Search results for "Nitro"

showing 10 items of 2762 documents

Progestogens stimulate prostacyclin production by human endothelial cells.

2005

BACKGROUND: The effects of progestogens on endothelial physiology are poorly studied. Prostacyclin is a potent vasodilator synthesized by two isoforms of cyclooxygenase (COX) in endothelium. We examined the effects of two clinically used progestogens, progesterone and medroxyprogesterone acetate (MPA), on prostacyclin production by cultured human umbilical vein endothelial cells (HUVEC) and the possible role of progesterone receptors and both COX enzymes. METHODS: Cells were exposed to 1-100 nmol/l of either progesterone or MPA and prostacyclin production was measured in culture medium. RESULTS: Both progestogens significantly increased prostacyclin release in a time- and dose-dependent man…

medicine.medical_specialtyUmbilical VeinsEndotheliumProstacyclinMedroxyprogesterone AcetateUmbilical veinInternal medicineProgesterone receptormedicineMedroxyprogesterone acetateHumansCyclooxygenase InhibitorsReceptorCells CulturedNitrobenzenesProgesteroneSulfonamidesbiologyCyclooxygenase 2 InhibitorsDose-Response Relationship DrugEstradiolRehabilitationObstetrics and GynecologyEndothelial CellsMembrane ProteinsEpoprostenolEndothelial stem cellMifepristoneEndocrinologymedicine.anatomical_structureReproductive MedicineCyclooxygenase 2Prostaglandin-Endoperoxide Synthasescardiovascular systembiology.proteinCyclooxygenase 1PyrazolesCyclooxygenaseEndothelium VascularProgestinsReceptors Progesteronemedicine.drugHuman reproduction (Oxford, England)
researchProduct

Is oxidative stress a therapeutic target in cardiovascular disease?

2010

An abnormal production of reactive oxygen species (ROS) and the subsequent decrease in vascular bioavailability of nitric oxide (NO) have long been proposed to be the common pathogenetic mechanism of the endothelial dysfunction, resulting from diverse cardiovascular risk factors such as hypercholesterolaemia, diabetes mellitus, chronic smoking, metabolic syndrome, and hypertension. Superoxide produced by the nicotinamide dinucleotide phosphate (NADPH) oxidase, mitochondrial sources, or the xanthine oxidase may react with NO, thereby resulting in excessive formation of peroxynitrite, a reactive nitrogen species that has been demonstrated to accelerate the atherosclerotic process by causing d…

medicine.medical_specialtyXanthine OxidaseAntioxidantmedicine.medical_treatmentmedicine.disease_causeArginineAntioxidantschemistry.chemical_compoundRisk FactorsInternal medicinemedicineHumansProspective StudiesEndothelial dysfunctionXanthine oxidaseReactive nitrogen specieschemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologybusiness.industrySuperoxideNADPH OxidasesPolyphenolsVitaminsmedicine.diseasePrognosisMitochondriaOxidative StressEndocrinologychemistryCardiovascular Diseasesbiology.proteinEndothelium VascularNitric Oxide SynthaseCardiology and Cardiovascular MedicinebusinessReactive Oxygen SpeciesOxidative stressEuropean heart journal
researchProduct

1203Post-ischemic stroke-induced myocardial dysfunction is associated with nitro-oxidative stress and sympathetic overactivity

2018

medicine.medical_specialtybusiness.industryInternal medicineIschemic strokeCardiologymedicineNitroCardiology and Cardiovascular Medicinemedicine.disease_causebusinessOxidative stressEuropean Heart Journal
researchProduct

280 Impact of ozone air pollution on ischemic cerebral and cardiac events in Dijon, France

2011

Background There is strong evidence that short-term exposure to ozone (O3) is associated with respiratory disease and death, but the effects of short-term exposure to ozone on ischemic heart and cerebrovascular disease have not been clearly established. Methods Daily levels of urban O3 pollution, the incidence of first-ever, recurrent, fatal and non-fatal ischemic cerebro-vascular events (ICVE) and myocardial infarction (MI) were compared using a bi-directional case-crossover design analysis. We analysed 1 574 ICVE and 913 MI that occurred in Dijon, France from 2001 to 2007. Sulphur dioxide (SO2), nitrogen dioxide (NO2), carbon monoxide (CO), particulate matter with an aerodiameter of 10μg/…

medicine.medical_specialtyeducation.field_of_studyOzonebusiness.industryIncidence (epidemiology)PopulationRespiratory diseaseConfoundingAir pollutionmedicine.diseasemedicine.disease_causechemistry.chemical_compoundchemistryInternal medicinemedicineCardiologyNitrogen dioxideMyocardial infarctioneducationbusinessCardiology and Cardiovascular MedicineArchives of Cardiovascular Diseases Supplements
researchProduct

Myogenic effects enhance norepinephrine constriction: Inhibition by nitric oxide and felodipine

1998

Myogenic effects enhance norepinephrine constriction: Inhibition by nitric oxide and felodipine. Myogenic, pressure-induced vasoconstriction may amplify the effects of circulating vasoconstrictors. Through intravital microscopy in cremaster arterioles (31 to 115 μm diameter), the relative contribution of myogenic responses (MR) to norepinephrine (NE)-induced constriction and the inhibitor potency of nitric oxide (NO) or a Ca2+ entry blocker (CEB), felodipine (F), were examined. In 24 anesthetized hamsters, a vessel occluder was placed around the aorta to control cremaster vessel inflow pressure (IP). NE infusion increased blood pressure (by 50 ± 2mm Hg) and induced significant constriction …

medicine.medical_specialtyendotheliumVasodilator AgentsmicrocirculationMyogenic mechanismBayliss effectBlood PressureNitric OxideNitroarginineMuscle Smooth VascularConstrictionNitric oxideMicrocirculationNorepinephrine (medication)Norepinephrinechemistry.chemical_compoundCricetinaeInternal medicineintravital microscopymedicineAnimalsVasoconstrictor AgentsBayliss effectAorta AbdominalcremasterFelodipineCapillariesArteriolesEndocrinologychemistryFelodipineNephrologyAnesthesiacalcium entry blockerInjections Intravenouscardiovascular systemmedicine.symptomVasoconstrictionmedicine.drugKidney International
researchProduct

Increased nitrotyrosine plasma levels in relation to systemic markers of inflammation and myeloperoxidase in chronic heart failure

2009

The presence of a reciprocal link between inflammation and oxidative/nitrosative stress has been postulated in chronic heart failure (CHF). We aimed to determine signs of nitrosative stress in serum/plasma of CHF patients. ELISA tests were used for quantification of serum/plasma levels of Nitrotyrosine (NT), H(2)O(2), total NO, nitrite (NO(2)(-)), myeloperoxidase (MPO), Tumor Necrosis Factor-alpha (TNFalpha) and pro-Brain Natriuretic Peptide (proBNP) in 66 CHF patients (9 in NYHA I, 34 NYHA II, 23 NYHA III) and in 14 age-matched healthy subjects. NT levels were higher in NYHA III CHF patients compared to NYHA II (p<0.05), NYHA I (p<0.03) and controls (p<0.02), whereas NO(2)(-) and total NO …

medicine.medical_specialtymedicine.drug_classInflammationSystemic inflammationGastroenterologyNITROSATIVE STRESSchemistry.chemical_compoundInternal medicineBlood plasmamedicineNatriuretic peptidecardiovascular diseasesOXIDATIVE STRESSEndothelial dysfunctionbiologybusiness.industryNitrotyrosinemedicine.diseasehumanitiesEndocrinologychemistryMyeloperoxidaseHeart failureENDOTHELIAL DYSFUNCTIONcardiovascular systembiology.proteinmedicine.symptomCardiology and Cardiovascular Medicinebusinesscirculatory and respiratory physiology
researchProduct

Nitric oxide and sensory afferent neurones modulate the protective effects of low-dose endotoxin on rat gastric mucosal damage

1995

Pretreatment (1 h) with low doses (5-40 micrograms/kg i.p.) of Escherichia coli endotoxin dose dependently reduced the gastric mucosal damage induced by a 10 min challenge with 1 ml ethanol (50% and 100%) in conscious rats. Treatment with the nitric oxide synthesis inhibitor, NG-nitro-L-arginine methyl ester (L-NAME, 5 and 10 mg/kg i.p.), significantly inhibited the protective effects of endotoxin (40 micrograms/kg i.p.). The actions of L-NAME were reversed by the prior administration of L-arginine (100 mg/kg i.p.). The protective effects of endotoxin were not influenced by pretreatment with dexamethasone (5 mg/kg s.c. twice) or indomethacin (5 mg/kg s.c.). However, ablation of sensory affe…

medicine.medical_treatmentIndomethacinPharmacologyArginineDexamethasoneNitric oxideRats Sprague-Dawleychemistry.chemical_compoundEscherichia colimedicineAnimalsNeurons AfferentEnzyme InhibitorsAntidoteDexamethasonePharmacologyAnalysis of VarianceEthanolEthanolSensory neuronRatsEndotoxinsNG-Nitroarginine Methyl Estermedicine.anatomical_structureMechanism of actionchemistryGastric MucosaCapsaicinAnesthesiaToxicityFemaleCapsaicinNitric Oxide Synthasemedicine.symptomInjections Intraperitonealmedicine.drugEuropean Journal of Pharmacology
researchProduct

Methodologies to study soil nitrogen Méthodes pour l'étude de l'azote dans le sol Metodologie per lo studio dell'azoto nel suolo

2011

Nitrogen (N) is a key element within terrestrial ecosystems and, thus, the whole knowledge of its transformation processes is crucial. Especially in soil, complex and heterogeneous system, N undergoes several transformations as it is indicated by its various oxidation states. Moreover, it mainly occurs under a number of organic forms, whose identification and quantification is still challenging. Here, we review some extraction methodologies of N from soil, but also the isotope dilution technique, which helps in assessing gross N mineralisation. Finally, we discuss the NanoSIMS technique, which shows a good potential for investigating soil N dynamics at nanoscale. Résumé L'azote (N) est un é…

methodology of analysiSettore AGR/13 - Chimica Agrarianitrogenδ15Nsoil
researchProduct

CCDC 2149627: Experimental Crystal Structure Determination

2022

Related Article: Adrian Laviós, Amparo Sanz-Marco, Carlos Vila, M. Carmen Muñoz, José R. Pedro, Gonzalo Blay|2022|Org.Lett.|24|2149|doi:10.1021/acs.orglett.2c00427

methyl (1R3S3aR8bR)-7-bromo-3-methoxy-3a-nitro-1-phenyl-233a8b-tetrahydro-1H-[1]benzofuro[23-c]pyrrole-1-carboxylateSpace GroupCrystallographyCrystal SystemCrystal StructureCell ParametersExperimental 3D Coordinates
researchProduct

Prevention of TNBS-induced colitis by probiotic supplement in mice

2014

micebusiness.industryProbioticsBioengineeringGeneral Medicinetrinitrobenzene sulfonic acidPharmacologyApplied Microbiology and Biotechnologylaw.inventionProbioticlawinflammationmicrobiotaMedicineProbiotics; microbiota; inflammation; trinitrobenzene sulfonic acid; micebusinessBiotechnologyTnbs colitis
researchProduct