Search results for "Neuropilin"

showing 8 items of 8 documents

Vascular endothelial growth factor expression in nasal polyps of aspirin-intolerant patients.

2012

Objective To study differences between aspirin-tolerant patients and aspirin-intolerant patients concerning vascular endothelial growth factor (VEGF) expression. Recent publications strongly suggest the involvement of VEGF and its receptors in the pathophysiologic process of nasal polyps. Design We subjected 43 polyp specimens to semiquantitative immunohistochemical analysis. We quantified VEGF and its receptors (Flk, Flt, and neuropilin) in all samples. To gain insight into potential VEGF-mediated cellular responses, we determined proliferative (Ki67) and apoptotic (caspase 3) indices. Patients Polyp samples were obtained from 22 aspirin-intolerant patients and from 21 aspirin-tolerant pat…

AdultMaleVascular Endothelial Growth Factor APathologymedicine.medical_specialtyAdolescentApoptosisStatistics Nonparametricchemistry.chemical_compoundNasal Polypsotorhinolaryngologic diseasesNeuropilinMedicineHumansNasal polypsReceptorAspirinAspirinbusiness.industryCaspase 3General MedicineMiddle Agedmedicine.diseaseImmunohistochemistryPathophysiologyVascular endothelial growth factorVascular endothelial growth factor AKi-67 AntigenReceptors Vascular Endothelial Growth FactorOtorhinolaryngologychemistryImmunohistochemistrySurgeryFemalebusinessmedicine.drugArchives of otolaryngology--headneck surgery
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Expression of semaphorin 3A and neuropilin 1 with clinicopathological features and survival in human tongue cancer

2012

Objective: To investigate the association between semaphorin 3A (SEMA 3A) and its receptor neuropilin 1 (NRP1) and the clinicopathologic characteristics of patients with tongue cancer. Study Design: Forty-three tongue squamous cell carcinoma specimens were included. Immunohistochemical staining of SEMA3A and NRP1 was performed on 15 normal tongue epithelium specimens and the 43 tumour specimens. Immunoreactivity was evaluated based on the staining intensity and distribution score. Statistical analyses were performed using Chi-squared and Spearman tests and Kaplan-Meier analysis. Results: SEMA3A was significantly down-regulated in tongue cancer compared with normal tongue (P=0.025), while NR…

MalePathologymedicine.medical_specialtyOdontologíaSemaphorinTongueNeuropilin 1medicineHumansGeneral DentistryOral Medicine and Pathologybusiness.industryCancerSemaphorin-3ASEMA3AMiddle AgedPrognosismedicine.disease:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludNeuropilin-1EpitheliumTongue NeoplasmsStainingSurvival Ratemedicine.anatomical_structureOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASCarcinoma Squamous CellImmunohistochemistryResearch-ArticleFemaleSurgerybusiness
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The impact of the receptor binding profiles of the vascular endothelial growth factors on their angiogenic features

2013

Abstract Background Vascular endothelial growth factors (VEGFs) are potential therapeutic agents for treatment of ischemic diseases. Their angiogenic effects are mainly mediated through VEGF receptor 2 (VEGFR2). Methods Receptor binding, signaling, and biological efficacy of several VEGFR2 ligands were compared to determine their characteristics regarding angiogenic activity and vascular permeability. Results Tested VEGFR2 ligands induced receptor tyrosine phosphorylation with different efficacy depending on their binding affinities. However, the tyrosine phosphorylation pattern and the activation of the major downstream signaling pathways were comparable. The maximal angiogenic effect stim…

Vascular Endothelial Growth Factor ASwineAngiogenesisBlotting WesternBiophysicsNeovascularization PhysiologicVascular permeabilityBiologyBiochemistryCapillary Permeabilitychemistry.chemical_compoundCell MovementHuman Umbilical Vein Endothelial CellsNeuropilinAnimalsHumansImmunoprecipitationPhosphorylationReceptorMolecular BiologyAortaCells CulturedCell Proliferationta1182Tyrosine phosphorylationrespiratory systemLigand (biochemistry)Vascular Endothelial Growth Factor Receptor-2Cell biologyVascular endothelial growth factorEndothelial stem cellchemistryBiochemistrycardiovascular systemEndothelium VascularPlasmidsSignal Transductioncirculatory and respiratory physiologyBiochimica et Biophysica Acta (BBA) - General Subjects
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Bone marrowVEGFCexpression is associated with multilineage dysplasia and several prognostic markers in adult acute myeloid leukemia, but not with sur…

2018

Vascular endothelial growth factor C (VEGFC) stimulates leukemia cell proliferation and survival, and promotes angiogenesis. We studied VEGFC expression in bone marrow samples from 353 adult acute myeloid leukemia (AML) patients and its relationship with several clinical, cytogenetic, and molecular variables. We also studied the expression of 84 genes involved in VEGF signaling in 24 patients. We found that VEGFC expression was higher in AML patients with myelodysplasia-related changes (AML-MRC) than in patients with non-AML-MRC. We also found an association between VEGFC expression and the patient cytogenetic risk group, with those with a worse prognosis having higher VEGFC expression leve…

AdultMale0301 basic medicineCancer ResearchAdolescentAngiogenesisVascular Endothelial Growth Factor CKaplan-Meier EstimateVEGFC expressionYoung Adult03 medical and health sciences0302 clinical medicineKDRBone Marrowhemic and lymphatic diseasesNeuropilin 1Biomarkers TumormedicineNRP1HumansGeneFLT1AgedChromosome AberrationsAcute myeloid leukemiaVascular Endothelial Growth Factor Receptor-1Cell growthbusiness.industryAdult Acute Myeloid LeukemiaHematologyVEGF signalingMiddle AgedPrognosismedicine.diseaseVascular Endothelial Growth Factor Receptor-2Neuropilin-1Leukemia Myeloid AcuteLeukemia030104 developmental biologymedicine.anatomical_structureOncologyVascular endothelial growth factor CMyelodysplastic Syndromes030220 oncology & carcinogenesisCancer researchFemaleBone marrowbusinessLeukemia & Lymphoma
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The VEGF/VEGF-R Axis in Sporadic Vestibular Schwannomas Correlates with Irradiation and Disease Recurrence

2012

<b><i>Background/Aims:</i></b> The molecular mechanisms downstream of mutated neurofibromatosis type 2 (NF2) gene resulting in the growth and development of vestibular schwannoma (VS) are controversial. Several lines of evidence suggest the involvement of the vascular endothelial growth factor (VEGF) pathway in VS development. Given that recent studies of VEGF blockade in patients with NF2-associated VS showed positive effects on VS growth control, we initiated this comprehensive study of the VEGF pathway in sporadic VS. <b><i>Methods:</i></b> A tissue microarray analysis of 182 sporadic VS was conducted. The expression of VEGF and its recepto…

AdultMaleVascular Endothelial Growth Factor APathologymedicine.medical_specialtyAdolescentYoung Adultchemistry.chemical_compoundNeuropilin 1medicineHumansNeurofibromatosis type 2ReceptorAgedCell ProliferationCell growthbusiness.industryNeuroma AcousticMiddle Agedmedicine.diseaseNeuromaImmunohistochemistryVascular Endothelial Growth Factor Receptor-2Neuropilin-1BlockadeVascular endothelial growth factorReceptors Vascular Endothelial Growth FactorOtorhinolaryngologychemistryTissue Array AnalysisImmunohistochemistryFemaleNeoplasm Recurrence LocalbusinessORL
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Liver-primed memory T cells generated under noninflammatory conditions provide anti-infectious immunity.

2013

SummaryDevelopment of CD8+ T cell (CTL) immunity or tolerance is linked to the conditions during T cell priming. Dendritic cells (DCs) matured during inflammation generate effector/memory T cells, whereas immature DCs cause T cell deletion/anergy. We identify a third outcome of T cell priming in absence of inflammation enabled by cross-presenting liver sinusoidal endothelial cells. Such priming generated memory T cells that were spared from deletion by immature DCs. Similar to central memory T cells, liver-primed T cells differentiated into effector CTLs upon antigen re-encounter on matured DCs even after prolonged absence of antigen. Their reactivation required combinatorial signaling thro…

T cellReceptors Antigen T-CellPriming (immunology)chemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationGeneral Biochemistry Genetics and Molecular BiologyMiceCross-PrimingAntigenCD28 AntigensmedicineAnimalslcsh:QH301-705.5Innate immune systemGene Expression ProfilingT-cell receptorReceptors Interleukin-12CD28Endothelial Cellshemic and immune systemsDendritic CellsAcquired immune systemListeria monocytogenesImmunity InnateNeuropilin-1Mice Inbred C57BLmedicine.anatomical_structurelcsh:Biology (General)LiverImmunologyImmunologic MemoryCD8Cell reports
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Noncanonical GLI1 signaling promotes stemness features and in vivo growth in lung adenocarcinoma

2016

Aberrant Hedgehog/GLI signaling has been implicated in a diverse spectrum of human cancers, but its role in lung adenocarcinoma (LAC) is still under debate. We show that the downstream effector of the Hedgehog pathway, GLI1, is expressed in 76% of LACs, but in roughly half of these tumors, the canonical pathway activator, Smoothened, is expressed at low levels, possibly owing to epigenetic silencing. In LAC cells including the cancer stem cell compartment, we show that GLI1 is activated noncanonically by MAPK/ERK signaling. Different mechanisms can trigger the MAPK/ERK/GLI1 cascade including KRAS mutation and stimulation of NRP2 by VEGF produced by the cancer cells themselves in an autocrin…

0301 basic medicineMAPK/ERK pathwayCancer ResearchLung NeoplasmsPyridinesPyridineMitogen-Activated Protein Kinase KinaseMice SCIDMiceCarcinoma Non-Small-Cell LungRNA Small InterferingNon-Small-Cell LungMolecular Biology; Genetics; Cancer ResearchTumorbiologyintegumentary systemHedgehog signaling pathwayCell biologyNeoplastic Stem CellsFemaleRNA InterferenceOriginal ArticleHumanXenograft Model Antitumor AssayAdenocarcinomaSCIDSmall InterferingZinc Finger Protein GLI1Cell LineProto-Oncogene Proteins p21(ras)Adenocarcinoma; Animals; Carcinoma Non-Small-Cell Lung; Cell Line Tumor; Female; Humans; Lung Neoplasms; Mice; Mice SCID; Mitogen-Activated Protein Kinase Kinases; Neoplastic Stem Cells; Neuropilin-2; Proto-Oncogene Proteins p21(ras); Pyridines; Pyrimidines; RNA Interference; RNA Small Interfering; Xenograft Model Antitumor Assays; Zinc Finger Protein GLI1; Molecular Biology; Genetics; Cancer Research03 medical and health sciencesParacrine signallingGeneticSettore MED/04 - PATOLOGIA GENERALEstem cellsCancer stem cellGLI1Cell Line TumorGeneticsAnimalsHumansAutocrine signallingMolecular BiologyMitogen-Activated Protein Kinase KinasesSettore MED/06 - ONCOLOGIA MEDICAAnimalCarcinomaXenograft Model Antitumor AssaysNeuropilin-2Lung Neoplasmlung cancer030104 developmental biologyPyrimidinesPyrimidineCancer cellbiology.proteinRNANeoplastic Stem CellSmoothened
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Presence of the Transmembrane Protein Neuropilin in Cytokine-induced Killer Cells

2020

Background/aim Cytokine-induced killer (CIK) cells are a heterogenous population of immune cells showing promising applications in immunotherapeutic cancer treatment. Neuropilin (NRP) proteins have been proven to play an important role in cancer development and prognosis. In this study, CIK cells were tested for expression of NRPs, transmembrane proteins playing a role in the proliferation and survival of cancer cells. Materials and methods CIK cells were analyzed at different time points via flow cytometry and quantitative real-time polymerase chain reaction for neuropilin expression. Results Phenotyping results showed CIK cells having developed properly, and low levels of NRP2 were detect…

Cancer ResearchImmunologyCellBiologyFlow cytometryCytokine-Induced Killer CellsImmune systemNeoplasmsNeuropilin 1medicineNeuropilinHumansNeuropilinsFlow cytometryNeuropilin.A549 cellmedicine.diagnostic_testCytokine-induced killer cellGeneral MedicinePrognosisNeuropilin-1Neuropilin-2Gene Expression Regulation NeoplasticBrain tumorCytokine-induced killer cellmedicine.anatomical_structureOncologyA549 CellsCancer cellCancer researchImmunotherapyLung cancerAnticancer Research
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