Search results for "Neovascularization"

showing 10 items of 351 documents

CD34+ progenitor to endothelial cell transition in post-pneumonectomy angiogenesis.

2012

In many species, pneumonectomy triggers compensatory lung growth that results in an increase not only in lung volume, but also in alveolar number. Whether the associated alveolar angiogenesis involves the contribution of blood-borne progenitor cells is unknown. To identify and characterize blood-borne progenitor cells contributing to lung growth after pneumonectomy in mice, we studied wild-type and wild-type/green fluorescence protein (GFP) parabiotic mice after left pneumonectomy. Within 21 days of pneumonectomy, a 3.2-fold increase occurred in the number of lung endothelial cells. This increase in total endothelial cells was temporally associated with a 7.3-fold increase in the number of …

Pulmonary and Respiratory MedicineTranscriptional ActivationPathologymedicine.medical_specialtyTime FactorsAngiogenesisCellular differentiationClinical BiochemistryGreen Fluorescent ProteinsCD34Neovascularization PhysiologicAntigens CD34Mice TransgenicBiologyMiceVasculogenesisCell MovementmedicineAnimalsRegenerationProgenitor cellPneumonectomyMolecular BiologyLungCell ProliferationStem CellsEndothelial CellsCell DifferentiationCell BiologyArticlesEndothelial stem cellVascular endothelial growth factor BMice Inbred C57BLGene Expression RegulationCancer researchStem cellAmerican journal of respiratory cell and molecular biology
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A new pyrazolo pyrimidine derivative inhibitor of cyclooxygenase-2 with anti-angiogenic activity

2003

In a previous study, we reported a new pyrazolo pyrimidine derivative, N(4)-benzyl-N(6),N(6)-dimethyl-1-1(tert-butyl)-1H-pyrazolo[3,4-d]pyrimidine-6,4-diamine (DPP), which inhibited potently cyclooxygenase-2 activity in intact cell assays with minor activity against cyclooxygenase-1 (IC(50)=0.9 nM for cyclooxygenase-2 versus IC(50)=59.6 nM for cyclooxygenase-1). In the present work, this behaviour was confirmed in vivo by using the 24-h zymosan-injected mouse air pouch model (ID(50)=1.36 nM/pouch for prostaglandin E(2) level). We also studied the possible beneficial effect of DPP in the angiogenesis-dependent murine air pouch granuloma and rat paw carrageenan-induced hyperalgesia models. DP…

Pyrimidinemedicine.medical_treatmentAngiogenesis InhibitorsPharmacologyCarrageenanDinoprostoneMicechemistry.chemical_compoundIn vivomedicineAnimalsEdemaCyclooxygenase InhibitorsRats WistarProstaglandin E2IC50NitrobenzenesPharmacologySulfonamidesGranulomaCyclooxygenase 2 InhibitorsNeovascularization PathologicbiologyTumor Necrosis Factor-alphaZymosanRatsIsoenzymesPyrimidinesEicosanoidchemistryBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesHyperalgesiabiology.proteinPyrazolesFemaleCyclooxygenasemedicine.symptomInterleukin-1Prostaglandin Emedicine.drugEuropean Journal of Pharmacology
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Endothelial cells and normal breast epithelial cells enhance invasion of breast carcinoma cells by CXCR-4-dependent up-regulation of urokinase-type p…

2008

Here we show the increase of invasion of three breast cancer cell lines (8701-BC, MDA-MB-231 and SKBR3) upon long-term co-incubation with culture medium of normal microvascular endothelial cells (MVEC) and normal breast epithelial cells (HB2). The enhancement of invasion relied on the interaction of microvascular endothelial cell and normal breast epithelial cell CXCL12 (SDF1) chemokine, whose expression by breast cancer cells was very low, with the cognate CXCR4 receptor of malignant cells, which resulted in over-expression of the urokinase-type plasminogen activator receptor (uPAR) on their surfaces. uPAR over-expression, showed by RT-PCR and Western blotting, was paralleled by increased …

Receptors CXCR4MAP Kinase Kinase 4AngiogenesisCellBreast NeoplasmsReceptors Cell SurfaceCell CommunicationBiologyCell LineReceptors Urokinase Plasminogen ActivatorPathology and Forensic MedicineMetastasisangiogenesisbreast cancerTumor Cells CulturedmedicineHumansNeoplasm InvasivenessBreastSettore BIO/06 - Anatomia Comparata E CitologiaPhosphorylationskin and connective tissue diseasesCXCR4Settore MED/04 - Patologia GeneraleNeovascularization PathologicReverse Transcriptase Polymerase Chain ReactionFibrinolysisEpithelial CellsCXCL12invasionmedicine.diseasemicroenvironmentChemokine CXCL12Neoplasm ProteinsUp-RegulationEndothelial stem cellUrokinase receptormedicine.anatomical_structureCulture Media ConditionedCancer cellCancer researchFemaleJNKEndothelium VascularBreast diseaseSDF1uPARPlasminogen activatorThe Journal of Pathology
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C-X-C Motif Chemokine Receptor 4 Blockade Promotes Tissue Repair After Myocardial Infarction by Enhancing Regulatory T Cell Mobilization and Immune-R…

2019

Background: Acute myocardial infarction (MI) elicits an inflammatory response that drives tissue repair and adverse cardiac remodeling. Inflammatory cell trafficking after MI is controlled by C-X-C motif chemokine ligand 12 (CXCL12) and its receptor, C-X-C motif chemokine receptor 4 (CXCR4). CXCR4 antagonists mobilize inflammatory cells and promote infarct repair, but the cellular mechanisms are unclear. Methods: We investigated the therapeutic potential and mode of action of the peptidic macrocycle CXCR4 antagonist POL5551 in mice with reperfused MI. We applied cell depletion and adoptive transfer strategies using lymphocyte-deficient Rag1 knockout mice; DEREG mice, which express a diphth…

Receptors CXCR4Regulatory T cellCXCR4 antagonistSus scrofaAnti-Inflammatory AgentsMyocardial InfarctionNeovascularization PhysiologicMice TransgenicInflammation030204 cardiovascular system & hematologyT-Lymphocytes RegulatoryVentricular Function Left03 medical and health sciencesChemokine receptor0302 clinical medicineImmune systemPhysiology (medical)medicineAnimalsMyocardial infarction030304 developmental biology0303 health sciencesMobilizationVentricular Remodelingbusiness.industryMyocardiumProteinsDendritic CellsRecovery of FunctionRegulatory T cellsTissue repairmedicine.diseaseMyocardial ContractionBlockadeMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureCancer researchmedicine.symptomCardiology and Cardiovascular MedicinebusinessSignal TransductionCirculation
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The topology of vitronectin: A complementary feature for neuroblastoma risk classification based on computer‐aided detection

2019

Tumors are complex networks of constantly interacting elements: tumor cells, stromal cells, immune and stem cells, blood/lympathic vessels, nerve fibers and extracellular matrix components. These elements can influence their microenvironment through mechanical and physical signals to promote tumor cell growth. To get a better understanding of tumor biology, cooperation between multidisciplinary fields is needed. Diverse mathematic computations and algorithms have been designed to find prognostic targets and enhance diagnostic assessment. In this work, we use computational digital tools to study the topology of vitronectin, a glycoprotein of the extracellular matrix. Vitronectin is linked to…

RiskCancer ResearchStromal celltopologyTumor Markers and SignaturesComputer scienceAngiogenesisTopologyTopologyvitronectinExtracellular matrixComputational biology03 medical and health sciencesNeuroblastoma0302 clinical medicinecomputational biologyNeuroblastNeuroblastomamedicineTumor MicroenvironmentHumansVitronectinCell ProliferationbiologyNeovascularization PathologicComplex networkmedicine.diseasePrognosisExtracellular MatrixOncology030220 oncology & carcinogenesisnetworksbiology.proteinVitronectinStem cellNetworksStromal CellsAlgorithmsInternational Journal of Cancer
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Inhibition of VEGF expression through blockade of Hif1a and STAT3 signalling mediates the anti-angiogenic effect of melatonin in HepG2 liver cancer c…

2013

Background: Hepatocellular carcinoma (HCC) growth relies on angiogenesis via vascular endothelial growth factor (VEGF) release. Hypoxia within tumour environment leads to intracellular stabilisation of hypoxia inducible factor 1 alpha (Hif1α) and signal transducer and activator of transcription (STAT3). Melatonin induces apoptosis in HCC, and shows anti-angiogenic features in several tumours. In this study, we used human HepG2 liver cancer cells as an in vitro model to investigate the anti-angiogenic effects of melatonin. Methods: HepG2 cells were treated with melatonin under normoxic or CoCl2-induced hypoxia. Gene expression was analysed by RT–qPCR and western blot. Melatonin-induced anti-…

STAT3 Transcription FactorTranscriptional ActivationVascular Endothelial Growth Factor ACancer ResearchCarcinoma HepatocellularTranscription GeneticAngiogenesisAngiogenesis InhibitorsApoptosismelatoninP300-CBP Transcription FactorsHif1αBiologyMelatoninSTAT3chemistry.chemical_compoundHypoxia-Inducible Factor 1-AlphamedicineHuman Umbilical Vein Endothelial CellsHumansp300-CBP Transcription FactorsSTAT3Promoter Regions GeneticTube formationNeovascularization PathologicLiver NeoplasmsCobaltHep G2 Cellshepatocellular carcinomaHypoxia-Inducible Factor 1 alpha SubunitVEGFCell Hypoxiadigestive system diseasesCyclic S-OxidesVascular endothelial growth factorGene Expression Regulation NeoplasticVascular endothelial growth factor AOncologychemistryCancer researchbiology.proteinTranslational Therapeuticsmedicine.drugSignal Transduction
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Channeled scaffolds implanted in adult rat brain.

2012

Scaffolds with aligned channels based on acrylate copolymers, which had previously demonstrated good com- patibility with neural progenitor cells were studied as coloniz- able structures both in vitro with neural progenitor cells and in vivo, implanted without cells in two different locations, in the cortical plate of adult rat brains and close to the subven- tricular zone. In vitro, neuroprogenitors colonize the scaffold and differentiate into neurons and glia within its channels. When implanted in vivo immunohistochemical analysis by confocal microscopy for neural and endothelial cells markers demonstrated that the scaffolds maintained continuity with the surrounding neural tissue and wer…

ScaffoldAgingMaterials scienceAngiogenesisbrainBiomedical EngineeringSubventricular zoneNeovascularization PhysiologicScaffold SeedingNeural tissue engineeringGlial scarScaffoldBiomaterialsangiogenesisbiocompatibilityImplants ExperimentalNeural Stem CellsIn vivomedicineAnimalsRats WistarCerebral CortexNeuronsTissue ScaffoldsMetals and AlloysBrainCell DifferentiationNeural stem cellRatsAdult Stem Cellsmedicine.anatomical_structureMicroscopy FluorescenceMAQUINAS Y MOTORES TERMICOSCeramics and CompositesMicroscopy Electron ScanningFemaleneural regenerationNeurogliaBiomedical engineeringStem Cell TransplantationJournal of biomedical materials research. Part A
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Non-cross-linked porcine-based collagen I-III membranes do not require high vascularization rates for their integration within the implantation bed: …

2012

There are conflicting reports concerning the tissue reaction of small animals to porcine-based, non-cross-linked collagen I-III membranes/matrices for use in guided tissue/bone regeneration. The fast degradation of these membranes/matrices combined with transmembrane vascularization within 4 weeks has been observed in rats compared with the slow vascularization and continuous integration observed in mice. The aim of the present study was to analyze the tissue reaction to a porcine-based non-cross-linked collagen I-III membrane in mice. Using a subcutaneous implantation model, the membrane was implanted subcutaneously in mice for up to 60 days. The extent of scaffold vascularization, tissue …

ScaffoldMaterials scienceBarrier membraneSus scrofaBiomedical EngineeringFibroinNeovascularization PhysiologicBiochemistryCollagen Type IBiomaterialsProsthesis ImplantationMicemedicineAnimalsBone regenerationMolecular BiologyPolytetrafluoroethyleneMembranesTissue ScaffoldsGranulation tissueMembranes ArtificialGeneral MedicineImmunohistochemistryTransmembrane proteinRatsmedicine.anatomical_structureMembraneCollagen Type IIICross-Linking ReagentsGiant cellBiophysicsMicroscopy Electron ScanningFemaleFibroinsBiotechnologyBiomedical engineeringActa biomaterialia
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Electrospun PHEA-PLA/PCL Scaffold for Vascular Regeneration: A Preliminary in Vivo Evaluation

2017

Abstract Background There is increasing interest in the development of vessel substitutes, and many studies are currently focusing on the development of biodegradable scaffolds capable of fostering vascular regeneration. We tested a new biocompatible and biodegradable material with mechanical properties similar to those of blood vessels. Methods The material used comprises a mixture of α,β-poly(N-2-hydroxyethyl)- d,l -aspartamide (PHEA) and polylactic acid (PLA), combined with polycaprolactone (PCL) by means of electrospinning technique. Low-molecular-weight heparin was also linked to the copolymer. A tubular PHEA-PLA/PCL sample was used to create an arteriovenous fistula in a pig model wit…

ScaffoldMaterials scienceBiocompatibilityPolymersSwinePolyesters0206 medical engineering02 engineering and technologySettore MED/22 - Chirurgia VascolareNeovascularizationchemistry.chemical_compoundPolylactic acidBlood vessel prosthesismedicineAnimalsTransplantationRegeneration (biology)Bioabsorbable scaffold Bioengineered vascular scaffold Experimental surgery021001 nanoscience & nanotechnology020601 biomedical engineeringBlood Vessel ProsthesisSettore MED/18 - Chirurgia GeneraleCoagulative necrosischemistrySettore CHIM/09 - Farmaceutico Tecnologico ApplicativoPolycaprolactoneSurgerymedicine.symptomPeptides0210 nano-technologyBiomedical engineeringTransplantation Proceedings
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The pre-vascularisation of a collagen-chondroitin sulphate scaffold using human amniotic fluid-derived stem cells to enhance and stabilise endothelia…

2015

Abstract A major problem in tissue engineering (TE) is graft failure in vivo due to core degradation in in vitro engineered constructs designed to regenerate thick tissues such as bone. The integration of constructs post-implantation relies on the rapid formation of functional vasculature. A recent approach to overcome core degradation focuses on the creation of cell-based, pre-engineered vasculature formed within the TE construct in vitro , prior to implantation in vivo . The primary objective of this study was to investigate whether an amniotic fluid-derived stem cell (AFSC)–human umbilical vein endothelial cell (HUVEC) co-culture could be used to engineer in vitro vasculature in a collag…

ScaffoldMaterials scienceBiomedical EngineeringNeovascularization PhysiologicBiochemistryUmbilical veinBiomaterialsTissue engineeringBlood vessel prosthesisIn vivoMaterials TestingHumansBone regenerationMolecular BiologyCells CulturedBioprosthesisTissue ScaffoldsStem CellsChondroitin SulfatesEndothelial CellsEquipment DesignGeneral MedicineAmniotic FluidBlood Vessel ProsthesisCapillariesCell biologyEquipment Failure AnalysisEndothelial stem cellCollagenStem cellStem Cell TransplantationBiotechnologyBiomedical engineeringActa Biomaterialia
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