off and twice while on successful treatment with 4-aminopyridine. All positron emission tomography scans of the patient showed a reduced cerebral glucose metabolism bilaterally in the region of the cerebellar tonsil and flocculus/paraflocculus when compared with a normal database of the whole brain. An additional region-of-interest analysis revealed that 4-aminopyridine treatment lessened the hypometabolism. This finding supports the hypothesis that the cerebellar tonsil and (para-) flocculus play a crucial role in downbeat nystagmus. The hypometabolism might reflect reduced inhibi…

Pathologymedicine.medical_specialtyCerebellumgenetic structuresEye MovementsNystagmusFlocculusNystagmus PathologicDownbeat nystagmusImaging Three-DimensionalVestibular nucleiFluorodeoxyglucose F18CerebellummedicinePotassium Channel BlockersHumans4-AminopyridineAgedFluorodeoxyglucosemedicine.diagnostic_testbusiness.industryGeneral Neurosciencemedicine.anatomical_structurePositron emission tomographyPositron-Emission TomographyCerebellar tonsilFemalemedicine.symptombusinessNeurosciencemedicine.drugNeuroreport
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Muscle degeneration in neuramindase 1 deficient mice results from infiltration of the muscle fibers by expanded connective tissue

2010

AbstractNeuraminidase 1 (NEU1) regulates the catabolism of sialoglycoconjugates in lysosomes. Congenital NEU1 deficiency in children is the basis of sialidosis, a severe neurosomatic disorder in which patients experience a broad spectrum of clinical manifestations varying in the age of onset and severity. Osteoskeletal deformities and muscle hypotonia have been described in patients with sialidosis. Here we present the first comprehensive analysis of the skeletal muscle pathology associated with loss of Neu1 function in mice. In this animal model, skeletal muscles showed an expansion of the epimysial and perimysial spaces, associated with proliferation of fibroblast-like cells and abnormal …

Pathologymedicine.medical_specialtyMuscle HypotoniaMuscle Fibers SkeletalNeuraminidaseConnective tissueApoptosisNEU1BiologyArticleMiceNecrosisNEU1SarcolemmaCell MovementSettore BIO/10 - BiochimicamedicineAnimalsSialidosisMuscular dystrophyMyopathyMolecular BiologySialidosiMetalloproteinaseCell ProliferationMice KnockoutMuscle biopsySialidosisECMmedicine.diagnostic_testSkeletal muscleFibroblastsMuscular Dystrophy Animalmedicine.diseaseLysosomeExtracellular MatrixMuscular Atrophymedicine.anatomical_structureConnective TissueImmunologyMolecular MedicineMuscle biopsymedicine.symptom
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Treatment of pemphigus vulgaris with mycophenolate mofetil

1997

different pattern of diffusion changes in a 51-year-old woman with chronic epilepsy and recurrent episodes of focal status epilepticus, for whom no aetiology could be established. Status consisted of clonic jerking of the right leg, which continued for 22 days and was followed by transient paresis. DWI during status showed decreased diffusion in the motor cortex of the right leg (relative decrease in ADC of 27%, see figure). Surprisingly, the diffusion was increased in the subcortical white matter (relative increase in ADC of 31%). On the T2-weighted image (not shown), both cortex and subcortical white matter of the corresponding region returned a high signal similar to previously reported …

Pathologymedicine.medical_specialtyPrednisoloneAnti-Inflammatory AgentsMycophenolateMycophenolic acidWhite matterIMP DehydrogenaseCortex (anatomy)medicineHumansPremovement neuronal activityParesisbusiness.industryRemission InductionPemphigus vulgarisGeneral MedicineMycophenolic Acidmedicine.diseasemedicine.anatomical_structureDrug Therapy Combinationmedicine.symptombusinessImmunosuppressive AgentsPemphigusmedicine.drugMotor cortexThe Lancet
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Targeting of multiple myeloma-related angiogenesis by miR-199a-5p mimics: in vitro and in vivo anti-tumor activity

2014

// Lavinia Raimondi 1 , Nicola Amodio 1 , Maria Teresa Di Martino 1 , Emanuela Altomare 1 , Marzia Leotta 1 , Daniele Caracciolo 1 , Annamaria Gulla 1 , Antonino Neri 2 , Simona Taverna 3 , Patrizia D’Aquila 4 , Riccardo Alessandro 3 , Antonio Giordano 5 , Pierosandro Tagliaferri 1 and Pierfrancesco Tassone 1,5 . 1 Department of Experimental and Clinical Medicine, Magna Graecia University and Medical Oncology Unit, T. Campanella Cancer Center, Salvatore Venuta University Campus, Catanzaro, Italy 2 Department of Medical Sciences University of Milan, Hematology1, IRCCS Policlinico Foundation, Milan, Italy 3 Department of Pathology and Forensic and Medical Biotechnology, Section of Biology and…

Pathologymedicine.medical_specialtyStromal cellAngiogenesisMultiple Myeloma; microRNA AngiogenesisBlotting WesternEnzyme-Linked Immunosorbent AssayMice SCIDIn Vitro TechniquesBiologyReal-Time Polymerase Chain ReactionTransfectionMicemiR-199-5pCell MovementMice Inbred NODSettore BIO/13 - Biologia ApplicataCell Line TumorCell AdhesionmedicineAnimalsHumansHypoxiaCell adhesionProtein kinase BCell ProliferationPlasma cell leukemiaNeovascularization PathologicmicroRNA AngiogenesisMicroRNATransfectionPlasma cell leukemiamedicine.diseaseXenograft Model Antitumor AssaysMolecular medicineCell HypoxiaMicroRNAsmedicine.anatomical_structureOncologyAngiogenesis; Hypoxia; Microenviroment; MicroRNA; miR-199-5p; MiRNA; Multiple myeloma; Plasma cell leukemiaCancer researchFemaleAngiogenesisBone marrowMicroenviromentMiRNAMultiple MyelomaResearch Paper
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A novel in vitro model for the study of plaque development in atherosclerosis

2006

SummaryFor the study of atherogenesis in vitro, coculture systems have been devised, in which two or more cell types can be cultured in close contact to each other. Herein, we describe a novel in vitro model that aims at the simulation of the morphology ofa normal muscular artery allowing for the study of the initial events in atherosclerosis. Usinga modified fibrin gel as a scaffold for the coculture of endothelial cells (ECs) and smooth muscle cells (SMCs), we generated an autologous in vitro model with a multilayer growth of SMCs (intima-like structure) covered by an endothelium. The production of extracellular matrix (ECM) could be visualized histologically and verified by (i) ascorbic-…

Pathologymedicine.medical_specialtyTime FactorsEndotheliumCellular differentiationMyocytes Smooth MuscleMonocytesMuscle Smooth VascularCell LineExtracellular matrixCell MovementLamininCell AdhesionmedicineHumansFoam cellFibrinDose-Response Relationship Drugbiologybusiness.industryEndothelial CellsCell DifferentiationHematologyAtherosclerosisCoculture TechniquesIn vitroExtracellular MatrixCell biologyLipoproteins LDLmedicine.anatomical_structureCell culturebiology.proteinbusinessGelsFoam CellsLipoproteinThrombosis and Haemostasis
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