Search results for "NB"

showing 10 items of 4401 documents

Relaxant effect of dopamine on the isolated rat uterus.

1988

The effect of dopamine was studied on the isolated uterus of diethylstilboestrol-treated rats. Dopamine, at concentrations (10(7)-10(-4) M) produced a concentration-dependent relaxation in the K+-depolarized rat uterus. On a molar basis, dopamine was about 500 times less potent than adrenaline in relaxing the uterus, the maximum degree of relaxation obtained with both drugs was the same. Pretreatment of the rats with reserpine (5 mg/kg) did not produce any modification of the dose-response curve to dopamine. Similarly, cocaine (3 x 10(-6) M) failed to modify the relaxant effect of dopamine. The dopamine induced relaxation was inhibited by propranolol (10(-9)-10(-7) M) in a dose-dependent ma…

medicine.medical_specialtyReserpineEpinephrineDopamineBiologyIn Vitro Techniqueschemistry.chemical_compoundUterine ContractionDopamineInternal medicinemedicinePrazosinAnimalsNeurotransmitterDiethylstilbestrolPharmacologyDose-Response Relationship DrugUterusRats Inbred StrainsGeneral MedicinePrazosinReserpineBenzazepinesPropranololRatsEndocrinologyEpinephrinechemistryDopamine receptorCatecholamineFemaleSulpirideSulpiridemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Effects of sodium fluoride on the mechanical activity in mouse gastric preparations.

2005

The aim of the present study was to investigate the responses induced by sodium fluoride (NaF) on gastric mechanical activity, using mouse whole-stomach preparations. The mechanical activity was recorded in vitro as changes of intraluminal pressure. In most of the preparations, NaF induced a tetrodotoxin-insensitive biphasic effect characterized by early relaxation followed by slowly developing contractile response. The contraction was dependent on the concentration of NaF, whereas the relaxation was observed at only 10–30 mmol/L NaF. The contractile effect was significantly reduced by nifedipine (an L-type Ca2+channel blocker), ryanodine or ruthenium red (inhibitors of Ca2+release from sar…

medicine.medical_specialtyRuthenium redPhysiologySettore BIO/09 - FisiologiaAdenylyl cyclasechemistry.chemical_compoundMiceNifedipinePhysiology (medical)Internal medicineSodium fluoridemedicineAnimalsChannel blockerEnzyme InhibitorsProtein Kinase CPharmacologyPhospholipase CRyanodine receptorStomachGastric mechanical activity Mouse stomach Smooth muscle Sodium fluorideMuscle SmoothGeneral MedicineNeomycinMice Inbred C57BLEndocrinologychemistryType C PhospholipasesAdenylyl Cyclase InhibitorsSodium FluorideCalciumExtracellular SpaceGastrointestinal Motilitymedicine.drugAdenylyl CyclasesMuscle ContractionCanadian journal of physiology and pharmacology
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Sarcosinaemia in a retarded, amaurotic child.

1986

A 9-month-old Turkish girl demonstrated an abnormal qualitative amino acid excretion pattern suggestive of sarcosinaemia. She was blind and had evidence of developmental and motor retardation. No other physical abnormalities were noted. Quantitative amino acid analysis revealed elevated serum and urine sarcosine levels. An oral sarcosine loading test showed an exaggerated response with a delayed conversion to glycine. Sarcosine was undetected in other family members.

medicine.medical_specialtySarcosinebusiness.industryGlycineInfantSarcosineUrineBlindnessAmino acid excretionElevated serumchemistry.chemical_compoundAmino acid analysisEndocrinologychemistryInternal medicineIntellectual DisabilityPediatrics Perinatology and Child HealthGlycineMedicineSarcosinaemiaHumansFemaleMotor retardationbusinessAmino Acid Metabolism Inborn ErrorsEuropean journal of pediatrics
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Modulation of hippocampal acetylcholine release after fimbria-fornix lesions and septal transplantation in rats

1997

Abstract Female Long–Evans rats sustained electrolytic lesions of the fimbria and the dorsal fornix causing a partial lesion of the septohippocampal pathway. Two weeks later, the rats received intra-hippocampal grafts of fetal septal cell suspensions. Nine to twelve months later, the release of acetylcholine (ACh) in the hippocampus of sham-operated, lesion-only and grafted rats was measured by microdialysis. The extent of cholinergic (re)innervation was determined by acetylcholinesterase (AChE) staining and densitometry. In both lesion-only and grafted rats, the ratio of ACh release to AChE staining intensity was increased as compared to sham-operated rats, indicating a loss of endogenous …

medicine.medical_specialtySciences du Vivant [q-bio]/Neurosciences [q-bio.NC]Microdialysis[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyScopolamineMuscarinic AntagonistsHippocampal formationBiologySerotonergicHippocampus03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineNeural PathwaysmedicineAnimalsBrain Tissue TransplantationCholinergic neuronNeurotransmitterComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciences8-Hydroxy-2-(di-n-propylamino)tetralinGeneral NeuroscienceFornixMuscarinic antagonistRats Inbred StrainsAcetylcholineRatsEndocrinologychemistryCholinergic FibersAnesthesiaReceptors SerotoninCholinergicRaphe NucleiFemaleSeptal Nuclei[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Receptors Serotonin 5-HT1030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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Muscarine receptors on the rat phrenic nerve, evidence for positive and negative muscarinic feedback mechanisms.

1987

Neuronal transmitter stores of the rat phrenic nerve were labelled by incubation with [3H]choline. Release of [3H]acetylcholine was elicited by electrical nerve stimulation (100 or 1500 pulses, 5 or 25 Hz) or by high potassium (27 mmol/l) and the effects of the muscarine receptor agonist oxotremorine and the antagonist scopolamine were investigated. Neither oxotremorine nor scopolamine affected the basal tritium efflux. A low concentration of oxotremorine (10 nmol/l) enhanced and a high concentration of oxotremorine (1 μol/l) reduced the electrically evoked [3H]acetylcholine release. Likewise, the high potassium-evoked [3H]acetylcholine release was reduced by a high concentration of oxotrem…

medicine.medical_specialtyScopolamineMotor nerveStimulationIn Vitro Techniqueschemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineAnimalsPharmacologyMuscarineChemistryOxotremorineRats Inbred StrainsGeneral MedicineReceptors MuscarinicAcetylcholineElectric StimulationNeostigmineRatsPhrenic NerveEndocrinologymedicine.anatomical_structurePeripheral nervous systemPotassiumAcetylcholineScopolamine Hydrobromidemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Characterization of endogenous noradrenaline release from intact and epithelium-denuded rat isolated trachea.

1991

1. Overflow of endogenous noradrenaline (NA) from the in vitro incubated rat trachea evoked by two periods of electrical field stimulation (S1, S2 at 3 or 15 Hz) or by high potassium (60 mM) was determined by high performance liquid chromatography (h.p.l.c.) with electrochemical detection. 2. In the presence of the neuronal uptake inhibitor desipramine, the alpha 2-adrenoceptor antagonist, yohimbine, enhanced the overflow of NA evoked by stimulation at 3 Hz by about 100% suggesting the presence of presynaptic inhibitory autoreceptors on the sympathetic nerves innervating the trachea. 3. When desipramine and yohimbine were present throughout the experiments, the overflow of NA evoked by the …

medicine.medical_specialtyScopolamineNeuromuscular transmissionStimulationTetrodotoxinIn Vitro TechniquesInhibitory postsynaptic potentialEpitheliumchemistry.chemical_compoundNorepinephrineDesipramineInternal medicinemedicineElectrochemistryAnimalsChromatography High Pressure LiquidPharmacologyOxotremorineDesipramineYohimbineMuscle SmoothRats Inbred StrainsEpitheliumElectric StimulationYohimbineRatsTracheaEndocrinologymedicine.anatomical_structurechemistryMuscle TonusTetrodotoxinAutoreceptorPotassiumFemalemedicine.drugResearch Article
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Impact of serotonin 2C receptor null mutation on physiology and behavior associated with nigrostriatal dopamine pathway function.

2009

The impact of serotonergic neurotransmission on brain dopaminergic pathways has substantial relevance to many neuropsychiatric disorders. A particularly prominent role has been ascribed to the inhibitory effects of serotonin 2C receptor (5-HT2CR) activation on physiology and behavior mediated by the mesolimbic dopaminergic pathway, particularly in the terminal region of the nucleus accumbens. The influence of this receptor subtype on functions mediated by the nigrostriatal dopaminergic pathway is less clear. Here we report that a null mutation eliminating expression of 5-HT2CRs produces marked alterations in the activity and functional output of this pathway. 5-HT2CR mutant mice displayed i…

medicine.medical_specialtySerotoninDopamineDopamine AgentsPhysiologySubstantia nigraStriatumBiologySettore BIO/09 - FisiologiaPiperazinesArticleMiceDopamine receptor D1Dopamine Uptake InhibitorsDopamineDopamine receptor D2Internal medicineNeural PathwaysmedicineReceptor Serotonin 5-HT2CAnimalsNeuronsBehavior AnimalPars compactaGeneral Neuroscience5-HT2CR substantia nigra pars compacta dorsal striatum dopamine extracellular recording in vivo patch clamp recording microdialysis Locomotor activity Stereotypic behaviorDopaminergicNeurobehavioral disordersBenzazepinesGroomingCorpus StriatumElectrophysiologyMice Inbred C57BLSubstantia NigraAmphetamineEndocrinologymedicine.anatomical_structureDopaminergic pathwaysDopamine AgonistsMutationAutoradiographyStereotyped BehaviorNeuroscienceLocomotionmedicine.drugThe Journal of neuroscience : the official journal of the Society for Neuroscience
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BDNF is essentially required for the early postnatal survival of nociceptors

2010

AbstractNeurotrophins promote the survival of specific types of neurons during development and ensure proper maintenance and function of mature responsive neurons. Significant effects of BDNF (Brain-Derived Neurotrophic Factor) on pain physiology have been reported but the contribution of this neurotrophin to the development of nociceptors has not been investigated. We present evidence that BDNF is required for the survival of a significant fraction of peptidergic and non-peptidergic nociceptors in dorsal root ganglia (DRG) postnatally. Bdnf homozygous mutant mice lose approximately half of all nociceptive neurons during the first 2 weeks of life and adult heterozygotes exhibit hypoalgesia …

medicine.medical_specialtySkin innervationCell SurvivalNeurotrophic factorMice Inbred StrainsNeuronal survivalMiceNeurotrophic factorsGanglia SpinalInternal medicineGlial cell line-derived neurotrophic factormedicineAnimalsGlial Cell Line-Derived Neurotrophic FactorNerve Growth FactorsDorsal root gangliaAutocrine signallingMolecular BiologyCells CulturedSensory neuronHypoalgesiabiologyBrain-Derived Neurotrophic FactorNociceptorsAnatomyCell BiologyBdnf knockout miceEmbryo MammalianSensory neuronmedicine.anatomical_structureEndocrinologynervous systemPeripheral nervous systembiology.proteinNociceptorNeurotrophinPeripheral nervous systemSignal TransductionNeurotrophinDevelopmental BiologyDevelopmental Biology
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Chronic heart damage following doxorubicin treatment is alleviated by lovastatin.

2014

The anticancer efficacy of anthracyclines is limited by cumulative dose-dependent early and delayed cardiotoxicity resulting in congestive heart failure. Mechanisms responsible for anthracycline-induced heart damage are controversially discussed and effective preventive measures are preferable. Here, we analyzed the influence of the lipid lowering drug lovastatin on anthracycline-induced late cardiotoxicity three month after treatment of C57BL/6 mice with five low doses of doxorubicin (5×3mg/kg BW; i.p.). Doxorubicin increased the cardiac mRNA levels of BNP, IL-6 and CTGF, while the expression of ANP remained unchanged. Lovastatin counteracted these persisting cardiac stress responses evoke…

medicine.medical_specialtyStatinCardiotonic AgentsAnthracyclinemedicine.drug_classBiologymedicine.disease_causeDNA MitochondrialInternal medicineNatriuretic Peptide Brainpolycyclic compoundsmedicineAnimalsDoxorubicinHSP70 Heat-Shock ProteinsLovastatinPharmacologyCardiotoxicityEjection fractionAntibiotics AntineoplasticInterleukin-6Gene Expression ProfilingMyocardiumConnective Tissue Growth Factormedicine.diseaseFibrosisCardiotoxicityMice Inbred C57BLEndocrinologyDoxorubicinHeart failureFemaleLovastatinOxidative stressmedicine.drugDNA DamagePharmacological research
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Corticotropin-Releasing Hormone-Mediated Induction of Intracellular Signaling Pathways and Brain-Derived Neurotrophic Factor Expression Is Inhibited …

2005

CRH receptor (CRHR) 1 and the cannabinoid receptor 1 (CB1) are both G protein-coupled receptors. Activation of CRHR1 leadstoincreasesincAMPproductionandphosphorylationof the transcription factor cAMP response element-binding protein (CREB). In contrast, CB1 is negatively coupled to the cAMP signaling cascade. In this study, we analyzed a putative interaction between these two systems focusing on the regulation of the expression of brain-derived neurotrophic factor (BDNF), a CREB-regulated gene. In situ hybridization revealed coexpression of CRHR1 and CB1 receptors in the granular layer of the cerebellum. Therefore, we analyzed the effects of CRH and the CB1 agonist WIN-55,212-2 on BDNF expr…

medicine.medical_specialtyTime FactorsCorticotropin-Releasing HormoneMorpholinesmedicine.medical_treatmentImmunoblottingEnzyme-Linked Immunosorbent AssayTropomyosin receptor kinase BNaphthalenesCREBModels BiologicalRats Sprague-DawleyMiceEndocrinologyNeurotrophic factorsCerebellumInternal medicineCannabinoid Receptor ModulatorsCyclic AMPmedicineAnimalsRNA MessengerCyclic AMP Response Element-Binding ProteinReceptorEgtazic AcidCells CulturedIn Situ HybridizationNeuronsBrain-derived neurotrophic factorSulfonamidesbiologyReverse Transcriptase Polymerase Chain ReactionBrain-Derived Neurotrophic FactorCalcium Channel BlockersIsoquinolinesEndocannabinoid systemBenzoxazinesRatsMice Inbred C57BLPyrimidinesEndocrinologynervous systembiology.proteinCalciumCannabinoidSignal transductionEndocannabinoidsProtein BindingSignal TransductionEndocrinology
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