Search results for "N-ACETYLCYSTEINE"

showing 10 items of 12 documents

Oxidative stress modulates rearrangement of endoplasmic reticulum-mitochondria contacts and calcium dysregulation in a Friedreich's ataxia model

2020

Friedreich ataxia (FRDA) is a neurodegenerative disorder characterized by neuromuscular and neurological manifestations. It is caused by mutations in the FXN gene, which results in loss of the mitochondrial protein frataxin. Endoplasmic Reticulum-mitochondria associated membranes (MAMs) are inter-organelle structures involved in the regulation of essential cellular processes, including lipid metabolism and calcium signaling. In the present study, we have analyzed in both, unicellular and multicellular models of FRDA, calcium management and integrity of MAMs. We observed that function of MAMs is compromised in our cellular model of FRDA, which was improved upon treatment with antioxidants. I…

0301 basic medicineAtaxiaClinical BiochemistryLipid peroxidationchemistry.chemical_elementMitochondrionCalciumEndoplasmic ReticulumBiochemistry03 medical and health sciences0302 clinical medicineMAMsmedicineAnimalsVitamin EMitochondrial calcium uptakelcsh:QH301-705.5Calcium signalinglcsh:R5-920biologyFrataxinEndoplasmic reticulumOrganic ChemistryN-acetylcysteineMitochondriaCell biologyOxidative StressDrosophila melanogaster030104 developmental biologychemistrylcsh:Biology (General)Friedreich AtaxiaFrataxinbiology.proteinCalciummedicine.symptomCellular modellcsh:Medicine (General)030217 neurology & neurosurgeryResearch PaperRedox Biology
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The Effects of N-Acetylcysteine on the Rat Mesocorticolimbic Pathway: Role of mGluR5 Receptors and Interaction with Ethanol

2021

N-acetylcysteine (NAC) is a prodrug that is marketed as a mucolytic agent and used for the treatment of acetaminophen overdose. Over the last few decades, evidence has been gathered that suggests the potential use of NAC as a new pharmacotherapy for alcohol use disorder (AUD), although its mechanism of action is already being debated. In this paper, we set out to assess both the potential involvement of the glutamate metabotropic receptors (mGluR) in the possible dual effect of NAC administered at two different doses and NAC’s effect on ethanol-induced activation. To this aim, 30 or 120 mg/kg of NAC was intraperitoneally administered to rats with the presence or absence of the negative allo…

0301 basic medicinePharmaceutical ScienceglutamatePharmacologyAcetylcysteine03 medical and health sciencesPharmacy and materia medica0302 clinical medicineNeurochemicalDrug Discoverymental disordersmedicinealcoholismMetabotropic glutamate receptor 5ChemistryCommunicationRGlutamate receptor<i>N</i>-acetylcysteineN-acetylcysteineRS1-441030104 developmental biologyMetabotropic receptorMTEPMechanism of actionMetabotropic glutamate receptorAlcoholismeMedicineMolecular Medicinemedicine.symptomTecnologia farmacèuticaMedicaments030217 neurology & neurosurgerymedicine.drugPharmaceuticals
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Rapid generation of hydrogen peroxide contributes to the complex cell death induction by the angucycline antibiotic landomycin E

2017

Landomycin E (LE) is an angucycline antibiotic produced by Streptomyces globisporus. Previously, we have shown a broad anticancer activity of LE which is, in contrast to the structurally related and clinically used anthracycline doxorubicin (Dx), only mildly affected by multidrug resistance-mediated drug efflux. In the present study, cellular and molecular mechanisms underlying the anticancer activity of landomycin E towards Jurkat T-cell leukemia cells were dissected focusing on the involvement of radical oxygen species (ROS). LE-induced apoptosis distinctly differed in several aspects from the one induced by Dx. Rapid generation of both extracellular and cell-derived hydrogen peroxide alr…

0301 basic medicinePoly (ADP-Ribose) Polymerase-1ApoptosisBiochemistryLandomycin EJurkat Cellschemistry.chemical_compoundSuperoxidesCaspaseCaspase-9chemistry.chemical_classificationCaspase 7Antibiotics AntineoplasticLeukemiabiologySuperoxideStreptomycesCaspase 9Respiratory burstMitochondriaBiochemistrySettore CHIM/03 - Chimica Generale E InorganicaReactive oxygen specieHumanJurkat CellCaspase 7Article03 medical and health sciencesPhysiology (medical)HumansReactive oxygen speciesAminoglycosideIntrinsic apoptosisApoptosiOxidative StreAnticancer drugHydrogen PeroxideMolecular biologyN-acetylcysteineSuperoxide radicalAcetylcysteineMulti-drug resistanceOxidative StressAminoglycosides030104 developmental biologychemistryStreptomyceApoptosisDoxorubicinbiology.proteinReactive Oxygen Species
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Chronic aspartame intake causes changes in the trans-sulphuration pathway, glutathione depletion and liver damage in mice

2017

No-caloric sweeteners, such as aspartame, are widely used in various food and beverages to prevent the increasing rates of obesity and diabetes mellitus, acting as tools in helping control caloric intake. Aspartame is metabolized to phenylalanine, aspartic acid, and methanol. Our aim was to study the effect of chronic administration of aspartame on glutathione redox status and on the trans-sulphuration pathway in mouse liver. Mice were divided into three groups: control; treated daily with aspartame for 90 days; and treated with aspartame plus N-acetylcysteine (NAC). Chronic administration of aspartame increased plasma alanine aminotransferase (ALT) and aspartate aminotransferase activities…

0301 basic medicinemedicine.medical_specialtyGlutamate-Cysteine LigaseClinical BiochemistryPhenylalanineBiochemistryMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineAspartic acidmedicineAnimalsHumansCysteineAspartamelcsh:QH301-705.5lcsh:R5-920S-adenosylmethionineMethioninebiologyAspartameChemistryOrganic ChemistryCystathionine gamma-LyaseMethionine AdenosyltransferaseGlutathioneGlutathioneCystathionine beta synthaseN-acetylcysteineAcetylcysteine030104 developmental biologyEndocrinologyGCLCGene Expression RegulationLiverlcsh:Biology (General)BiochemistrySweetening Agents030220 oncology & carcinogenesisbiology.proteinChemical and Drug Induced Liver Injurylcsh:Medicine (General)Research PaperCysteineRedox Biology
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Inhibitory effects of N-acetylcysteine on the functional responses of human eosinophils in vitro

2007

Background Oxidative stress appears to be relevant in the pathogenesis of inflammation in allergic diseases like bronchial asthma. Eosinophils are oxidant-sensitive cells considered as key effectors in allergic inflammation. Objective The aim of this work was to study the effects of the clinically used antioxidant N-acetyl-L-cysteine (NAC) on the functional responses of human-isolated eosinophils. Methods Human eosinophils were purified from the blood of healthy donors by a magnetic bead separation system. The effects of NAC were investigated on the generation of reactive oxygen species (chemiluminescence and flow cytometry), Ca2+ signal (fluorimetry), intracellular glutathione (GSH; flow c…

Chemokine CCL11EotaxinLuminescenceImmunologyhuman eosinophilsBiologyPharmacologymedicine.disease_causeTranslocation GeneticAllergic inflammationAcetylcysteinechemistry.chemical_compoundmedicineHumansImmunology and Allergychemistry.chemical_classificationreactive oxygen speciesEosinophil cationic proteinReactive oxygen speciesCell DeathEosinophil Cationic ProteinNADPH OxidasesFree Radical ScavengersGlutathioneEosinophilPhosphoproteinsGlutathioneN-acetylcysteineAcetylcysteineEosinophilsN-Formylmethionine Leucyl-Phenylalaninemedicine.anatomical_structurechemistryBiochemistryChemokines CCeosinophil cationic proteinCalciumReactive Oxygen SpeciesOxidative stressp47(phox)medicine.drugp67(phox)
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Can PBDEs affect the pathophysiologic complex of epithelium in lung diseases?

2020

Brominated flame-retardant (BFRs) exposure promotes multiple adverse health outcomes involved in oxidative stress, inflammation, and tissues damage. We investigated BFR effects, known as polybrominated diphenyl ethers (PBDEs) (47, 99 and 209) in an air-liquid-interface (ALI) airway tissue derived from A549 cell line, and compared with ALI culture of primary human bronchial epithelial cells (pHBEC). The cells, exposed to PBDEs (47, 99 and 209) (0.01-1 mu M) for 24 h, were studied for IL-8, Muc5AC and Muc5B (mRNAs and proteins) production, as well as NOX-4 (mRNA) expression. Furthermore, we evaluated tight junction (TJ) integrity by Trans-Epithelial Electrical Resistance (TEER) measurements, …

Lung DiseasesHealth Toxicology and Mutagenesis0208 environmental biotechnology02 engineering and technology010501 environmental sciencesMucin 5ACBROMINATED FLAME RETARDANTSmedicine.disease_cause01 natural sciencesPolybrominated diphenyl ethersPARTICULATE MATTERElectric ImpedanceHalogenated Diphenyl EthersFlame RetardantsInhalationTight junctionAIRWAY MUCUSChemistryGeneral Medicinerespiratory systemPollutionMucin-5BINTRACELLULAR GLUTATHIONEPolybrominated diphenyl ethers; Inflammation; Oxidative stress; Mucins; Epithelial barrier integrity; Rheological propertiesmedicine.anatomical_structureNADPH Oxidase 4medicine.symptomEnvironmental EngineeringInflammationBronchiEXPOSURE SYSTEMTight JunctionsAndrologymedicineEnvironmental ChemistryHumansRheological propertiesPolybrominated diphenyl ether0105 earth and related environmental sciencesAgedInflammationEpithelial barrier integrityPOLYBROMINATED DIPHENYL ETHERSMucinInterleukin-8MucinsPublic Health Environmental and Occupational HealthEpithelial CellsGeneral ChemistryN-ACETYLCYSTEINEEpithelium020801 environmental engineeringrespiratory tract diseasesOxidative StressA549 CellsMucinEX-VIVO MODELOxidative streRespiratory epitheliumAEROSOL-PARTICLESOxidative stressChemosphere
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N-Acetylcysteine Amide Exerts Possible Neuroprotective Effects in Newborn Pigs after Perinatal Asphyxia

2016

&lt;b&gt;&lt;i&gt;Background:&lt;/i&gt;&lt;/b&gt; Perinatal asphyxia and ensuing reoxygenation change the antioxidant capacity of cells and organs. &lt;b&gt;&lt;i&gt;Objectives:&lt;/i&gt;&lt;/b&gt; To analyze the neuroprotective effect of the antioxidant N-acetylcysteine amide (NACA) after perinatal hypoxia-reoxygenation with an emphasis on proinflammatory cytokines and the transcription factor NF-&amp;#x03BA;B in the prefrontal cortex of neonatal pigs. &lt;b&gt;&lt;i&gt;Methods:&lt;/i&gt;&lt;/b&gt; Twenty-nine newborn pigs, aged 12-36 h, were subjected to global hypoxia and hypercapnia. One sham-operated group (n = 5) and 2 experimental groups (n = 12) were exposed to 8% oxygen, until the …

Male0301 basic medicinecongenital hereditary and neonatal diseases and abnormalitiesTime FactorsSwineInterleukin-1betaPharmacologyNeuroprotection03 medical and health sciences0302 clinical medicinemedicineAnimalsN-Acetylcysteine amideHypoxiaskin and connective tissue diseasesreproductive and urinary physiologyAsphyxia NeonatorumTumor Necrosis Factor-alphabusiness.industryNF-kappa BBrainmedicine.diseaseAcetylcysteinePerinatal asphyxiaOxygenAntioxidant capacityNeuroprotective Agents030104 developmental biologyAnimals NewbornAnesthesiaPediatrics Perinatology and Child Healthpopulation characteristicsFemalesense organsbusinessBiomarkers030217 neurology & neurosurgeryDevelopmental BiologyNeonatology
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N-Acetylcysteine for the Management of Non-Acetaminophen Drug-Induced Liver Injury in Adults: A Systematic Review

2022

Introduction: Idiosyncratic drug-induced liver injury (DILI) is a rare adverse reaction to drugs and other xenobiotics. DILI has different grades of severity and may lead to acute liver failure (ALF), for which there is no effective therapy. N-acetylcysteine (NAC) has been occasionally tested for the treatment of non-acetaminophen drug-induced ALF. However, limited evidence for its efficacy and safety is currently available. Our aim was to elucidate the benefit and safety of NAC in DILI and evaluate its hepatoprotective effect.Methods: We conducted a systematic review to evaluate the management and prevention focused on NAC in idiosyncratic DILI. The main outcomes included mortality due to …

PharmacologyPharmacology (medical)DILInon-acetaminophenacute liver failureacute liver injuryN-acetylcysteine
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Premedication with oral solution of simethicone plus N-acetylcysteine to improve the quality upper GI endoscopy: Initial experience

2020

Introduction: To assess the efficacy and safety of simethicone with or without N-acetylcysteine (NAC) as premedications before gastroscopy. Materials and methods: Patients were randomized into 5 groups. Endoscopic visibility was evaluated in 4 districts (esophagus, gastric body, fundus and antrum) using a visual scale, graduating from 1 to 4 points. Results: There was no significant difference on the rate of positive findings when comparing simethicone with simethicone plus NAC and with water, respectively. Simethicone plus NAC showed better total mucosal visibility score than simethicone alone. Both simethicone plus NAC and simethicone alone offer more benefit than water. The procedure tim…

PremedicationGastroscopySimethiconeQualityN-acetylcysteine
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Oxidative Stress and Cognitive Alterations Induced by Cancer Chemotherapy Drugs: A Scoping Review

2021

Cognitive impairment is one of the most deleterious effects of chemotherapy treatment in cancer patients, and this problem sometimes remains even after chemotherapy ends. Common classes of chemotherapy-based regimens such as anthracyclines, taxanes, and platinum derivatives can induce both oxidative stress in the blood and in the brain, and these effects can be reproduced in neuronal and glia cell cultures. In rodent models, both the acute and repeated administration of doxorubicin or adriamycin (anthracyclines) or cisplatin impairs cognitive functions, as shown by their diminished performance in different learning and memory behavioural tasks. Administration of compounds with strong antiox…

cognition0301 basic medicineAntioxidantPhysiologymedicine.medical_treatmentClinical BiochemistryReviewRM1-950Pharmacologymedicine.disease_causeBiochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineMedicine<i>N-</i>acetylcysteineDoxorubicinplatinumCaffeic acid phenethyl esterMolecular BiologyMesnaanthracyclinesCisplatinChemotherapybusiness.industryCancerclinical trialCell Biologymedicine.diseaseN-acetylcysteinetaxanes030104 developmental biologychemistry030220 oncology & carcinogenesisbiomarkerTherapeutics. PharmacologybusinessOxidative stressmedicine.drugAntioxidants
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