Search results for "Muscarinic Agonist"

showing 10 items of 23 documents

Anti-inflammatory and antioxidant effects of muscarinic acetylcholine receptor (mAChR) activation in the rat hippocampus

2019

AbstractRecently we found that acute treatment with Oxotremorine (Oxo), a non-selective mAChRs agonist, up-regulates heat shock proteins and activates their transcription factor heat shock factor 1 in the rat hippocampus. Here we aimed to investigate: a) if acute treatment with Oxo may regulate pro-inflammatory or anti-inflammatory cytokines and oxidative stress in the rat hippocampus; b) if chronic restraint stress (CRS) induces inflammatory or oxidative alterations in the hippocampus and whether such alterations may be affected by chronic treatment with Oxo. In the acute experiment, rats were injected with single dose of Oxo (0.4 mg/kg) and sacrificed at 24 h, 48 h and 72 h. In the CRS ex…

MaleHydrocortisonemedicine.medical_treatmentInterleukin-1betaNeuroimmunologyAnti-Inflammatory Agentslcsh:MedicinePharmacologymedicine.disease_causeHippocampusSettore BIO/09 - FisiologiaAntioxidantsSuperoxide Dismutase-1Muscarinic acetylcholine receptorPhosphorylationlcsh:Sciencechemistry.chemical_classificationMultidisciplinarybiologyneurodegenerationAlzheimer's diseaseReceptors MuscarinicNeuroprotective AgentsCytokineSignal Transductionmedicine.drugRestraint PhysicalAgonistmedicine.drug_classScopolaminemuscarinic acetylcholine receptorMuscarinic AgonistsArticleOxotremorine anti-inflammatory cytokinesSuperoxide dismutaseHeat shock proteinOxotremorinemedicineAnimalsRats WistarInflammationReactive oxygen speciesInterleukin-6Superoxide DismutaseOxotremorinelcsh:RTranscription Factor RelARatsOxidative Stresschemistrybiology.proteinlcsh:QReactive Oxygen SpeciesProtein Processing Post-TranslationalOxidative stressScientific Reports
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The use of pilocarpine in opioid-induced xerostomia

2001

Oral dryness can be a symptom of asystemic disease, an adverse effect of anticholin-ergic, antiadrenergic or cytotoxic drug treatment, orit can be due to local radiotherapy. Opioid use isstrongly associated with xerostomia, although themechanism for this remains unclear; in one studypatients receiving morphine were four times morelikely to have a dry mouth than patients taking otherdrugs known to cause xerostomia.

MaleNarcoticsmedicine.medical_specialtyPalliative caremedicine.medical_treatmentAdministration OralPainMuscarinic AgonistsXerostomiaGastroenterologyMuscarinic Agonist03 medical and health sciences0302 clinical medicinestomatognathic system030502 gerontologyNeoplasmsInternal medicinemedicineHumansAdverse effectAgedChemotherapybusiness.industryPilocarpinefood and beveragesGeneral MedicineMiddle AgedDry mouthstomatognathic diseasesTreatment OutcomeAnesthesiology and Pain MedicineOpioidPilocarpineNarcotic030220 oncology & carcinogenesisAnesthesiaToxicityMorphineNeoplasmFemalemedicine.symptom0305 other medical sciencebusinessHumanmedicine.drugPalliative Medicine
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Switching between persistent firing and depolarization block in individual rat CA1 pyramidal neurons

2018

The hippocampal formation plays a role in mnemonic tasks and epileptic discharges in vivo. In vitro, these functions and malfunctions may relate to persistent firing (PF) and depolarization block (DB), respectively. Pyramidal neurons of the CA1 field have previously been reported to engage in either PF or DB during cholinergic stimulation. However, it is unknown whether these cells constitute disparate populations of neurons. Furthermore, it is unclear which cell-specific peculiarities may mediate their diverse response properties. However, it has not been shown whether individual CA1 pyramidal neurons can switch between PF and DB states. Here, we used whole cell patch clamp in the current …

MalePotassium ChannelsPatch-Clamp Techniquesantagonists & inhibitors [TRPC Cation Channels]physiology [Electrophysiological Phenomena]Cognitive Neurosciencepharmacology [Muscarinic Agonists]metabolism [TRPC Cation Channels]drug effects [Pyramidal Cells]HippocampusStimulationMuscarinic AgonistsIn Vitro TechniquesHippocampal formation050105 experimental psychologyMembrane Potentialspharmacology [Carbachol]03 medical and health sciences0302 clinical medicineCurrent clampAnimalsRats Long-Evans0501 psychology and cognitive sciencesddc:610Patch clampCA1 Region HippocampalTRPC Cation Channelsphysiology [CA1 Region Hippocampal]Dose-Response Relationship Drugphysiology [Pyramidal Cells]ChemistryPyramidal Cells05 social sciencescytology [CA1 Region Hippocampal]drug effects [Membrane Potentials]Depolarizationmetabolism [Potassium Channels]drug effects [Electrophysiological Phenomena]Potassium channelElectrophysiological PhenomenaRatsdrug effects [CA1 Region Hippocampal]CholinergicCarbacholFemaleNeuroscience030217 neurology & neurosurgeryHippocampus
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Adrenoceptor- and cholinoceptor-mediated mechanisms in the regulation of 5-hydroxytryptamine release from isolated tracheae of newborn rabbits

1996

Abstract 1. Isolated tracheae of newborn rabbits were incubated in vitro and the outflow of 5-hydroxytryptamine (5-HT) was determined by h.p.l.c. with electrochemical detection. Evidence has previously been provided that this 5-HT outflow derives from neuroendocrine epithelial (NEE) cells of the airway mucosa. 2. Phenylephrine (1, 10 and 30 microM) enhanced the outflow of 5-HT by 80, 290 and 205%, respectively. 5-HT outflow evoked by 10 microM phenylephrine was not affected by the presence of the neurotoxin tetrodotoxin (1 microM). 3. Rauwolscine, ARC 239 (an alpha(2B)-adrenoceptor preferring antagonist), yohimbine and prazosin antagonized the effect of 10 microM phenylephrine in a concentr…

MaleSerotoninmedicine.medical_specialtyRauwolscineIn Vitro TechniquesMuscarinic Agonistschemistry.chemical_compoundIsoprenalineInternal medicineReceptors Adrenergic betaCyclic AMPmedicinePrazosinAnimalsReceptors CholinergicPhenylephrinePharmacologyForskolinMuscarineHydroxyindoleacetic AcidReceptors AdrenergicYohimbineTracheaEndocrinologyAnimals NewbornchemistryFemaleHexamethoniumRabbitsResearch Articlemedicine.drugBritish Journal of Pharmacology
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Gastric relaxation induced by apigenin and quercetin: Analysis of the mechanism of action

2009

Abstract Aims Recently, flavonoids have been shown to cause murine gastric relaxation. In the present study we examined the mechanism of action underlying gastric relaxation induced by apigenin and quercetin in isolated mouse stomach. Main methods The mechanical activity from the whole stomach was detected as changes in the endoluminal pressure and the response to increasing concentrations of both flavonoids were tested before and after different pharmacological treatments. Key findings Apigenin and quercetin-induced a concentration-dependent gastric relaxation, apigenin being more potent than quercetin. The responses were unaffected by 2′5′dideoxyadenosine, an inhibitor of adenylate cyclas…

Malemedicine.medical_specialtyCarbacholNifedipineMuscle Relaxationchemistry.chemical_elementCalcium antagonistIn Vitro TechniquesMuscarinic AgonistsCalciumPharmacologySettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyPotassium ChlorideMicechemistry.chemical_compoundSmooth muscleInternal medicineCyclic AMPmedicineAnimalsheterocyclic compoundsApigeninGeneral Pharmacology Toxicology and PharmaceuticsCyclic GMPCyclic nucleotide phosphodiesteraseChemistryRyanodine receptorStomachMuscle SmoothGeneral MedicineCalcium Channel BlockersMice Inbred C57BLEndocrinologyMechanism of actionGastric toneApigeninFlavonoidCalciumCarbacholQuercetinmedicine.symptomQuercetinIntracellularMuscle Contractionmedicine.drugLife Sciences
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In vitro tracheal hyperresponsiveness to muscarinic receptor stimulation by carbachol in a rat model of bleomycin-induced pulmonary fibrosis

2006

Summary 1 Bleomycin-induced lung injury is widely used as an experimental model to investigate the pathophysiology of pulmonary fibrosis but the alterations in the pharmacological responsiveness of airways isolated from bleomycin-exposed animals has been scarcely investigated. The aim of this study was to examine the in vitro tracheal responses to muscarinic receptor stimulation with carbachol in a rat bleomycin model. 2 Concentration–response curves to carbachol (10 nm to 0.1 mm) were obtained in tracheal rings isolated from Sprague–Dawley rats 14 days after endotracheal bleomycin or saline. The intracellular calcium signal in response to carbachol (10 μm) was measured by epifluorescence m…

Malemedicine.medical_specialtyCarbacholPulmonary FibrosisStimulationIn Vitro TechniquesMuscarinic AgonistsLung injuryCalcium in biologyProinflammatory cytokineRats Sprague-DawleyBleomycinFibrosisInternal medicinePulmonary fibrosisMuscarinic acetylcholine receptormedicineAnimalsCalcium SignalingPharmacologyDose-Response Relationship DrugTumor Necrosis Factor-alphabusiness.industryMuscle Smoothrespiratory systemmedicine.diseaseReceptors MuscarinicRatsTracheaDisease Models AnimalEndocrinologyCarbacholBronchial HyperreactivitybusinessInterleukin-1Muscle Contractionmedicine.drugAutonomic and Autacoid Pharmacology
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Muscarinic inhibition of endogenous noradrenaline release from rabbit isolated trachea: receptor subtype and receptor reserve.

1994

The aim of the present study was to characterize putative muscarine receptors on sympathetic nerve terminals in the rabbit trachea. Release of endogenous noradrenaline from in vitro incubated rabbit trachea was evoked by electrical field stimulation (3 Hz, 540 pulses) and quantified by high performance liquid chromatography with electrochemical detection. The muscarine receptor agonist oxotremorine inhibited the evoked release of noradrenaline completely at 1 mumol/l (EC50: 64 nmol/l). The concentration response curve was very steep (Hill coefficient of 2.3). Scopolamine shifted the concentration response curve of oxotremorine to the right (-log KB 8.48) demonstrating specific, inhibitory m…

Malemedicine.medical_specialtyIndomethacinCholinergic AgentsEndogenyMuscarinic AntagonistsIn Vitro TechniquesMuscarinic AgonistsHigh-performance liquid chromatographyReceptor subtypechemistry.chemical_compoundNorepinephrineAdrenergic AgentsInternal medicineMuscarinic acetylcholine receptormedicineAnimalsReceptorPharmacologyMuscarineBinding SitesPhenoxybenzamineChemistryRabbit (nuclear engineering)General Medicinerespiratory systemReceptors MuscarinicIn vitroElectric StimulationTracheaEndocrinologyFemaleRabbitsNaunyn-Schmiedeberg's archives of pharmacology
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Muscarinic modulation of acetylcholine release: Receptor subtypes and possible mechanisms

1989

The release of acetylcholine from central and peripheral neurones can be inhibited and facilitated by muscarine autoreceptors, i.e. receptors located on the cholinergic neurone. In the last few years evidence has accumulated that muscarine receptors are heterogeneous. This chapter describes attempts that have been made to classify the muscarine autoreceptors. In addition, some possible mechanisms behind the neuronal muscarine receptors are examined.

MuscarineMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2Muscarinic agonistchemistry.chemical_compoundnervous systemchemistryMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4NeuroscienceAcetylcholinemedicine.drug
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Validation of Fenoterol to Study β<sub>2</sub>-Adrenoceptor Function in the Rat Urinary Bladder

2021

Fenoterol is a β<sub>2</sub>-adrenoceptor (AR)-selective agonist that is commonly used to investigate relaxation responses mediated by β<sub>2</sub>-AR in smooth muscle preparations. Some data have questioned this because fenoterol had low potency in the rat urinary bladder when a muscarinic agonist was used as a pre-contraction agent and because some investigators proposed that fenoterol may act in part via β<sub>3</sub>-AR. We designed the present study to investigate whether fenoterol is a proper pharmacological tool to study β<sub>2</sub>-AR-mediated relaxation responses in the rat urinary bladder. Firstly, we have compared the effect of p…

PharmacologyAgonistCarbacholUrinary bladderChemistrymedicine.drug_classAntagonistGeneral Medicinerespiratory systemPharmacologyRat Urinary BladderMuscarinic agonistmedicine.anatomical_structuremedicinePotencyFenoterolmedicine.drugPharmacology
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Differential effects of anandamide on acetylcholine release in the guinea-pig ileum mediated via vanilloid and non-CB1 cannabinoid receptors

2001

The effects of anandamide on [3H]-acetylcholine release and muscle contraction were studied on the myenteric plexus-longitudinal muscle preparation of the guinea-pig ileum preincubated with [3H]-choline. Anandamide increased both basal [3H]-acetylcholine release (pEC50 6.3) and muscle tone (pEC50 6.3). The concentration-response curves for anandamide were shifted to the right by 1 μM capsazepine (pKB 7.5 and 7.6), and by the combined blockade of NK1 and NK3 tachykinin receptors with the antagonists CP99994 plus SR142801 (each 0.1 μM). The CB1 and CB2 receptor antagonists, SR141716A (1 μM) and SR144528 (30 nM), did not modify the facilitatory effects of anandamide. Anandamide inhibited the e…

Pharmacologymedicine.medical_specialtyCannabinoid receptormedicine.medical_treatmentTRPV1AnandamideMuscarinic agonistchemistry.chemical_compoundEndocrinologychemistryInternal medicinemedicineCannabinoidCapsazepineTachykinin receptorAcetylcholinemedicine.drugBritish Journal of Pharmacology
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