Search results for "Neurotoxin"

showing 10 items of 53 documents

Reactive neurogenesis during regeneration of the lesioned medial cerebral cortex of lizards

1995

Abstract This study reports that lesion of the adult lizard medial cortex (lizard hipocampal fascia dentata) induces a short period of intensive neurogenesis which we have termed reactive neurogenesis; a cell proliferation event that occurs in the subjacent ependyma. Specific lesion of the medial cortex was achieved by intraperitoneal injection of the neurotoxin 3-acetylpyridine and proliferating cells were detected using tritiated thymidine or 5-bromodeoxiuridine pulse labelling. After lesion, granule neurons in the lizard medial cortex cell layer appeared pyknotic and died; they were then removed and progressively replaced by a set of new neurons. These neurons were mostly generated from …

Pathologymedicine.medical_specialtyPyridinesMedial cortexNeurotoxinsPodarcis hispanicaLesionNeuroblastmedicineAnimalsCerebral CortexbiologyGeneral NeuroscienceNeurogenesisLizardsDNAAnatomybiology.organism_classificationImmunohistochemistryNerve RegenerationMicroscopy Electronmedicine.anatomical_structureCerebral cortexAutoradiographyFascia dentatamedicine.symptomEpendymaCell DivisionNeuroscience
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Temperature dependence of the toxic effects of phenytoin on peripheral neuromuscular function of the rat tail.

1990

We studied the acute effects of a single dose of phenytoin (250 mg/kg) on peripheral neuromuscular function. The evoked muscle action potentials of the dorsal segmental muscles in the rat tail, and the conduction velocity of the dorsal nerve trunk which innervates them, were measured before and after the intraperitoneal injection of phenytoin. The experiments were performed at different temperatures, 27 (physiological tail temperature), 36 and 37 degrees C (physiological central temperature) in different groups of animals. The amplitudes of the evoked muscle action potentials in the treated groups showed no significant modifications at 27 degrees C, at 36 degrees C a small nonsignificant de…

PhenytoinMaleTailmedicine.medical_specialtyTime Factorsmedicine.medical_treatmentIntraperitoneal injectionCentral nervous systemNeurotoxinsNeural ConductionNeuromuscular JunctionAction PotentialsToxicologyNerve conduction velocityCellular and Molecular NeuroscienceDevelopmental NeuroscienceReference ValuesInternal medicinemedicineAnimalsEvoked PotentialsChemistryMusclesTemperatureRats Inbred StrainsRatsElectrophysiologyEndocrinologyAnticonvulsantmedicine.anatomical_structureAnesthesiaPeripheral nervous systemPhenytoinToxicitymedicine.drugNeurotoxicology and teratology
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Excitotoxin-induced changes in transglutaminase during differentiation of cerebellar granule cells

2002

Excitotoxicity induced by NMDA receptor stimulation is able to increase the activity of many enzymes involved in neuronal cell death. Primary cultures of rat cerebellar granule cells were used to elucidate the role of transglutaminase reaction in the excitotoxic cell response, and to evaluate the role of glutamate receptors in cell survival and degeneration. Granule neurons, maintained in vitro for two weeks, were exposed to NMDA at different stages of differentiation. Following NMDA receptor activation, increases in transglutaminase activity were observed in cell cultures. The levels of enzyme activity were higher in cells at 5 days in vitro than in those at 8-9 or 13-14 days in vitro. Mor…

Programmed cell deathN-MethylaspartateTime FactorsCell SurvivalTissue transglutaminaseNeurotoxinsClinical BiochemistryExcitotoxicityStimulationmedicine.disease_causeReceptors N-Methyl-D-AspartateBiochemistryCerebellummedicineAnimalsRats WistarNeuronsTransglutaminasesbiologyOrganic ChemistryGlutamate receptorCell DifferentiationIn vitroRatsCell biologyAnimals Newbornnervous systemApoptosisNerve Degenerationbiology.proteinNMDA receptorTransglutaminase – Excitotoxicity – Neurodegenerative diseases – Apoptosis – Glutamate – Cerebellar granule neuronsAmino Acids
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Inhibition in vivo of the activity of botulinum neurotoxin A by small molecules selected by virtual screening

2012

To search for small molecular size inhibitors of botulinum neurotoxin A (BoNT/A) endopeptidase activity, we have screened the NCI library containing about 1 million structures against the substrate binding pocket of BoNT/A. Virtual screening (VS) was performed with the software Glide (Grid-based ligand docking energetics) and the findings were confirmed by AutoDock. Ten compounds were found that had favorable energetic and glide criteria and 5 of these compounds were selected for their ability to protect mice in vivo against a lethal dose of BoNT/A. Each compound was incubated at different molar excesses with a lethal dose of the toxin and then the mixture injected intravenously into mice. …

Protein ConformationToxinChemistryNeurotoxinsLethal doseComputational BiologyAutoDockPharmacologyProtective AgentsToxicologymedicine.disease_causeSmall moleculeToxicologyMiceEndopeptidase activityDocking (molecular)In vivoToxicitymedicineAnimalsBotulinum Toxins Type ASequence AlignmentSoftwareToxicon
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Minireview: Nicotinic Acetylcholine Receptors on Hippocampal Neurons: Distribution on the Neuronal Surface and Modulation of Receptor Activity

1997

The recent development of a technique that uses infrared microscopy for the visualization of well-defined areas on the surface of neurons, and a computerized system of micromanipulators led to the discovery that functional nicotinic acetylcholine receptors (nAChRs) are expressed at higher density on the dendrites than on the soma of rat hippocampal neurons. The finding that the expression of alpha-bungarotoxin-sensitive, alpha 7-bearing, nAChRs and dihydro-beta-erythroidine-sensitive, alpha 4 beta 2 nAChRs tends to increase along the dendritic length suggests that these receptors may be highly involved in the integration of synaptic functions in hippocampal neurons. The present report also …

SerotoninMicrocystinsBacterial ToxinsNeurotoxinsReceptors NicotinicHippocampal formationPharmacologyHippocampusModels BiologicalBiochemistryGanglion type nicotinic receptormedicineAnimalsReceptorEvoked PotentialsMolecular Biologygamma-Aminobutyric AcidAcetylcholine receptorNeuronsCyanobacteria ToxinsChemistryCell BiologyAcetylcholineRatsmedicine.anatomical_structureNicotinic agonistnervous systemMarine ToxinsSomaAlpha-4 beta-2 nicotinic receptorInfrared microscopyNeuroscienceJournal of Receptors and Signal Transduction
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Vulnerability of peripheral catecholaminergic neurons to MPTP is not regulated by alpha-synuclein.

2010

Although generally considered a prototypical movement disorder, Parkinson's disease is commonly associated with a broad-spectrum of non-motor symptoms, including autonomic dysfunctions caused by significant alterations in catecholaminergic neurons of the peripheral sympathetic nervous system. Here we present evidence that alpha-synuclein is highly expressed by sympathetic ganglion neurons throughout embryonic and postnatal life and that it is found in tyrosine hydroxylase-positive sympathetic fibers innervating the heart of adult mice. However, mice deficient in alpha-synuclein do not exhibit any apparent alterations in sympathetic development. Sympathetic neurons isolated from mouse embryo…

Sympathetic nervous system1-Methyl-4-phenylpyridiniumα-Synuclein knockoutTyrosine 3-MonooxygenaseNeurotoxinsNeurotrophic factorSubstantia nigraBiologylcsh:RC321-571chemistry.chemical_compoundMiceCatecholaminesSympathetic Fibers PostganglionicParkinsonian DisordersNeurotrophic factorsmedicineNeurotoxinAutonomic gangliaAnimalslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCells CulturedNeuronsGanglia SympatheticCell DeathMPTPSympathetic ganglionMice Mutant Strainsnervous system diseasesMPP+medicine.anatomical_structureNeurologychemistrynervous system1-Methyl-4-phenyl-1236-tetrahydropyridinePeripheral nervous systemSympathetic nervous systemNerve Degenerationalpha-SynucleinCatecholaminergic cell groupsPeripheral nervous systemNeuroscienceNeurobiology of disease
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Recent progress in optical and electrochemical biosensors for sensing of Clostridium botulinum neurotoxin

2018

Abstract Botulinum toxin is a neurotoxic protein which produced from Clostridium botulinum and related species and it block acetylcholine release from presynaptic nerve terminals at the neuromuscular junctions. This toxin is life threatening for millions of people and growing menace to society since causing human botulism. Enzymatic activity of Botulinum neurotoxin within the cell made it hazardous and lead to flaccid paralysis. However, there isn't any reliable and precise remedy for this toxin. Therefore, there is an urgent need for early detection of this toxin in a fast and meticulous way with a robust and cost-effective relationship for real-time monitoring of Botulinum neurotoxin. Sev…

Web of sciencebusiness.industry010401 analytical chemistryEarly detection02 engineering and technology021001 nanoscience & nanotechnologymedicine.diseasemedicine.disease_cause01 natural sciencesBotulinum toxinBotulinum neurotoxin0104 chemical sciencesAnalytical ChemistryElectrochemical biosensorNeurotoxinMedicineClostridium botulinumBotulism0210 nano-technologybusinessNeuroscienceSpectroscopymedicine.drugTrAC Trends in Analytical Chemistry
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A synthetic hexapeptide (Argireline) with antiwrinkle activity

2008

Botulinum neurotoxins (BoNTs) represent a revolution in cosmetic science because of their remarkable and long-lasting antiwrinkle activity. However, their high neurotoxicity seriously limits their use. Thus, there is a need to design and validate non-toxic molecules that mimic the action of BoNTs. The hexapeptide Ac-EEMQRR-NH2 (coined Argireline) was identified as a result of a rational design programme. Noteworthy, skin topography analysis of an oil/water (O/W) emulsion containing 10% of the hexapeptide on healthy women volunteers reduced wrinkle depth up to 30% upon 30 days treatment. Analysis of the mechanism of action showed that Argireline significantly inhibited neurotransmitter relea…

chemistry.chemical_classificationAgingRational designNeurotoxicityPharmaceutical SciencePeptideDermatologymedicine.diseaseExocytosisColloid and Surface ChemistryMechanism of actionchemistryBiochemistryChemistry (miscellaneous)In vivoDrug DiscoveryToxicitymedicineNeurotoxinmedicine.symptomInternational Journal of Cosmetic Science
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First evidence of antimicrobial activity of neurotoxin 2 from anemonia sulcata (Cnidaria)

2014

International audience; We investigated the antibacterial activity of Anemonia sulcata (Cnidaria, Anthozoa) tentacle and body acidic extracts. Biochemical purification consisted of first step on solid phase Sep-Pak C8 column followed by several HPLC runs on C18 column using different conditions. Anti-Micrococcus lysodeikticus activity has been detected in 40 % acetonitrile fractions. The resulting purified molecule from tentacles had a molecular mass determined by MALDI-TOF mass spectrum of 4946,299 Da and has been completely sequenced. Its aa sequence revealed identity with the Neurotoxin 2 (ATX-II), a Na + channel blocking toxins. Consequently, ATX-II appeared to display a dual role as to…

lcsh:Biology (General)antimicrobial peptideMicrococcus lysodeikticusneurotoxin[SDU.STU.HY]Sciences of the Universe [physics]/Earth Sciences/Hydrologylcsh:QH301-705.5antimicrobial peptide; Anemonia sulcata; ATX II; neurotoxin; Micrococcus lysodeikticusATX IIAnemonia sulcata
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The CB1 cannabinoid receptor mediates excitotoxicity-induced neural progenitor proliferation and neurogenesis.

2007

Endocannabinoids are lipid signaling mediators that exert an important neuromodulatory role and confer neuroprotection in several types of brain injury. Excitotoxicity and stroke can induce neural progenitor (NP) proliferation and differentiation as an attempt of neuroregeneration after damage. Here we investigated the mechanism of hippocampal progenitor cell engagement upon excitotoxicity induced by kainic acid administration and the putative involvement of the CB1 cannabinoid receptor in this process. Adult NPs express kainate receptors that mediate proliferation and neurosphere generation in vitro via CB1 cannabinoid receptors. Similarly, in vivo studies showed that excitotoxicity-induce…

medicine.medical_specialtyKainic acidCannabinoid receptorNeurotoxinsExcitotoxicityKainate receptorBiologymedicine.disease_causeBiochemistryNeuroprotectionHippocampuschemistry.chemical_compoundMiceReceptor Cannabinoid CB1Epidermal growth factorInternal medicinemedicineAnimalsMolecular BiologyCell ProliferationMice KnockoutNeuronsKainic AcidStem CellsNeurogenesisCell BiologyEndocannabinoid systemCell biologyNerve RegenerationEndocrinologynervous systemchemistrylipids (amino acids peptides and proteins)Fibroblast Growth Factor 2The Journal of biological chemistry
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