Search results for "Nitroarginine"

showing 10 items of 111 documents

Nitric oxide and brain hyperexcitability.

2004

Nitric oxide (NO) is a gaseous messenger involved in atypical forms of intercellular communications, able to exert a strong functional modulation of several neurotransmitter systems. In particular, NO heavily influences the excitatory neurotransmitter glutamate, mainly through NMDA receptors, and the inhibitory neurotransmitter GABA, mainly through GABA A receptors. Due to the involvement of glutamate and GABA in a delicate balance conditioning the functional status of the neural cells, this interaction suggests a role for NO in regulating neuronal excitability and its transition towards hyperexcitability phenomena. This article reviews the main knowledge about the relationships existing be…

Disease Models AnimalEpilepsyNG-Nitroarginine Methyl EsterAnimalsBrainGlutamic AcidHumansNitric oxide glutamate GABA epilepsy reviewNervous System DiseasesNitric OxideSettore BIO/09 - Fisiologiagamma-Aminobutyric AcidIn vivo (Athens, Greece)
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Mechanisms underlying diabetes enhancement of endothelin-1-induced contraction in rabbit basilar artery

2004

The influence of alloxan-induced diabetes on the reactivity of rabbit basilar artery to endothelin-1 was examined. Endothelin-1 induced concentration-dependent contraction of basilar arteries that was higher in diabetic than in control rabbits. Endothelium removal produced a higher enhancement of the endothelin-1-induced contraction in control than in diabetic rabbits. N(G)-nitro-L-arginine (L-NOArg) enhanced the maximal contraction induced by endothelin-1 in control rabbits and potentiated this response in diabetic rabbits. Endothelin ETA receptor antagonist, cyclo(D-Asp-Pro-D-Val-Leu-D-Trp) (BQ-123), inhibited endothelin-1-induced contraction in both rabbit groups. Endothelin ETB receptor…

Endothelin Receptor AntagonistsMaleNitroprussidemedicine.medical_specialtyContraction (grammar)Vascular smooth muscleEndotheliumEndothelin A Receptor AntagonistsVasodilator AgentsEndothelin B Receptor AntagonistsNitroargininePeptides CyclicMuscle Smooth VascularDiabetes Mellitus ExperimentalPiperidinesIsometric Contractionmedicine.arteryInternal medicinemedicineBasilar arteryAnimalsEnzyme InhibitorsAntihypertensive AgentsPharmacologyDiabetisEndothelin-1Artèriesbusiness.industryEndoteli vascularReceptor Endothelin AReceptor Endothelin BEndothelin 1Òxid nítricEndothelin A Receptor AntagonistsEndothelin B Receptor AntagonistsEndocrinologymedicine.anatomical_structureBasilar Arterycardiovascular systemRabbitsbusinessEndothelin receptorOligopeptidesEuropean Journal of Pharmacology
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β2- and β1-Adrenoceptor Expression Exhibits a Common Regulatory Pattern With GRK2 and GRK5 in Human and Animal Models of Cardiovascular Diseases.

2015

To explore if genic expression of β(1)- or β(2)-adrenoceptors (ARs) exhibits a common regulatory pattern with G protein-coupled receptor kinase (GRK) 2, GRK3, or GRK5 expression, we determined messenger RNA levels for these genes in different tissues from human and animal models of cardiovascular disease. We measured genic expression by qRT polymerase chain reaction in the left and right ventricles or peripheral blood mononuclear cells from healthy (n = 21), hypertensive (n = 20), heart failure (n = 24), and heart transplanted patients (n = 17) or in left ventricle, peripheral blood mononuclear cells, and kidney from spontaneously hypertensive rats or L-N-methyl-arginine-induced hypertensiv…

G-Protein-Coupled Receptor Kinase 5medicine.medical_specialtyG-Protein-Coupled Receptor Kinase 2Heart DiseasesBiologyPeripheral blood mononuclear cellRats Inbred WKYInternal medicineRats Inbred SHRmedicineAnimalsHumansRNA MessengerReceptorGenePharmacologyRegulation of gene expressionMessenger RNAG protein-coupled receptor kinaseKinaseBeta adrenergic receptor kinaseDisease Models AnimalEndocrinologyNG-Nitroarginine Methyl EsterGene Expression RegulationOrgan SpecificityCase-Control StudiesHypertensionbiology.proteinReceptors Adrenergic beta-2Receptors Adrenergic beta-1Cardiology and Cardiovascular MedicineJournal of cardiovascular pharmacology
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Differential sensitivity of rat hepatocyte CYP isoforms to self-generated nitric oxide.

2001

AbstractEarly loss of P450 in rat hepatocyte cultures appears directly related to nitric oxide (NO) overproduction. This study investigates the influence of endogenously generated NO (or NO-derived species) on the relative expression of cytochrome P450 (CYP) isoforms in rat hepatocytes. Our results support the view that loss of P450 holoenzyme in culture is the ultimate consequence of a NO driven process, activated during the common hepatocyte isolation procedure, that leads to an accelerated and selective degradation of specific CYP apoproteins. Under conditions in which NO and peroxynitrite formation is operative, changes in the level of specific CYP isoforms result in a significant alter…

Gene isoformMaleTime FactorsBlotting WesternBiophysicsNitric OxideBiochemistryDexamethasoneNitric oxideRats Sprague-Dawleychemistry.chemical_compoundP450 contentApoenzymesCytochrome P-450 Enzyme Systembeta-NaphthoflavoneStructural BiologyGeneticsmedicineAnimalsInducerOverproductionMolecular BiologyCells CulturedDrug metabolismbiologyCytochrome P450Cell BiologyCytochrome P450 inductionCell biologyRatsIsoenzymesmedicine.anatomical_structureNG-Nitroarginine Methyl EsterBiochemistrychemistryHepatocyteEnzyme Inductionbiology.proteinHepatocytesNitric Oxide SynthaseCytochrome P450 isoformRat hepatocyte cultureHoloenzymesPeroxynitriteDrug metabolismFEBS letters
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Potential Functional Significance of Brain-Type and Muscle-Type Nitric Oxide Synthase I Expressed in Adventitia and Media of Rat Aorta

1999

Abstract —Skeletal muscle and myocardium express μNOS I, an elongated splice variant of neuronal-type nitric oxide (NO) synthase (NOS I), and NOS III, endothelial-type NO synthase, respectively. This study was designed to elucidate whether vascular smooth muscle also contains a constitutively expressed NO synthase isoform. In the rat, μNOS I contains an insert of 102 nucleotides after nucleotide 2865 of the cDNA, yielding a protein of 164 kd. Reverse transcription-polymerase chain reaction with primers flanking this insert and with insert-specific primers indicated that endothelium-denuded rat aorta expresses both brain-type NOS I and μNOS I. RNase protection analyses with an antisense RNA…

Gene isoformPathologymedicine.medical_specialtyDNA ComplementaryVascular smooth muscleNitric Oxide Synthase Type IIIBlotting WesternAorta ThoracicNitric Oxide Synthase Type INitroarginineGene Expression Regulation EnzymologicMuscle Smooth VascularMembrane PotentialsPotassium ChlorideNitric oxideImmunoenzyme TechniquesRats Sprague-DawleyNorepinephrinechemistry.chemical_compoundmedicine.arteryAdventitiamedicineAnimalsVasoconstrictor AgentsAorta AbdominalRNA MessengerMuscle SkeletalMessenger RNAAortabiologyBrainSkeletal muscleMolecular biologyRatsNitric oxide synthaseAntisense Elements (Genetics)medicine.anatomical_structurechemistryVasoconstrictionbiology.proteinCalciumFemaleNitric Oxide SynthaseTunica MediaCardiology and Cardiovascular MedicineArteriosclerosis, Thrombosis, and Vascular Biology
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Involvement of neuronal processes and nitric oxide in the inhibition by endotoxin of pentagastrin-stimulated sastric acid secretion

1994

Administration of E. coli endotoxin (1 mg kg-1, i.v.) abolished the acid response induced by the i.v. infusion of pentagastrin (8 micrograms kg-1 h-1) in the continuously perfused stomach of the anaesthetized rat. Local serosal application of tetrodotoxin (36 ng per rat) completely restored acid responses to pentagastrin in endotoxin-treated rats. However, pretreatment with atropine (0.5 mg kg-1, s.c.), capsaicin (20, 30, and 50 mg kg-1, s.c. 2 weeks before the study) or guanethidine (16 mg kg-1, s.c. 3 and 16h before) did not influence the inhibitory effects of endotoxin. Continuous i.v. infusion with NG-nitro arginine methyl ester (L-NAME, 10 mg kg-1 h-1) restored the secretory responses …

LipopolysaccharidesMalemedicine.medical_specialtyTetrodotoxinPeptide hormoneBiologyArginineNitric OxideNitric oxideGastric AcidPhenylephrinechemistry.chemical_compoundInternal medicineEscherichia colimedicineAnimalsDrug InteractionsRats WistarInfusions IntravenousGuanethidineNeuronsPharmacologyStomachGeneral MedicineRatsEndotoxinsPentagastrinNG-Nitroarginine Methyl EsterEndocrinologyMechanism of actionchemistryGastrointestinal hormoneGastric MucosaCapsaicinTetrodotoxinFemalePentagastrinmedicine.symptommedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Effects of asymmetric dimethylarginine on renal arteries in portal hypertension and cirrhosis

2016

AIM. To evaluate the effects of asymmetric dimethylarginine (ADMA) in renal arteries from portal hypertensive and cirrhotic rats. METHODS. Rat renal arteries from Sham (n = 15), pre-hepatic portal hypertension (PPVL; n = 15) and bile duct ligation and excision-induced cirrhosis (BDL; n = 15) were precontracted with norepinephrine, and additional contractions were induced with ADMA (10-6-10-3 mol/L), an endogenous inhibitor of nitric oxide (NO) synthase. Concentration-response curves to acetylcholine (1 × 10-9-3 × 10-6 mol/L) were determined in precontracted renal artery segments with norepinephrine in the absence and in the presence of ADMA. Kidneys were collected to determine the protein e…

Liver CirrhosisMale0301 basic medicineAsymmetric dimethylarginineCirrhosisArginineKidneyurologic and male genital diseasesRats Sprague-DawleyNorepinephrinechemistry.chemical_compoundRenal ArteryVasoconstrictor AgentsEnzyme InhibitorsPortal hypertensionKidneyGastroenterologyGeneral MedicineBasic StudyDimethylarginine dimethylaminohydrolaseNG-Nitroarginine Methyl Estermedicine.anatomical_structureCirrhosisCardiologyPortal hypertensionmedicine.symptommedicine.medical_specialtyCirrosi hepàticaEndotheliumArginineNitric OxidePressió sanguíniaAmidohydrolases03 medical and health sciencesInternal medicinemedicine.arteryHypertension PortalmedicineAnimalsHumansEndotheliumRenal arterybusiness.industrymedicine.diseaseAcetylcholineRats030104 developmental biologychemistryVasoconstrictionNitric oxide inhibitorsNitric Oxide SynthaseAsymmetric dimethylargininebusinessVasoconstriction
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The expression mechanism of the residual LTP in the CA1 region of BDNF k.o. mice is insensitive to NO synthase inhibition

2011

Abstract BDNF and nitric oxide signaling both contribute to long-term potentiation (LTP) at glutamatergic synapses, but to date, few studies analyzed the interaction of both signaling cascades in the same synaptic pathway. Here we addressed the question whether the residual LTP in the CA1 region of hippocampal slices from heterozygous BDNF knockout mice (BDNF +/− ) is dependent on nitric oxide (NO) signaling. Extracellular recording of synaptic field potentials elicited by presynaptic Schaffer collateral stimulation was performed in the CA1 region of hippocampal slices of 4- to 6-week-old mice, and LTP was induced by a theta burst stimulation protocol. Application of the nitric oxide inhibi…

Long-Term PotentiationBiophysicsTropomyosin receptor kinase BIn Vitro TechniquesBiologyNitric oxideMicechemistry.chemical_compoundmedicineAnimalsEnzyme InhibitorsCA1 Region HippocampalMolecular BiologyMice KnockoutBrain-derived neurotrophic factorBrain-Derived Neurotrophic Factormusculoskeletal neural and ocular physiologyGeneral NeuroscienceExcitatory Postsynaptic PotentialsLong-term potentiationElectric StimulationCell biologyMice Inbred C57BLNG-Nitroarginine Methyl EsterSynaptic fatiguemedicine.anatomical_structureAnimals Newbornnervous systemchemistrySchaffer collateralSynaptic plasticityRetrograde signalingNeurology (clinical)Nitric Oxide SynthaseNeuroscienceDevelopmental BiologyBrain Research
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Inhibitory responses to exogenous adenosine in murine proximal and distal colon”

2006

The aims of the present study were firstly, to characterize pharmacologically the subtypes of P(1) purinoreceptors involved in the inhibitory effects induced by exogenous adenosine in longitudinal smooth muscle of mouse colon, and secondly, to examine differences in the function and distribution of these receptors between proximal and distal colon. Adenosine (100 microM-3 mM) caused a concentration-dependent reduction of the amplitude of spontaneous contractions in the proximal colon, and muscular relaxation in the distal colon. In the proximal colon, adenosine effects were antagonized by a selective A(1) receptor antagonist, 1,3-dipropyl-8-cyclopentylxanthine (DPCPX, 10 nM), but were not m…

MaleAdenosineNitric Oxide Synthase Type IIIColonmouse colonadenosine A2B receptorNitric OxideSettore BIO/09 - FisiologiaMiceP1 purinoreceptorAnimalsadenosine A3 receptorEnzyme InhibitorsDose-Response Relationship Drugadenosine A1 receptorReceptors Purinergic P1Muscle SmoothTriazolesnitrergic nervesMice Inbred C57BLNG-Nitroarginine Methyl Esteradenosine A2 receptorPurinergic P1 Receptor AntagonistsXanthinesPapersQuinazolinesTheobrominemechanical activityMuscle ContractionSignal Transduction
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Altered tachykinergic influence on gastric mechanical activity in mdx mice

2006

Abstract This study investigated whether alterationsin gastric activity in dystrophic mdx mouse can beattributed to dysfunctions of tachykinins. Endolumi-nal pressure was recorded and the expression ofneuronal nitric oxide synthase (nNOS), NK1 and NK2neurokinin receptors was investigated by immunoh-istochemistry. SR48968, NK2 receptor antagonist, butnot SR140333, NK1 receptor antagonist, decreased thetone only in mdx gastric preparations. In the presenceof N x -nitro- L -arginine methyl ester ( L -NAME), inhib-itor of NOS, SR48968 reduced the tone also in normalstomach. [Sar 9 , Met(O 2 ) 11 ]-SP, agonist of NK1 recep-tors, caused tetrodotoxin-sensitive relaxations, antag-onized by SR140333…

MaleAgonistQuinuclidinesmedicine.medical_specialtymdx mouseManometryPhysiologymedicine.drug_classNitric Oxide Synthase Type ISettore BIO/09 - FisiologiaNitric oxideMicechemistry.chemical_compoundimmunohistochemistry mdx mouse nitric oxide stomach tachykininsOrgan Culture TechniquesNeurokinin-1 Receptor AntagonistsPiperidinesTachykininsInternal medicinemedicineAnimalsEnzyme InhibitorsReceptorbiologyEndocrine and Autonomic SystemsStomachStomachGastroenterologyAntagonistMuscle SmoothReceptors Neurokinin-2Receptors Neurokinin-1musculoskeletal systemImmunohistochemistryMuscular Dystrophy DuchenneNitric oxide synthaseDisease Models AnimalNG-Nitroarginine Methyl Estermedicine.anatomical_structureEndocrinologychemistryMuscle TonusBenzamidesMice Inbred mdxbiology.proteinNK1 receptor antagonistGastrointestinal MotilityMuscle Contraction
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