Search results for "Neural cell"

showing 10 items of 97 documents

Characterization and isolation of immature neurons of the adult mouse piriform cortex

2015

Physiological studies indicate that the piriform or primary olfactory cortex of adult mammals exhibits a high degree of synaptic plasticity. Interestingly, a subpopulation of cells in the layer II of the adult piriform cortex expresses neurodevelopmental markers, such as the polysialylated form of neural cell adhesion molecule (PSA-NCAM) or doublecortin (DCX). This study analyzes the nature, origin, and potential function of these poorly understood cells in mice. As previously described in rats, most of the PSA-NCAM expressing cells in layer II could be morphologically classified as tangled cells and only a small proportion of larger cells could be considered semilunar-pyramidal transitiona…

0301 basic medicinebiologyNeurogenesisDoublecortinCell biology03 medical and health sciencesCellular and Molecular NeurosciencePrimary olfactory cortex030104 developmental biology0302 clinical medicinenervous systemDevelopmental NeuroscienceAntigenNeuroblastPiriform cortexSynaptic plasticitybiology.proteinNeural cell adhesion moleculeNeuroscience030217 neurology & neurosurgeryDevelopmental Neurobiology
researchProduct

Effects of Dopamine on the Immature Neurons of the Adult Rat Piriform Cortex

2020

The layer II of the adult piriform cortex (PCX) contains a numerous population of immature neurons. Interestingly, in both mice and rats, most, if not all, these cells have an embryonic origin. Moreover, recent studies from our laboratory have shown that they progressively mature into typical excitatory neurons of the PCX layer II. Therefore, the adult PCX is considered a “non-canonical” neurogenic niche. These immature neurons express the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a molecule critical for different neurodevelopmental processes. Dopamine (DA) is a relevant neurotransmitter in the adult CNS, which also plays important roles in neural development and …

0301 basic medicinedopamine D2 receptorPSA-NCAMPopulationBiologylcsh:RC321-57103 medical and health scienceschemistry.chemical_compoundpiriform cortex0302 clinical medicineDopaminePiriform cortexDopamine receptor D2medicineeducationNeurotransmitterlcsh:Neurosciences. Biological psychiatry. Neuropsychiatryeducation.field_of_studyGeneral NeuroscienceDopaminergicBrief Research ReportCell biology030104 developmental biologychemistrynervous systemplasticityNeural cell adhesion moleculedopamineNeural development030217 neurology & neurosurgeryNeurosciencemedicine.drug
researchProduct

Effects of PSA Removal from NCAM on the Critical Period Plasticity Triggered by the Antidepressant Fluoxetine in the Visual Cortex.

2016

Neuronal plasticity peaks during critical periods of postnatal development and is reduced towards adulthood. Recent data suggests that windows of juvenile-like plasticity can be triggered in the adult brain by antidepressant drugs such as Fluoxetine. Although the exact mechanisms of how Fluoxetine promotes such plasticity remains unknown, several studies indicate that inhibitory circuits play an important role. The polysialylated form of the neural cell adhesion molecules (PSA-NCAM) has been suggested to mediate the effects of Fluoxetine and it is expressed in the adult brain by mature interneurons. Moreover, the enzymatic removal of PSA by neuroaminidase-N not only affects the structure of…

0301 basic medicinegenetic structuresPSA-NCAMta3112lcsh:RC321-571critical period plasticity03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineparvalbumin interneuronsSYNAPTIC PLASTICITYNeuroplasticitymedicinevisual plasticityMONOCULAR DEPRIVATIONlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryREGULATES PLASTICITYOriginal ResearchbiologyMEDIAL PREFRONTAL CORTEXPOLYSIALIC ACID3112 NeurosciencesCELLULAR AND MOLECULAR NEUROSCIENCEfluoxetineLong-term potentiationSciences bio-médicales et agricoles3. Good healthOCULAR DOMINANCE PLASTICITYMonocular deprivation030104 developmental biologyVisual cortexmedicine.anatomical_structureSTRUCTURAL PLASTICITYnervous systemCELL-ADHESION MOLECULESynaptic plasticitybiology.proteinNeural cell adhesion moleculeLONG-TERM POTENTIATIONPsychologyNeuroscience030217 neurology & neurosurgeryParvalbuminNeuroscienceNEUROTROPHIC FACTORFOSB
researchProduct

Occurrence of new neurons in the piriform cortex

2015

In a recent mini-review (Yuan et al., 2015), support is given to the idea that neurons are generated during adulthood in the mammalian piriform cortex (PC), their periventricular origin being also discussed. It is known since long time that a subpopulation of cortical layer II cells in the adult PC of rodents express immature neuronal markers such as polysialylated NCAM (PSA-NCAM; Seki and Arai, 1991; Bonfanti et al., 1992) and doublecortin (DCX; Nacher et al., 2002). These immature neurons have been found in most mammals studied so far, their occurrence being restricted to the paleocortex in rodents (Seki and Arai, 1991; Bonfanti et al., 1992; Nacher et al., 2002), and extended to neocorti…

Adult neurogenesis; Doublecortin; Piriform cortex; PSA-NCAM; Structural plasticity; Anatomy; Neuroscience (miscellaneous); Cellular and Molecular NeuroscienceOlfactory systembiologyGeneral CommentaryPSA-NCAMNeurogenesisNeuroscience (miscellaneous)Embryonic stem cellstructural plasticityOlfactory bulbDoublecortinadult neurogenesispiriform cortexCellular and Molecular Neurosciencenervous systemdoublecortinPiriform cortexBrain sizebiology.proteinNeural cell adhesion moleculeAnatomyNeuroscienceNeuroscienceFrontiers in Neuroanatomy
researchProduct

PSA-NCAM expression in the human prefrontal cortex.

2006

The prefrontal cortex (PFC) of adult rodents is capable of undergoing neuronal remodeling and neuroimaging studies in humans have revealed that the structure of this region also appears affected in different psychiatric disorders. However, the cellular mechanisms underlying this plasticity are still unclear. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) may mediate these structural changes through its anti-adhesive properties. PSA-NCAM participates in neurite outgrowth and synaptogenesis and changes in its expression occur parallel to neuronal remodeling in certain regions of the adult brain. PSA-NCAM is expressed in the hippocampus and temporal cortex of adult hum…

AdultCalbindinsNeuropilInterneuronHippocampusFluorescent Antibody TechniquePrefrontal CortexNeural Cell Adhesion Molecule L1RodentiaCellular and Molecular NeuroscienceS100 Calcium Binding Protein GSpecies SpecificityInterneuronsNeuroplasticityNeuropilmedicineCell AdhesionAnimalsHumansPrefrontal cortexAgedTemporal cortexDepressive DisorderNeuronal PlasticitybiologyDendritesMiddle AgedAxonsDoublecortinmedicine.anatomical_structurenervous systembiology.proteinSialic AcidsNeural cell adhesion moleculePsychologyNeuroscienceJournal of chemical neuroanatomy
researchProduct

Expression of adhesion factors and degrading proteins in primary and secondary glioblastomas and their precursor tumors.

2000

In tumor tissue specimens of 27 primary and 17 secondary glioblastomas and the precursor lesions, the immunohistochemical expression patterns of the membrane protein CD44s, the basal lamina proteins laminin, collagen IV, and fibronectin, the lectin galectin-3 recognizing tenascin and N-CAM as well as of the matrix-degrading enzymes matrix metalloproteinase MMP-2 and MMP-9, and cathepsin D were studied. Besides expression of basal lamina proteins in vessels, all glioblastomas and the precursor lesions showed strong immunoreactivity of CD44s, tenascin, galectin-3, and N-CAM which were restricted to solid tumor masses. Present in solid tumor areas, MMP-2, MMP-9 and cathepsin D were also strong…

AdultCancer Researchanimal structuresGalectin 3TenascinCathepsin DBiologyAstrocytomaCathepsin DLamininGliomamedicineHumansCell adhesionNeural Cell Adhesion MoleculesBrain NeoplasmsMiddle Agedmedicine.diseaseMolecular biologyAntigens DifferentiationImmunohistochemistryMatrix MetalloproteinasesFibronectinsFibronectinmedicine.anatomical_structureHyaluronan ReceptorsMembrane proteinMatrix Metalloproteinase 9biology.proteinMatrix Metalloproteinase 2Basal laminaCollagenLamininNeoplasm Recurrence LocalGlioblastomaInvasionmetastasis
researchProduct

Cells expressing markers of immature neurons in the amygdala of adult humans

2012

The polysialylated form of the neuronal cell adhesion molecule (PSA-NCAM) is expressed by immature neurons in the amygdala of adult mammals, including non-human primates. In a recent report we have also described the presence of PSA-NCAM-expressing cells in the amygdala of adult humans. Although many of these cells have been classified as mature interneurons, some of them lacked mature neuronal markers, suggesting the presence of immature neurons. We have studied, using immunohistochemistry, the existence and distribution of these immature neurons using post mortem material. We have also analysed the presence of proliferating cells and the association between immature neurons and specialise…

AdultDoublecortin Domain ProteinsMaleNeural Cell Adhesion Molecule L1AmygdalaWhite matterNeural Stem CellsAntigenParenchymamedicineAnimalsHumansSaimiriAgedNeuronsCATSbiologyGeneral NeuroscienceNeuropeptidesNeurogenesisMiddle AgedAmygdalaDoublecortinAdult Stem CellsKi-67 Antigenmedicine.anatomical_structurenervous systemAstrocytesCatsSialic Acidsbiology.proteinFemaleMicrotubule-Associated ProteinsNeuroscienceNeuronal Cell Adhesion MoleculeBiomarkersEuropean Journal of Neuroscience
researchProduct

Calf Blood Compound (CFC) and Homeopathic Drug Induce Differentiation of Primary Human Skeletal Muscle Cells.

2019

AbstractThe use of injections to treat structural muscle injuries is controversially discussed. In our controlled in vitro study, we investigated the biological impact of Actovegin and Traumeel alone and in combination on primary human skeletal muscle cells. Cells were characterized by immunofluorescence staining for myogenic factor 5 (Myf5) and MyoD, and cultured with or without Actovegin and / or Traumeel. The effects of these agents were assayed by cell viability and gene expression of the specific markers MyoD, Myf5, neural adhesion molecule (NCAM), and CD31. Myotube formation was determined by myosin staining. Neither Actovegin nor Traumeel showed toxic effects or influenced cell viabi…

AdultMaleCell SurvivalMuscle Fibers SkeletalDown-RegulationPhysical Therapy Sports Therapy and RehabilitationHeme030204 cardiovascular system & hematologyPharmacologyMyoD03 medical and health sciences0302 clinical medicineIn vivoGene expressionMyosinmedicineHumansOrthopedics and Sports MedicineViability assayCells CulturedAgedMyoD ProteinMineralsDose-Response Relationship DrugChemistryPlant ExtractsSkeletal muscleCell Differentiation030229 sport sciencesMiddle Agedmusculoskeletal systemCD56 AntigenPlatelet Endothelial Cell Adhesion Molecule-1medicine.anatomical_structureNeural cell adhesion moleculeMYF5Myogenic Regulatory Factor 5International journal of sports medicine
researchProduct

Neural antigens in oligodendrogliomas and dysembryoplastic neuroepithelial tumors

1997

Oligodendrogliomas and dysembryoplastic neuroepithelial tumors (DNT) are frequently associated with epilepsies and share the presence of oligodendroglia-like cells with small round nuclei and optically empty perinuclear halos. The two entities may be difficult to discriminate in small surgical specimens and the origin and differentiation of the oligodendroglia-like cells has been controversial. To better characterize and distinguish the two entities we examined 25 oligodendrogliomas and 16 DNT immunohistochemically for the presence of the proliferation-associated Ki-67 antigen and the following neural antigens: the alpha 1 subunit of the GABAA receptor (GABAR), N-methyl-D-aspartate receptor…

AdultMalePathologymedicine.medical_specialtyOligodendrogliomaGlutamate decarboxylaseNerve Tissue ProteinsPathology and Forensic MedicineDiagnosis DifferentialCellular and Molecular NeuroscienceAntigenAntigens NeoplasmBiomarkers TumormedicineHumansNerve TissueAgedEpilepsybiologyGlial fibrillary acidic proteinTeratomaMiddle Agedmedicine.diseaseImmunohistochemistryNeoplasms Neuroepithelialnervous systembiology.proteinSynaptophysinImmunohistochemistryFemaleNeural cell adhesion moleculeNeurology (clinical)OligodendrogliomaNeuNActa Neuropathologica
researchProduct

Apoptosis-related Proteins in Skeletal Muscle Fibers of Spinal Muscular Atrophy

1997

There is evidence that apoptosis in spinal muscular atrophies (SMA) is not restricted to motor neurons but also affects muscle fibers. Studying the expression of several apoptosis-associated proteins we found constant expression of bax in muscle fibers, which promoted cell death. The expression of bax correlated with defective innervation of muscle fibers was also indicated by upregulation of N-CAM. While in early-onset SMA atrophic as well as normo- and hypertrophic muscle fibers displayed expression of bax, muscle fibers in late-onset SMA and peripheral neuropathies showed bax-expression only in atrophic fibers. Other investigated apoptosis-associated factors comprised interleukin-1 beta …

AdultMaleProgrammed cell deathPathologymedicine.medical_specialtyMuscle Fibers Skeletalbcl-X ProteinMuscle ProteinsApoptosisBiologyMicrofilamentPathology and Forensic MedicineMuscular Atrophy SpinalCellular and Molecular NeuroscienceReference ValuesProto-Oncogene ProteinsmedicineHumansMyocyteMuscle SkeletalActinAgedbcl-2-Associated X ProteinCaspase 1InfantPeripheral Nervous System DiseasesGeneral MedicineSpinal muscular atrophyMiddle AgedSMA*Spinal muscular atrophiesmedicine.diseaseCell biologyCysteine EndopeptidasesProto-Oncogene Proteins c-bcl-2NeurologyFemaleNeural cell adhesion moleculeNeurology (clinical)Journal of Neuropathology and Experimental Neurology
researchProduct