Search results for "NORADRENALINE"

showing 10 items of 16 documents

Plasma noradrenaline and the pressor action of exogenous noradrenaline in normotensive subjects and patients with essential hypertension.

1978

1. An inverse relationship was found between plasma noradrenaline and reactivity to exogenous noradrenaline in normotensive subjects. 2. The relationship between plasma noradrenaline and reactivity was disturbed in age-matched patients with essential hypertension. 3. A multiple-regression analysis showed a highly significant correlation between adrenergic activity and reactivity to noradrenaline and the mean arterial blood pressure level (r = 0·91). The results suggest that adrenergic activity and pressor response to noradrenaline combined are important determinants of arterial blood pressure. 4. An inverse relationship could also be demonstrated between plasma renin activity and reactivity…

AdultMalemedicine.medical_specialtyPlasma noradrenalinebusiness.industryAngiotensin IIAdrenergicBlood PressureGeneral MedicineEssential hypertensionmedicine.diseasePlasma renin activityAngiotensin IINorepinephrineEndocrinologyBlood pressurePressor responseInternal medicineHypertensionReninmedicineHumansFemalebusinessClinical science and molecular medicine. Supplement
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Sympathetic nervous activity and the pressor effect of noradrenaline under chronic?-?-adrenoceptor blockade with labetalol in hypertension

1983

In 14 patients with essential hypertension, the influence of the alpha- and beta-adrenoceptor blocking drug labetalol on blood pressure, heart rate, plasma renin, plasma noradrenaline and pressor effect of exogenous noradrenaline was investigated during long-term treatment. During the initial four weeks of treatment, labetalol at a dose of 400 mg/day showed a slight effect only on supine blood pressure, whereas upright blood pressure was already lowered effectively after the second week of treatment (p less than 0.01). An increase in the mean dose to 850 mg/day had an additional blood pressure-lowering effect (p less than 0.001), whereby a preferential decrease of the orthostatic blood pres…

Adultmedicine.medical_specialtySympathetic Nervous SystemSupine positionPostureAlpha (ethology)Blood PressureEssential hypertensionPlasma renin activityPlacebosNorepinephrineInternal medicineReceptors Adrenergic betaReninDrug DiscoveryHeart ratemedicineHumansDrug InteractionsLabetalolLabetalolGenetics (clinical)Plasma noradrenalineDose-Response Relationship Drugbusiness.industryGeneral MedicineMiddle AgedReceptors Adrenergic alphamedicine.diseaseReceptors AdrenergicEndocrinologyBlood pressureEthanolaminesChronic DiseaseHypertensionMolecular Medicinebusinesscirculatory and respiratory physiologymedicine.drugKlinische Wochenschrift
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The analysis of 51 genes in DSM-IV combined type attention deficit hyperactivity disorder: association signals in DRD4, DAT1 and 16 other genes.

2006

Contains fulltext : 35205.pdf (Publisher’s version ) (Closed access) Attention deficit hyperactivity disorder (ADHD) is a common neurodevelopmental disorder, starting in early childhood and persisting into adulthood in the majority of cases. Family and twin studies have demonstrated the importance of genetic factors and candidate gene association studies have identified several loci that exert small but significant effects on ADHD. To provide further clarification of reported associations and identify novel associated genes, we examined 1,038 single-nucleotide polymorphisms (SNPs) spanning 51 candidate genes involved in the regulation of neurotransmitter pathways, particularly dopamine, nor…

Candidate geneGenetics and epigenetic pathways of disease [NCMLS 6]MedizinReceptors NicotinicTryptophan HydroxylaseNeuroinformatics [DCN 3]0302 clinical medicinePerception and Action [DCN 1]Determinants in Health and Disease [EBP 1]ChildOncogene ProteinsGenetics0303 health sciencesbiologyDNA POOLING ANALYSISPedigree3. Good healthserotoninPsychiatry and Mental healthConduct disorderChild Preschool/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingMonoamine oxidase AdopaminePsychologyFunctional Neurogenomics [DCN 2]Genetic MarkersAdolescentSynaptosomal-Associated Protein 25Single-nucleotide polymorphismassociation studyPolymorphism Single NucleotideMental health [NCEBP 9]Genetic determinismGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceMONOAMINE-OXIDASE-ACognitive neurosciences [UMCN 3.2]SDG 3 - Good Health and Well-beingmental disordersmedicineHumansAttention deficit hyperactivity disorderADHDGenetic Predisposition to Disease5-HT1B RECEPTOR GENEddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersMonoamine OxidaseMolecular Biology030304 developmental biologyGenetic associationDopamine Plasma Membrane Transport ProteinsSEROTONIN TRANSPORTER GENEDOPAMINE-BETA-HYDROXYLASESiblingsReceptors Dopamine D4candidate genemedicine.diseaseTwin studyPREFERENTIAL TRANSMISSIONHaplotypesCATECHOL-O-METHYLTRANSFERASEAttention Deficit Disorder with HyperactivityCONDUCT DISORDERbiology.proteinnoradrenalineDEFICIT/HYPERACTIVITY DISORDERNO EVIDENCE030217 neurology & neurosurgerylinkage disequilibriumMolecular Psychiatry
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Modification of the monoaminergic neurotransmitters in frontal cortex and hippocampus by dietary trans alpha linolenic acid in piglets

2002

International audience; The effect of partial isomerization of dietary α-linolenic acid on the monoaminergic neurotransmitters in piglets was studied. After feeding the animals for 14 days with diets containing or not trans α-linolenic acid, neurotransmitters related to the monoaminergic function were quantified in the frontal cortex and in the hippocampus. The partial isomerization of dietary α-linolenic acid resulted in increasing endogenous monoamine levels in the frontal cortex (+55% for dopamine) and was related to a very low incorporation of trans polyunsaturated fatty acids. However, feeding animals with a diet in which the imbalance generated by the isomerization of α-linolenic acid…

DIETARY TRANSPOLYUNSATURATED FATTY ACIDSDOPAMINENORADRENALINEHIPPOCAMPUSFRONTAL CORTEX[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]NEW BORN PIGLET
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From the Golgi-Cajal mapping to the transmitter-based characterization of the neuronal networks leading to two modes of brain communication: Wiring a…

2007

After Golgi-Cajal mapped neural circuits, the discovery and mapping of the central monoamine neurons opened up for a new understanding of interneuronal communication by indicating that another form of communication exists. For instance, it was found that dopamine may be released as a prolactin inhibitory factor from the median eminence, indicating an alternative mode of dopamine communication in the brain. Subsequently, the analysis of the locus coeruleus noradrenaline neurons demonstrated a novel type of lower brainstem neuron that monosynaptically and globally innervated the entire CNS. Furthermore, the ascending raphe serotonin neuron systems were found to globally innervate the forebrai…

DopamineTortuosityBrain functionWiring transmissionSynaptic TransmissionDiffusionDual probe microdialysisMicrofluorimetrychemistry.chemical_compoundCatecholaminesPressure gradientsVolume transmissionHistofluorescenceLocus coeruleusExtracellular spaceNeurological and mental disordersNeurotransmitterNeuronsNeurotransmitter AgentsGeneral NeuroscienceBrain5-HydroxytryptamineAmygdalamedicine.anatomical_structure5-Hydroxytryptamine; Amygdala; Brain function; Brain uncoupling protein-2; Catecholamines; CA turnover; Clearance; Diffusion; Dopamine; Dorsal raphe; Dual probe microdialysis; Extracellular space; Extrasynaptic receptors; Histofluorescence; Local circuits; Locus coeruleus; Mapping of monoamine neurons; Microdensitometry; Microfluorimetry; Neurological and mental disorders; Noradrenaline; Nucleus accumbens; Pressure gradients; Receptor mosaics; Receptor–receptor interactions; Substantia nigra; Thermal gradients; Tortuosity; Transmitter–receptor mismatches; Volume fraction; Volume transmission; Wiring transmissionClearanceNucleus accumbensCA turnoverLocal circuitsReceptor–receptor interactionsSilver StainingMapping of monoamine neuronsModels NeurologicalNeurotransmissionBiologySerotonergicSubstantia nigramedicineBiological neural networkAnimalsHumansThermal gradientsTransmitter–receptor mismatchesVolume fractionExtrasynaptic receptorsMonoamine neurotransmitterchemistryReceptor mosaicsForebrainNoradrenalineLocus coeruleusBrain uncoupling protein-2Neurology (clinical)NeuronNerve NetMicrodensitometry5-Hydroxytryptamine Amygdala Brain function Brain uncoupling protein-2 Catecholamines CA turnover Clearance DiffusionNeuroscienceDorsal raphe
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Prenatal diazepam exposure functionally alters the GABA(A) receptor that modulates [3H]noradrenaline release from rat hippocampal synaptosomes.

2002

In rats, exposure to diazepam (DZ) during the last week of gestation is associated with behavioral alterations (in some cases sexually dimorphic) that appear when the animals reach adulthood. This study was conducted to evaluate the effects of prenatal DZ exposure on the function of the gamma-aminobutyric (GABA)(A) receptor complex. The method used - perfusion of rat hippocampal nerve terminals labeled with [3H]noradrenaline (NA) - allowed us to evaluate the effects of DZ on a specific native GABA(A) receptor subtype which is located on hippocampal noradrenergic nerve endings and mediates the release of NA. Muscimol stimulated synaptosomal release of [3H]NA in a concentration-dependent mann…

Fetal ProteinsMaleBaclofenNerve Tissue ProteinsPregnanoloneBicucullinein uteroHippocampusGABA AntagonistsNorepinephrineAllosteric RegulationPregnancyAnimalsPicrotoxinRats WistarGABA AgonistsDiazepam In utero [3H]Noradrenaline release Synaptosomes GABAA receptor Allosteric modulationallosteric modulationDiazepamMental DisordersGABAA receptorReceptors GABA-ARatsProtein SubunitsPrenatal Exposure Delayed EffectsSettore BIO/14 - FarmacologiaFemaleSynaptosomesDevelopmental neuroscience
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Modulation of high impulsivity and attentional performance in rats by selective direct and indirect dopaminergic and noradrenergic receptor agonists

2011

Rationale Impulsivity is associated with a number of psychiatric disorders, most notably attention deficit/hyperactivity disorder (ADHD). Drugs that augment catecholamine function (e.g. methylphenidate and the selective noradrenaline reuptake inhibitor atomoxetine) have clinical efficacy in ADHD, but their precise mechanism of action is unclear. Objective The objective of this study is to investigate the relative contribution of dopamine (DA) and noradrenaline (NA) to the therapeutic effects of clinically effective drugs in ADHD using rats selected for high impulsivity on the five-choice serial reaction time task (5CSRTT). Methods We examined the effects of direct and indirect DA and NA rec…

MaleImpulsivityQuinpiroleDopamineSerial LearningAtomoxetine HydrochlorideImpulsivityChoice BehaviorPiperazines03 medical and health sciences0302 clinical medicineQuinpiroleDopaminemental disordersAnimals Outbred StrainsReaction TimemedicineAnimalsAttentionOriginal InvestigationPharmacologyPropylaminesMethylphenidateDopaminergicAtomoxetineGBR-12909Adrenergic AgonistsGuanfacineRats030227 psychiatry3. Good healthGuanfacineSumaniroleFive-choice serial reaction time taskAtomoxetine; Dopamine; Five-choice serial reaction time task; GBR-12909; Guanfacine; Impulsivity; Methylphenidate; Noradrenaline; Quinpirole; Sumanirole; Adrenergic Agonists; Animals; Animals Outbred Strains; Atomoxetine Hydrochloride; Attention; Benzimidazoles; Choice Behavior; Dopamine Agonists; Guanfacine; Impulsive Behavior; Male; Methylphenidate; Piperazines; Propylamines; Quinpirole; Rats; Reaction Time; Serial Learning; PharmacologyAnesthesiaDopamine AgonistsImpulsive BehaviorNoradrenalineAtomoxetineMethylphenidateBenzimidazolesmedicine.symptomPsychologyNeuroscience030217 neurology & neurosurgerymedicine.drugAtomoxetine hydrochloride
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Role of calcineurin in Ca2+-induced release of catecholamines and neuropeptides

1998

Neurotransmission requires rapid docking, fusion, and recycling of neurotransmitter vesicles. Several of the proteins involved in this complex Ca2+-regulated mechanism have been identified as substrates for protein kinases and phosphatases, e.g., the synapsins, synaptotagmin, rabphilin3A, synaptobrevin, munc18, MARCKS, dynamin I, and B-50/GAP-43. So far most attention has focused on the role of kinases in the release processes, but recent evidence indicates that phosphatases may be as important. Therefore, we investigated the role of the Ca2+/calmodulin-dependent protein phosphatase calcineurin in exocytosis and subsequent vesicle recycling. Calcineurin-neutralizing antibodies, which blocke…

MaleSynaptobrevinCYCLOSPORINE-APhosphataseCalcineurin InhibitorsB-50 GAP-43Biologydynamin IBiochemistryBRAIN NERVE-TERMINALSExocytosisSynaptotagmin 1SincalidephosphataseGeneeskundeCellular and Molecular NeuroscienceNorepinephrineBacterial ProteinsPERMEATED SYNAPTOSOMESAnimalsratNEUROTRANSMITTER RELEASEMARCKSEnzyme InhibitorsRats WistarPROTEIN-KINASE-CDynaminCalcineurinTRANSMITTER RELEASEDYNAMIN-ISynapsinPhosphoric Monoester HydrolasesRatsINDUCED NORADRENALINE RELEASECalcineurinBiochemistryImmunoglobulin GStreptolysinsCalciumexocytosisCALMODULIN-BINDINGSynaptosomes
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Nicotine receptors do not modulate the 3H-noradrenaline release from the isolated rat heart evoked by sympathetic nerve stimulation.

1982

Isolated rat hearts with the right sympathetic nerves attached were perfused at a constant flow rate of 7 ml/min with Tyrode's solution. (-)-3H-Noradrenaline (final concentration 10–13.9 nM) was infused for 10 min to label the noradrenaline stores. After wash-out the sympathetic nerves were stimulated electrically (3 Hz, 180 impulses, 1 ms, 20–30 mA) three times (S1–S3) at intervals of 15 min. 3H-Noradrenaline and its metabolites were determined by liquid scintillation counting according to Graefe et al. (1973). Both, nicotine 50 μM and p-aminophenethyltrimethylammonium (PAPETA) 30 μM, enhanced the 3H-noradrenaline overflow in the absence of nerve stimulation. The effect of PAPETA was bipha…

Malemedicine.medical_specialtyNicotineSympathetic Nervous SystemSympathetic nerveStimulationIn Vitro TechniquesReceptors NicotinicTritiumReuptakeMethoxyhydroxyphenylglycol3h noradrenalineNicotinechemistry.chemical_compoundNorepinephrineInternal medicinemedicineAnimalsReceptors CholinergicReceptorPharmacologyNeuronsMyocardiumHeartRats Inbred StrainsGeneral MedicineRat heartElectric StimulationRatsQuaternary Ammonium CompoundsEndocrinologychemistryMandelic AcidsHexamethoniumFemalemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Die Wirkung von Ouabain auf die Elimination von Noradrenalin aus der Perfusionsfl�ssigkeit des isolierten Kaninchenherzens

1966

Isolated rabbit hearts were perfused with Tyrode solution containing 10 ng/ml (-)-noradrenaline for 20 min. The uptake by the heart of noradrenaline was calculated by estimating the percentage of amine removed from the perfusion fluid during a single passage through the heart. Previous experiments have shown that the amount of noradrenaline removed from the perfusion fluid corresponds well to the amount retained by the heart. In control experiments, the mean removal (elimination) of noradrenaline was 50±1.7% of the amount infused as measured biologically and 46±2.2% as measured fluorimetrically. Ouabain was added to the perfusion fluid at concentrations ranging from 2.74·10−7 to 2.0·10−6 mo…

PharmacologyInotropemedicine.medical_specialtyPotassiumNoradrenaline transportSodiumchemistry.chemical_elementGeneral Medicinemedicine.diseaseOuabainCell membraneEndocrinologymedicine.anatomical_structurechemistryInternal medicineMolemedicineAtrioventricular blockmedicine.drugNaunyn-Schmiedebergs Archiv f�r Pharmakologie und Experimentelle Pathologie
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