Search results for "Neuron"

showing 10 items of 2611 documents

Acid sphingomyelinase – a regulator of canonical transient receptor potential channel 6 (TRPC6) activity

2019

Recent investigations propose the acid sphingomyelinase (ASM)/ceramide system as a novel target for antidepressant action. ASM catalyzes the breakdown of the abundant membrane lipid sphingomyelin to the lipid messenger ceramide. This ASM‐induced lipid modification induces a local shift in membrane properties, which influences receptor clustering and downstream signaling. Canonical transient receptor potential channels 6 (TRPC6) are non‐selective cation channels located in the cell membrane that play an important role in dendritic growth, synaptic plasticity and cognition in the brain. They can be activated by hyperforin, an ingredient of the herbal remedy St. John’s wort for treatment of de…

0301 basic medicineCeramideMedizinCeramidesPC12 CellsBiochemistryFIASMATRPC603 medical and health sciencesCellular and Molecular NeuroscienceTransient receptor potential channelchemistry.chemical_compound0302 clinical medicineddc:570medicineAnimalsInstitut für Biochemie und BiologieIon channelTRPC Cation ChannelsNeuronsRatsCell biologySphingomyelin Phosphodiesterase030104 developmental biologychemistryLipid modificationAcid sphingomyelinaseSphingomyelin030217 neurology & neurosurgerymedicine.drugJournal of Neurochemistry
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Brain Distribution and Modulation of Neuronal Excitability by Indicaxanthin From Opuntia Ficus Indica Administered at Nutritionally-Relevant Amounts

2018

Several studies have recently investigated the role of nutraceuticals in complex pathophysiological processes such as oxidative damages, inflammatory conditions and excitotoxicity. In this regard, the effects of nutraceuticals on basic functions of neuronal cells, such as excitability, are still poorly investigated. For this reason, the possible modulation of neuronal excitability by phytochemicals (PhC) could represent an interesting field of research given that excitotoxicity phenomena are involved in neurodegenerative alterations leading, for example, to Alzheimer's disease. The present study was focused on indicaxanthin from Opuntia ficus indica, a bioactive betalain pigment, with a pro…

0301 basic medicineCerebellumAgingCognitive NeuroscienceExcitotoxicityHippocampusindicaxanthinBiologyHippocampal formationmedicine.disease_causeNeuroprotectionmicroiontophoresisbrain localizationlcsh:RC321-57103 medical and health scienceschemistry.chemical_compound0302 clinical medicineexcitabilitymedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchnutraceuticalselectrophysiologyCortex (botany)brain localization; electrophysiology; excitability; indicaxanthin; microiontophoresis; neuroprotection; nutraceuticals030104 developmental biologymedicine.anatomical_structurechemistrynervous systemmicroiontophoresineuroprotectionNeuronIndicaxanthinNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Aging Neuroscience
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26th Annual Computational Neuroscience Meeting (CNS*2017): Part 2

2017

International audience; No abstract available

0301 basic medicineCerebellumComputer science[SDV]Life Sciences [q-bio]General Neurosciencelcsh:QP351-495Meeting Abstractslcsh:RC321-57103 medical and health sciencesCellular and Molecular Neurosciencelcsh:Neurophysiology and neuropsychology030104 developmental biologymedicine.anatomical_structuremedicineNeuronlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNeuroscienceComputingMilieux_MISCELLANEOUScomputational neuroscienceBMC Neuroscience
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Opposing Effects of CREBBP Mutations Govern the Phenotype of Rubinstein-Taybi Syndrome and Adult SHH Medulloblastoma

2018

Recurrent mutations in chromatin modifiers are specifically prevalent in adolescent or adult patients with Sonic hedgehog-associated medulloblastoma (SHH MB). Here, we report that mutations in the acetyltransferase CREBBP have opposing effects during the development of the cerebellum, the primary site of origin of SHH MB. Our data reveal that loss of Crebbp in cerebellar granule neuron progenitors (GNPs) during embryonic development of mice compromises GNP development, in part by downregulation of brain-derived neurotrophic factor (Bdnf). Interestingly, concomitant cerebellar hypoplasia was also observed in patients with Rubinstein-Taybi syndrome, a congenital disorder caused by germline mu…

0301 basic medicineCerebellumCrebbp protein mousemetabolism [Cerebellar Neoplasms]acetyltransferase; cerebellum; CREBBP; development; Rubinstein-Taybi syndrome; SHH medulloblastomagenetics [Hedgehog Proteins]MiceNeurotrophic factorsmetabolism [CREB-Binding Protein]Mice KnockoutNeuronsRubinstein-Taybi Syndromepathology [Rubinstein-Taybi Syndrome]CREBBPCREB-Binding ProteinPhenotypegenetics [CREB-Binding Protein]3. Good healthpathology [Cerebellar Neoplasms]acetyltransferasePhenotypemedicine.anatomical_structuregenetics [Rubinstein-Taybi Syndrome]Femalemetabolism [Hedgehog Proteins]Signal TransductionSHH medulloblastomaAdultcerebellumBiologyGeneral Biochemistry Genetics and Molecular BiologyCREBBP; Rubinstein-Taybi syndrome; SHH medulloblastoma; acetyltransferase; cerebellum; development.03 medical and health sciencesGermline mutationAcetyltransferasesmetabolism [Medulloblastoma]medicineAnimalsHumansgenetics [Cerebellar Neoplasms]Hedgehog Proteinsddc:610Cerebellar NeoplasmsdevelopmentMolecular BiologyMedulloblastomaRubinstein–Taybi syndromegenetics [Medulloblastoma]metabolism [Rubinstein-Taybi Syndrome]pathology [Medulloblastoma]Cell Biologymedicine.disease030104 developmental biologyMutationphysiology [CREB-Binding Protein]Cancer researchSHH protein humanCerebellar hypoplasia (non-human)metabolism [Acetyltransferases]CREBBP protein humanMedulloblastomaDevelopmental BiologyCongenital disorderDevelopmental Cell
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Enhanced autophagic-lysosomal activity and increased BAG3-mediated selective macroautophagy as adaptive response of neuronal cells to chronic oxidati…

2019

Oxidative stress and a disturbed cellular protein homeostasis (proteostasis) belong to the most important hallmarks of aging and of neurodegenerative disorders. The proteasomal and autophagic-lysosomal degradation pathways are key measures to maintain proteostasis. Here, we report that hippocampal cells selected for full adaptation and resistance to oxidative stress induced by hydrogen peroxide (oxidative stress-resistant cells, OxSR cells) showed a massive increase in the expression of components of the cellular autophagic-lysosomal network and a significantly higher overall autophagic activity. A comparative expression analysis revealed that distinct key regulators of autophagy are upregu…

0301 basic medicineClinical BiochemistryLFQ Label-free quantificationLETM Leucine zipper and EF-hand containing transmembrane proteinmedicine.disease_causeBiochemistryCHX Cycloheximide0302 clinical medicineBNIP3 Bcl-2 interacting protein 3RAPA RapamycinPIK3C3 Class III PI3‐kinasePhosphorylationlcsh:QH301-705.5Neuronslcsh:R5-920PolyUB PolyubiquitinChemistryBAG3OPA1 Optic atrophy 1TOR Serine-Threonine KinasesWIPI1 WD repeat domain phosphoinositide-interacting protein 1ATG Autophagy relatedTFEB Transcription factor EBCell biologyMitochondriasiRNA Small interfering RNADLP1 Dynamin-like protein 1LAMP1 Lysosomal‐associated membrane protein 1PURO Puromycinlcsh:Medicine (General)Protein homeostasisResearch PaperBafA1 Bafilomycin A1LAMP2 Lysosomal‐associated membrane protein 2Proteasome Endopeptidase ComplexRAB18 Member RAS oncogeneTUB TubulinLC3 Light chain 3 proteinOxidative phosphorylationBAG3CTSD Cathepsin DModels BiologicalCell Line03 medical and health sciencesDownregulation and upregulationMacroautophagymedicineAutophagyHumansAdaptationBAG1 Bcl-2-associated athanogene 1BECN1 Beclin1PI3K/AKT/mTOR pathwayAdaptor Proteins Signal TransducingTEM Transmission electron microscopyHsp70 Heat shock protein 70Organic ChemistryAutophagyAutophagosomesmTOR Mammalian target of rapamycinHsp70Oxidative Stress030104 developmental biologyProteostasislcsh:Biology (General)CV CanavanineBAG3 Bcl-2-associated athanogene 3MTT (3-(45-Dimethylthiazol-2-yl)-25-Diphenyltetrazolium Bromide)Apoptosis Regulatory ProteinsLysosomes030217 neurology & neurosurgeryOxidative stressRedox Biology
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The NG2 Protein Is Not Required for Glutamatergic Neuron-NG2 Cell Synaptic Signaling.

2014

NG2 glial cells (as from now NG2 cells) are unique in receiving synaptic input from neurons. However, the components regulating formation and maintenance of these neuron–glia synapses remain elusive. The transmembrane protein NG2 has been considered a potential mediator of synapse formation and alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor (AMPAR) clustering, because it contains 2 extracellular Laminin G/Neurexin/Sex Hormone-Binding Globulin domains, which in neurons are crucial for formation of transsynaptic neuroligin– neurexin complexes. NG2 is connected via Glutamate Receptor-Interacting Protein with GluA2/3-containing AMPARs, thereby possibly mediating receptor clus…

0301 basic medicineCognitive NeuroscienceNeurexinSynaptogenesisGlutamic AcidNeuroliginMice TransgenicBiologyNeurotransmissionHippocampusSynaptic Transmission03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicinePostsynaptic potentialAnimalsReceptors AMPAAntigensNeuronsMembrane Proteins030104 developmental biologynervous systemSynaptic plasticitySynapsesProteoglycansSynaptic signalingNeurosciencePostsynaptic densityNeuroglia030217 neurology & neurosurgeryCerebral cortex (New York, N.Y. : 1991)
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Synaptic Phospholipid Signaling Modulates Axon Outgrowth via Glutamate-dependent Ca2+-mediated Molecular Pathways.

2015

Abstract Altered synaptic bioactive lipid signaling has been recently shown to augment neuronal excitation in the hippocampus of adult animals by activation of presynaptic LPA2-receptors leading to increased presynaptic glutamate release. Here, we show that this results in higher postsynaptic Ca2+ levels and in premature onset of spontaneous neuronal activity in the developing entorhinal cortex. Interestingly, increased synchronized neuronal activity led to reduced axon growth velocity of entorhinal neurons which project via the perforant path to the hippocampus. This was due to Ca2+-dependent molecular signaling to the axon affecting stabilization of the actin cytoskeleton. The spontaneous…

0301 basic medicineCognitive NeuroscienceNeuronal OutgrowthHippocampusGlutamic AcidAxon hillockSynaptic Transmission03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicinePostsynaptic potentialmedicinePremovement neuronal activityAnimalsbioactive phospholipidsCalcium SignalingAxonearly synchronized activityCells CulturedPhospholipidsChemistryOriginal ArticlesEntorhinal cortexPerforant pathActin cytoskeletonAxonsCell biologyCa2+-signalingentorhinal–hippocampal formation030104 developmental biologymedicine.anatomical_structureaxon outgrowthnervous systemCalcium030217 neurology & neurosurgeryMetabolic Networks and PathwaysCerebral cortex (New York, N.Y. : 1991)
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L’adaptation optimale du mouvement humain au milieu gravitaire

2017

International audience

0301 basic medicineComputer scienceGeneral MedicineGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences030104 developmental biology0302 clinical medicine[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathology[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Humanities030217 neurology & neurosurgeryComputingMilieux_MISCELLANEOUS[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Network-Wide Adaptive Burst Detection Depicts Neuronal Activity with Improved Accuracy

2017

Neuronal networks are often characterized by their spiking and bursting statistics. Previously, we introducedan adaptive burst analysis methodwhich enhances the analysis power for neuronal networks with highly varying firing dynamics. The adaptation is based on single channels analyzing each element of a network separately. Such kind of analysis was adequate for the assessment of local behavior, where the analysis focuses on the neuronal activity in the vicinity of a single electrode. However, the assessment of the whole network may be hampered, if parts of the network are analyzed using different rules. Here, we test how using multiple channels and measurement time points affect adaptive b…

0301 basic medicineComputer scienceNeuroscience (miscellaneous)Interval (mathematics)Machine learningcomputer.software_genreta3112lcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceBursting0302 clinical medicineMoving averageHistogramMethodsCluster analysislcsh:Neurosciences. Biological psychiatry. Neuropsychiatryta113network classificationbusiness.industryEmphasis (telecommunications)Pattern recognition217 Medical engineeringlaskennallinen neurotiede113 Computer and information sciencesPower (physics)030104 developmental biologymicroelectrode arraysburst detectionburst synchronySpike (software development)Artificial intelligenceneuronal networksbusinesscomputer030217 neurology & neurosurgeryNeurosciencecomputational neuroscienceFrontiers in Computational Neuroscience
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Spectral entropy based neuronal network synchronization analysis based on microelectrode array measurements

2016

Synchrony and asynchrony are essential aspects of the functioning of interconnected neuronal cells and networks. New information on neuronal synchronization can be expected to aid in understanding these systems. Synchronization provides insight in the functional connectivity and the spatial distribution of the information processing in the networks. Synchronization is generally studied with time domain analysis of neuronal events, or using direct frequency spectrum analysis, e.g., in specific frequency bands. However, these methods have their pitfalls. Thus, we have previously proposed a method to analyze temporal changes in the complexity of the frequency of signals originating from differ…

0301 basic medicineComputer scienceNeuroscience (miscellaneous)ta3112Radio spectrumSynchronizationlcsh:RC321-571Correlation03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineBiological neural networkMethodsTime domainlcsh:Neurosciences. Biological psychiatry. NeuropsychiatrySimulationEvent (probability theory)rat cortical cellsMEAmicroelectrode array213 Electronic automation and communications engineering electronicsspectral entropyInformation processingCorrectiondeveloping neuronal networksMultielectrode array217 Medical engineering030104 developmental biologycorrelationmouse cortical cellsBiological systemsynchronization030217 neurology & neurosurgeryNeuroscienceFrontiers in Computational Neuroscience
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