Search results for "Myocardial Contraction"

showing 10 items of 102 documents

Effects of amsacrine (m-AMSA), a new aminoacridine antitumor drug, on the rabbit heart.

1983

There is emerging clinical evidence that amsacrine (m-AMSA) administration may be associated with cardiotoxic effects such as severe, even fatal, ventricular arrhythmias and impairment of the inotropic performance of the heart. Information on the cardiac effects of m-AMSA in animals is scanty. Studies on mice, dogs, and monkeys have not evidenced the cardiotoxicity of the compound. The data presented in this paper show that m-AMSA causes acute ECG alterations in normal rabbits and a dose-related negative inotropic effect on the isolated rabbit heart, suggesting that this species may be a useful model for the study of the cardiac actions of this antiblastic.

AmsacrineDose-Response Relationship DrugAminoacridinesHeart VentriclesAntineoplastic AgentsArrhythmias CardiacHeartModels BiologicalMyocardial ContractionCardiotoxicityElectrocardiographym-Amsaantitumor drugDepression ChemicalHeart Function Testscancer.AnimalsRabbitsCancer treatment reports
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Antimuscarinic action of quinidine on the heart? A study in myocardial preparations from cat hearts

1984

Quinidine exerts anticholinergic effects which have been ascribed to atropine-like properties of the drug. We have examined the effects of acetylcholine on the force of contraction in isolated heart muscle preparations from cats and compared the inhibitory effects of atropine with those of quinidine. The effects of acetylcholine were antagonized competitively in the presence of atropine. The Schild-plot yielded a straight line; the slope was not significantly different from unity. In the presence of quinidine, the concentration-response curve of acetylcholine was shifted to the right as with atropine, however, the Schild-plot yielded a regression line which was not linear; the slope was sta…

AtropineMaleQuinidineInotropemedicine.medical_specialtymedicine.drug_classAction PotentialsIn Vitro TechniquesPharmacologyParasympatholyticInternal medicinemedicineAnticholinergicAnimalsPhosphodiesterase inhibitorPharmacologyPapaverineChemistryCell MembraneParasympatholyticsMyocardial ContractionQuinidineAcetylcholineElectrophysiologyAtropineEndocrinologyCatsFemaleAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Spontaneous Cardiomyocyte Differentiation From Adipose Tissue Stroma Cells

2004

Cardiomyocyte regeneration is limited in adult life. Thus, the identification of a putative source of cardiomyocyte progenitors is of great interest to provide a usable model in vitro and new perspective in regenerative therapy. As adipose tissues were recently demonstrated to contain pluripotent stem cells, the emergence of cardiomyocyte phenotype from adipose-derived cells was investigated. We demonstrated that rare beating cells with cardiomyocyte features could be identified after culture of adipose stroma cells without addition of 5-azacytidine. The cardiomyocyte phenotype was first identified by morphological observation, confirmed with expression of specific cardiac markers, immunocy…

AtropineMalemedicine.medical_specialtyStromal cellPhysiologyCellular differentiationHeart VentriclesCholinergic AgentsAdipose tissueAdipose tissueCardiomyocytes ; Adipose tissue ; Differentiation ; Stem cells ; Cell therapyStem cellsBiologyCell therapyCell therapyMiceAdrenergic Agents:CIENCIAS MÉDICAS ::Medicina interna [UNESCO]Internal medicinemedicineAnimalsMyocytes CardiacHeart AtriaProgenitor cellInduced pluripotent stem cellCells CulturedUNESCO::CIENCIAS MÉDICAS ::Medicina internaCardiomyocytesRegeneration (biology)Multipotent Stem CellsIsoproterenolCell Differentiation:CIENCIAS MÉDICAS [UNESCO]Myocardial ContractionPropranololCell biologyClone CellsMice Inbred C57BLEndocrinologyPhenotypeAdipose TissueDifferentiationUNESCO::CIENCIAS MÉDICASRNACarbacholStem cellStromal CellsCardiology and Cardiovascular MedicineMyoblasts Cardiac
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Subtypes of muscarinic receptor on cholinergic nerves and atrial cells of chicken and guinea-pig hearts

1988

1. Electrically driven chicken and guinea-pig atria were used to investigate the negative inotropic effects of the muscarinic agonists methacholine and acetylcholine (ACh). The release of ACh from isolated hearts into the perfusate in response to (preganglionic) vagal or (pre- and postganglionic) field stimulation was bioassayed on the guinea-pig ileum or determined by labelling with [3H]-choline. 2. Concentration-response curves for the negative inotropic effect of methacholine were shifted to the right by pirenzepine in various concentrations (0.03 to 10 mumol l-1). The pA2 values were 7.76 in chicken atria and 6.53 in guinea-pig atria. Pirenzepine and atropine antagonized the negative in…

Atropinemedicine.medical_specialtyGuinea PigsTubocurarineStimulationIn Vitro TechniquesBiologyNeuroeffector junctionParasympathetic Nervous SystemInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsPharmacologyHeartVagus NervePirenzepineMyocardial ContractionReceptors MuscarinicPirenzepineAcetylcholineElectric StimulationVagus nerveAtropineEndocrinologyMethacholineChickensAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Beta-blockers: Historical Perspective and Mechanisms of Action

2019

Beta-blockers are widely used molecules that are able to antagonize β-adrenergic receptors (ARs), which belong to the G protein-coupled receptor family and receive their stimulus from endogenous catecholamines. Upon β-AR stimulation, numerous intracellular cascades are activated, ultimately leading to cardiac contraction or vascular dilation, depending on the relevant subtype and their location. Three subtypes have been described that are differentially expressed in the body (β1-, β2- and β3-ARs), β1 being the most abundant subtype in the heart. Since their discovery, β-ARs have become an important target to fight cardiovascular disease. In fact, since their discovery by James Black in the …

Beta-adrenergic blocking agentbusiness.industryMyocardiumAdrenergic beta-AntagonistsHeartStimulationVasodilationGeneral MedicinePropranolol030204 cardiovascular system & hematologyStimulus (physiology)PharmacologyMyocardial Contraction03 medical and health sciences0302 clinical medicineCardiovascular DiseasesReceptors Adrenergic betaHeart ratemedicineAnimalsHumansbusinessReceptormedicine.drugG protein-coupled receptorRevista Española de Cardiología (English Edition)
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Polyunsaturated fatty acids in cultured cardiomyocytes : effect on physiology and beta-adrenoceptor function

1992

This study was carried out to investigate the influence of the membrane fatty acid composition on the basal electrical and contractile activities and the response to beta-adrenergic stimulation of rat cardiac muscle cells in culture. Cells were grown for 3 days in a conventional serum culture medium and then incubated for 24 h in synthetic media containing either n-6 or n-3 as the sole source of polyunsaturated fatty acids (PUFA). The n-6/n-3 ratio in the phospholipids was 0.9 in the n-3 cells and 13.1 in the n-6 cells compared with 6.3 in controls cells. Such modifications did not alter action potentials and the main parameters related to contraction, although shortening was slightly acce…

Chronotropicmedicine.medical_specialtyContraction (grammar)PhysiologyPhospholipidStimulation030204 cardiovascular system & hematologyBiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineHeart RatePhysiology (medical)Internal medicineReceptors Adrenergic betamedicineMyocyteAnimalsCells CulturedComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciences[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyPOTENTIEL ELECTRIQUEMyocardiumFatty AcidsCardiac muscleIsoproterenolHeartMyocardial ContractionElectrophysiologyEndocrinologymedicine.anatomical_structureACIDE GRAS POLYINSATURE N-6chemistryCell cultureFatty Acids UnsaturatedACIDE GRAS POLYINSATURE N-3RATCardiology and Cardiovascular Medicine[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyPolyunsaturated fatty acid
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Study on the histamine-like activity of guanfacine

1990

Abstract The effects of guanfacine have been studied on guinea-pig isolated atria and diethylstilboestrol-treated rat isolated uterus to determine whether it possesses histamine-like activity. Guanfacine produced a concentration-dependent negative chronotropic effect which was not modified by ranitidine (0.1 μM). In rat isolated uterus contracted by KCl, clonidine (5–5000 μM) produced concentration-dependent relaxation which was blocked by ranitidine (0.1 μM), but guanfacine only produced relaxation at high concentrations (100–1000 μM), and this was not affected by ranitidine (0.1 μM). It is concluded that guanfacine, unlike clonidine, does not produce effects due to activation of H2-recept…

Chronotropicmedicine.medical_specialtyGuinea PigsUterusPharmaceutical SciencePharmacologyClonidinePotassium ChlorideGuinea pigRanitidineUterine Contractionchemistry.chemical_compoundInternal medicinemedicineAnimalsHeart AtriaDiethylstilbestrolPharmacologyDose-Response Relationship Drugbusiness.industryRats Inbred StrainsMyocardial ContractionGuanfacineRatsGuanfacineClonidineEndocrinologymedicine.anatomical_structureMechanism of actionchemistryFemalemedicine.symptombusinessAdrenergic alpha-AgonistsHistaminemedicine.drugJournal of Pharmacy and Pharmacology
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Effects of the Phosphodiesterase Inhibitor Enoximone on the Autonomic Innervation of the Isolated Heart

1989

Enoximone is a selective inhibitor of a low Km, cyclic AMP-specific type of phosphodiesterase (PDE III). In guinea pig and chicken atria, enoximone (0.1-100 mumol/L) caused a weak increase in the force of contraction. The heart rate was slightly enhanced or was unchanged (chicken). Enoximone (30 mumol/L) also failed to shift the concentration-response curves for the positive inotropic and chronotropic effects of norepinephrine in guinea pig atria. Under almost the same conditions, enoximone and the nonselective PDE inhibitor 3-isobutyl-1-methylxanthine (IBMX) markedly potentiated the forskolin-induced mobilization of choline from phospholipids. The concentrations of IBMX (100 mumol/L) and o…

Chronotropicmedicine.medical_specialtyIBMXPhosphodiesterase InhibitorsGuinea PigsIn Vitro TechniquesAutonomic Nervous SystemGuinea pigContractilitychemistry.chemical_compoundHeart Rate1-Methyl-3-isobutylxanthineInternal medicinemedicineAnimalsEnoximonePhosphodiesterase inhibitorEnoximonePharmacologyChemistryMyocardiumColforsinImidazolesPhosphodiesteraseHeartMyocardial ContractionAcetylcholineElectric StimulationEndocrinologyCardiology and Cardiovascular MedicineChickensAcetylcholinemedicine.drugJournal of Cardiovascular Pharmacology
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Ouabain enhances release of acetylcholine in the heart evoked by unilateral vagal stimulation.

1986

The aim of the study was to elucidate peripheral effects of ouabain on the parasympathetic innervation of the heart, effects that could contribute to the experimentally and clinically well established “vagal effect of cardiac glycosides”. The experiments were carried out with ouabain concentrations of 3×10−7 and 10−6 mol/l, which were considered “therapeutic”, as they increased force of contraction and did not elicit arrhythmias in incubated chicken atria. In atrial preparations of chickens and guinea-pigs the negative chronotropic and inotropic effects of acetylcholine (ACh) were not altered by 3×10−7 mol/l ouabain. Resting efflux of ACh from perfused chicken hearts was increased by ouabai…

Chronotropicmedicine.medical_specialtyStimulationIn Vitro TechniquesOuabainParasympathetic nervous systemHeart RateInternal medicinemedicineAnimalsOuabainPharmacologyDenervationbusiness.industryHeartVagus NerveGeneral MedicineDenervationMyocardial ContractionAcetylcholineElectric StimulationVagus nervemedicine.anatomical_structureEndocrinologymedicine.symptombusinessChickensAcetylcholinemedicine.drugMuscle contractionNaunyn-Schmiedeberg's archives of pharmacology
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Acute myocardial effects of mitoxantrone in the rabbit

1987

Some clinical studies that were performed for the purpose of assessing the potential cardiotoxicity of mitoxantrone (DHAD) have shown that repeated administrations of the drugs in some patients cause a mild impairment of cardiac functions and morphological changes in the myocardial cells qualitatively similar to those elicited by anthracyclines. Since doxorubicin has been reported to cause acute cardiac effects, probably related to its chronic cardiotoxicity, experiments were carried out on the rabbit heart to investigate whether DHAD is also able to induce acute cardiac effects. Our results show that this drug caused a reversible dose-related impairment of cardiac contractility on the isol…

ElectrocardiographyDHADDrug Evaluation PreclinicalAnimalsHeartRabbitsIn Vitro TechniquesMitoxantroneantitumor drugsMyocardial ContractionCardiotoxicity
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