Search results for "Nitric oxide synthase"

showing 10 items of 531 documents

Contractile responses of human thyroid arteries to vasopressin

2013

Abstract Aims In the present study we investigated the intervention of nitric oxide and prostacyclin in the responses to vasopressin of isolated thyroid arteries obtained from multi-organ donors. Main methods Paired artery rings from glandular branches of the superior thyroid artery, one normal and the other deendothelised, were mounted in organ baths for isometric recording of tension. Concentration–response curves to vasopressin were determined in the absence and in the presence of either the vasopressin V 1 receptor antagonist d(CH 2 ) 5 Tyr(Me)AVP (10 − 8  M), the nitric oxide synthase inhibitor N G -monomethyl- l -arginine ( L -NMMA, 10 − 4  M), or the inhibitor of prostaglandins indom…

AdultMaleVasopressinmedicine.medical_specialtyEndotheliumVasopressinsIndomethacinThyroid GlandProstacyclinIn Vitro TechniquesGeneral Biochemistry Genetics and Molecular BiologyNitric oxideSuperior thyroid arterychemistry.chemical_compoundmedicine.arteryInternal medicinemedicineHumansDrug InteractionsGeneral Pharmacology Toxicology and PharmaceuticsAgedVasopressin receptoromega-N-MethylarginineDose-Response Relationship DrugbiologyChemistryThyroidArteriesGeneral MedicineMiddle AgedArginine VasopressinNitric oxide synthaseEndocrinologymedicine.anatomical_structureVasoconstrictionbiology.proteinFemalehormones hormone substitutes and hormone antagonistsmedicine.drugLife Sciences
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Simultaneous assessment of endothelial function, nitric oxide synthase activity, nitric oxide-mediated signaling, and oxidative stress in individuals…

2007

Abstract Background: Endothelial function is impaired in hypercholesterolemia and atherosclerosis. Based on mostly indirect evidence, this impairment is attributed to reduced synthesis or impaired biological activity of endothelium-derived nitric oxide (NO). It was the aim of this study to directly estimate and compare whole-body NO production in normo- and hypercholesterolemia by applying a nonradioactive stable isotope dilution technique in vivo. Methods: We enrolled 12 normocholesterolemic and 24 hypercholesterolemic volunteers who were all clinically healthy. To assess whole-body NO synthesis, we intravenously administered l-[guanidino-(15N2)]-arginine and determined the urinary excreti…

AdultMalemedicine.medical_specialtyAdolescentNitric Oxide Synthase Type IIIClinical BiochemistryHypercholesterolemiaVasodilationEndothelial NOSArginineDinoprostNitric OxideNitric oxideExcretionchemistry.chemical_compoundSex FactorsInternal medicinemedicineHumansCyclic GMPAgedNitratesbiologyBiochemistry (medical)Nitric Oxide Synthase Type IIIMiddle AgedNitric oxide synthaseOxidative StressEndocrinologychemistrybiology.proteinFemaleEndothelium VascularNitric Oxide SynthaseAsymmetric dimethylarginineLipoproteinClinical chemistry
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Association between polymorphisms of endothelial nitric oxide synthase gene (NOS3) and left posterior wall thickness (LPWT) of the heart in Fabry dis…

2008

Fabry disease is an X-chromosomal storage disorder due to loss-of-function mutations of the GLA gene encoding the lysosomal enzyme α-galactosidase A. Accumulating glycosphingolipid deposits disturb the function of various cells, in particular that of myocytes, arterial smooth-muscle cells, and vascular endothelium. Hypertrophic cardiomyopathy, for example measured by left posterior wall thickness (LPWT) of the heart, represents a major component of Fabry disease morbidity in adult patients. Endothelium-derived nitric oxide (eNO), produced by eNO synthase (eNOS), is a key regulator of vessel wall function and cardiovascular homeostasis. We analysed the effect of the polymorphisms c.894G > T …

AdultMalemedicine.medical_specialtyAdolescentNitric Oxide Synthase Type IIIMutation MissenseMinisatellite RepeatsYoung AdultSex FactorsGene FrequencyInternal medicineGermanyGenotypeGeneticsMedicineHumansGenetic Predisposition to DiseaseAlleleChildAllele frequencyGenetics (clinical)AgedUltrasonographyGeneticsAlpha-galactosidasePolymorphism Geneticbiologybusiness.industryMyocardiumHaplotypeHypertrophic cardiomyopathyNitric Oxide Synthase Type IIIExonsCardiomyopathy HypertrophicMiddle Agedmedicine.diseaseFabry diseaseIntronsEndocrinologyPhenotypeHaplotypesCase-Control Studiesalpha-Galactosidasebiology.proteinFabry DiseaseRegression AnalysisFemaleHypertrophy Left VentricularbusinessJournal of inherited metabolic disease
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Endothelium-dependent responses in human isolated thyroid arteries from donors

2004

The functional properties of the endothelium of human thyroid arteries remain unexplored. We investigated the intervention of nitric oxide (NO), prostacyclin (PGI(2)) and endothelium-derived hyperpolarizing factor (EDHF) in the responses to acetylcholine and noradrenaline in isolated thyroid arteries obtained from multi-organ donors. Artery rings were suspended in organ baths for isometric recording of tension. The contribution of NO, PGI(2) and EDHF to endothelium-dependent relaxation was determined by the inhibitory effects of N(G)-monomethyl-L-arginine (L-NMMA), indomethacin, and K(+) channel inhibitors respectively. Acetylcholine induced concentration-dependent relaxation; this effect w…

AdultMalemedicine.medical_specialtyCharybdotoxinCharybdotoxinEndotheliumEndocrinology Diabetes and MetabolismIndomethacinThyroid GlandProstacyclinNitric OxideApaminOuabainNitric oxideBiological FactorsNorepinephrinechemistry.chemical_compoundEndocrinologyCulture TechniquesInternal medicinePotassium Channel BlockersmedicineHumansomega-N-MethylarginineDose-Response Relationship DrugArteriesMiddle AgedEpoprostenolAcetylcholineEndocrinologymedicine.anatomical_structureApaminchemistryVasoconstrictioncardiovascular systemFemaleEndothelium VascularNitric Oxide SynthaseSodium-Potassium-Exchanging ATPasePeptidesAcetylcholinemedicine.drugArteryJournal of Endocrinology
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Effects of exhaustive stretch-shortening cycle exercise on muscle blood flow during exercise

2006

Aim:  The influence of exhaustive stretch-shortening cycle exercise (SSC) on skeletal muscle blood flow (BF) during exercise is currently unknown. Methods:  Quadriceps femoris (QF) BF was measured in eight healthy men using positron emission tomography before and 3 days after exhaustive SSC exercise. The SSC protocol consisted of maximal and submaximal drop jumps with one leg. Needle biopsies of the vastus lateralis muscles were taken immediately and 2 days after SSC for muscle endothelial nitric oxide synthase (eNOS) and interleukin-1-beta (IL-1β) mRNA level determinations. Results:  All subjects reported subjective muscle soreness after SSC (P < 0.001), which was well in line with a decre…

AdultMalemedicine.medical_specialtyNitric Oxide Synthase Type IIIPhysiologyBiopsyHemodynamicsPhysical exerciseIsometric exerciseStatistics NonparametricStretch shortening cycleEnosIsometric ContractionInternal medicinemedicineHumansRNA MessengerMuscle SkeletalCreatine KinaseExercisebiologyReverse Transcriptase Polymerase Chain ReactionChemistrySkeletal muscleBlood flowbiology.organism_classificationSurgeryOxygenmedicine.anatomical_structureRegional Blood FlowPositron-Emission TomographyMuscle FatigueCardiologyPerfusionInterleukin-1Acta Physiologica
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Influence of risk factors on nitric oxide metabolites at the initial stage of juvenile acute myocardial infarction.

2009

Few data are accessible about the nitric oxide (NO) stable end-products (nitrite/NO2 − and nitrate/NO3 − :N O x) in acute coronary syndromes. An increase in inducible NO synthase (iNOS) was found during experimental myocardial infarction [13] and this increase persisted for 2 weeks. In experimental models of acute myocardial infarction (AMI) other authors [2] observed a NOx increase, a correlation between NOx level and iNOS activity and an inhibitory action carried out by S-methylisothiourea, that is an iNOS inhibitor [5]. The NOx level was also measured in a small group of patients with myocardial infarction in whom the peak of NOx elevation occurred 2 and 3 days after the onset of symptom…

AdultMalemedicine.medical_specialtyPhysiologyMyocardial InfarctionNitric Oxide Synthase Type IIEssential hypertensionNitric OxideNitric oxideCoronary artery diseasechemistry.chemical_compoundYoung AdultGriess testRisk FactorsPhysiology (medical)Internal medicinemedicineJuvenile myocardial infarction cardiovascular risk factors nitrite nitrateHumansMyocardial infarctionNitriteNOxbusiness.industryHematologyVenous bloodMiddle Agedmedicine.diseaseSurgerychemistryCase-Control StudiesCardiologyFemaleCardiology and Cardiovascular MedicinebusinessClinical hemorheology and microcirculation
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Nitric oxide mediates abnormal responsiveness of thyroid arteries in methimazole-treated patients.

2005

Objective: We studied the intervention of nitric oxide (NO), prostacyclin and endothelium-derived hyperpolarizing factor (EDHF) in mediating responses to acetylcholine in thyroid arteries from euthyroid and methimazole-treated (MT) patients. Design and methods: Branches of the superior thyroid artery were obtained from 19 euthyroid patients and 17 MT patients (euthyroid at the time of surgery) undergoing total thyroidectomy or hemithyroidectomy. Artery rings were suspended in organ baths for isometric recording of tension. Results and conclusions: Acetylcholine caused endothelium-dependent relaxation of greater magnitude in arteries from MT patients (pD2 (−log EC50) values were 7.68±0.19 in…

AdultMalemedicine.medical_specialtyPotassium ChannelsCharybdotoxinEndocrinology Diabetes and MetabolismMuscle RelaxationThyroid GlandApaminNitric OxideHyperthyroidismMuscle Smooth VascularSuperior thyroid arterychemistry.chemical_compoundBiological FactorsNorepinephrineEndocrinologyAntithyroid AgentsInternal medicinemedicine.arterymedicinePotassium Channel BlockersHumansEuthyroidEnzyme InhibitorsMethimazoleomega-N-Methylargininebusiness.industryThyroidGeneral MedicineArteriesMiddle AgedAcetylcholinemedicine.anatomical_structureEndocrinologychemistryApaminCirculatory systemPotassiumCalciumFemaleEndothelium VascularNitric Oxide SynthasebusinessAcetylcholinemedicine.drugBlood vesselArteryEuropean journal of endocrinology
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Influence of Endothelial Nitric Oxide on Adrenergic Contractile Responses of Human Cerebral Arteries

1996

The present study was designed to investigate the influence of the endothelium and that of the L-arginine pathway on the contractile responses of isolated human cerebral arteries to electrical field stimulation (EFS) and norepinephrine. Rings of human middle cerebral artery were obtained during autopsy of 19 patients who had died 3–8 h before. EFS (1–8 Hz) induced frequency-dependent contractions that were abolished by tetrodotoxin, prazosin, and guanethidine (all at 10-6 M). The increases in tension were of greater magnitude in arteries denuded of endothelium. NG-monomethyl L-arginine (L-NMMA 10-4 M) potentiated the contractile response to EFS in artery rings with endothelium but did not …

AdultMalemedicine.medical_specialtySympathetic Nervous SystemEndotheliumIndomethacinCerebral arteriesIn Vitro TechniquesNitric Oxide030218 nuclear medicine & medical imagingNitric oxideNorepinephrine03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicine.arteryInternal medicinePrazosinmedicineHumansGuanethidineAgedAged 80 and overomega-N-Methylargininebusiness.industryCerebral ArteriesMiddle AgedElectric Stimulationmedicine.anatomical_structureEndocrinologyNeurologychemistryVasoconstrictionAnesthesiaMiddle cerebral arterycardiovascular systemFemaleEndothelium VascularNeurology (clinical)Nitric Oxide SynthaseCardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgerymedicine.drugBlood vesselArteryJournal of Cerebral Blood Flow &amp; Metabolism
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Plasma concentration of asymmetric dimethylarginine, an endogenous inhibitor of nitric oxide synthase, is elevated in hyperthyroid patients.

2002

Cardiovascular manifestations are frequent findings in patients with thyroid hormone disorders. Nitric oxide (NO) plays a key role in vascular, endothelial-mediated relaxation. NO is synthesized from L-arginine by NO synthase, an enzyme inhibited by endogenous compounds, mainly asymmetric dimethylarginine [asymmetric N(G),N(G)-dimethyl-L-arginine (ADMA)]. The aim of our work was to investigate whether plasma L-arginine and dimethylarginine concentrations and NO production are altered in hypo- and hyperthyroid patients, compared with control subjects. L-arginine, ADMA and symmetric dimethylarginine were analyzed by HPLC. NO was measured as plasma nitrite plus nitrate (NO(x)) concentration by…

AdultMalemedicine.medical_specialtyendocrine system diseasesEndotheliumEndocrinology Diabetes and MetabolismClinical BiochemistryArginineNitric OxideBiochemistryHyperthyroidismNitric oxidechemistry.chemical_compoundEndocrinologyHypothyroidismGriess testReference ValuesInternal medicineBlood plasmamedicineHumansNitriteNitritesAgedNitratesbiologyBiochemistry (medical)Osmolar ConcentrationMiddle AgedNitric oxide synthaseEndocrinologymedicine.anatomical_structurechemistrybiology.proteinFemaleNitric Oxide SynthaseAsymmetric dimethylarginineHormoneThe Journal of clinical endocrinology and metabolism
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Nitric oxide production and endothelium-dependent vasorelaxation ameliorated by N1-methylnicotinamide in human blood vessels.

2012

N 1 -methylnicotinamide (MNA + ) has until recently been thought to be a biologically inactive product of nicotinamide metabolism in the pyridine nucleotides pathway. However, the latest observations imply that MNA + may exert antithrombotic and anti-inflammatory effects through direct action on the endothelium. We examined both in vivo and in vitro whether the compound might induce vasorelaxation in human blood vessels through the improvement of nitric oxide (NO) bioavailability and a reduction of oxidative stress mediated by endothelial NO synthase (eNOS) function. MNA + treatment (100 mg/m 2 orally) in healthy normocholesterolemic and hypercholesterolemic subjects increased the l-argini…

AdultNiacinamidemedicine.medical_specialtyEndotheliumBrachial ArteryNitric Oxide Synthase Type IIIHypercholesterolemiachemistry.chemical_elementCalciumIn Vitro Techniquesmedicine.disease_causeNitric OxideNitric oxidechemistry.chemical_compoundN^{1}-methylnicotinamideDouble-Blind MethodEnosnitric oxideInternal medicineInternal MedicinemedicineHumansflow-mediated dilationCalcimycinCells Culturedendothelial nitric oxide synthaseoxidized low-density lipoproteinbiologyDose-Response Relationship DrugChemistrySuperoxidebiology.organism_classificationendothelial cellsAcetylcholineOxygenVasodilationOxidative Stressmedicine.anatomical_structureEndocrinologysuperoxideEndothelium VascularAcetylcholineOxidative stressmedicine.drugLipoproteinHypertension (Dallas, Tex. : 1979)
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