Search results for "Nectin"

showing 10 items of 355 documents

Metabolic syndrome pathophysiology: The role of adipose tissue

2006

Several pathophysiological explanations for the metabolic syndrome have been proposed involving insulin resistance, chronic inflammation and ectopic fat accumulation following adipose tissue saturation. However, current concepts create several paradoxes, including limited cardiovascular risk reduction with intensive glucose control in diabetics, therapies that result in weight gain (PPAR agonists), and presence of some of the metabolic traits among some lipodystrophies. We propose the functional failure of an organ, in this case, the adipose tissue as a model to interpret its manifestations and to reconcile some of the apparent paradox. A cornerstone of this model is the failure of the adip…

MalePerilipin-1medicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMedicine (miscellaneous)AdipokineAdipose tissueBiologySeverity of Illness IndexPPAR agonistInsulin resistanceRisk FactorsHyperinsulinismInternal medicinemedicineAnimalsHumansObesityMetabolic SyndromeNutrition and DieteticsAdiponectinInsulinPhosphoproteinsPrognosismedicine.diseaseDisease Models AnimalEndocrinologyAdipose TissuePerilipinFemaleAdiponectinInsulin ResistanceMetabolic syndromeCarrier ProteinsCardiology and Cardiovascular MedicineNutrition, Metabolism and Cardiovascular Diseases
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Visceral adiposity index (VAI) is predictive of an altered adipokine profile in patients with type 2 diabetes.

2014

Aims Although there is still no clear definition of “adipose tissue dysfunction” or ATD, the identification of a clinical marker of altered fat distribution and function may provide the needed tools for early identification of a condition of cardiometabolic risk. Our aim was to evaluate the correlations among various anthropometric indices [BMI, Waist Circumference (WC), Hip Circumference (HC), Waist/Hip ratio (WHR), Body Adiposity Index (BAI) and Visceral adiposity Index (VAI)] and several adipocytokines [Visfatin, Resistin, Leptin, Soluble leptin receptors (sOB-R), Adiponectin, Ghrelin, Adipsin, PAI-1, vascular endothelial growth factor (VEGF), Hepatocyte growth factor (HGF) TNF-α, hs-CRP…

MalePhysiologyEpidemiologylcsh:MedicineType 2 diabetesSettore MED/13 - EndocrinologiaEndocrinologyImmune PhysiologyMedicine and Health SciencesCluster AnalysisClinical Epidemiologylcsh:ScienceAdiposityMultidisciplinaryAnthropometryLeptinVAI adipokine diabetesMiddle AgedLipidsType 2 DiabetesPhysiological ParametersResearch DesignCytokinesFemaleAnatomyResearch Articlemedicine.medical_specialtyWaistClinical Research DesignImmunologyAdipokineEndocrine SystemIntra-Abdominal FatBody adiposity indexResearch and Analysis MethodsAdipokinesInternal medicineDiabetes MellitusmedicineHumansObesityAgedNutritionDiabetic EndocrinologyAdiponectinbusiness.industryBody Weightlcsh:RBiology and Life SciencesMolecular Developmentmedicine.diseaseEndocrinologyDiabetes Mellitus Type 2ROC CurveImmune SystemMetabolic DisordersClinical ImmunologyResistinlcsh:QMetabolic syndromebusinessDevelopmental BiologyPLoS ONE
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Inhibition of Protein S by Autoantibodies in Patients with Acquired Protein S Deficiency

1996

SummaryThis study was undertaken to analyze antibodies to protein S (PS) in patients with an acquired PS deficiency. Plasma from symptomatic patients with acquired (n = 14) or congenital (n = 10) PS deficiency and 10 healthy donors was screened for PS antibodies by immunoblotting and for anti-phospholipid antibodies. PS antibodies (IgG) were detected in five of the patients with acquired PS deficiency. These antibodies belonged to the G1 and G4 immunoglobulin subclasses. IgG fractions from the same 5 patients were shown to inhibit PS activity. The inhibition of PS activity by the 5 IgG fractions was shown to be time-and dose-dependent and was abolished following incubation with purified PS,…

MaleProtein S DeficiencyProtein SAbsorptionProtein Schemistry.chemical_compoundCoagulopathymedicineCardiolipinHumansAutoantibodiesDose-Response Relationship Drugbiologybusiness.industryAutoantibodyHematologymedicine.diseaseMolecular biologyBlood proteinschemistryCase-Control StudiesImmunoglobulin GImmunologyMonoclonalAntibodies Antiphospholipidbiology.proteinFemaleVitronectinAntibodybusinessThrombosis and Haemostasis
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Gene expression profiling of human stenotic aorto-coronary bypass grafts by cDNA array analysis

2003

Objective: Aorto-coronary bypass graft disease with its increasing clinical signification represents an unsolved problem in cardiological and heart surgery practice. Late occlusion of autologous saphenous vein grafts is due to medial and neointimal thickening secondary to migration and proliferation of smooth muscle cells (SMCs) and the subsequent formation of atherosclerotic plaques. This study is aimed at identifying differentially expressed genes in human stenotic bypass grafts to detect unknown pathomechanism and to identify novel targets for prophylactic treatment options. Methods: Stenotic saphenous aorto-coronary bypass grafts ðn ¼ 5Þ were retrieved during re-do aortocoronary bypass …

MaleReoperationPulmonary and Respiratory MedicineNeointimaPathologymedicine.medical_specialtyReceptor ErbB-3Proto-Oncogene Proteins c-junIn situ hybridizationProto-Oncogene MasCoronary RestenosisProto-Oncogene Proteins c-mycDownregulation and upregulationGene expressionmedicineHumansHSP70 Heat-Shock ProteinsSaphenous VeinCoronary Artery BypassVeinAgedOligonucleotide Array Sequence Analysismedicine.diagnostic_testbusiness.industryGene Expression ProfilingGeneral Medicinemedicine.diseaseFibronectinsGene expression profilingStenosismedicine.anatomical_structureFemaleSurgeryCardiology and Cardiovascular MedicinebusinessFluorescence in situ hybridizationEuropean Journal of Cardio-Thoracic Surgery
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Liver and pancreatic fat content and metabolism in healthy monozygotic twins with discordant physical activity

2011

Background & Aims: Ectopic fat in muscle and liver is linked to obesity and type 2 diabetes. Recently, pancreatic lipid accumulation has also been associated with beta-cell dysfunction and reduced insulin production, leading to the development of type 2 diabetes. Physical exercise training has been shown to attenuate beta-cell dysfunction in patients, but little is known about its effects on pancreatic and hepatic fat accumulation. In this study, we validated in-vivo proton magnetic resonance spectroscopy ((1)H MRS) in pancreatic fat measurement with biochemical measurements in a pig model. Thereafter, the effects of increased physical activity on the amounts of pancreatic and liver fat…

MaleSwinemedicine.medical_treatmentSus scrofaAdipose tissueMonozygotic twinACID UPTAKEType 2 diabetesFatty Acids NonesterifiedFat Measurement0302 clinical medicineFatty AcidsHEPATIC INSULIN-RESISTANCEMagnetic Resonance Imagingmedicine.anatomical_structureADIPOSE-TISSUEAdipose TissueLiverModels AnimalSwine Miniature030211 gastroenterology & hepatologyPancreasMonozygotic twinsAdultmedicine.medical_specialty030209 endocrinology & metabolismDEPENDENT DIABETES-MELLITUSMotor ActivityBiologyta3111HISPANIC ADOLESCENTSYoung Adult03 medical and health sciencesInsulin resistanceBETA-CELL FUNCTIONInternal medicineMagnetic resonance spectroscopymedicineMAGNETIC-RESONANCE SPECTROSCOPYAnimalsHumansADIPONECTIN CONCENTRATIONSPancreasPLASMA ADIPONECTINHepatologyPhysical activityInsulinTRIGLYCERIDE CONTENTTwins MonozygoticLipid Metabolismmedicine.diseaseObesityEndocrinologyInsulin ResistanceJournal of Hepatology
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Engineered microenvironments for synergistic VEGF - Integrin signalling during vascularization

2017

We have engineered polymer-based microenvironments that promote vasculogenesis both in vitro and in vivo through synergistic integrin-growth factor receptor signalling. Poly(ethyl acrylate) (PEA) triggers spontaneous organization of fibronectin (FN) into nanonetworks which provide availability of critical binding domains. Importantly, the growth factor binding (FNIII12-14) and integrin binding (FNIII9-10) regions are simultaneously available on FN fibrils assembled on PEA. This material platform promotes synergistic integrin/VEGF signalling which is highly effective for vascularization events in vitro with low concentrations of VEGF. VEGF specifically binds to FN fibrils on PEA compared to …

MaleVascular Endothelial Growth Factor AIntegrinsBiophysicsNeovascularization PhysiologicBioengineeringpoly(ethyl acrylate)ArticleBiomaterialsHuman Umbilical Vein Endothelial CellsImage Processing Computer-AssistedAnimalsHumansPhosphorylationExtracellular Signal-Regulated MAP KinasesFibronectinTissue EngineeringPhospholipase C gammaProtein assemblyVascularizationVEGFFibronectinsMice Inbred C57BLCellular MicroenvironmentMechanics of MaterialsFocal Adhesion Protein-Tyrosine KinasesFISICA APLICADAMutationCeramics and CompositesINGENIERIA ELECTRICAGrowth factorsProtein BindingSignal Transduction
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Perlecan-Induced Suppression of Smooth Muscle Cell Proliferation Is Mediated Through Increased Activity of the Tumor Suppressor PTEN

2004

We were interested in the elucidation of the interaction between the heparan sulfate proteoglycan, perlecan, and PTEN in the regulation of vascular smooth muscle cell (SMC) growth. We verified serum-stimulated DNA synthesis, and Akt and FAK phosphorylation were significantly reduced in SMCs overexpressing wild-type PTEN. Our previous studies showed perlecan is a potent inhibitor of serum-stimulated SMC growth. We report in the present study, compared with SMCs plated on fibronectin, serum-stimulated SMCs plated on perlecan exhibited increased PTEN activity, decreased FAK and Akt activities, and high levels of p27, consistent with SMC growth arrest. Adenoviral-mediated overexpression of cons…

MaleVascular smooth musclePhysiology:CIENCIAS MÉDICAS ::Farmacodinámica [UNESCO]Aorta ThoracicBasement MembraneCulture Media Serum-FreeMuscle Smooth VascularRats Sprague-DawleyMicePhosphorylationCells CulturedGlycosaminoglycansbiologyProtein-Tyrosine KinasesCell cycle:CIENCIAS MÉDICAS [UNESCO]musculoskeletal systemUNESCO::CIENCIAS MÉDICAS ::FarmacodinámicaUNESCO::CIENCIAS MÉDICAScardiovascular systemPhosphorylationSmooth muscle cell proliferationCardiology and Cardiovascular MedicineCell DivisionDNA ReplicationBasement membraneRecombinant Fusion ProteinsPerlecanProtein Serine-Threonine KinasesVascular injurySmooth muscle cell proliferation ; Restenosis ; Vascular injury ; Vascular development ; Basement membraneCatheterizationProto-Oncogene ProteinsAnimalsPTENProtein kinase BRestenosisCell growthVascular developmentOligonucleotides AntisenseFibronectinsRatsFibronectinFocal Adhesion Kinase 1Focal Adhesion Protein-Tyrosine Kinasesbiology.proteinCancer researchHeparitin SulfateCarotid Artery InjuriesProtein Processing Post-TranslationalProto-Oncogene Proteins c-aktHeparan Sulfate ProteoglycansCirculation Research
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Matrix metalloproteinase-12 cleaved fragment of titin as a predictor of functional capacity in patients with heart failure and preserved ejection fra…

2020

Serum levels of matrix metalloproteinase-12 cleaved fragment of titin (TIM), a novel circulatory biomarker specific for cardiac titin degradation, has emerged as a potential biomarker in cardiovascular diseases. In this work, we aimed to evaluate the association between TIM and maximal functional capacity assessed by the percentage of predicted peak exercise oxygen uptake (pp-peakVO2) in patients with heart failure and preserved ejection fraction (HFpEF). Design. In this post-hoc study, we included 46 stable symptomatic (New York Heart Association II-III) HFpEF patients enrolled in the TRAINING-HF study (NCT02638961). pp-peak-VO2 was calculated from baseline values. Baseline circulating lev…

Maleanimal structuresfunctional capacity030204 cardiovascular system & hematologyMatrix (biology)Matrix metalloproteinase03 medical and health sciences0302 clinical medicineMatrix Metalloproteinase 12HumansMedicineConnectin030212 general & internal medicineExerciseAgedAged 80 and overHeart FailureEjection fractionbiologybusiness.industryStroke Volumetitin degradationmusculoskeletal systemmedicine.diseaseMolecular biologyheartfailure with preservedejection fractionHeart failureembryonic structuresCirculatory systemcardiovascular systembiology.proteinBiomarker (medicine)FemaleTitinCardiology and Cardiovascular MedicinebusinessHeart failure with preserved ejection fractiontissuesBiomarkers
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Association of adiponectin with adverse outcome in coronary artery disease patients: results from the AtheroGene study

2008

In primary prevention, the adipocytokine adiponectin seems to be protective against diabetes mellitus and cardiovascular disease. Data in patients with manifest coronary artery disease (CAD) are scant stimulating the investigation of the association of adiponectin concentrations and cardiovascular outcome in a prospective CAD cohort.In 1890 consecutive patients with documented CAD [1130 with stable angina (SAP) and 760 with acute coronary syndrome (ACS)] baseline concentrations of adiponectin were measured by enzyme-linked immuno assay. During a median follow-up of 2.5 years cardiovascular events were registered (cardiovascular deaths 70; non-fatal myocardial infarction 46). Baseline adipon…

Malemedicine.medical_specialtyAdiponectinbusiness.industryCoronary Artery DiseaseMiddle AgedBrain natriuretic peptidemedicine.diseaseAngina PectorisCoronary artery diseaseDiabetes mellitusInternal medicinemedicineCardiologyHumansPopulation studyFemaleAdiponectinMyocardial infarctionAcute Coronary SyndromeRisk factorEpidemiologic MethodsCardiology and Cardiovascular MedicinebusinessComplicationEuropean Heart Journal
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Coagulation factors and proteinase inhibitors in the plasma of children with acute lymphoblastic leukoses. Behaviour before and during treatment acco…

1984

The thrombocyte count, the factor XIII (F XIII) activity, the concentration of fibrinogen (F I), prothrombin (F II), fibronectin (CIG), albumin and the proteinase inhibitors antithrombin III (AT III), alpha 2-macroglobulin (A2M), alpha 1-antitrypsin (A1A) and Cl-esterase inactivator (Cl-INA) were determined in ten children with acute lymphoblastic leukaemia (ALL). Changes due to the disease and to therapy were observed. Before the start of treatment the patients had thrombocytopenia secondary to the disease, and the proteinase inhibitors--especially Cl-INA and A1A--were raised. During the induction phase the thrombocyte count rose but there was also a marked increase in the concentration of…

Malemedicine.medical_specialtyAdolescentAntithrombin IIIAlpha (ethology)Complement C1 Inactivator ProteinsFibrinogenMaintenance therapyInternal medicineDrug DiscoveryAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansProtease Inhibitorsalpha-MacroglobulinsChildGenetics (clinical)Factor XIIIbusiness.industryAntithrombinAlbuminFibrinogenGeneral MedicineFactor XIIIMolecular medicineBlood Coagulation FactorsFibronectinsLeukemia LymphoidEndocrinologyCoagulationChild Preschoolalpha 1-AntitrypsinImmunologyMolecular MedicineFemaleProthrombinbusinessmedicine.drugKlinische Wochenschrift
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