Search results for "Neurologi"

showing 10 items of 1189 documents

Axonal control of the adult neural stem cell niche.

2014

SummaryThe ventricular-subventricular zone (V-SVZ) is an extensive germinal niche containing neural stem cells (NSCs) in the walls of the lateral ventricles of the adult brain. How the adult brain’s neural activity influences the behavior of adult NSCs remains largely unknown. We show that serotonergic (5HT) axons originating from a small group of neurons in the raphe form an extensive plexus on most of the ventricular walls. Electron microscopy revealed intimate contacts between 5HT axons and NSCs (B1) or ependymal cells (E1) and these cells were labeled by a transsynaptic viral tracer injected into the raphe. B1 cells express the 5HT receptors 2C and 5A. Electrophysiology showed that acti…

Cellular differentiationMessengerRegenerative MedicineMedical and Health SciencesImmunoenzyme TechniquesLateral ventriclesMice0302 clinical medicineNeural Stem CellsReceptor Serotonin 5-HT2C5-HT2CStem Cell NicheNeurons0303 health sciencesMicroscopyBlottingReverse Transcriptase Polymerase Chain ReactionNeurogenesisBrainCell DifferentiationAnatomyBiological SciencesNeural stem cellCell biologySerotonin Receptor AgonistsElectrophysiologyNeurologicalMolecular MedicineStem Cell Research - Nonembryonic - Non-HumanWesternReceptorSerotoninEpendymal CellNeurogenesis1.1 Normal biological development and functioningBlotting WesternBiologySerotonergicReal-Time Polymerase Chain ReactionElectronArticle03 medical and health sciencesUnderpinning researchGeneticsAnimalsRNA Messenger030304 developmental biologyCell ProliferationRapheNeurosciencesCell BiologyStem Cell ResearchAxonsMicroscopy Electronnervous systemRaphe NucleiRNARaphe nuclei030217 neurology & neurosurgeryDevelopmental Biology
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Can Alzheimer disease be a form of type 3 diabetes?

2012

Alzheimer disease (AD) and metabolic syndrome are two highly prevalent pathological conditions of Western society due to incorrect diet, lifestyle, and vascular risk factors. Recent data have suggested metabolic syndrome as an independent risk factor for AD and pre-AD syndrome. Furthermore, biological plausibility for this relationship has been framed within the “metabolic cognitive syndrome” concept. Due to the increasing aging of populations, prevalence of AD in Western industrialized countries will rise in the near future. Thus, new knowledge in the area of molecular biology and epigenetics will probably help to make an early molecular diagnosis of dementia. An association between metabo…

Central Nervous SystemAgingmedicine.medical_specialtySingle-nucleotide polymorphismType 2 diabetesBiologyBioinformaticsPolymorphism Single NucleotideSHIP2 ADAlzheimer DiseaseRisk FactorsDiabetes mellitusInternal medicinemedicineDiabetes MellitusDementiaHumansInsulinEpigeneticsRisk factorLife StyleAgedSettore MED/04 - Patologia GeneraleMetabolic SyndromeInositol Polyphosphate 5-PhosphatasesSyndromeModels Theoreticalmedicine.diseasePhosphoric Monoester HydrolasesEndocrinologySettore MED/26 - NeurologiaGeriatrics and GerontologyAlzheimer's diseaseMetabolic syndromeCognition DisordersSignal TransductionRejuvenation research
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Gray Matter NG2 Cells Display Multiple Ca2+-Signaling Pathways and Highly Motile Processes

2011

NG2 cells, the fourth type of glia in the mammalian CNS, receive synaptic input from neurons. The function of this innervation is unknown yet. Postsynaptic changes in intracellular Ca(2+)-concentration ([Ca(2+)](i)) might be a possible consequence. We employed transgenic mice with fluorescently labeled NG2 cells to address this issue. To identify Ca(2+)-signaling pathways we combined patch-clamp recordings, Ca(2+)-imaging, mRNA-transcript analysis and focal pressure-application of various substances to identified NG2-cells in acute hippocampal slices. We show that activation of voltage-gated Ca(2+)-channels, Ca(2+)-permeable AMPA-receptors, and group I metabotropic glutamate-receptors provo…

Central Nervous SystemAnatomy and PhysiologyVesicular glutamate transporter 1Glycobiologylcsh:MedicineHippocampal formationBiochemistryIon ChannelsTransmembrane Transport ProteinsMice0302 clinical medicinePostsynaptic potentialBiomacromolecule-Ligand Interactionslcsh:ScienceCells CulturedMembrane potential0303 health sciencesMultidisciplinarybiologyReverse Transcriptase Polymerase Chain ReactionDepolarizationNeurochemistryNeurotransmittersCell biologyElectrophysiologymedicine.anatomical_structureNeurologyNeurogliaMedicineProteoglycansNeurochemicalsGlutamateNeurogliaResearch ArticleNervous System PhysiologySignal TransductionCell PhysiologyMotilityNeuroimagingMice TransgenicNeurological System03 medical and health sciencesNeuropharmacologymedicineAnimalsHumansddc:610Biology030304 developmental biologyEndoplasmic reticulumlcsh:RProteinsGamma-Aminobutyric AcidTransmembrane ProteinsLuminescent ProteinsMicroscopy Electronnervous systemMicroscopy FluorescenceSynapsesVesicular Glutamate Transport Protein 1biology.proteinNervous System Componentslcsh:QCalciumPhysiological Processes030217 neurology & neurosurgeryNeurosciencePLoS ONE
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Immune regulatory neural stem/precursor cells protect from central nervous system autoimmunity by restraining dendritic cell function.

2009

Background: The systemic injection of neural stem/precursor cells (NPCs) provides remarkable amelioration of the clinicopathological features of experimental autoimmune encephalomyelitis (EAE). This is dependent on the capacity of transplanted NPCs to engage concurrent mechanisms of action within specific microenvironments in vivo. Among a wide range of therapeutic actions alternative to cell replacement, neuroprotective and immune modulatory capacities of transplanted NPCs have been described. However, lacking is a detailed understanding of the mechanisms by which NPCs exert their therapeutic plasticity. This study was designed to identify the first candidate that exemplifies and sustains …

Central Nervous SystemEncephalomyelitis Autoimmune ExperimentalCell Transplantationmedicine.medical_treatmentScienceAutoimmunityNeurological Disorders/Multiple Sclerosis and Related DisordersBiologyMiceImmune systemPrecursor cellmedicineotorhinolaryngologic diseasesAnimalsLymph nodeInflammationNeuronsMultidisciplinaryStem CellsExperimental autoimmune encephalomyelitisMesenchymal stem cellQRStem-cell therapyDendritic cellDendritic Cellsmedicine.diseaseCell biologyDevelopmental Biology/Stem CellsMicroscopy Electronstomatognathic diseasesmedicine.anatomical_structureImmune SystemImmunologyBone Morphogenetic ProteinsMedicineFemaleLymph NodesStem cellNeuroscience/Neurobiology of Disease and RegenerationResearch ArticlePLoS ONE
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Elevated cerebrospinal fluid and plasma homocysteine levels in ALS

2009

BACKGROUND: High cerebrospinal fluid (CSF) and plasma levels of homocysteine (HC) have been reported in certain neurodegenerative disorders, such as Alzheimer's, Parkinson's diseases and, recently, amyotrophic lateral sclerosis (ALS). OBJECTIVES: To assay the CSF and plasma levels of HC in ALS patients and controls, and to evaluate the relationship between HC levels and clinical variables of the disease. METHODS: Cerebrospinal fluid from sixty-nine (M/F 1.87) and plasma from sixty-five ALS patients (M/F 1.83) were taken and stored at -80 degrees C until use. Controls (CSF = 55; plasma = 67) were patients admitted to our hospital for neurological disorders with no known relationship to HC ch…

Central Nervous SystemMaleAmyotrophic Lateral SclerosisHyperhomocysteinemiaComorbidityMiddle Agedcerebrospinal fluidPredictive Value of TestsHumansamyotrophic lateral sclerosiSettore MED/26 - NeurologiaFemaleHomocysteineplasmaBiomarkersAged
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Polysialic acid is required for dopamine D2 receptor-mediated plasticity involving inhibitory circuits of the rat medial prefrontal cortex.

2011

Decreased expression of dopamine D2 receptors (D2R), dysfunction of inhibitory neurotransmission and impairments in the structure and connectivity of neurons in the medial prefrontal cortex (mPFC) are involved in the pathogenesis of schizophrenia and major depression, but the relationship between these changes remains unclear. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a plasticity-related molecule, may serve as a link. This molecule is expressed in cortical interneurons and dopamine, via D2R, modulates its expression in parallel to that of proteins related to synapses and inhibitory neurotransmission, suggesting that D2R-targeted antipsychotics/antidepressants…

Central Nervous SystemMaleAnatomy and Physiologylcsh:MedicineRats Sprague-DawleyNeural PathwaysMolecular Cell BiologyNeurobiology of Disease and Regenerationlcsh:SciencePsychiatryMicroscopy ConfocalNeuronal PlasticityMultidisciplinaryNeuronal MorphologybiologyGlutamate Decarboxylasemusculoskeletal neural and ocular physiologyNeurotransmittersAnatomyImmunohistochemistryMental Healthmedicine.anatomical_structureNeurologyDopamine AgonistsMedicineNcamResearch Articlemedicine.drugNeural NetworksInterneuronSynaptophysinNeurophysiologyPrefrontal CortexNeuropsychiatric DisordersNeural Cell Adhesion Molecule L1NeurotransmissionNeurological SystemNeuropharmacologyDopamineDopamine receptor D2NeuroplasticityCell AdhesionNeuropilmedicineAnimalsBiologyMood DisordersReceptors Dopamine D2lcsh:RRatsNeuroanatomynervous systemCellular NeuroscienceSynapsesSchizophreniaSialic Acidsbiology.proteinNeural cell adhesion moleculelcsh:QNeuroscienceParvalbuminNeurosciencePLoS ONE
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Risk factors for central nervous system tumors in children: New findings from a case-control study

2017

BACKGROUND: Central nervous system tumors (CNS) are the most frequent solid tumor in children. Causes of CNS tumors are mainly unknown and only 5% of the cases can be explained by genetic predisposition. We studied the effects of environmental exposure on the incidence of CNS tumors in children by subtype, according to exposure to industrial and/or urban environment, exposure to crops and according to socio-economic status of the child. METHODS: We carried out a population-based case-control study of CNS tumors in Spain, covering 714 incident cases collected from the Spanish Registry of Childhood Tumors (period 1996-2011) and 4284 controls, individually matched by year of birth, sex, and au…

Central Nervous SystemMaleUrban PopulationSocial Scienceslcsh:MedicineNervous SystemUrban EnvironmentsCentral Nervous System NeoplasmsFamilies0302 clinical medicineSociologyRisk FactorsEpidemiology of cancerMedicine and Health SciencesMedicine030212 general & internal medicinelcsh:ScienceChildNeurological TumorsChildrenGeographic AreasAir PollutantsMultidisciplinaryGeographyAgricultureGliomaPlantsTerrestrial EnvironmentsOncologyNeurology030220 oncology & carcinogenesisChild PreschoolFemaleAnatomyInfantsResearch ArticleUrban Areasmedicine.medical_specialtyAdolescentCropsAstrocytomaFruits03 medical and health sciencesHumansSocial StratificationPesticidesSistema nerviós centralbusiness.industryEcology and Environmental Scienceslcsh:ROrganismsCase-control studyInfant NewbornBiology and Life SciencesCancers and NeoplasmsCancerInfantEnvironmental Exposuremedicine.diseaseSocial ClassAge GroupsCentral nervous systemFamily medicineCase-Control StudiesPeople and PlacesEarth SciencesPopulation Groupingslcsh:QbusinessCrop Science
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Single administration of tripeptide α-MSH(11-13) attenuates brain damage by reduced inflammation and apoptosis after experimental traumatic brain inj…

2013

Following traumatic brain injury (TBI) neuroinflammatory processes promote neuronal cell loss. Alpha-melanocyte-stimulating hormone (α-MSH) is a neuropeptide with immunomodulatory properties, which may offer neuroprotection. Due to short half-life and pigmentary side-effects of α-MSH, the C-terminal tripeptide α-MSH(11-13) may be an anti-inflammatory alternative. The present study investigated the mRNA concentrations of the precursor hormone proopiomelanocortin (POMC) and of melanocortin receptors 1 and 4 (MC1R/MC4R) in naive mice and 15 min, 6, 12, 24, and 48 h after controlled cortical impact (CCI). Regulation of POMC and MC4R expression did not change after trauma, while MC1R levels incr…

Central Nervous SystemMaleendocrine systemAnatomy and PhysiologyPro-OpiomelanocortinMouseScienceAnti-Inflammatory AgentsGene ExpressionApoptosisNeurological SystemImmunomodulationMiceModel OrganismsNeurorehabilitation and TraumaAnimalsMelanocyte-Stimulating HormonesBiologyCalcium-Binding ProteinsMicrofilament ProteinsQRBrainAnimal ModelsPeptide FragmentsMice Inbred C57BLHead InjuryNeurologyImmune SystemBrain InjuriesNervous System ComponentsCytokinesReceptor Melanocortin Type 4MedicineClinical ImmunologyMicrogliaInflammation MediatorsReceptor Melanocortin Type 1hormones hormone substitutes and hormone antagonistsResearch ArticleNervous System PhysiologyPLoS ONE
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Morphological Characterization of the Entire Interneuron Population Reveals Principles of Neuromere Organization in the Ventral Nerve Cord ofDrosophi…

2011

Decisive contributions to our understanding of the mechanisms underlying the development of the nervous system have been made by studies performed at the level of single, identified cells in the fruit flyDrosophila. While all the motor neurons and glial cells in thoracic and abdominal segments of theDrosophilaembryo have been individually identified, few of the interneurons, which comprise the vast majority of cells in the CNS, have been characterized at this level. We have applied a single cell labeling technique to carry out a detailed morphological characterization of the entire population of interneurons in abdominal segments A1–A7. Based on the definition of a set of spatial parameters…

Central Nervous SystemNervous systemCell typeInterneuronCD8 AntigensGreen Fluorescent ProteinsLIM-Homeodomain ProteinsModels NeurologicalStatistics as TopicPopulationCell CountBiologyFunctional LateralityAnimals Genetically ModifiedInterneuronsNeural PathwaysmedicineAnimalsDrosophila ProteinsAmino Acidseducationeducation.field_of_studyGeneral NeurosciencefungiArticlesNeuromereAxonsmedicine.anatomical_structureVentral nerve cordDrosophilaAxon guidanceNeuroscienceDrosophila ProteinTranscription FactorsThe Journal of Neuroscience
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IL-17 and related cytokines involved in the pathology and immunotherapy of multiple sclerosis: Current and future developments.

2014

Multiple sclerosis (MS), an autoimmune neurological disorder, is driven by self-reactive T helper (Th) cells. Research on the role of Th17 lymphocytes in MS pathogenesis has made significant progress in identifying various immunological as well as environmental factors that induce the differentiation and expansion of these cells, different subsets of Th17 cells with varying degrees of pathogenicity, and the role of the secreted effector cytokines. While approved therapies for MS offer significant benefit to patients, there remain unmet needs. Ongoing clinical trials aim to translate the advanced knowledge of Th17 cytokines to improved therapies. This review discusses the current status and …

Central Nervous SystemPathologymedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisEndocrinology Diabetes and Metabolismmedicine.medical_treatmentImmunologyAutoimmunityNeurological disorderGeneral Biochemistry Genetics and Molecular BiologyUnmet needsPathogenesisMicemedicineImmunology and AllergyAnimalsHumansEffectorbusiness.industryMultiple sclerosisInterleukin-17Cell DifferentiationImmunotherapyInterferon-betamedicine.diseaseClinical trialImmunologyTh17 CellsInterleukin 17ImmunotherapyInflammation MediatorsbusinessCytokinegrowth factor reviews
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