Search results for "Neurofibromatosis."

showing 10 items of 42 documents

Desmoplastic melanoma may mimic a cutaneous peripheral nerve sheath tumor: Report of 3 challenging cases

2017

Desmoplastic melanoma (DM) and cutaneous malignant peripheral nerve sheath tumors (MPNST) reveal histological and immunohistochemical similarities, including S100 positivity and negative staining for conventional melanocytic markers. We present 3 cases of cutaneous S100-positive spindle cell tumors in elderly patients, in which first findings led to initial misdiagnoses as cutaneous MPNST and benign peripheral sheath nerve tumor (neurofibroma). The identification of adjacent atypical melanocytic hyperplasia in the overlying skin along with tumor cell proliferation, also in the superficial dermis, the neurotropic component and the absence of any relationship between the tumor and a major ner…

Desmoplastic melanomaPathologymedicine.medical_specialtyHistologyintegumentary systembusiness.industryMelanomaDermatologymedicine.diseasePathology and Forensic MedicineBenign tumor030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicinemedicine.anatomical_structureDermis030220 oncology & carcinogenesismedicineNeurofibromaImmunohistochemistryNeurofibromatosisbusinessPeripheral Nerve SheathJournal of Cutaneous Pathology
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Management of large dorsal diffuse plexiform neurofibroma

2014

Abstract Neurofibromas are considered one of the hallmark signs of neurofibromatosis type 1(NF1). Diffuse plexiform neurofibroma is a congenital NF1-associated tumor, characterized by overgrowth and interference with function of the affected area. The rich vascular plexus associated with neurofibromas, together with their infiltrative pattern, makes them difficult to eradicate. Complications of neurofibromas are rare but include malignant transformation and potentially lifethreatening hemorrhages. The use of the term “giant” to define a neurofibroma is controversial because there is no clear consensus and descriptions are limited to few case reports. We report a case of a large dorsal diffu…

DorsumPathologymedicine.medical_specialtyNeurofibromaPerioperative managementbusiness.industryArterial EmbolizationSettore MED/19 - Chirurgia Plasticamedicine.diseasePlexiform neurofibromaVascular plexusmedicineNeurofibromaSurgeryNeurofibromatosisbusiness
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Small bowel gastrointestinal stromal tumor presenting with gastrointestinal bleeding in patient with type 1 Neurofibromatosis: Management and laparos…

2021

Highlights • A multidisciplinary team is mandatory for the correct management of hemorrhagic GIST and its complications. • There is a well-known association between type 1 Neurofibromatosis and GIST. • Type 1 Neurofibromatosis-GIST and sporadic GIST have different behaviour. • In case of localised and resectable GIST surgical treatment is the mainstay. • Laparoscopic approach, if performed correctly, is safe and effective with better short-term outcomes then open surgery.

Laparoscopic surgerymedicine.medical_specialtyGastrointestinal bleedingLower gastrointestinal bleedingProximal jejunal GISTmedicine.medical_treatmentLaparoscopic surgery03 medical and health sciences0302 clinical medicineMelenaCase reportmedicineMedical historyStromal tumorneoplasmsType 1 neurofibromatosisGiSTmedicine.diagnostic_testbusiness.industryEsophagogastroduodenoscopyGeneral surgerySmall bowel GISTmedicine.diseasedigestive system diseasesGI bleeding030220 oncology & carcinogenesis030211 gastroenterology & hepatologySurgerymedicine.symptombusinessInternational Journal of Surgery Case Reports
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Mutation spectrum of NF1 gene in Italian patients with neurofibromatosis type 1 using Ion Torrent PGM™ platform

2017

Neurofibromatosis type 1 (NF1) is caused by mutations of the NF1 gene and is one of the most common human autosomal dominant disorders. The patient shows different signs on the skin and other organs from early childhood. The best known are six or more café au lait spots, axillary or inguinal freckling, increased risk of developing benign nerve sheath tumours and plexiform neurofibromas. Mutation detection is complex, due to the large gene size, the large variety of mutations and the presence of pseudogenes. Using Ion Torrent PGM™ Platform, 73 mutations were identified in 79 NF1 Italian patients, 51% of which turned out to be novel mutations. Pathogenic status of each variant was classifi…

Male0301 basic medicineDNA Mutational Analysismedicine.disease_causeChildGenetics (clinical)Sanger sequencingGeneticsMutationNeurofibromin 1biologyMosaicismCafe-au-Lait SpotsNeurofibromatosis type 1; Legius's syndrome; Next generation sequencingGeneral MedicineMiddle AgedItalyChild PreschoolsymbolsMedical geneticsFemalemedicine.symptomHumanAdultmedicine.medical_specialtyNeurofibromatosis 1AdolescentPseudogeneDNA Mutational Analysi03 medical and health sciencessymbols.namesakeGeneticNext generation sequencingCafé au lait spotSkin AbnormalitieGeneticsmedicineHumansCafe-au-Lait SpotNeurofibromatosisLegius's syndromeInfantSequence Analysis DNAIon semiconductor sequencingmedicine.diseaseNeurofibromin 1030104 developmental biologyMutationSkin Abnormalitiesbiology.proteinNeurofibromatosis type 1European Journal of Medical Genetics
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SPRED1, a RAS MAPK pathway inhibitor that causes Legius syndrome, is a tumour suppressor downregulated in paediatric acute myeloblastic leukaemia

2013

Constitutional dominant loss-of-function mutations in the SPRED1 gene cause a rare phenotype referred as neurofibromatosis type 1 (NF1)-like syndrome or Legius syndrome, consisted of multiple café-au-lait macules, axillary freckling, learning disabilities and macrocephaly. SPRED1 is a negative regulator of the RAS MAPK pathway and can interact with neurofibromin, the NF1 gene product. Individuals with NF1 have a higher risk of haematological malignancies. SPRED1 is highly expressed in haematopoietic cells and negatively regulates haematopoiesis. SPRED1 seemed to be a good candidate for leukaemia predisposition or transformation. We performed SPRED1 mutation screening and expression status i…

MaleCancer ResearchAdolescentLoss of HeterozygosityFrameshift mutationGene productLoss of heterozygosityPrecursor B-Cell Lymphoblastic Leukemia-Lymphomahemic and lymphatic diseasesGeneticsmedicineHumansGenes Tumor SuppressorNeurofibromatosisChildMolecular BiologyAdaptor Proteins Signal TransducingLegius syndromeNeurofibromin 1biologyCafe-au-Lait SpotsInfant NewbornIntracellular Signaling Peptides and ProteinsMacrocephalyInfantMembrane Proteinsmedicine.diseaseNeurofibromin 1Gene Expression Regulation NeoplasticLeukemia Myeloid AcuteHaematopoiesisGenes rasChild PreschoolMutationCancer researchbiology.proteinFemalemedicine.symptomOncogene
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Solitary intraosseous neurofibroma of the mandible. Apropos of a case

2010

Neurofibroma is a benign neoplasm derived from peripheral nerves. Most of these are associated with Neurofibromatosis but may also occur as solitary lesions. When found on the head and neck they are generally located in the soft tissue. Intraosseous location is very rare. The following report describes a case of an intraosseous neurofibroma located in the left mandibular ramus of a 14-year-old child. The patient did not had clinical evidence of the lesion and it was found on a routine radiographic examination. Surgical excision of the lesion was scheduled and the sample was submitted to histopathological study. Representative sample cuts were studied using conventional techniques of hematox…

MalePathologymedicine.medical_specialtyNeurofibromaAdolescentbusiness.industryRadiographyMandibleSoft tissue:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseLesionMandibular NeoplasmsImmunophenotypingOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASmedicineHumansNeurofibromaSurgerymedicine.symptomDifferential diagnosisNeurofibromatosisbusinessGeneral Dentistry
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Childhood microphthalmic neurofibromatosis.

1989

We present an atypical case of neurofibromatosis marked by young age at onset, rapid invasiveness of the tumor, the presence of curious autonomic symptoms, cranial malformation and congenital microphthalmus, a combination that suggests a new syndrome, which might be called childhood microphthalmic neurofibromatosis.

Malemedicine.medical_specialtyPediatricsNeurologyNeurofibromatosis 1AdolescentGeneral NeuroscienceDermatologyGeneral MedicineSyndromemedicine.diseaseMagnetic Resonance ImagingDevelopmental psychologyPsychiatry and Mental healthYoung agemedicineAutonomic symptomsHumansMicrophthalmosNeurology (clinical)NeurosurgeryNeurofibromatosisPsychologyNeuroradiologyItalian journal of neurological sciences
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Neurofibromatosis of the nipple-areolar area: a case series

2010

Abstract Introduction Neurofibromatosis type 1 is an autosomal dominant disorder that occurs across all ethnic groups and affects approximately one in 4000 individuals. One of the most noticeable characteristics of the disease is the development of neurofibromas. Case presentation A total of 258 patients (131 women, 127 men) with neurofibromatosis type 1 were evaluated between 1994 and 2004 in our hospital's dermatology department. Nine patients (3.45%, 95% confidence limits 1.22 to 5.68) had neurofibromas of the breast. One of these nine patients presented with an extensive congenital plexiform neurofibroma in the outer quadrants of her right breast, extending to the nipple-areolar complex…

Medicine(all)medicine.medical_specialtyPathologybusiness.industryCase reportlcsh:Rmedicinelcsh:MedicineGeneral MedicineNeurofibromatosismedicine.diseasebusinessDermatologyJournal of Medical Case Reports
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Neuroendocrine neoplasms in the context of inherited tumor syndromes: a reappraisal focused on targeted therapies

2022

Purpose Neuroendocrine neoplasms can occur as part of inherited disorders, usually in the form of well-differentiated, slow-growing tumors (NET). The main predisposing syndromes include: multiple endocrine neoplasias type 1 (MEN1), associated with a large spectrum of gastroenteropancreatic and thoracic NETs, and type 4 (MEN4), associated with a wide tumour spectrum similar to that of MEN1; von Hippel-Lindau syndrome (VHL), tuberous sclerosis (TSC), and neurofibromatosis 1 (NF-1), associated with pancreatic NETs. In the present review, we propose a reappraisal of the genetic basis and clinical features of gastroenteropancreatic and thoracic NETs in the setting of inherited syndromes with a s…

Neuroendocrine neoplasmMEN4EndocrinologyMEN1Endocrinology Diabetes and MetabolismVon Hippel–Lindau (VHL) syndromeTargeted therapieVon Hippel–Lindau (VHL) syndrome.Neurofibromatosis 1 (NF-1)Tuberous sclerosis (TSC)
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Current standing and frontiers of gene therapy for meningiomas

2013

Meningiomas are among the most common intracranial tumors. The treatment of choice for these lesions is complete resection, but in 50% of cases it is not achieved due to tumor location and/or surgical morbidities. Moreover, benign meningiomas have high recurrence rates of up to 32% in long-term follow-up. Molecular analyses have begun to uncover the genetics behind meningiomas, giving rise to potential genetics-based treatments, including gene therapy. The authors performed a literature review on the most relevant genes associated with meningiomas and both current and potential gene therapy strategies to treat these tumors. Wild-type NF2 gene insertion, oncolytic viruses, and transfer of si…

Neurofibromatosis 2medicine.medical_specialtyAngiogenesisGenetic enhancementGenetic TherapyGeneral MedicineBiologymedicine.diseaseOncolytic virusMeningiomaClinical trialBenign MeningiomaMeningeal Neoplasmsotorhinolaryngologic diseasesmedicineCancer researchHumansSurgeryNeurology (clinical)NeurosurgeryMeningiomaGeneSignal TransductionNeurosurgical Focus
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