Search results for "Muscarinic"

showing 10 items of 263 documents

Effects of ouabain on human bronchial muscle in vitro

2003

The effects of ouabain, an inhibitor of the plasmalemmal Na(+)/K(+)-ATPase activity, were examined in human isolated bronchus. Ouabain produced concentration-dependent contraction with -logEC(50)=7.16+/-0.11 and maximal effect of 67+/-4% of the response to acetylcholine (1 mM). Ouabain (10 microM)-induced contraction was epithelium-independent and was not depressed by inhibitors of cyclooxygenase and lipoxygenase, antagonists of muscarinic, histamine H(1)-receptors and alpha-adrenoceptors, or neuronal Na(+) channel blockade. The inhibition of ouabain contraction in tissues bathed in K(+)-free medium, and the inhibition by ouabain of the K(+)-induced relaxation confirm that the contractile a…

NitroprussideCromakalimmedicine.medical_specialtySodium-Hydrogen ExchangersTime FactorsInositol PhosphatesMuscle RelaxationVasodilator AgentsBronchiIn Vitro TechniquesOuabainMembrane Potentialschemistry.chemical_compoundSodium Potassium Chloride Symporter InhibitorsInternal medicineMuscarinic acetylcholine receptormedicineHumansVasoconstrictor AgentsNa+/K+-ATPaseOuabainInositol phosphateProtein Kinase CPharmacologychemistry.chemical_classificationForskolinColforsinIsoproterenolMuscle SmoothGeneral MedicineCalcium Channel BlockersAcetylcholineAmilorideEndocrinologychemistryCalciumSodium-Potassium-Exchanging ATPaseHistamineAcetylcholineHistaminemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Economic impact of mepolizumab in uncontrolled severe eosinophilic asthma, in real life

2021

Abstract Background and aims Severe asthma is burdened by frequent exacerbations and use of oral corticosteroids (OCS) which worsen patients’ health and increase healthcare spending. Aim of this study was to assess the clinical and economic effect of adding mepolizumab (MEP) for the treatment of these patients. Methods Patients >18 years old, referred to 8 asthma clinics, starting MEP between May 2017 and December 2018, were enrolled and followed-up for 12 months. Information in the 12 months before mepolizumab were collected retrospectively. The evaluation parameters included: OCS use, number of exacerbations/hospitalizations, concomitant therapies, comorbidity, and annual number of workin…

OR Odds RatioPediatricsSevere asthmaExacerbationAnti IL-5; Comorbidities; Mepolizumab; OCS; Pharmacoeconomics; Severe asthmagastroesophageal reflux diseaseSettore MED/10 - MALATTIE DELL'APPARATO RESPIRATORIOICS inhaled corticosteroidRate ratioOCS Oral Corticosteroidslaw.inventionComorbiditiesLAMA long acting muscarinic antagonist0302 clinical medicineRandomized controlled trialfractional nitric oxideInterquartile rangelawlong acting beta 2 agonistOdds RatioImmunology and AllergyRR Rate Ratio030223 otorhinolaryngologyPharmacoeconomicLOS Length of stayLOSIQRLAMAMEP MepolizumabORCISD Standard DeviationMEPPharmacoeconomicsACT Asthma Control TestComorbiditieCI Confidence Intervalsmedicine.druglcsh:Immunologic diseases. AllergyPulmonary and Respiratory Medicinemedicine.medical_specialtyinterquartile rangelong acting muscarinic antagonistImmunologyLABALABA long acting beta 2 agonistComorbidities Mepolizumab OCS Pharmacoeconomics Severe asthma Anti IL-5RRArticleRate Ratiochronic obstructive pulmonary disease03 medical and health sciencesPharmacoeconomicsOCS Oral CorticosteroidAsthma Control TestConfidence IntervalsFeNO fractional nitric oxideRCTs Randomized Controlled TrialmedicineCOPDGERD gastroesophageal reflux diseaseFeNOIQR interquartile rangeMepolizumabSDAsthmaRCTsOral Corticosteroidsbusiness.industryGERDmedicine.diseaseICS inhaled corticosteroidsACTComorbidityRandomized Controlled TrialsCI Confidence IntervalRCTs Randomized Controlled TrialsOCSCOPD chronic obstructive pulmonary disease030228 respiratory systemICSStandard DeviationLength of stayAnti IL-5inhaled corticosteroidslcsh:RC581-607businessMepolizumab
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Novel modes of rhythmic burst firing at cognitively-relevant frequencies in thalamocortical neurons.

2008

It is now widely accepted that certain types of cognitive functions are intimately related to synchronized neuronal oscillations at both low (alpha/theta) (4-7/8-13 Hz) and high (beta/gamma) (18-35/30-70 Hz) frequencies. The thalamus is a key participant in many of these oscillations, yet the cellular mechanisms by which this participation occurs are poorly understood. Here we describe how, under appropriate conditions, thalamocortical (TC) neurons from different nuclei can exhibit a wide array of largely unrecognised intrinsic oscillatory activities at a range of cognitively-relevant frequencies. For example, both metabotropic glutamate receptor (mGluR) and muscarinic Ach receptor (mAchR) …

Periodicity* Cognition* Acetylcholine; * Metabotropic glutamate receptor; * Lateral geniculate nucleus; * Intralaminar nucleus; * Oscillations; * EEG; * Cognition; * Perception; * Memory* EEGAction PotentialsSettore BIO/09 - FisiologiaIon ChannelsArticle* PerceptionBurstingThalamusBiological Clocks* Lateral geniculate nucleuMuscarinic acetylcholine receptorNeural PathwaysmedicineAnimalsHumans* Metabotropic glutamate receptorMolecular BiologyCerebral CortexNeurons* OscillationChemistryGeneral Neuroscience* Intralaminar nucleuGlutamate receptorReceptors NeurotransmitterElectrophysiology* MemoryMetabotropic receptormedicine.anatomical_structure* AcetylcholineMetabotropic glutamate receptorWakefulnessNeurology (clinical)NeuronNeuroscienceDevelopmental Biology
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Validation of Fenoterol to Study β<sub>2</sub>-Adrenoceptor Function in the Rat Urinary Bladder

2021

Fenoterol is a β<sub>2</sub>-adrenoceptor (AR)-selective agonist that is commonly used to investigate relaxation responses mediated by β<sub>2</sub>-AR in smooth muscle preparations. Some data have questioned this because fenoterol had low potency in the rat urinary bladder when a muscarinic agonist was used as a pre-contraction agent and because some investigators proposed that fenoterol may act in part via β<sub>3</sub>-AR. We designed the present study to investigate whether fenoterol is a proper pharmacological tool to study β<sub>2</sub>-AR-mediated relaxation responses in the rat urinary bladder. Firstly, we have compared the effect of p…

PharmacologyAgonistCarbacholUrinary bladderChemistrymedicine.drug_classAntagonistGeneral Medicinerespiratory systemPharmacologyRat Urinary BladderMuscarinic agonistmedicine.anatomical_structuremedicinePotencyFenoterolmedicine.drugPharmacology
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Muscarinic acetylcholine receptor trafficking in streptolysin O-permeabilized MDCK cells.

1996

We investigated the validity of streptolysin O (SLO)-permeabilized Madin-Darbin canine kidney (MDCK) cells which express muscarinic acetylcholine receptors (mAChRs) coupled to pertussis toxin-sensitive guanine nucleotide-binding proteins (G proteins) for the study of the molecular machinery that regulated mAChR internalization and recycling. Exposure of SLO-permeabilized cells to carbachol-reduced cell surface receptor number by up to 40% without changing total receptor number. The kinetics and maximal extent of receptor internalization as well as the potency of carbachol to induce receptor internalization were almost identical in SLO-permeabilized and non-permeabilized cells. Using this se…

PharmacologyG protein-coupled receptor kinasemedia_common.quotation_subjectB-cell receptorMuscarinic acetylcholine receptor M3General MedicineMuscarinic acetylcholine receptor M1BiologyKidneyReceptors MuscarinicPermeabilityCell biologyAdenosine TriphosphateDogsBacterial ProteinsCell surface receptorGTP-Binding ProteinsGuanosine 5'-O-(3-Thiotriphosphate)Muscarinic acetylcholine receptor M5StreptolysinsEnzyme-linked receptorAnimalsInternalizationCells Culturedmedia_commonNaunyn-Schmiedeberg's archives of pharmacology
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Murine embryonic stem cell line CGR8 expresses all subtypes of muscarinic receptors and multiple nicotinic receptor subunits: Down-regulation of α4- …

2015

Non-neuronal acetylcholine mediates its cellular effects via stimulation of the G-protein-coupled muscarinic receptors and the ligand-gated ion channel nicotinic receptors. The murine embryonic stem cell line CGR8 synthesizes and releases non-neuronal acetylcholine. In the present study a systematic investigation of the expression of nicotinic receptor subunits and muscarinic receptors was performed, when the stem cells were grown in the presence or absence of LIF, as the latter condition induces early differentiation. CGR8 cells expressed multiple nicotinic receptor subtypes (α3, α4, α7, α9, α10, β1, β2, β3, β4, γ, δ, e) and muscarinic receptors (M1, M3, M4, M5); M2 was detected only in 2 …

PharmacologyImmunologyMuscarinic acetylcholine receptor M3Down-RegulationMuscarinic acetylcholine receptor M2Cell DifferentiationMuscarinic acetylcholine receptor M1BiologyReceptors NicotinicReceptors MuscarinicCell biologyCell LineMiceProtein SubunitsNicotinic agonistGanglion type nicotinic receptorGene Expression RegulationMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptorImmunology and AllergyAnimalsAlpha-4 beta-2 nicotinic receptorEmbryonic Stem CellsInternational immunopharmacology
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The variation of acetylcholine release from myenteric neurones with stimulation frequency and train length. Role of presynaptic muscarine receptors.

1983

1. The effects of scopolamine on the release of 3H-acetylcholine (ACh) from the guinea-pig myenteric plexus were studied at different stimulation frequencies (0.03–10 Hz) and train lengths (1–180 pulses). Release of 3H-ACh was measured in the absence of cholinesterase inhibitors as the outflow of tritium from myenteric plexus-longitudinal muscle preparations preloaded with 3H-choline. 2. In control experiments the volley output of 3H-ACh declined with increasing train length and increasing stimulation frequency. Stimulation by one pulse produced the highest volley output. 3. Scopolamine facilitated the evoked output of 3H-ACh via blockade of presynaptic muscarine receptors. A significant in…

PharmacologyMuscarinePulse (signal processing)Guinea PigsScopolamineMyenteric PlexusStimulationGeneral MedicineInhibitory postsynaptic potentialReceptors MuscarinicAcetylcholineElectric StimulationFeedbackchemistry.chemical_compoundchemistryNegative feedbackMuscarinic acetylcholine receptormedicineAnimalsNeuroscienceAcetylcholineMyenteric plexusmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Acetylcholine release at motor endplates and autonomic neuroeffector junctions: a comparison.

1996

Acetylcholine released at motor endplates and at autonomic neuroeffector junctions binds to nicotinic and muscarinic receptors to affect the activity of the corresponding target cells. Additionally, nicotonic and muscarinic receptors modulate various intracellular regulatory pathways (second messengers, gene expression) and mediate trophic effects. To maintain homeostasis of the individual cell and of the whole organism the release of acetylcholine has to be strictly controlled within both nervous systems. The basic events of synthesis, storage, and release are comparable at motoneurones and autonomic neurones, but mechanisms regulating transmitter release appear to differ. The motor endpla…

PharmacologyMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2BiologyMotor EndplateReceptors MuscarinicAcetylcholineNeuroeffector junctionNicotinic agonistMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptorMuscarinic acetylcholine receptor M4medicineNeuroeffector JunctionAnimalsNeuroscienceAcetylcholinemedicine.drugPharmacological research
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The cholinergic 'pitfall': acetylcholine, a universal cell molecule in biological systems, including humans.

1999

1. Acetylcholine (ACh) represents one of the most exemplary neurotransmitters. In addition to its presence in neuronal tissue, there is increasing experimental evidence that ACh is widely expressed in pro- and eukaryotic non-neuronal cells. Thus, ACh has been detected in bacteria, algae, protozoa, tubellariae and primitive plants, suggesting an extremely early appearance of ACh in the evolutionary process. 2. In humans, ACh and/or the synthesizing enzyme, choline acetyltransferase, has been demonstrated in epithelial cells (airways, alimentary tract, urogenital tract, epidermis), mesothelial (pleura, pericardium) and endothelial and muscle cells. In addition, immune cells express the non-ne…

PharmacologyNeuronsPhysiologyCellular differentiationBiologyCholine acetyltransferaseAcetylcholineCell biologyEvolution MolecularParacrine signallingNicotinic agonistBiochemistryPhysiology (medical)Muscarinic acetylcholine receptormedicineCholinergicAnimalsCholinesterasesHumansAcetylcholinemedicine.drugCalcium signalingClinical and experimental pharmacologyphysiology
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Acetylcholine beyond neurons: the non-neuronal cholinergic system in humans

2008

Animal life is controlled by neurons and in this setting cholinergic neurons play an important role. Cholinergic neurons release ACh, which via nicotinic and muscarinic receptors (n- and mAChRs) mediate chemical neurotransmission, a highly integrative process. Thus, the organism responds to external and internal stimuli to maintain and optimize survival and mood. Blockade of cholinergic neurotransmission is followed by immediate death. However, cholinergic communication has been established from the beginning of life in primitive organisms such as bacteria, algae, protozoa, sponge and primitive plants and fungi, irrespective of neurons. Tubocurarine- and atropine-sensitive effects are obser…

PharmacologyNicotinic agonistMuscarinic acetylcholine receptormedicineCholinergicBiologySignal transductionCholinergic neuronReceptorNeuroscienceAcetylcholineAcetylcholine receptormedicine.drugBritish Journal of Pharmacology
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