Search results for "Mutation."

showing 10 items of 2808 documents

Systematic search for neutropenia should be part of the first screening in patients with poikiloderma.

2011

Poikiloderma occurs in a number of hereditary syndromes, the best known of which is Rothmund-Thomson syndrome (RTS). Differential diagnoses include Dyskeratosis Congenita (DC) with high genetic heterogeneity and Clericuzio-type Poikiloderma with Neutropenia (CPN) due to mutations in the C16orf57 gene. Mutations in the RECQL4 gene are only observed in two thirds of RTS patients. In this study, 10 patients referred for syndromic poikiloderma and negative for RECQL4 sequencing analysis were investigated for C16orf57 mutations. Two C16orf57 heterozygous nonsense mutations (p.W81X and p.Y89X) were identified in a 5-year-old female child presenting with generalized poikiloderma, dental dysplasia,…

medicine.medical_specialtyHeterozygoteNeutropeniaNonsense mutationPoikilodermaNeutropeniaDiagnosis DifferentialGeneticsmedicineHumansAbnormalities MultipleGenetic TestingGenetics (clinical)Genetic testingRetrospective StudiesGeneticsmedicine.diagnostic_testRecQ HelicasesGenetic heterogeneitybusiness.industryRothmund-Thomson SyndromeGeneral Medicinemedicine.diseaseDermatologyPedigreePalmoplantar keratodermaCodon NonsenseChild PreschoolAbsolute neutrophil countErythrocyte CountFemalebusinessDyskeratosis congenitaEuropean journal of medical genetics
researchProduct

Identification of a novel mutation of MTP gene in a patient with abetalipoproteinemia.

2011

Abetalipoproteinemia (ABL), or Bassen-Kornzweig syndrome, is a rare autosomal recessive disorder of lipoprotein metabolism, characterized by fat malabsorption, hypocholesterolemia retinitis pigmentosa, progressive neuropathy and acanthocytosis from early infancy. We describe the clinical and molecular characterization of a 6-month-old infant born of consanguineous, apparently healthy parents from Iran. The patient was hospitalized because of failure to thrive, greasy stool and vomiting. The patient's serum lipid profile, the clinical phenotype and the duodenal histology suggested the clinical diagnosis of ABL. The MTP gene analysis by direct sequencing revealed a novel homozygous mutation (…

medicine.medical_specialtyHeterozygoteSettore MED/09 - Medicina InternaDuodenumSpecialties of internal medicineInternal medicineRetinitis pigmentosamedicineHumansgeneticsFamily HealthMTP gene mutations.ABLHepatologymedicine.diagnostic_testApoB-containing lipoproteins.business.industryAbetalipoproteinemiaInfantAbetalipoproteinemia.Heterozygote advantageGeneral Medicinemedicine.diseaseLipidsAbetalipoproteinemiaFat malabsorptionHypocholesterolemiaEndocrinologyPhenotypeRC581-951Failure to thriveFemaleHypocholesterolemia.medicine.symptomLipid profilebusinessCarrier Proteins
researchProduct

Genetic variants in the MTHFR are not associated with fatty liver disease.

2020

The common missense sequence variants of methylenetetrahydrofolate reductase (MTHFR), rs1801131 (c.A1298C) and rs1801133 (c.C677T), favour the development of hyperhomocysteinemia and diminished DNA methylation. Previous studies, carried out in small series and with suboptimal characterization of the hepatic phenotype, tested the association of these genetic variants with fatty liver disease (FLD), with conflicting results. Here, we assessed the association of rs1801131 and rs1801133 with hepatic phenotype in the Liver Biopsy Cross-Sectional Cohort, a large cohort (n=1375 from Italy and 411 from Finland) of European individuals with suspect FLD associated with dysmetabolism. A total of 1786 …

medicine.medical_specialtyHyperhomocysteinemiaGenotypeGastroenterologyPolymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineNAFLDInternal medicinesteatosisMedicineMissense mutationHumansGenetic Predisposition to DiseaseFinlandMethylenetetrahydrofolate Reductase (NADPH2)Hepatologymedicine.diagnostic_testbiologybusiness.industryFatty liverNASHmedicine.diseaseFatty LiverCross-Sectional StudiesItaly030220 oncology & carcinogenesisLiver biopsyMethylenetetrahydrofolate reductaseCase-Control StudiesMTHFRDNA methylationCohortbiology.proteinfatty liver disease030211 gastroenterology & hepatologySteatosisfibrosibusinessLiver international : official journal of the International Association for the Study of the LiverREFERENCES
researchProduct

Arterial thrombosis in Philadelphia-negative myeloproliferative neoplasms predicts second cancer: a case-control study.

2020

Abstract Patients with Philadelphia-negative myeloproliferative neoplasm (MPN) are prone to the development of second cancers, but the factors associated with these events have been poorly explored. In an international nested case-control study, we recruited 647 patients with carcinoma, nonmelanoma skin cancer, hematological second cancer, and melanoma diagnosed concurrently or after MPN diagnosis. Up to 3 control patients without a history of cancer and matched with each case for center, sex, age at MPN diagnosis, date of diagnosis, and MPN disease duration were included (n = 1234). Cases were comparable to controls for MPN type, driver mutations and cardiovascular risk factors. The freque…

medicine.medical_specialtyImmunologyKaplan-Meier EstimateGene mutationBiochemistryGastroenterologyMyeloproliferative neoplasms03 medical and health sciences0302 clinical medicineSDG 3 - Good Health and Well-beingInternal medicineCarcinomaMedicineHumansPhiladelphia ChromosomeMyeloproliferative neoplasmMyeloproliferative Disordersbusiness.industryCase-control studyCancerfood and beveragesMyeloproliferative neoplasmssecond cancersarterial eventsNeoplasms Second PrimaryThrombosisCell BiologyHematologyOdds ratioArteriesmedicine.diseasesecond cancersThrombosisSettore MED/15 - MALATTIE DEL SANGUEarterial events030220 oncology & carcinogenesisCase-Control StudiesMultivariate Analysis/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingSkin cancerbusiness030215 immunologyFollow-Up StudiesBlood
researchProduct

Clinicogenetic Risk Models in Patients Randomized to Receive Consolidative Autologous Stem-Cell Transplantation after Frontline R-CHOP for Advanced F…

2018

Abstract Introduction: Advanced follicular lymphoma (FL) is a clinically and molecularly heterogeneous disease. About 20% of patients have early progression of disease (POD) and short overall survival (OS). We have previously shown that integration of lymphoma-specific gene mutations and clinical factors improves pretreatment risk stratification (Pastore, 2015) and prediction of early POD (i.e., within 24 months, POD24; Jurinovic, 2016). Recently, we have shown that high-dose therapy (HDT) followed by autologous stem-cell transplantation (ASCT) is an effective treatment option for eligible patients with high-risk disease as defined by POD24 (Jurinovic, 2018). Here, we aimed to explore wheth…

medicine.medical_specialtyIntention-to-treat analysisbusiness.industryImmunologyFollicular lymphomaCell BiologyHematologyGene mutationmedicine.diseaseBiochemistryTransplantationAutologous stem-cell transplantationInternational Prognostic IndexInternal medicineCohortmedicineIn patientbusinessBlood
researchProduct

A leptin missense mutation associated with hypogonadism and morbid obesity

1998

medicine.medical_specialtyLeptin DeficiencyInsulin bloodGenetics of obesityLeptinBiologymedicine.diseaseObesityMorbid obesityEndocrinologyInternal medicineMutation (genetic algorithm)GeneticsmedicineMissense mutationNature Genetics
researchProduct

The deubiquitinating enzyme CYLD regulates the differentiation and maturation of thymic medullary epithelial cells.

2014

The cross talk between thymocytes and the thymic epithelium is critical for T-cell development and the establishment of central tolerance. Medullary thymic epithelial cells (mTECs) are located in the thymic medulla and mediate the elimination of self-reactive thymocytes, thereby preventing the onset of autoimmunity. Previous studies identified the deubiquitinating enzyme CYLD as a critical regulator of T-cell development by activating proximal T-cell receptor signaling during the transition of double-positive to single-positive thymocytes. Here we evaluated the impact of the naturally occurring short-splice variant of the cyld gene (sCYLD) on the development and maturation of mTECs. We foun…

medicine.medical_specialtyMedullary cavityImmunologyReceptors Antigen T-CellCell CountThymus GlandBiologyDeubiquitinating Enzyme CYLDImmunophenotypingMiceInternal medicinemedicineImmunology and AllergyAnimalsMice KnockoutThymocytesUbiquitinationCell DifferentiationEpithelial CellsCell BiologyCell biologyDeubiquitinating Enzyme CYLDCysteine EndopeptidasesEndocrinologyPhenotypeAntigens SurfaceMutationFemaleSignal TransductionImmunology and cell biology
researchProduct

Pharmacological Characterization of Loss of Function Mutations of the Human Melanocortin 1 Receptor That Are Associated with Red Hair

2004

Variation in skin color is the major host risk factor for melanoma and other forms of skin cancer. Individuals with red hair show an increased ratio of phaeomelanin to eumelanin in both hair and skin. This ratio is regulated by the melanocortin (MC) 1 receptor. There are several common point mutations in the human MC1 receptor that are overrepresented in North European red-heads, and in individuals with pale skin. In order to determine the functional significance of these mutations, we expressed the Asp84Glu, Val92Met, Arg163Gln, and Asp294His variants of the human MC1 receptors in eukaryotic cells and determined their ability to bind alpha-melanocyte stimulating hormone (MSH) peptides and …

medicine.medical_specialtyMelanocyte-stimulating hormoneMolecular Sequence DataDermatologyBiologyKidneymedicine.disease_causeBiochemistrypolymorphismStructure-Activity RelationshipGPCRInternal medicineCyclic AMPmedicineHumansPoint MutationpigmentationAmino Acid SequencemelanocortinHair ColorReceptorMSHMolecular BiologyCells CulturedG protein-coupled receptorMutationintegumentary systemMelanomaPoint mutationCell Biologymedicine.diseaseProtein Structure TertiaryEndocrinologyalpha-MSHMelanocortinReceptor Melanocortin Type 1Melanocortin 1 receptorJournal of Investigative Dermatology
researchProduct

2015

Ocular signs in Fabry disease have generally been regarded to be primarily of diagnostic value. We explored whether ocular findings, alone or in particular in combination with the α-galactosidase A gene mutation, have predictive value for disease severity. Data from the Fabry Outcome Survey (FOS), a large, global database sponsored by Shire, were selected for adult patients who had undergone ophthalmological examination. Three ocular signs were assessed: cornea verticillata, tortuous conjunctival and/or retinal vessels, and cataract. Fabry disease severity was measured using FOS Mainz Severity Score Index and modifications thereof. Ophthalmological data were available for 1203 (699 female, …

medicine.medical_specialtyMultidisciplinarybusiness.industryAge adjustmentmedicine.diseaseFabry diseaseeye diseasesmedicine.anatomical_structureCataractsOphthalmologyInternal medicineCorneaGenotypemedicineMissense mutationCornea verticillatasense organsEye Findingmedicine.symptombusinessPLOS ONE
researchProduct

Mutational analysis of E-cadherin, β-catenin and APC genes in synovial sarcomas

2010

medicine.medical_specialtyMutationPathologyHistologyCadherinCell adhesion moleculeCancerAnatomical pathologyGeneral MedicineBiologymedicine.disease_causemedicine.diseaseSynovial sarcomaPathology and Forensic MedicineCateninmedicineCancer researchGeneHistopathology
researchProduct