Search results for "Mutant"

showing 10 items of 670 documents

Bacteroides vulgatus protects against escherichia coli-induced colitis in gnotobiotic interleukin-2-deficient mice

2003

Abstract Background & Aims: The microflora plays a crucial role in inflammatory bowel diseases (IBDs). Specific pathogen-free (SPF), but not germ-free, interleukin (IL)-2-deficient (IL-2−/−) mice develop colitis. The colitogenicity of commensal bacteria was determined. Methods: Gnotobiotic IL-2−/− and IL-2+/+ mice were colonized with Escherichia coli mpk, Bacteroides vulgatus mpk, or both bacterial strains, or with E. coli strain Nissle 1917. DNA arrays were used to characterize E. coli mpk. Colitis was analyzed by histology and real-time reverse-transcription polymerase chain reaction (RT-PCR) for interferon (IFN)-γ, tumor necrosis factor (TNF)-α, IL-10, and CD14 messenger RNA (mRNA) expre…

MaleGene Expressionmedicine.disease_causeMicrobiologyFecesMiceInterferonEscherichia colimedicineAnimalsBacteroidesGerm-Free LifeColitisEscherichia coliBacteroidaceaeEscherichia coli InfectionsSpecific-pathogen-freeHepatologybiologyGastroenterologyInterleukinColitismedicine.diseasebiology.organism_classificationEnterobacteriaceaeMice Mutant StrainsSpecific Pathogen-Free OrganismsIntestinesMice Inbred C57BLInterleukin-2FemaleTumor necrosis factor alphamedicine.drugGastroenterology
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ALDH1A3 Mutations Cause Recessive Anophthalmia and Microphthalmia

2013

Anophthalmia and microphthalmia (A/M) are early-eye-development anomalies resulting in absent or small ocular globes, respectively. A/M anomalies occur in syndromic or nonsyndromic forms. They are genetically heterogeneous, some mutations in some genes being responsible for both anophthalmia and microphthalmia. Using a combination of homozygosity mapping, exome sequencing, and Sanger sequencing, we identified homozygosity for one splice-site and two missense mutations in the gene encoding the A3 isoform of the aldehyde dehydrogenase 1 (ALDH1A3) in three consanguineous families segregating A/M with occasional orbital cystic, neurological, and cardiac anomalies. ALDH1A3 is a key enzyme in the…

MaleGenetic LinkageRetinoic acidGenes RecessiveBiologymedicine.disease_causeMicrophthalmiachemistry.chemical_compoundsymbols.namesakeChromosome SegregationReportmedicineGeneticsFood and NutritionHumansMicrophthalmosMissense mutationGenetics(clinical)Genetics (clinical)Exome sequencingSanger sequencingGeneticsMutationAnophthalmiaHomozygoteAnophthalmosExonsSequence Analysis DNAAldehyde DehydrogenaseDisease gene identificationmedicine.diseaseAldehyde OxidoreductasesMolecular biologyIntronseye diseasesPedigreeHEK293 CellschemistryAlimentation et NutritionMutationsymbolsFemaleMutant Proteinssense organsThe American Journal of Human Genetics
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Ehlers-Danlos syndrome type VII: phenotype and genotype

1994

A patient suffering from a severe form of Ehlers-Danlos syndrome is presented (EDS type VII). The presence of bilateral congenital hip dislocation, generalized joint hypermobility and a soft hyperelastic skin with abnormal scarring suggested a specific collagen type I defect. SDS-PAGE analysis of collagens secreted into the medium of fibroblast cultures showed a retarded migration of more than half of the alpha 2(I) chains. CNBr peptide mapping of the HPLC-purified altered chain localized the mutant locus to the N-terminal region of the protein. cDNA analysis of the corresponding gene COL1A2 revealed, in addition to the expected collagen sequence, a transcript missing the entire exon 6. Thi…

MaleGeneticsSplice site mutationBase SequenceGenotypeChemistryMolecular Sequence DataMutantIntronLocus (genetics)ExonsDermatologyGeneral Medicinemedicine.diseaseCollagen type I alpha 1ExonPhenotypeEhlers–Danlos syndromeChild PreschoolMutationmedicineHumansEhlers-Danlos SyndromeCollagenGeneArchives of Dermatological Research
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A novel KCNQ3 mutation in familial epilepsy with focal seizures and intellectual disability

2015

Mutations in the KCNQ2 gene encoding for voltage-gated potassium channel subunits have been found in patients affected with early onset epilepsies with wide phenotypic heterogeneity, ranging from benign familial neonatal seizures (BFNS) to epileptic encephalopathy with cognitive impairment, drug resistance, and characteristic electroencephalography (EEG) and neuroradiologic features. By contrast, only few KCNQ3 mutations have been rarely described, mostly in patients with typical BFNS. We report clinical, genetic, and functional data from a family in which early onset epilepsy and neurocognitive deficits segregated with a novel mutation in KCNQ3 (c.989G>T; p.R330L). Electrophysiological stu…

MaleGenotype-phenotype correlationmedicine.medical_specialtyNeurologyBenign familial neonatal seizuresMutantGenotype-phenotype correlationsmedicine.disease_causeMutagenesiKCNQ3 Potassium ChannelEpilepsyKCNQBenign Familial Neonatal Seizures KCNQ cognitive impairment voltage-gated potassium channels epilepsy mutagenesis genotype-phenotype correlationsSeizuresSettore M-PSI/08 - Psicologia ClinicaIntellectual DisabilityIntellectual disabilitymedicineHumansKCNQ2 Potassium ChannelVoltage-gated potassium channelBenign familial neonatal seizuresGenetic Predisposition to DiseaseGenetic TestingChildGenetic testingGeneticsMutationEpilepsymedicine.diagnostic_testGenetic heterogeneitybusiness.industryMedicine (all)Benign familial neonatal seizures; Cognitive impairment; Epilepsy; Genotype-phenotype correlations; KCNQ; Mutagenesis; Voltage-gated potassium channels; Child; Female; Genetic Testing; Humans; Intellectual Disability; KCNQ2 Potassium Channel; KCNQ3 Potassium Channel; Male; Mutation; Pedigree; Seizures; Genetic Predisposition to Disease; Neurology (clinical); Neurology; Medicine (all)Benign familial neonatal seizuremedicine.diseaseSeizureSettore MED/39 - Neuropsichiatria InfantilePedigreeCognitive impairmentNeurologyMutagenesisMutationFemaleNeurology (clinical)businessVoltage-gated potassium channelsHuman
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Functional analysis of a rare HBV deletion mutant in chronically infected children.

2003

Liver damage caused by chronic hepatitis B virus (HBV) infection may be enhanced through the selection of deleted HBV preS mutants by intracellular accumulation of viral proteins and subsequent cell death. However, the prevalence and impact of such mutants on the clinical course of infection have not yet been studied in children. Serum samples from 60 children (mean age 9.8 y) were investigated by means of PCR and direct sequencing of the entire preS region. Only one patient (1.5%) was found with a mixed HBV population of a deletion spanning 183 nucleotides and wild-type sequences. This mutation alters the HBV large-surface protein and removes the small-surface promoter. To clarify the sign…

MaleHepatitis B virusAdolescentPopulationMutantmedicine.disease_causeEndoplasmic ReticulumPolymerase Chain ReactionVirusHepatitis B ChronicOrthohepadnavirusmedicineHumanseducationChildDNA PrimersSequence DeletionHepatitis B viruseducation.field_of_studyMutationExpression vectorbiologyBase SequenceInfantbiology.organism_classificationVirologyHepadnaviridaeChild PreschoolPediatrics Perinatology and Child HealthFemalePediatric research
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Compound heterozygosity in the SPG4 gene causes hereditary spastic paraplegia

2008

The SPG4 gene is frequently mutated in autosomal dominant form of hereditary spastic paraplegia (HSP). We report that the compound heterozygous sequence variants S44L, a known polymorphism, and c.1687G>A, a novel mutation in SPG4 cause a severe form of HSP in a patient. The family members carrying solely c.1687G>A mutation are asymptomatic for HSP. The reverse transcriptase-polymerase chain reaction (RT-PCR) analysis revealed that the c.1687G>A mutation is a splice site mutation and causes skipping of the exon 15 of spastin. Furthermore, quantification of RT-PCR products by sequencing and quantification of allele-specific expression by pyrosequencing assay revealed that c.1687G>A is a leaky…

MaleHeterozygoteSpastinHereditary spastic paraplegiaDNA Mutational AnalysisMolecular Sequence DataMutantIntracellular SpaceBiologyCompound heterozygositySpastinPolymorphism Single NucleotideWhite PeopleLoss of heterozygosity03 medical and health sciencesExon0302 clinical medicineGermanyGeneticsmedicineHumansRNA MessengerAllelesGenetics (clinical)030304 developmental biologyAdenosine TriphosphatasesRegulation of gene expressionGenetics0303 health sciencesSplice site mutationBase SequenceSpastic Paraplegia HereditaryComputational BiologyExonsmedicine.diseasePedigreeProtein TransportAmino Acid SubstitutionGene Expression RegulationMutationFemaleRNA Splice Sites030217 neurology & neurosurgeryHeLa CellsClinical Genetics
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Diminished Contact Hypersensitivity Response in IL‐4 Deficient Mice at a Late Phase of the Elicitation Reaction

1997

Contact hypersensitivity (CHS) is thought to depend on the activation of T cells of Th1 and/or Tc1 type. The role of Th2/Tc2 cells in the contact allergic reaction is not clear. The aim of this study was to analyse the functional contribution of Th2/Tc2 cells in CHS using the interleukin-4 (IL-4) deficient mouse model. Interleukin-4 deficient (IL4T) and control (wt) mice were sensitized by epicutaneous application of 2,4-dinitrofluorobenzene. The ear swelling response measured 24 h after challenge was similar in IL4T and control mice. However, from 48 h onwards, ear swelling values were significantly reduced in IL4T mice. The stimulatory capacity of freshly isolated as well as 3-day culture…

MaleImmunologyPopulationCellCell CountBiologyDermatitis ContactLymphocyte ActivationFlow cytometryMiceT-Lymphocyte SubsetsmedicineAnimalseducationInterleukin 4SkinMice Inbred BALB Ceducation.field_of_studymedicine.diagnostic_testEpidermis (botany)EffectorT-cell receptorContact hypersensitivityReceptors Antigen T-Cell gamma-deltaDendritic CellsGeneral MedicineFlow CytometryMolecular biologyMice Mutant StrainsMice Inbred C57BLmedicine.anatomical_structureLangerhans CellsImmunologyFemaleInterleukin-4EpidermisScandinavian Journal of Immunology
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Mutant K-ras2 in serum

2003

Mutant tumour derived DNA has been detected in the sera of colorectal cancer patients. We investigated if mutant serum KRAS2 was detectable preoperatively in a large group of patients with colorectal neoplasia. A prospective study of 94 patients who underwent putative curative resection for colorectal carcinoma (CRC) was performed to ascertain if serum mutant KRAS2 could be used postoperatively as a disease marker.Preoperative sera from 78 patients were analysed (group A). Sera from 94 patients were obtained three monthly for up to three years during the postoperative period (group B). Codon 12 and 13 KRAS2 mutations were analysed in matched tumour and serum samples.In the preoperative grou…

MaleLetterColorectal cancervirusesMutantDNA Mutational AnalysisBioinformaticsProto-Oncogene Proteins p21(ras)03 medical and health sciencesCollaborative group0302 clinical medicineProto-Oncogene ProteinsMedicineHumansRas2neoplasmsGene030304 developmental biologyAged0303 health sciencesbusiness.industryPoint mutationGastroenterologyDNA NeoplasmMiddle Agedmedicine.diseasePrognosis3. Good healthProto-Oncogene Proteins p21(ras)Molecular analysisCarcinoembryonic AntigenEpidemiologic Studies030220 oncology & carcinogenesisCancer researchras ProteinsFemaleNeoplasm Recurrence LocalbusinessColorectal NeoplasmsBiomarkers
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Forward genetics inWolbachia: Regulation ofWolbachiaproliferation by the amplification and deletion of an addictive genomic island

2021

Copyright: © 2021 Duarte et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

MaleLife CyclesCancer ResearchPhysiologyEggsMutantForward geneticsQH426-470LarvaeReproductive PhysiologyTiter regulationGenomic islandreproductive and urinary physiologyGenetics (clinical)Gene EditingGenetics0303 health sciencesbiologyEukaryotaAnimal ModelsGenomicsPhenotype3. Good healthInsectsPhenotypeDrosophila melanogasterExperimental Organism SystemsDicistroviridaeOctomomFemaleDrosophilaWolbachiaDrosophila melanogasterWolbachiaResearch ArticleGenomic IslandsArthropodaLongevityGenomicsResearch and Analysis MethodsInvertebrate genomics03 medical and health sciencesModel Organismsparasitic diseasesGeneticsAnimalsSymbiosisMolecular BiologyEcology Evolution Behavior and Systematics030304 developmental biologyBacteria030306 microbiologyHost (biology)OrganismsBiology and Life SciencesSingle nucleotide polymorphismsbiochemical phenomena metabolism and nutritionbiology.organism_classificationInvertebratesBacterial LoadForward geneticsAnimal GenomicsAnimal StudiesbacteriaZoologyEntomologyGenome BacterialDevelopmental BiologyGenetic screen
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IL-5 Enhances in Vitro and in Vivo Antigen-Specific IgA Production in MHC Genetically Determined Low IL-5 Responder Mice

1995

Lymphonode cells from BALB/k mice, but not from BALB/c mice, immunized with picryl chloride (PCl) produce IL-5 when stimulated with the specific antigen in vitro and this correlates with picryl-specific IgA levels in vivo, which are 6 to 10 times higher in BALB/k mice. B lymphocytes from BALB/k mice cultured with PCl-immune T cells from BALB/k produce in vivo anti-PCl-IgA, while B lymphocytes from BALB/c mice, cultured with T cells from BALB/c mice, fail to produce appreciable amounts of anti-PCl IgA, unless IL-5 is added to cultures. B lymphocytes from both strains of mice produce similar amounts of total IgA antibodies when stimulated in vitro with lipopolysaccharide. In vivo administrati…

MaleLipopolysaccharideImmunologyPicryl ChlorideMajor histocompatibility complexMajor Histocompatibility ComplexPicryl chlorideEpitopesMicechemistry.chemical_compoundAntigenIn vivoAnimalsInterleukin 5Cells CulturedMice Inbred BALB CbiologyMolecular biologyMice Mutant StrainsRecombinant ProteinsIn vitroImmunoglobulin Achemistrybiology.proteinInterleukin-5AntibodyInjections IntraperitonealCellular Immunology
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