Search results for "Neuronal"

showing 10 items of 556 documents

Neural Compensation Within the Human Triceps Surae During Prolonged Walking

2011

During human walking, muscle activation strategies are approximately constant across consecutive steps over a short time, but it is unknown whether they are maintained over a longer duration. Prolonged walking may increase tendinous tissue (TT) compliance, which can influence neural activation, but the neural responses of individual muscles have not been investigated. This study investigated the hypothesis that muscle activity is up- or down-regulated in individual triceps surae muscles during prolonged walking. Thirteen healthy subjects walked on a treadmill for 60 min at 4.5 km/h, while triceps surae muscle activity, maximal muscle compound action potentials, and kinematics were recorded…

AdultMalemedicine.medical_specialtyPhysiologyPhysical ExertionWalkingPhysical medicine and rehabilitationmedicineHumansMuscle SkeletalGaitNeuronal Plasticitybusiness.industryGeneral NeuroscienceMotor controlMuscle activationAdaptation PhysiologicalGaitDuration (music)Physical EnduranceFemalemedicine.symptombusinessAnkle JointMuscle ContractionMuscle contractionJournal of Neurophysiology
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Analysis of thiamine transporter genes in sporadic beriberi

2014

Abstract Objective Thiamine or vitamin B 1 deficiency diminishes thiamine-dependent enzymatic activity, alters mitochondrial function, impairs oxidative metabolism, and causes selective neuronal death. We analyzed for the first time, the role of all known mutations within three specific thiamine carrier genes, SLC19 A2, SLC19 A3 , and SLC25 A19 , in a patient with atrophic beriberi, a multiorgan nutritional disease caused by thiamine deficiency. Methods A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema, a subacute sensorimotor neuropathy, and incontinence. Despite normal vitamin B 1 serum levels, his clinical picture was rapidly reverted by high-dose in…

AdultMalemedicine.medical_specialtySLC19 A- SLC25 A19SLC19 AEndocrinology Diabetes and MetabolismGene mutationBeriberimedicine.disease_causeMitochondrial Membrane Transport Proteinslaw.inventionBeriberilawInternal medicineGenotypemedicineThiamine transporterObjective: Thiamine or vitamin B1 deficiency diminishes thiamine-dependent enzymatic activity alters mitochondrial function impairs oxidative metabolism and causes selective neuronal death. We analyzed for the first time the role of all known mutations within three specific thiamine carrier genes SLC19 A2 SLC19 A3 and SLC25 A19 in a patient with atrophic beriberi a multiorgan nutritional disease caused by thiamine deficiency. Methods: A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema a subacute sensorimotor neuropathy and incontinence. Despite normal vitamin B1 serum levels his clinical picture was rapidly reverted by high-dose intramuscular thiamine treatment suggesting a possible genetic resistance. We used polymerase chain reaction followed by amplicon sequencing to study all the known thiamine-related gene mutations identified within the Human Gene Mutation Database. Results: Thirty-seven mutations were tested: 29 in SLC19 A2 6 in SLC19 A3 and 2 in SLC25 A19. Mutational analyses showed a wild-type genotype for all sequences investigated. Conclusion: This is the first genetic study in beriberi disease. We did not detect any known mutation in any of the three genes in a sporadic dry beriberi patient. We cannot exclude a role for other known or unknown mutations in the same genes or in other thiamine-associated genes in the occurrence of this nutritional neuropathy.HumansThiamineGenePolymerase chain reactionGeneticsMutationNutrition and DieteticsbiologyMembrane Transport ProteinsThiamine Deficiencymedicine.diseaseAlcoholismEndocrinologyMutationbiology.proteinThiamineMutations
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Neural drive preservation after detraining following neuromuscular electrical stimulation training

2006

The purpose of the study was to investigate the behaviour of the central nervous system when 5 weeks of neuromuscular electrical stimulation (NMES) training was followed by 5 weeks of detraining. Nineteen males were divided into the neuromuscular electrostimulated group (EG, n=12) and the control group (CG, n=7). The training program consisted of 15 sessions of isometric NMES over a 5-week period. The EG subjects were tested before training (PRE), after 5 weeks of NMES training (POST) and after 5 weeks of detraining (DE) while CG subjects were only tested at PRE and at POST. Soleus (SOL) and gastrocnemii (GAS) maximal H-reflex and M-wave potentials were evoked at rest (i.e., H(max) and M(ma…

AdultMalemedicine.medical_specialtyTime FactorsPhysical ExertionStimulationIsometric exercisePlantar flexionH-ReflexVoluntary contractionPhysical medicine and rehabilitationHumansMedicineMuscle SkeletalNeuronal Plasticitybusiness.industryGeneral NeuroscienceMuscle activationEvoked Potentials MotorAdaptation PhysiologicalElectric StimulationSupramaximal stimulationPhysical FitnessAnesthesiaH-reflexTraining programbusinesshuman activitiesAnkle JointMuscle ContractionNeuroscience Letters
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Loss of the Ca2+/calmodulin-dependent protein kinase type IV in dopaminoceptive neurons enhances behavioral effects of cocaine.

2008

The persistent nature of addiction has been associated with activity-induced plasticity of neurons within the striatum and nucleus accumbens (NAc). To identify the molecular processes leading to these adaptations, we performed Cre/loxP-mediated genetic ablations of two key regulators of gene expression in response to activity, the Ca 2+ /calmodulin-dependent protein kinase IV (CaMKIV) and its postulated main target, the cAMP-responsive element binding protein (CREB). We found that acute cocaine-induced gene expression in the striatum was largely unaffected by the loss of CaMKIV. On the behavioral level, mice lacking CaMKIV in dopaminoceptive neurons displayed increased sensitivity to cocai…

AdultMalemedicine.medical_specialtymedia_common.quotation_subjectMice TransgenicStriatumBiologyNucleus accumbensCREBPolymorphism Single NucleotideCocaine-Related DisordersMiceInternal medicineGene expressionmedicineAnimalsHumansProtein kinase ACyclic AMP Response Element-Binding Proteinmedia_commonRegulation of gene expressionNeuronsAnalysis of VarianceMultidisciplinaryNeuronal PlasticityAddictionGene Expression ProfilingBiological SciencesMolecular biologyImmunohistochemistryConditioned place preferenceCorpus StriatumEndocrinologyGene Expression Regulationbiology.proteinFemaleBrazilCalcium-Calmodulin-Dependent Protein Kinase Type 4Gene DeletionProceedings of the National Academy of Sciences of the United States of America
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Passive exposure to speech sounds modifies change detection brain responses in adults

2019

In early life auditory discrimination ability can be enhanced by passive sound exposure. In contrast, in adulthood passive exposure seems to be insufficient to promote discrimination ability, but this has been tested only with a single short exposure session in humans. We tested whether passive exposure to unfamiliar auditory stimuli can result in enhanced cortical discrimination ability and change detection in adult humans, and whether the possible learning effect generalizes to different stimuli. To address these issues, we exposed adult Finnish participants to Chinese lexical tones passively for 2 h per day on 4 consecutive days. Behavioral responses and the brain's event-related potenti…

AdultMalemedicine.medical_specialtyoppiminenCognitive NeuroscienceMismatch negativityhavaitseminenAudiologyperceptual learningevent-related potentialsta3112050105 experimental psychology03 medical and health sciencesSound exposureP3aYoung Adultäänteet0302 clinical medicinePerceptual learningEvent-related potentialP3bspeech soundsmedicineotorhinolaryngologic diseasespuhe (ilmiöt)HumansSpeech0501 psychology and cognitive sciencesAttentionaivotutkimusLatency (engineering)ta515Neuronal Plasticitypassive exposure05 social sciencesBrainContrast (music)Event-Related Potentials P300kuuloNeurologyAcoustic StimulationEvoked Potentials AuditorySpeech PerceptionFemalePsychology030217 neurology & neurosurgeryNeuroImage
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The effect of paired associative stimulation on fatigue resistance

2015

Paired associative stimulation (PAS) is a non-invasive stimulation method developed to induce bidirectional changes in the excitability of the cortical projections to the target muscles. However, very few studies have shown an association between changes in motor evoked potentials (MEP) after PAS and behavioral changes in healthy subjects. In the present study we hypothesized that the functional relevance of PAS can be seen during fatiguing exercise, since there is always a central contribution to the development of fatigue. Transcranial magnetic stimulation was applied over the motor cortex to measure changes in the MEPs of the soleus muscle before and after PAS. Furthermore, fatigue resis…

AdultMalepaired associative stimulationmedicine.medical_treatmentStimulationIsometric exerciseta3112Young AdultPaired associative stimulationFatigue resistancemotor cortextranscranial magnetic stimulationmedicineHumansddc:796Muscle Skeletalta315skin and connective tissue diseasesSoleus muscleNeuronal PlasticityGeneral NeuroscienceHealthy subjectsFatigue; Central fatigue; Motor cortex; Transcranial magnetic stimulation; Paired associative stimulationGeneral MedicineEvoked Potentials Motorcentral fatigueTranscranial magnetic stimulationmedicine.anatomical_structureMuscle FatigueFemalefatiguesense organsPsychologyNeuroscienceMotor cortex
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Morphologic diagnosis in neuronal ceroid lipofuscinosis.

1997

Morphologic pathology in NCL is marked by two processes, the interaction of which has not yet been completely clarified: 1) degeneration of nerve cells, foremost in the cerebral cortex, resulting in considerable cerebral atrophy in early childhood forms, likely responsible for clinical and neuroradiological findings; 2) widespread accumulation of autofluorescent lysosomal lipopigments of varying ultrastructure, the demonstration of which is still largely responsible for diagnostic recognition of an individual patient's NCL. Numerous tissues and organs are available for biopsy, among them brain (historical), rectum (still favoured by some), skeletal muscle and peripheral nerves (largely by c…

AdultPathologymedicine.medical_specialtyConjunctivaAdolescentBiopsyAutopsyAtrophyNeuronal Ceroid-LipofuscinosesBiopsyMedicineHumansTissue DistributionChildCerebral atrophyCerebral Cortexmedicine.diagnostic_testbusiness.industryInfantGeneral MedicinePigments Biologicalmedicine.diseaseLipidsmedicine.anatomical_structureCerebral cortexChild PreschoolPediatrics Perinatology and Child HealthNerve DegenerationNeuronal ceroid lipofuscinosisNeurology (clinical)Morphologic diagnosisAtrophybusinessLysosomesNeuropediatrics
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Electrophysiological findings of neuronal ceroid lipofuscinosis in heterozygotes.

1988

Nineteen obligate heterozygotes, 8 individuals at risk of being heterozygote, and 10 patients afflicted with four different forms of neuronal ceroid lipofuscinosis were examined electrophysiologically. The group of obligate heterozygotes was compared to age-matched control groups. Statistically significant differences were found between scotopic b-wave amplitudes, P-ERG amplitudes, and EOG light peaks of the obligate carriers of the juvenile type and the control subjects. The photopic L-ERGs and the latencies of the VEPs were mostly within the normal range. The findings represent the first evidence of functional ophthalmological changes in obligate carriers of neuronal ceroid lipofuscinosis…

AdultPathologymedicine.medical_specialtyHeterozygotegenetic structuresAdolescentPhysiologyBiologyCellular and Molecular NeuroscienceNeuronal Ceroid-LipofuscinosesRisk FactorsmedicineElectroretinographyHumansScotopic visionChildmedicine.diagnostic_testObligateHeterozygote advantageElectrooculographymedicine.diseaseSensory SystemsOphthalmologyElectrophysiologyElectrooculographyChild PreschoolEvoked Potentials VisualNeuronal ceroid lipofuscinosissense organsElectroretinographyPhotopic visionGraefe's archive for clinical and experimental ophthalmology = Albrecht von Graefes Archiv fur klinische und experimentelle Ophthalmologie
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Cytokeratin 20 Is a General Marker of Cutaneous Merkel Cells While Certain Neuronal Proteins Are Absent

1995

Merkel cells are difficult to identify in tissue sections. Previous studies have used cytokeratins (CK) 8, 18, and 19 as histologic markers of Merkel cells. However, these CKs are also expressed in some outer root sheath keratinocytes and some early fetal epidermal cells and thus are not truly specific of Merkel cells in general. Using selective antibodies against a newly described CK, number 20--originally found in intestinal epithelium and Merkel cell carcinomas--in comparison to a key protein of neuroendocrine cells, chromogranin A, we established CK 20 as a specific Merkel cell marker in skin of humans, pigs, and mice. CK 20 seems to be an even more general and sensitive Merkel cell mar…

AdultPathologymedicine.medical_specialtySwineCellHuman skinNerve Tissue ProteinsDermatologyKeratin-20BiologyOuter root sheathBiochemistryCytokeratinMiceFetusIntermediate Filament ProteinsmedicineAnimalsHumansMolecular BiologySkinintegumentary systemChromogranin APeripherinEpithelial CellsCell BiologyMolecular biologyImmunohistochemistrymedicine.anatomical_structurebiology.proteinMerkel cellNeuronal Cell Adhesion MoleculeBiomarkersHairJournal of Investigative Dermatology
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Current state of clinical and morphological features in human NCL.

2004

The neuronal ceroid lipofuscinoses (NCL) are large group of autosomal recessive lysosomal storage disorders with both enzymatic deficiency and structural protein dysfunction. Previously, diagnosis of (NCL) was based on age at onset clinicopathological (C‐P) findings described 4 forms, classified as infantile (INCL) (2), late‐infantile (LINCL) (5), juvenile (JNCL) (6), and adult (ANCL) most patients with NCL have progressive ocular and cerebral dysfunvtion, including cognitive/motor dysfunction and uncontrolled seizures. After reviewing 520 patients with NCL, we found that about 104 (20%) did not fit this classification of NCL With further research, 4 additional forms have been recognized: F…

AdultPathologymedicine.medical_specialtymedicine.disease_causeArticlePathology and Forensic MedicineEpilepsyNeuronal Ceroid-LipofuscinosesGenotypemedicineHumansPalmitoyl protein thioesteraseAge of OnsetChildInclusion BodiesMutationbiologyTripeptidyl-Peptidase 1General NeurosciencePPT1Infantmedicine.diseasePhenotypeCLN8Child PreschoolMutationbiology.proteinNeurology (clinical)Age of onsetBrain pathology (Zurich, Switzerland)
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