Search results for "NEPHRITIS"

showing 10 items of 101 documents

Per-protocol repeat kidney biopsy portends relapse and long-term outcome in incident cases of proliferative lupus nephritis

2019

Abstract Objectives In patients with LN, clinical and histological responses to treatment have been shown to be discordant. We investigated whether per-protocol repeat kidney biopsies are predictive of LN relapses and long-term renal function impairment. Methods Forty-two patients with incident biopsy-proven active proliferative (class III/IV±V) LN from the database of the UCLouvain were included in this retrospective study. Per-protocol repeat biopsies were performed after a median [interquartile range (IQR)] time of 24.3 (21.3–26.2) months. The National Institutes of Health activity index (AI) and chronicity index (CI) scores were assessed in all biopsies. Results Despite a moderate corre…

MaleBiopsy030232 urology & nephrologyKidneyGastroenterologychemistry.chemical_compound0302 clinical medicinesystemic lupus erythematosusRecurrenceInterquartile rangePharmacology (medical)Proteinuriamedicine.diagnostic_testHazard ratioPrognosisLupus NephritisProteinuriaKidney TubulesCreatinineDisease ProgressionhistopathologyFemaleRenal biopsymedicine.symptomRituximabImmunosuppressive AgentsAdultlong-term outcomemedicine.medical_specialtyRenal functionMethylprednisoloneYoung Adult03 medical and health sciencesrenal biopsyRheumatologyInternal medicineBiopsymedicineHumansImmunologic FactorsCyclophosphamideGlucocorticoidsProportional Hazards ModelsRetrospective Studieslupus nephritisrepeat biopsy030203 arthritis & rheumatologyCreatininebusiness.industryrenal functionMycophenolic AcidchemistryPulse Therapy DrugHistopathologybusinessRheumatology
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Proteinase-3 mRNA expressed by glomerular epithelial cells correlates with crescent formation in Wegener's granulomatosis

2000

Proteinase-3 mRNA expressed by glomerular epithelial cells correlates with crescent formation in Wegener's granulomatosis. Background Wegener's granulomatosis (WG) is characterized by systemic vasculitis with crescentic glomerulonephritis (CGN) and circulating autoantibodies directed against neutrophil cytoplasmic antigens (ANCA). Proteinase 3 (PR-3), a neutral serine proteinase in neutrophils implicated in the growth control of myeloid cells, has been identified as the target antigen for ANCA in WG. Since the kidneys are frequently involved in WG, we studied the in situ expression of PR-3 by renal parenchymal cells. Methods We assessed the expression of PR-3 in kidney biopsies of 15 patien…

AdultMalePathologymedicine.medical_specialtyBiopsyMyeloblastinKidney GlomerulusIn situ hybridizationBiologyurologic and male genital diseasesKidneyvasculitisAntigenProteinase 3medicineRapidly progressive glomerulonephritisHumanscrescent glomerulonephritisNorthern blotRNA Messengerrapidly progressive glomerulonephritisCells CulturedAgedKidneyANCAurogenital systemSerine EndopeptidasesGranulomatosis with PolyangiitisEpithelial CellsMiddle Agedmedicine.diseasekidney parenchymal cellsmedicine.anatomical_structureKidney TubulesNephrologyImmunohistochemistryFemaleSystemic vasculitisKidney International
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Thrombotic thrombocytopenic purpura (TTP) leading to pseudotumour's autoimmune pancreatitis (AIP): A case report

2012

International audience; Introduction: Autoimmune pancreatitis is an idiopathic inflammatory disease that produces pancreatic masses and ductal strictures. This benign disease can be associated with extrapancreatic manifestations including cholangitis, sialadenitis, inflammatory bowel disease or retroperitoneal fibrosis, mediastinal adenopathy, interstitial nephritis mainly due to immunoglobulin G4 (Ig G4), and occasional association with other auto-immune diseases. Observation: We report a 57-year-old woman who developed thrombotic thrombocytopenic purpura (UP) and pseudo-tumour's seronegative autoimmune pancreatitis (ATP) type 1. The patient was initially treated with pulse corticosteroids…

medicine.medical_specialtyVON-WILLEBRAND-FACTOREndocrinology Diabetes and Metabolismmedicine.medical_treatmentInterstitial nephritisAnti-Inflammatory AgentsThrombotic thrombocytopenic purpuraRetroperitoneal fibrosisGastroenterologyInflammatory bowel diseaseDISEASEAutoimmune DiseasesAntibodies Monoclonal Murine-Derived03 medical and health sciences0302 clinical medicineThrombotic thrombocytopenic purpuraInternal medicine[SDV.IDA]Life Sciences [q-bio]/Food engineeringmedicineHumans[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringSYSTEMIC-LUPUS-ERYTHEMATOSUSAutoimmune pancreatitisAutoimmune pancreatitisPurpura Thrombotic ThrombocytopenicHepatologybusiness.industryENTITYGastroenterologyMiddle Agedmedicine.diseaseSialadenitis3. Good healthPancreatitis030220 oncology & carcinogenesisImmunologyFemale030211 gastroenterology & hepatologyRituximabPlasmapheresismedicine.symptombusinessRituximabmedicine.drug
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Immunoregulatory role of Jα281 T cells in aged mice developing lupus-like nephritis

2007

Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the emergence of autoreactive T cells. Humans and mice with SLE have reduced numbers of CD1d-restricted invariant natural killer T (iNKT) cells, suggesting a key role for these cells in its immunopathogenesis. This subset uses an invariant TCR constituted by Valpha14 Jalpha281 chains paired with some Vbeta domains. The regulatory role for iNKT cells in non-autoimmune mice was suggested by our previous results showing that aged Jalpha281 knockout (KO) mice produce anti-dsDNA. Here we show that old Jalpha281 KO mice have proteinuria and antibodies against dsDNA and cardiolipin. Histological analysis of Jalpha281 KO m…

AgingImmunologyReceptors Antigen T-CellEnzyme-Linked Immunosorbent AssayLymphocyte Activationmedicine.disease_causeAutoimmunity Knockout NKT cellsAutoimmunityMicemedicineAnimalsLupus Erythematosus SystemicImmunology and AllergyAutoantibodiesMice KnockoutSettore MED/04 - Patologia GeneraleB-LymphocytesSystemic lupus erythematosusbiologyT-cell receptorAutoantibodyNatural killer T cellMarginal zonemedicine.diseaseImmunohistochemistryLupus NephritisKiller Cells NaturalImmunologybiology.proteinAntibodyNephritisSpleenEuropean Journal of Immunology
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Acanthocyturia—A characteristic marker for glomerular bleeding

1991

Acanthocyturia—A characteristic marker for glomerular bleeding. Erythrocyte morphology by phase contrast microscopic examination (PCM) of the urine is widely employed in distinguishing glomerular from nonglomerular bleeding. The proposed percentages of dysmorphic red cells are significant for glomerular bleeding in the range of 10 to 80% in the literature, because there is no clear cut definition of “dysmor-phism.” In the present study midstream urine samples of 351 patients with hematuria (> 8 erythrocytes/μl) and of 33 healthy controls were examined. The various dysmorphic red cells were analyzed by PCM according to a detailed hematological classification. Most of the dysmorphic red cells…

Pathologymedicine.medical_specialtyRenal glomerulusKidney GlomerulusEchinocyteAcanthocytesHemorrhageUrineurologic and male genital diseasesBiopsymedicineHumansUrine cytologyKidneymedicine.diagnostic_testRed Cellurogenital systembusiness.industryOsmolar ConcentrationGlomerulonephritisHydrogen-Ion Concentrationmedicine.diseasefemale genital diseases and pregnancy complicationsProteinuriamedicine.anatomical_structureNephrologyKidney DiseasesAcanthocytebusinessBiomarkersKidney International
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Reflux nephropathy: a clinico-pathological study of 16 cases.

1991

The pathologic features of 16 nephrectomy specimens obtained from patients with a radiological diagnosis of reflux nephropathy were analyzed. Chronic pyelonephritis was diagnosed in 7 cases, renal dysplasia in 5 and segmental atrophy in 4. Clear correlations between pathologic pictures and pathogenic mechanisms involved in the development of renal lesions in kidneys with reflux are difficult to establish. These results show, however, that 'reflux nephropathy' is a broad term, which encompasses both congenital and acquired renal lesions.

AdultMalemedicine.medical_specialtyPathologyAdolescentUrologymedicine.medical_treatmentKidneyVesicoureteral refluxGastroenterologyNephropathyInternal medicinemedicineHumansChronic pyelonephritisChildReflux nephropathyVesico-Ureteral RefluxPyelonephritisbusiness.industryMiddle Agedmedicine.diseaseRenal dysplasiaNephrectomyChild PreschoolChronic DiseaseEtiologyClinico pathologicalFemaleKidney DiseasesAtrophybusinessUrologia internationalis
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Temporary warm ischaemia, 5/6 nephrectomy and single uranylnitrate administration — comparison of three models intended to cause renal fibrosis in ra…

2001

In patients the progression of pathologic renal processes after the treatment of primary disease is a problem of increasing importance and therapeutic strategies are insufficient till now. The aim of this paper was to search for rat models of interstitial fibrosis as a basis for testing therapeutic strategies to prevent end-stage renal failure. Experiments were done on adult female Wistar rats (Han:Wist) to investigate long-term consequences of temporary warm ischaemia, 5/6 nephrectomy (5/6 NX) and single uranyl nitrate (UN) administration (0.3 or 0.5 mg/ 100 g body wt. intraperitoneally). Observation time was 20 weeks after injury in each group. Creatinine clearance, urinary protein excret…

medicine.medical_specialtyPathologyTime Factorsmedicine.medical_treatmentUrologyRenal functionKidneyToxicologyNephrectomyPathology and Forensic MedicineExcretionFibrosismedicineRenal fibrosisAnimalsRats WistarKidneybusiness.industryKidney metabolismCell BiologyGeneral Medicinemedicine.diseaseFibrosisNephrectomyRatsDisease Models AnimalHydroxyprolineProteinuriamedicine.anatomical_structureCreatinineReperfusion InjuryUranyl NitrateNephritis InterstitialFemalebusinessKidney diseaseExperimental and Toxicologic Pathology
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Therapy with lamivudine and steroids in a patient with acute hepatitis B and rapidly progressive glomerulonephritis.

2006

To the Editor: Tang et al.1 recently reported that lamivudine (LAM) improves renal outcome in patients with chronic hepatitis B virus (HBV) infection and membranous nephropathy. We would like to add our experience on acute hepatitis B and rapidly progressive glomerulonephritis.

business.industryvirus diseasesLamivudinemedicine.diseasedigestive system diseasesVirusMembranous nephropathyChronic hepatitisNephrologyHepatitis B virus | Glomerulonephritis | membranous nephropathyImmunologymedicineRapidly progressive glomerulonephritisIn patientAcute hepatitis Bbusinessmedicine.drug
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Anti-C1q Autoantibodies in Lupus Nephritis: Prevalence and Clinical Significance

2005

Recently, anti-C1q autoantibodies have been proposed as a useful marker in systemic lupus erythematosus (SLE) since their occurrence correlates with renal involvement and, possibly, with nephritic activity. We aimed to evaluate the prevalence of anti-C1q antibodies in patients with SLE, with and without renal involvement, and to correlate these markers' presence and levels with the activity of the disease and nephropathy. We studied 61 patients with SLE, 40 of whom had biopsy-proven lupus nephritis; 35 patients with other connective tissue diseases; and 54 healthy controls. In addition, 18 lupus nephritis patients were followed up during the disease time course. Anti-C1q antibodies were mea…

BiopsySLELupus nephritisEnzyme-Linked Immunosorbent AssaySystemic lupus erythematosuAnti-DNA antibodieSeverity of Illness IndexGeneral Biochemistry Genetics and Molecular BiologyFollow-Up StudieNephropathyCohort StudiesHistory and Philosophy of Scienceimmune system diseasesAutoimmune diseasePrevalencemedicineHumansLupus Erythematosus SystemicConnective Tissue DiseasesGlomerulonephritiskin and connective tissue diseasesRenal flareConnective Tissue DiseaseAutoantibodiesAutoimmune diseaseBiochemistry Genetics and Molecular Biology (all)Systemic lupus erythematosusbusiness.industryLupus nephritiComplement C1qGeneral NeuroscienceAutoantibodyGlomerulonephritisBiomarkermedicine.diseaseLupus NephritisAutoantibodieAntibodies Anti-IdiotypicItalyAntibodies AntinuclearImmunologyAnti-C1q antibodieCohort StudiebusinessNephritisBiomarkersFollow-Up StudiesHumanAnti-SSA/Ro autoantibodiesAnnals of the New York Academy of Sciences
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TD-09 IL-34 promotes macrophage-mediated lupus nephritis in MRL-Faslpr mice

2018

Background Nephritis is the major cause of mortality and morbidity in patients with lupus. Macrophages (Mo) are central to kidney destruction in lupus-prone mice and patients. CSF-1, and the newly identified IL-34, mediate Mo survival and proliferation. However, IL-34 and CSF-1 differ during development and disease. While CSF-1 and IL-34 share the CSF-1 receptor (cFMS), expressed by Mo, IL-34 binds to a second receptor, Protein-Tyrosine Phosphatase ζ (PTPRZ) in inflamed kidneys. Intra-renal IL-34, cFMS, and PTPRZ are increased during the progression of lupus nephritis in MRL-Faslpr mice. Therefore, we hypothesized that IL-34 is a potential therapeutic target for lupus nephritis. Methods and…

KidneySystemic lupus erythematosusbusiness.industryLupus nephritisMonocyte proliferationmedicine.diseasePathogenesismedicine.anatomical_structureImmunologymedicineBone marrowbusinessReceptorNephritisTissue Damage
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