Search results for "Neuropathy"

showing 10 items of 213 documents

Nervenläsionen bei der Hüft-TEP-Implantation - Strategien zur Lähmungsprophylaxe

2003

Aim Nerve lesions are a serious complication of total hip arthroplasty. The incidence of lesions of large nerves exceeds those of luxation and septic loosening. An analysis of damage causes should lead to prophylactic strategies and improvement of operative technique. Patients are more aware of this complication and malpractice cases are increasing. Between 1977 and 2001 the medical association of North Rhine dealt with 126 cases of nerve lesions. Method All potential intraoperative causes for nerve damage are investigated. Results Incorrectly placed Hohmann retractors offer the largest damage potential. Electro surgery and self-cutting screws have a hazardous potential. Sharp injuries, ret…

Hip surgerymedicine.medical_specialtybusiness.industrymedicine.medical_treatmentmedicine.diseaseArthroplastySurgeryRetractorPeripheral neuropathyMalpracticeOrthopedic surgerymedicineOrthopedics and Sports MedicineSurgerybusinessComplicationTrauma surgeryZeitschrift für Orthopädie und ihre Grenzgebiete
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Diabetic foot syndrome: Immune-inflammatory features as possible cardiovascular markers in diabetes

2015

Diabetic foot ulcerations have been extensively reported as vascular complications of diabetes mellitus associated with a high degree of morbidity and mortality. Diabetic foot syndrome (DFS), as defined by the World Health Organization, is an "ulceration of the foot (distally from the ankle and including the ankle) associated with neuropathy and different grades of ischemia and infection". Pathogenic events able to cause diabetic foot ulcers are multifactorial. Among the commonest causes of this pathogenic pathway it's possible to consider peripheral neuropathy, foot deformity, abnormal foot pressures, abnormal joint mobility, trauma, peripheral artery disease. Several studies reported how …

Inflammationmedicine.medical_specialtyPathologySettore MED/09 - Medicina InternaDiabetic foot syndrome; Inflammation; Cytokines; Cardiovascular risk; Markerbusiness.industryType 2 Diabetes MellitusType 2 diabetesDiseaseDiabetic foot syndromeMarkerCardiovascular riskmedicine.diseaseDiabetic footInsulin resistancePeripheral neuropathyInternal medicineDiabetes mellitusmedicineOrthopedics and Sports MedicineTopic HighlightbusinessCytokineFoot deformityWorld Journal of Orthopedics
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A novel compound heterozygous mutation in GALC associated with adult-onset Krabbe disease: case report and literature review

2021

Krabbe disease (KD) is a rare autosomal recessive lipid storage leukodystrophy. It is caused by deficient enzyme activity resulting from mutations of the β-galactocerebrosidase (GALC) gene. KD is distinguished into subtypes based on the age of onset; these are early infantile, late infantile, juvenile, and adult-onset. We report a case of a 47-year-old Caucasian man with a 2-year history of muscle atrophy and weakness in both hands associated with pyramidal signs and mild spasticity in the lower limbs. An extensive work-up led this motor neuron disease-like disorder to be diagnosed as adult-onset KD. The patient was found to be compound heterozygous for two GALC mutations (p.G286D and p.Y49…

MaleCellular and Molecular NeuroscienceHeterozygoteMutationGeneticsHumansSettore MED/26 - NeurologiaMiddle AgedGenetics (clinical)Compound heterozygous mutation GALC Adult-onset Krabbe disease Peripheral neuropathyGalactosylceramidaseLeukodystrophy Globoid Cell
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A very mild phenotype of Charcot-Marie-Tooth disease type 4H caused by two novel mutations in FGD4

2019

Abstract Background Mutations in the FGD4 gene cause an autosomal recessive demyelinating peripheral neuropathy referred to as CMT4H, characterized by its onset in infancy or early-childhood and its slow progression. Methods The clinical and genetic status of two patients with CMT4H was studied, performing genetic testing with a panel of genes and analysing FGD4 mRNA expression by quantitative PCR. Results Two novel FGD4 variants (c.514delG and c.2211dupA) were identified in two mildly affected Spanish siblings with CMT4H, and with disease onset in late adolescence/adulthood (one of them remaining asymptomatic at 20). On examination, foot deformity was observed without weakness or sensory i…

MaleCharcot-Marie-ToothPathologymedicine.medical_specialtyAdolescentFGD4medicine.disease_causeAsymptomaticYoung Adult03 medical and health sciences0302 clinical medicineCharcot-Marie-Tooth DiseaseCharcot-Marie-Tooth disease type 4HCMT4HmedicineHumans030212 general & internal medicineAlleleFrameshift MutationGeneAllelesGenetic testingMutationmedicine.diagnostic_testbusiness.industrySiblingsCMTMicrofilament Proteinsmedicine.diseasePhenotypePedigreeNeuropathyPhenotypePeripheral neuropathyNeurologyFemaleNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryJournal of the Neurological Sciences
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SPG10 is a rare cause of spastic paraplegia in European families.

2008

Contains fulltext : 71099.pdf (Publisher’s version ) (Closed access) BACKGROUND: SPG10 is an autosomal dominant form of hereditary spastic paraplegia (HSP), which is caused by mutations in the neural kinesin heavy chain KIF5A gene, the neuronal motor of fast anterograde axonal transport. Only four mutations have been identified to date. OBJECTIVE: To determine the frequency of SPG10 in European families with HSP and to specify the SPG10 phenotype. PATIENTS AND METHODS: 80 index patients from families with autosomal dominant HSP were investigated for SPG10 mutations by direct sequencing of the KIF5A motor domain. Additionally, the whole gene was sequenced in 20 of these families. RESULTS: Th…

MaleDNA Mutational AnalysisKinesinsHEREDITARYmedicine.disease_cause0302 clinical medicineSpasticPerception and Action [DCN 1]Missense mutationKIF5AAge of OnsetChildFrameshift MutationMUTATIONGenes DominantGeneticsNeurologic Examination0303 health sciencesMutationSplice site mutationSITEExonsMiddle AgedAnterograde axonal transport3. Good healthPedigreeEuropePsychiatry and Mental healthPhenotypeATAXIASChild PreschoolFemaleChromosome DeletionMOTORFunctional Neurogenomics [DCN 2]AdultNeuromuscular diseaseGenotypeHereditary spastic paraplegiaMutation Missense03 medical and health sciencesCognitive neurosciences [UMCN 3.2]medicineHumansGait Disorders Neurologic030304 developmental biologyChromosome Aberrationsbusiness.industrySpastic Paraplegia HereditarySequence Analysis DNAmedicine.diseaseGENEPeripheral neuropathyGenetics PopulationSurgeryNeurology (clinical)RNA Splice Sitesbusiness030217 neurology & neurosurgeryJournal of neurology, neurosurgery, and psychiatry
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The influence of local capsaicin treatment on small nerve fibre function and neurovascular control in symptomatic diabetic neuropathy.

2002

Topical treatment with capsaicin cream has been shown to be successful in the treatment of different symptomatic nerve disorders like diabetic neuropathy. Conflicting data exist on the effect of capsaicin on nerve function and neurovascular control especially in patients with diabetic neuropathy. The aim of this pilot study was to investigate the impact of topical capsaicin application on small nerve fibre function and neurovascular control. Capsaicin cream was applied to the feet of 13 patients with symptomatic diabetic neuropathy over a period of 8 weeks. Before and during the treatment period, we investigated the total symptoms score, the vibration, thermal (heat and cold) and pain perce…

MaleDiabetic neuropathyEndocrinology Diabetes and MetabolismAdministration TopicalSubstance PPilot Projectschemistry.chemical_compoundRoute of administrationEndocrinologyNerve FibersDiabetic NeuropathiesInternal MedicineMedicineHumansbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseNeurovascular bundlePeripheral neuropathymedicine.anatomical_structureDiabetes Mellitus Type 1chemistryDiabetes Mellitus Type 2CapsaicinAnesthesiaSensory ThresholdsNociceptorFemaleCapsaicinbusinessSensory nervePhytotherapyActa diabetologica
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Giant axonal neuropathy and leukodystrophy

1991

Abstract An 11-year-old Persian boy, born to consanguineous parents, manifested a progressive gait abnormality beginning at 5 years of age. A severe cerebellar disorder developed with associated dysfunction of the peripheral nervous system, but no sign of mental impairment. The sensory and motor nerve conduction velocities were greatly reduced, especially in the lower extremities. Cerebrospinal fluid protein was normal. Computed tomography and magnetic resonance imaging revealed leukoencephalopathy, especially in the cerebellum, but also in periventricular areas. The diagnosis of giant axonal neuropathy was established by biopsy of the sural nerve. The few previous histologic examinations h…

MaleIntermediate FilamentsMotor nerveGenes RecessiveSural nerveCerebral VentriclesLeukoencephalopathyConsanguinityDevelopmental NeuroscienceCerebellummedicineHumansCerebellar disorderGliosisPeripheral NervesChildMyelin SheathSpinocerebellar DegenerationsGiant axonal neuropathybusiness.industryLeukodystrophyAnatomymedicine.diseaseMagnetic Resonance ImagingAxonsMicroscopy Electronmedicine.anatomical_structurenervous systemNeurologyPeripheral nervous systemPediatrics Perinatology and Child HealthGait abnormalityNeurology (clinical)medicine.symptomHereditary Sensory and Motor NeuropathybusinessPediatric Neurology
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Waterjet Dissection of Peripheral Nerves: An Experimental Study of the Sciatic Nerve of Rats

2010

BACKGROUND: Although waterjet dissection has been well evaluated in intracranial pathologies, little is known of its qualities in peripheral nerve surgery. Theoretically, the precise dissection qualities could support the separation of nerves from adjacent tissues and improve the preservation of nerve integrity in peripheral nerve surgery. OBJECTIVE: To evaluate the potential of the new waterjet dissector in peripheral nerve surgery. METHODS: Waterjet dissection with pressures of 20 to 80 bar was applied on the sciatic nerves of 101 rats. The effect of waterjet dissection on the sciatic nerve was evaluated by clinical tests, neurophysiological examinations, and histopathological studies up …

MaleMicrosurgerySciatic Neuropathymedicine.medical_treatmentDissection (medical)Neurosurgical ProceduresRats Sprague-DawleyPressuremedicineAnimalsIntraoperative ComplicationsTherapeutic Irrigationbusiness.industryDissectionEquipment DesignAnatomyNerve injuryMicrosurgerySurgical Instrumentsmedicine.diseaseSciatic NerveNerve RegenerationRatsPeripheralmedicine.anatomical_structurePeripheral nervous systemModels AnimalSurgeryOccipital nerve stimulationNeurology (clinical)Sciatic nerveSciatic Neuropathymedicine.symptombusinessOperative Neurosurgery
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Sensory neuropathy and signs of central sensitization in patients with peripheral arterial disease.

2006

Patients with peripheral arterial disease (PAD) may develop a broad range of peripheral nerve dysfunctions including pain and sensory deficiencies due to chronic ischemia mostly involving the lower limbs. To investigate the degree of sensory abnormalities in such patients quantitative sensory testing (QST) might be a useful tool. Forty-five patients and 20 controls were enrolled in the present study and underwent QST according to the protocol of the German Research Network on Neuropathic Pain. PAD was graded according to the Rutherford classification. PAD patients were divided into two groups: 16 patients with critical limb ischemia (severe PAD) and 29 patients with intermittent claudicatio…

MalePain ThresholdIschemiaSensationSensationmedicineHumansThermosensingAgedPain MeasurementNeurologic ExaminationPeripheral Vascular DiseasesAnalysis of Variancebusiness.industryPeripheral Nervous System DiseasesCritical limb ischemiamedicine.diseaseIntermittent claudicationbody regionsAnesthesiology and Pain MedicinePeripheral neuropathyAllodyniamedicine.anatomical_structureNeurologyAnesthesiaCase-Control StudiesNeuropathic painSensation DisordersFemaleNeurology (clinical)medicine.symptombusinessSensory nervePain
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Thermal Thresholds Predict Painfulness of Diabetic Neuropathies

2004

OBJECTIVE—Pathophysiology explaining pain in diabetic neuropathy (DN) is still unknown. RESEARCH DESIGN AND METHODS—Thirty patients with peripheral DN (17 men and 13 women; mean age 52.4 ± 2.5 years) were investigated. Fifteen patients had neuropathic pain, and 15 patients were free of pain. Patients were followed over 2 years and examined at the beginning and thereafter every 6 months. Clinical severity and painfulness of the DN were assessed by the neuropathy impairment score and visual analog scales (VASs). Cold and warm perception thresholds as well as heat pain thresholds were obtained for evaluation of Aδ- and C-fibers. Nerve conduction velocities (NCVs) and vibratory thresholds were …

MalePain Thresholdmedicine.medical_specialtyHot TemperatureDiabetic neuropathyVisual analogue scaleEndocrinology Diabetes and MetabolismElectromyographyRisk AssessmentSeverity of Illness IndexDiabetic NeuropathiesPredictive Value of TestsReference ValuesDiabetes mellitusSeverity of illnessInternal MedicinemedicineHumansHeart rate variabilityThermosensingPain MeasurementProbabilityAdvanced and Specialized Nursingmedicine.diagnostic_testElectromyographybusiness.industryMiddle AgedPrognosismedicine.diseaseCold TemperatureElectrophysiologyDiabetes Mellitus Type 1Diabetes Mellitus Type 2HyperalgesiaCase-Control StudiesSensory ThresholdsAnesthesiaPredictive value of testsNeuropathic painPhysical therapyFemalebusinessDiabetes Care
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